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1  Barrett's esophagus segments and persistent gastroesophageal reflux.
2  frequently performed surgical procedure for gastroesophageal reflux.
3 fundoplication for the surgical treatment of gastroesophageal reflux.
4  choanal atresia repair, and/or treatment of gastroesophageal reflux.
5 ergone a fundoplication for the treatment of gastroesophageal reflux.
6 The symptoms are often confused for those of gastroesophageal reflux.
7 esophageal diseases result from uncontrolled gastroesophageal reflux.
8 hincter relaxation is the main mechanism for gastroesophageal reflux.
9 sociated with a two-to threefold increase in gastroesophageal reflux.
10 n, poor overall health, home oxygen use, and gastroesophageal reflux.
11 n, many of the symptoms of EE mimic those of gastroesophageal reflux.
12  esophagus and may work synergistically with gastroesophageal reflux.
13  criteria and two with findings secondary to gastroesophageal reflux.
14  leaks and strictures, gastric dilation, and gastroesophageal reflux.
15  point), obstructive sleep apnea (+1 point), gastroesophageal reflux (+1 point), and depression (+1 p
16 omorbidities were allergic rhinitis (62.4%), gastroesophageal reflux (42.1%), sinusitis (37.9%), nasa
17 dies were selected by using the search terms gastroesophageal reflux, adenocarcinoma, and Barrett's e
18      Although hemifundoplication may prevent gastroesophageal reflux after esophagomyotomy for achala
19 ocephaly, developed intellectual disability, gastroesophageal reflux and a seizure disorder.
20            There were 7 million diagnoses of gastroesophageal reflux and almost 4 million diagnoses o
21 tissue remodeling is associated with chronic gastroesophageal reflux and constitutes a premalignant l
22                BE is associated with chronic gastroesophageal reflux and esophageal cancer.
23 because of both mechanical effects promoting gastroesophageal reflux and nonmechanical effects.
24 also significant improvements in symptoms of gastroesophageal reflux and rhinitis, bronchial reversib
25 wn a positive correlation between pathologic gastroesophageal reflux and those with hiatal hernia or
26 index greater than 4 on pH monitoring and/or gastroesophageal reflux and/or herniated wrap on upper g
27                             Infantile colic, gastroesophageal reflux, and constipation are the most c
28 trition because of uncoordinated swallowing, gastroesophageal reflux, and constipation.
29 855 demonstrated that esophagitis was due to gastroesophageal reflux, and Hirsch in 1900 diagnosed an
30 y causes symptoms similar to those seen with gastroesophageal reflux, and is characterized by increas
31 pse include neck flexion, airway secretions, gastroesophageal reflux, and sleep deprivation.
32 ralized anxiety disorder, diabetes mellitus, gastroesophageal reflux, bacterial infection, and bone m
33 is used variably to prevent complications of gastroesophageal reflux, but its effectiveness is unprov
34 ment of patients who fail medical therapy of gastroesophageal reflux, but recurrent gastroesophageal
35                                              Gastroesophageal reflux causes inflammation and intestin
36 trophy (SMA) notes that patients suffer from gastroesophageal reflux, constipation and delayed gastri
37 ies, including sleep apnea, hypoventilation, gastroesophageal reflux, degenerative joint disease, uri
38 p of these patients is quite important since gastroesophageal reflux, developmental delay, chronic lu
39 ng bowel habit (15%), constipation (13%) and gastroesophageal reflux disease (12%).
40 lipidemia (24.5%), hypertension (21.9%), and gastroesophageal reflux disease (17.3%).
41                              The presence of gastroesophageal reflux disease (26%), esophageal strict
42 ere rhinitis (84.0%), sinusitis (47.8%), and gastroesophageal reflux disease (46.3%).
