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1 peptide antagonist for the cholecystokinin-B/gastrin receptor.
2 g to the G-protein-coupled cholecystokinin-B/gastrin receptor.
3 s, but the release is not mediated via CCK-B/gastrin receptor.
4 ) that expresses the human cholecystokinin-B/gastrin receptor.
5 ased expression of cholecystokinin B (CCK-B)/gastrin receptors.
6 LD-1 expressed both functional PPARgamma and gastrin receptors.
7 s gastroprotection is negated by blockade of gastrin receptors.
8 or the presence of cholecystokinin-B (CCK-B)/gastrin receptors, (2) the effect of gastrin on D-myo-In
9 ither cholecystokinin-A or cholecystokinin-B/gastrin receptor absence, body weight as a function of a
10 examine the role of cholecystokinin 2 (CCK2)/gastrin receptor and histamine H2-receptor signaling in
11 hormone that binds to the cholecystokinin B-gastrin receptor and regulates the activity of L-histidi
12 somatostatin (10(-10) mol/L) but not by the gastrin receptor antagonist L365,260 at 10(-6) mol/L.
13 d with Helicobacter felis and given the CCK2/gastrin receptor antagonist YF476 and/or the histamine H
14 (400 ppm in drinking water) alone, the CCK2/gastrin receptor antagonist YM022 (45 mg/kg/wk) alone, a
18 ansmembrane domains of the cholecystokinin-B/gastrin receptor (CCK-BR) comprise a putative ligand bin
20 ectopic expression of the cholecystokinin B/gastrin receptor (CCK-BR) is widely reported in human co
22 nist binding to the cholecystokinin-2 (CCK2)/gastrin receptor (CCK2R), a G-protein-coupled receptor,
24 thesis, summarizing current understanding of gastrin receptors, describing the regulation of gastrin
27 al cells that express cholecystokinin (CCK)B/gastrin receptors have been used for analysis of intrace
28 e variants of the cholecystokinin-2 (CCK(2))/gastrin receptor; however, their relative contributions
29 ings demonstrate the importance of the CCK-B/gastrin receptor in maintaining the normal cellular comp
32 active mutant of the human cholecystokinin-B/gastrin receptor, Leu325 --> Glu, offers the potential t
35 Both cholecystokinin-A and cholecystokinin-B/gastrin receptor-/- mice maintained normal body weight w
37 olorectal adenomas express cholecystokinin B/gastrin receptor mRNA, and thus hypergastrinemia may inc
39 ported the presence of novel progastrin (PG)/gastrin receptors on normal and cancerous intestinal cel
40 is unclear how FAK receives signals from the gastrin receptor or other G-protein-coupled receptors th
42 growth in BDL rats by interacting with CCK-B/gastrin receptors through a signal transduction pathway
44 ptors and is not due to occupation of CCK-2 (gastrin) receptors, which also are expressed in the inte
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