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1 he middle and distal parts, and three at the gastroesophageal junction).
2 metastatic adenocarcinoma of the stomach and gastroesophageal junction.
3 correlated with the (18)F-FDG uptake at the gastroesophageal junction.
4 asia seen in some cases, particularly at the gastroesophageal junction.
5 system delivers radiofrequency energy to the gastroesophageal junction.
6 ination after radiofrequency ablation at the gastroesophageal junction.
7 d marker and target of transformation at the gastroesophageal junction.
8 t from passive mechanical distraction of the gastroesophageal junction.
9 d genomic dosage in an adenocarcinoma of the gastroesophageal junction.
10 resectable adenocarcinoma of the stomach or gastroesophageal junction.
11 endoscopic biopsies above, at, and below the gastroesophageal junction; 2) esophageal motility; and 3
12 nts to radiofrequency energy delivery to the gastroesophageal junction (35 patients) or to a sham pro
13 patients with untreated advanced gastric or gastroesophageal junction adenocarcinoma to evaluate act
14 ction of stage IB through IV (M0) gastric or gastroesophageal junction adenocarcinoma were randomly a
15 gnosis in patients with distal esophageal or gastroesophageal junction adenocarcinoma who have receiv
16 Patients with histologic proof of gastric or gastroesophageal junction adenocarcinoma with a Karnofsk
17 rpose After curative resection of gastric or gastroesophageal junction adenocarcinoma, Intergroup Tri
18 ion After a curative resection of gastric or gastroesophageal junction adenocarcinoma, postoperative
19 rs with stage IB through IV (M0) gastric and gastroesophageal junction adenocarcinoma, previously ran
21 radiochemotherapy for high risk gastric and gastroesophageal junction adenocarcinoma: Demonstrated s
22 veness in reducing mortality from esophageal/gastroesophageal junction adenocarcinomas has not been e
23 ed by gastroesophageal reflux disease in the gastroesophageal junction and associated with tumorigene
25 the type and condition of the mucosa at the gastroesophageal junction and its relation to gastroesop
26 velopment of metaplasia and neoplasia at the gastroesophageal junction and suggests practical guideli
27 The RCs for recognizing the location of the gastroesophageal junction and the diaphragmatic hiatus w
28 py of local and locally advanced esophageal, gastroesophageal junction, and gastric adenocarcinomas,
29 ry site (distal one-third, middle one-third, gastroesophageal junction, and proximal one-third), Laur
31 plasia in an endoscopically normal-appearing gastroesophageal junction are histologic indicators of g
32 ined as eradication of IM (in esophageal and gastroesophageal junction biopsy specimens), documented
33 ence in adenocarcinomas of the esophagus and gastroesophageal junction by relatively low resolution g
36 Therapy of patients with advanced gastric or gastroesophageal junction cancer should provide symptom
37 reviously untreated metastatic esophageal or gastroesophageal junction cancer were randomly assigned
39 otherapy for locally advanced esophageal and gastroesophageal junction cancers is based on a few posi
40 results with a large series of patients with gastroesophageal junction cancers, TTE can be performed
42 to assume that intestinal metaplasia at the gastroesophageal junction develops as a result of chroni
43 T1 adenocarcinoma of the distal esophagus or gastroesophageal junction from January 1985 to December
44 chemotherapy regimen for advanced gastric or gastroesophageal junction (GEJ) adenocarcinoma and is as
46 iate esophageal versus gastric epithelium in gastroesophageal junction (GEJ) biopsies, the histology
50 g to primary tumor location into two groups: gastroesophageal junction (GEJ)/cardia and distal gastri
51 were more frequent in proximal (esophageal, gastroesophageal junction [GEJ], and cardia) tumors than
55 patients with carcinoma of the esophagus or gastroesophageal junction has been surgery, although pri
56 ith adenocarcinoma of the gastric cardia and gastroesophageal junction have no history of reflux.
57 pathogenesis of intestinal metaplasia at the gastroesophageal junction have yielded contradictory res
59 the lower frequency of tumors arising at the gastroesophageal junction in comparison with distal gast
60 carcinomas arising in the stomach or in the gastroesophageal junction in patients with HER2-positive
61 o III cancer of the mid-/distal-esophagus or gastroesophageal junction, measurable disease, and Easte
63 s epithelium and 3 biopsy specimens from the gastroesophageal junction of 3 patients without Barrett'
64 axation nadir also typically occurred before gastroesophageal junction opening (median, 2.1 seconds;
65 hincter relaxation onset invariably preceded gastroesophageal junction opening (median, 5.0 seconds;
66 ent lower esophageal sphincter relaxation to gastroesophageal junction opening in an unsedated human
67 Transformation associated with reflux at the gastroesophageal junction reflects activation by bile ac
68 lthough progression to adenocarcinoma at the gastroesophageal junction reflects exposure to acid and
70 of intestinal metaplasia located only at the gastroesophageal junction than it has been in patients w
71 rough studies showed a pocket of acid at the gastroesophageal junction that escaped the buffering eff
72 thoracic esophageal carcinoma, including the gastroesophageal junction tumors (Siewert I), were rando
73 ch, carina, and one vertebral body above the gastroesophageal junction was 13.9, 14.3, and 15.1 mm, r
74 xtent of the endoscopic BE segment above the gastroesophageal junction were 0.95 and 0.94, respective
75 th resected adenocarcinoma of the stomach or gastroesophageal junction were randomly assigned to surg
77 currence of adenocarcinoma of the stomach or gastroesophageal junction who have undergone curative re
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