43 of diseases such as functional dyspepsia and gastroesophageal reflux disease (e.g. vomiting, disorder
44 gus (n = 1059) with those from subjects with gastroesophageal reflux disease (gastroesophageal reflux
45 .1; 95% confidence interval [CI], 2.9-12.9), gastroesophageal reflux disease (GERD) (RR, 1.9; 95% CI,
46 lin, leptin, and ghrelin are associated with gastroesophageal reflux disease (GERD) and Barrett's eso
47  We examined the incidence and predictors of gastroesophageal reflux disease (GERD) and dyspepsia and
48              Skinner to the understanding of gastroesophageal reflux disease (GERD) and its complicat
49  might represent an alternative treatment of gastroesophageal reflux disease (GERD) and may provide d
50  has enormous potential for the treatment of gastroesophageal reflux disease (GERD) and other esophag
51     The advent of the Montreal definition of gastroesophageal reflux disease (GERD) and the Rome III
52 agement of extraesophageal manifestations of gastroesophageal reflux disease (GERD) and to compare th
53   Lung transplant recipients with documented gastroesophageal reflux disease (GERD) are at increased
54                 Current diagnostic tests for gastroesophageal reflux disease (GERD) are suboptimal an
55 hageal eosinophilia who were thought to have gastroesophageal reflux disease (GERD) but who did not r
56                                Management of gastroesophageal reflux disease (GERD) commonly starts w
57 t clear why only a minority of patients with gastroesophageal reflux disease (GERD) develop Barrett's
58 .4)] were included; 70% had been treated for gastroesophageal reflux disease (GERD) during infancy.
59                                     However, gastroesophageal reflux disease (GERD) has remained the
60 The histologic changes associated with acute gastroesophageal reflux disease (GERD) have not been stu
61         The aim was to compare recurrence of gastroesophageal reflux disease (GERD) in children rando
62 RAs) are frequently used in the treatment of gastroesophageal reflux disease (GERD) in children; howe
63 ull-thickness plication for the treatment of gastroesophageal reflux disease (GERD) in comparison wit
64 n (TF) can decrease or eliminate features of gastroesophageal reflux disease (GERD) in some patients
65                                              Gastroesophageal reflux disease (GERD) is a chronic diso
66                                              Gastroesophageal reflux disease (GERD) is a common diagn
67                                              Gastroesophageal reflux disease (GERD) is a common medic
68                                              Gastroesophageal reflux disease (GERD) is a risk factor
69                   It has been suggested that gastroesophageal reflux disease (GERD) is a risk factor
70                                              Gastroesophageal reflux disease (GERD) is a very common
71                                              Gastroesophageal reflux disease (GERD) is associated wit
72                          The pathogenesis of gastroesophageal reflux disease (GERD) is complex and in
73  illustrated by the Montreal classification, gastroesophageal reflux disease (GERD) is much more than
74                                              Gastroesophageal reflux disease (GERD) is prevalent worl
75 ump inhibitor (PPI) therapy in patients with gastroesophageal reflux disease (GERD) is reported in up
76                                              Gastroesophageal reflux disease (GERD) is the most commo
77                                              Gastroesophageal reflux disease (GERD) is the most preva
78                                              Gastroesophageal reflux disease (GERD) is the strongest
79      Exclusion criteria included symptoms of gastroesophageal reflux disease (GERD) more than once a
80                        Many individuals with gastroesophageal reflux disease (GERD) never visit their
81 f an underlying acid peptic disorder such as gastroesophageal reflux disease (GERD) nor should it pre
82 is name, currently used for the treatment of gastroesophageal reflux disease (GERD) or completing Hel
83                           Most patients with gastroesophageal reflux disease (GERD) report that stres
84  has been developed for use in patients with gastroesophageal reflux disease (GERD) symptoms despite
85  discrepancy reflects a higher prevalence of gastroesophageal reflux disease (GERD) symptoms or a hig
86 assessed the utility of NBI in patients with gastroesophageal reflux disease (GERD) symptoms.
