ライフサイエンスコーパス: gastric pH

1 he others because the caseins clotted at the gastric pH.

2 potentially useful therapeutics to regulate gastric pH.

3 onstrate equivalency and ability to increase gastric pH.

4 estion of a meal and maintain homeostasis of gastric pH.

5 pH paper are not interchangeable measures of gastric pH.

6 -secreting parietal cells and an increase in gastric pH.

7 Neither rHSP nor ITF altered gastric pH.

8 PEP 1:2 ratio inversely correlates with gastric pH.

9 ils, H. pylori-infected gerbils had a higher gastric pH, a higher incidence of gastric ulcers, and a

10 24-h median [IQ] percentages of time with gastric pH above 3 and 4 were higher with rabeprazole th

11 a basic center imparting high solubility at gastric pH, addressing the dissolution limitation observ

12 Gastric pH affected the composition of the aspirate micr

13 fter omeprazole was first administered, mean gastric pH after omeprazole was started, and the lowest

14 omeprazole mean gastric pH was 7.1, the mean gastric pH after starting omeprazole was 6.8, and the me

15 n terms of histologic healing and increasing gastric pH and had a larger overall treatment effect.

16 is of the mice included measurement of basal gastric pH and plasma gastrin levels.

17 er endoscopic hemostasis, and in controlling gastric pH and protecting against upper gastrointestinal

18 e; in the remaining 16 subjects, low vs high gastric pH changed between tests.

19 received <3 mg/kg/day of ranitidine had poor gastric pH control as compared with 19% who received a m

20 ric drug references resulted in unsuccessful gastric pH control in a high percentage of pediatric int

21 Gastric pH control with ranitidine was considered unsucc

22 after omeprazole was started, and the lowest gastric pH during omeprazole therapy.

23 alkaline pH, and excystation in response to gastric pH followed by alkaline pH and protease.

24 The median basal gastric pH for the cohort after stabilization on therapy

25 nt mice exhibited a marked increase in basal gastric pH (from 3.2 to 5.2) and an approximately 10-fol

26 y (0.05 mg x kg(-1) x hr(-1)) until reaching gastric pH > or =4 for > or =75% of a 24-hr period, afte

27 ate ranitidine concentration associated with gastric pH > or =4 was 287 +/- 133 ng/mL.

28 ial ranitidine dosage regimen that targets a gastric pH > or =4.

29 termittent secretion, and 11% for consistent gastric pH higher than 3.5.

30 nzymes and bile salts, as well as the higher gastric pH in the infant model.

31 ngle intravenous dose (1.52 +/- 0.47 mg/kg), gastric pH increased from 1.6 +/- 1.0 to 5.1 +/- 1.1 (p

32 had more severe gastric inflammation, higher gastric pH, increased parietal cell loss, increased gast

33 Secondary efficacy measures were gastric pH measured 4 hrs after omeprazole was first adm

34 per gastrointestinal bleeding and maintained gastric pH of > 5.5 in mechanically ventilated critical

35 itidine and the dose should be titrated to a gastric pH of > or =4.

36 e than intravenous cimetidine in maintaining gastric pH of >4 in critically ill patients.

37 - 8.4 with a median pentagastrin stimulated gastric pH of 1.60 (range 1.0 - 8.2).

38 For Salmonella, transient exposure to gastric pH prepares invading bacteria for the stresses o

39 Thereafter, gastric pH should be monitored and the dose of ranitidin

40 onal status but it inversely correlated with gastric pH (Spearman's rho = -0.34; P = 0.0001).

41 location of the pH step-up point (shift from gastric pH to a pH >4) was also measured before and afte

42 Gastric pH values were lower in sst2 (-/-) mice (3.8 +/-

43 Median gastric pH was > or =6 on all trial days with omeprazole

44 In the omeprazole suspension group, median gastric pH was >4 on each trial day in 95% of patients.

45 The 4-hr postomeprazole mean gastric pH was 7.1, the mean gastric pH after starting o

46 Gastric pH was determined at the end of the ranitidine d

47 Gastric pH was monitored for 24 hours on three separate

48 Gastric pH was monitored hourly via nasogastric pH probe

49 Poor control of gastric pH was not associated with feeding, intubation s

50 Gastric pH was poorly controlled in 36% of patients.

51 ritional status, smoking, alcohol intake and gastric pH were also analysed.