87                                Patients with gastroesophageal reflux disease (GERD) who are not respo
88 phagus (cases) were matched to subjects with gastroesophageal reflux disease (GERD) without Barrett's
89 tailed critique of objective measurements of gastroesophageal reflux disease (GERD) would improve man
90 BS), 3.0; dyspepsia, 1.8; constipation, 3.9; gastroesophageal reflux disease (GERD), 9.7.
91                                              Gastroesophageal reflux disease (GERD), a condition comm
92 cluding Crohn disease (CrD), celiac disease, gastroesophageal reflux disease (GERD), and eosinophilic
93 frequency of irritable bowel syndrome (IBS), gastroesophageal reflux disease (GERD), and overactive b
94 e upper airways dysfunction syndrome (RUDS), gastroesophageal reflux disease (GERD), and rare cases o
95 rm use of PPIs for three common indications: gastroesophageal reflux disease (GERD), Barrett's esopha
96 sophagus has been proposed for patients with gastroesophageal reflux disease (GERD), but there is lit
97 n several pathophysiological states, such as gastroesophageal reflux disease (GERD), functional dyspe
98                                              Gastroesophageal reflux disease (GERD), functional dyspe
99       The association of body mass index and gastroesophageal reflux disease (GERD), including its co
100         Barrett esophagus, a complication of gastroesophageal reflux disease (GERD), predisposes pati
101 ients, who had a previous fundoplication for gastroesophageal reflux disease (GERD), underwent reoper
102                                       Unlike gastroesophageal reflux disease (GERD), whose symptoms c
103 n accurate method for diagnosing adults with gastroesophageal reflux disease (GERD).
104 s for a range of medical disorders including gastroesophageal reflux disease (GERD).
105 only used in the diagnosis and management of gastroesophageal reflux disease (GERD).
106 ctional gastrointestinal disorders (FGD) and gastroesophageal reflux disease (GERD).
107 e existing endoscopy-based interventions for gastroesophageal reflux disease (GERD).
108  a significant increase in the prevalence of gastroesophageal reflux disease (GERD).
109 in the past year has centered on surgery for gastroesophageal reflux disease (GERD).
110 on atypical/extraesophageal manifestation of gastroesophageal reflux disease (GERD).
111 one combination vs omeprazole monotherapy in gastroesophageal reflux disease (GERD).
112 s and many do not report typical symptoms of gastroesophageal reflux disease (GERD).
113 the relaxed EGJ was altered in patients with gastroesophageal reflux disease (GERD).
114 en fundoplication (CNF) for the treatment of gastroesophageal reflux disease (GERD).
115 dal anti-inflammatory drugs) and symptoms of gastroesophageal reflux disease (GERD).
116 urgical option for patients with early-stage gastroesophageal reflux disease (GERD).
117  procedure of choice for obese patients with gastroesophageal reflux disease (GERD).
118 y risk factors for both BE and EA is chronic gastroesophageal reflux disease (GERD).
119 responders and treatment-naive patients with gastroesophageal reflux disease (GERD).
120 cations in adults were dysphagia (70.1%) and gastroesophageal reflux disease (GERD)/heartburn (27.1%)
121            We also included 10 patients with gastroesophageal reflux disease (GERD; age, 32-60 y; 7 w
122 n 2002 and 2005 were matched to persons with gastroesophageal reflux disease (GERD; n = 316) and to p
123 nths after fundoplication for PPI-refractory gastroesophageal reflux disease (n = 14 270 degrees LPF
124 (n = 296 cases) were matched to persons with gastroesophageal reflux disease (n = 308) without Barret
125 ad EoE (n = 17), indeterminate EoE (n = 15), gastroesophageal reflux disease (n = 7), or normal esoph
126  Glasgow Coma Scale score <9 (p = .021), and gastroesophageal reflux disease (p = .033).
127 ether patient-identified domains (dysphagia, gastroesophageal reflux disease [GERD], nausea/vomiting,
128 uency, severity, and duration of symptoms of gastroesophageal reflux disease among randomly selected
129 er improvement in quality of life related to gastroesophageal reflux disease and a 50% or greater red
130 rs to result in decreased prevalence of both gastroesophageal reflux disease and adenocarcinoma of th
131 and surgical techniques in the management of gastroesophageal reflux disease and constipation also ma
132 patterns from biopsies of patients with EoE, gastroesophageal reflux disease and controls.
133 11,945 patients aged 18 years and older with gastroesophageal reflux disease and erosive esophagitis.
134 allenges to diagnosis, including the role of gastroesophageal reflux disease and proton pump inhibito
135  75-year-old woman with a medical history of gastroesophageal reflux disease and type II diabetes pre
136 e mechanisms involved in the pathogenesis of gastroesophageal reflux disease are complex and multifac
137   The high reoperation rate and incidence of gastroesophageal reflux disease are concerning.
138 pump inhibitors and has focused attention on gastroesophageal reflux disease as a causative factor in
139  We prospectively assessed 100 patients with gastroesophageal reflux disease before and after sphinct
140 iteria consisted of a confirmed diagnosis of gastroesophageal reflux disease by an abnormal esophagea
141 , screening 50-year-old men with symptoms of gastroesophageal reflux disease by Cytosponge is cost ef
142  emerging disease that is distinguished from gastroesophageal reflux disease by the expression of a u
143 ished before their place in the treatment of gastroesophageal reflux disease can be determined.
144  suppression, have been confirmed in EoE and gastroesophageal reflux disease cell cultures.
145                                              Gastroesophageal reflux disease complicated by Barrett e
146                                              Gastroesophageal reflux disease complicated by Barrett's
147 search into the use of these technologies in gastroesophageal reflux disease continues to accelerate,
148  95% confidence interval [CI]: 1.04-2.67) or gastroesophageal reflux disease controls (OR = 1.61; 95%
149 bjects with gastroesophageal reflux disease (gastroesophageal reflux disease controls, n = 1332), and
150       Early aggressive surgical treatment of gastroesophageal reflux disease decreases the rate of br
151 es for detailed drugs (lipid-lowering drugs, gastroesophageal reflux disease drugs, antihypertensive
152 ugs in 8 drug classes (lipid-lowering drugs, gastroesophageal reflux disease drugs, diabetes drugs, a
153 ecome available in the last 2 years for many gastroesophageal reflux disease endotherapies, providing
154                           Most patients with gastroesophageal reflux disease experience symptomatic r
155 rm outcome of ARS in pediatric patients with gastroesophageal reflux disease have shown good to excel
156  months improved in patients with normal GE (Gastroesophageal Reflux Disease Health-Related Quality o
157 imary laparoscopic antireflux surgery due to gastroesophageal reflux disease in adults (>18 years).
158           BMI is associated with symptoms of gastroesophageal reflux disease in both normal-weight an
159 re of the height in meters - and symptoms of gastroesophageal reflux disease in persons of normal wei
160  related to the acidic environment caused by gastroesophageal reflux disease in the gastroesophageal
161                                              Gastroesophageal reflux disease is a common clinical pro
162                                              Gastroesophageal reflux disease is a highly prevalent ch
163                                              Gastroesophageal reflux disease is a highly prevalent di
164                                              Gastroesophageal reflux disease is a prevalent disorder
165                                              Gastroesophageal reflux disease is common in adults of a
166 w endoscopic therapies have emerged to treat gastroesophageal reflux disease itself.
167  esophageal sphincter (LES) in patients with gastroesophageal reflux disease often has a low resting
168 is (EoE) was historically distinguished from gastroesophageal reflux disease on the basis of histolog
169 ated with EoE (or indeterminate EoE) but not gastroesophageal reflux disease or normal esophagus and
170  intravenous therapy in patients with severe gastroesophageal reflux disease or the Zollinger-Ellison
171                      We randomly assigned 64 gastroesophageal reflux disease patients to radiofrequen
172 esents a new option for selected symptomatic gastroesophageal reflux disease patients who are intoler
173 5), and more had a >50% improvement in their gastroesophageal reflux disease quality of life score (n
174                     Endoscopic therapies for gastroesophageal reflux disease represent a minimally in
175 ted with a relatively high rate of recurrent gastroesophageal reflux disease requiring treatment, dim
176       Symptoms improved in group A, with the Gastroesophageal Reflux Disease Symptom Assessment Scale
177 uency energy delivery significantly improved gastroesophageal reflux disease symptoms and quality of
178 racticed, its value in patients with chronic gastroesophageal reflux disease symptoms is of unproven
179 ng upper endoscopy for patients with chronic gastroesophageal reflux disease symptoms to assess for B
180  men in the United Kingdom with histories of gastroesophageal reflux disease symptoms, assuming the p
181 inistration recently cleared new endoluminal gastroesophageal reflux disease treatments; however, no
182                               The history of gastroesophageal reflux disease was highly prevalent in
183                                              Gastroesophageal reflux disease was identified as an imp
184                                      De novo gastroesophageal reflux disease was reported in 43.8%.
185                                Patients with gastroesophageal reflux disease who have a partial respo
186 structive sleep apnea may be associated with gastroesophageal reflux disease, a strong risk factor fo
187 allergic rhinitis, chronic rhinitis, asthma, gastroesophageal reflux disease, adenotonsillitis, sleep
188              Risk factors evaluated included gastroesophageal reflux disease, alcohol consumption, sm
189 cent had symptoms of dyspnea, 87 percent had gastroesophageal reflux disease, and 54 percent had nasa
190 lization for asthma, mechanical ventilation, gastroesophageal reflux disease, and aspiration or other
191 lation), and prior diagnoses (eg, pneumonia, gastroesophageal reflux disease, and other comorbidities
192  the individual to irritable bowel syndrome, gastroesophageal reflux disease, and peptic ulcer diseas
193 treat a variety of gastric disorders such as gastroesophageal reflux disease, autoimmune gastritis, g
194  factors for EAC have been identified-mainly gastroesophageal reflux disease, Barrett's esophagus, ob
195 shown to be a safe and effective therapy for gastroesophageal reflux disease, but its effect on the L
196           Symptoms frequently mimic those of gastroesophageal reflux disease, but the diseases are di
197                         Risk factors include gastroesophageal reflux disease, central obesity, and sm
198 sing and treating chest pain associated with gastroesophageal reflux disease, correlates abnormal amb
199  terms: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
200 lowing: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
201 l gastrointestinal complications of obesity: gastroesophageal reflux disease, erosive esophagitis, Ba
202   This issue provides a clinical overview of gastroesophageal reflux disease, focusing on diagnosis,
203 of the lower esophagus epithelium related to gastroesophageal reflux disease, is the strongest known
204 a diligent search for an esophageal etiology-gastroesophageal reflux disease, motility abnormalities,
205 itant chemical carcinogen treatment leads to gastroesophageal reflux disease, multilayered epithelium
206               With improved understanding of gastroesophageal reflux disease, newer developments in d
207 on airway disease, including rhinosinusitis, gastroesophageal reflux disease, obesity and dysfunction
208 , including history of peptic ulcer disease, gastroesophageal reflux disease, or gastrointestinal ble
209                  This article is a review of gastroesophageal reflux disease, other types of esophagi
210                    Too much acid can lead to gastroesophageal reflux disease, peptic ulcer disease, a
211  premorbid prevalence of anxiety, headaches, gastroesophageal reflux disease, sleep apnea, and infect
212  44 patients (52%) had objective findings of gastroesophageal reflux disease, such as esophagitis.
213                                 Triggered by gastroesophageal reflux disease, the origin of this meta
214 ic conditions (hypertension, hyperlipidemia, gastroesophageal reflux disease, thyroid disease, diabet
215                                   Except for gastroesophageal reflux disease, which showed a higher r
216 for 5.2 million visits annually, followed by gastroesophageal reflux disease, with 4.5 million visits
217 l adenocarcinoma (BEAC) is a complication of gastroesophageal reflux disease, with no effective chemo
218 ministered a modified version of a validated gastroesophageal reflux disease-specific QOL tool to pat
219             Quality of life, measured by the gastroesophageal reflux disease-specific QOL tool, and r
220 llow-up after ARS in pediatric patients with gastroesophageal reflux disease.
221 180-degree LAF for the surgical treatment of gastroesophageal reflux disease.
222 st frequently performed surgical therapy for gastroesophageal reflux disease.
223 ved cells is a novel potential treatment for gastroesophageal reflux disease.
224 drome (IBS) frequently overlap with those of gastroesophageal reflux disease.
225 gents widely prescribed for the treatment of gastroesophageal reflux disease.
226  to evaluate for the presence of concomitant gastroesophageal reflux disease.
227  thereby conclusively distinguishing it from gastroesophageal reflux disease.
228 fundoplication is an effective treatment for gastroesophageal reflux disease.
229  increasingly recognized disease that mimics gastroesophageal reflux disease.
230 ders, including irritable bowel syndrome and gastroesophageal reflux disease.
231 ct patients from developing complications of gastroesophageal reflux disease.
232 itis is a new disease commonly confused with gastroesophageal reflux disease.
233  esophageal motor disorders and treatment of gastroesophageal reflux disease.
234  the association between BMI and symptoms of gastroesophageal reflux disease.
235 relation to the occurrence and management of gastroesophageal reflux disease.
236  and obese persons are at increased risk for gastroesophageal reflux disease.
237 actors usually indicative of severe types of gastroesophageal reflux disease.
238 d-mediated event and a reliable indicator of gastroesophageal reflux disease.
239  directions in the research and treatment of gastroesophageal reflux disease.
240 eading to duodenal ulcer, gastric ulcer, and gastroesophageal reflux disease.
241 ithout Barrett's esophagus or other types of gastroesophageal reflux disease.
242 ery effective and long-lasting treatment for gastroesophageal reflux disease.
243         Obesity is associated with a risk of gastroesophageal reflux disease.
244 the AGA Institute on "Endoscopic Therapy for Gastroesophageal Reflux Disease." It was approved by the
245 is a laryngeal symptom that can be caused by gastroesophageal reflux disease; however, treatment outc
246 cough may have abnormal proximal exposure to gastroesophageal reflux documented by HMII that would ha
247 ers, including nonalcoholic steatohepatitis, gastroesophageal reflux, gallstones, and increased risk
248 oplication on the incidence of postoperative gastroesophageal reflux (GER) after Heller myotomy.
249                          The contribution of gastroesophageal reflux (GER) and vagal function to esop
250          Dental erosion is a complication of gastroesophageal reflux (GER) in adults; in children, it
251                                              Gastroesophageal reflux (GER) is a disorder that is comm
252              The prevalence of abnormal acid gastroesophageal reflux (GER) is higher in patients with
253                                 Asymptomatic gastroesophageal reflux (GER) is prevalent in children w
254 f a disease engendered more controversy than gastroesophageal reflux (GER), a highly prevalent condit
255 symptom relief, prevalence of post-treatment gastroesophageal reflux (GER), and complications.
256 , the symptoms of EE were often confused for gastroesophageal reflux (GER).
257 sts with erosive tooth wear have significant gastroesophageal reflux (GERD), despite minor reflux sym
258 n the temporal association between cough and gastroesophageal reflux (GOR) in patients in whom non-GO
259  laparoscopically but failure with recurrent gastroesophageal reflux has resulted in the development
260 tocompatibility locus antigen antibodies and gastroesophageal reflux have been associated with poor o
261  sphincter (UES) to simulated or spontaneous gastroesophageal reflux have shown conflicting results.
262                 Subjects reporting nocturnal gastroesophageal reflux in both 1999 and 2010 had more N
263            For the first time, mechanisms of gastroesophageal reflux in critically ill mechanically v
264 rtburn with and without regurgitation due to gastroesophageal reflux in the absence of esophageal muc
265 py of gastroesophageal reflux, but recurrent gastroesophageal reflux is a persistent problem that req
266                                              Gastroesophageal reflux is common among patients with po
267 he diagnosis of cough, LPR, or asthma due to gastroesophageal reflux is difficult, as no criterion st
268                                              Gastroesophageal reflux is the main risk factor for esop
269  allograft injury from donor-directed Abs or gastroesophageal reflux led to new ColV and KAT Abs post
270 = 8), leiomyomatosis (n = 5), and refractory gastroesophageal reflux (n = 2) who underwent gastric tr
271 discussed: rhinitis, chronic rhinosinusitis, gastroesophageal reflux, obstructive sleep apnoea, vocal
272                                           As gastroesophageal reflux persists or recurs in 43% of chi
273             There is significant though mild gastroesophageal reflux postoperatively in 46% of patien
274 rative complications, sleep difficulties and gastroesophageal reflux progressively worsened during fo
275 in the postoperative period from SSc-related gastroesophageal reflux, renal impairment, and skin fibr
276 ireflux surgery, 17.7% experienced recurrent gastroesophageal reflux requiring long-term medication u
277                                              Gastroesophageal reflux scores were higher in overweight
278 , sex, percent predicted FEV1, self-reported gastroesophageal reflux, St. George's Respiratory Questi
279 presenting with extra-esophageal symptoms of gastroesophageal reflux such as chronic cough and asthma
280 based on the modified Rome III criteria) and gastroesophageal reflux symptoms (GERS) in a population-
281                           Apoptosis pathway, gastroesophageal reflux symptoms (reflux), higher body m
282 eep apnea and to determine whether nocturnal gastroesophageal reflux symptoms affect the relationship
283 estioned regarding severity of their typical gastroesophageal reflux symptoms and presence of nocturn
284 nal GERD in 1999 (>/=3 episodes of nocturnal gastroesophageal reflux symptoms per week) had an OR of
285  Barrett's esophagus patients with nocturnal gastroesophageal reflux symptoms should be further evalu
286 's esophagus was associated with more severe gastroesophageal reflux symptoms, and nocturnal reflux s
287 al reflux symptoms and presence of nocturnal gastroesophageal reflux symptoms.
288                                  The rate of gastroesophageal reflux was comparable with prior studie
289                                              Gastroesophageal reflux was defined by abnormal pH-testi
290                                 At 6 months, gastroesophageal reflux was evaluated by 24-hour pH test
291          The physiologic link between HH and gastroesophageal reflux was made at the second half of t
292                                              Gastroesophageal reflux was reported in 50.0% (40 vs 27
293                        Objective evidence of gastroesophageal reflux was seen in 46% patients postope
294                                              Gastroesophageal reflux was the most common GI diagnosis
295                        Chronic sinusitis and gastroesophageal reflux were also associated with exacer
296         From 1995 to 2003, 461 patients with gastroesophageal reflux were enrolled in 4 randomized co
297 pump inhibitors (PPIs) are popular drugs for gastroesophageal reflux, which are now available for lon
298  BACKGROUND & AIMS: Central obesity promotes gastroesophageal reflux, which may be related to increas
299 s have failed to demonstrate that preventing gastroesophageal reflux with antireflux surgery halts th
300              Six studies have suggested that gastroesophageal reflux worsens after cholecystectomy.

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