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1 Barrett's esophagus segments and persistent gastroesophageal reflux.
2 frequently performed surgical procedure for gastroesophageal reflux.
3 fundoplication for the surgical treatment of gastroesophageal reflux.
4 choanal atresia repair, and/or treatment of gastroesophageal reflux.
5 ergone a fundoplication for the treatment of gastroesophageal reflux.
6 The symptoms are often confused for those of gastroesophageal reflux.
7 esophageal diseases result from uncontrolled gastroesophageal reflux.
8 hincter relaxation is the main mechanism for gastroesophageal reflux.
9 sociated with a two-to threefold increase in gastroesophageal reflux.
10 n, poor overall health, home oxygen use, and gastroesophageal reflux.
11 n, many of the symptoms of EE mimic those of gastroesophageal reflux.
12 esophagus and may work synergistically with gastroesophageal reflux.
13 criteria and two with findings secondary to gastroesophageal reflux.
14 leaks and strictures, gastric dilation, and gastroesophageal reflux.
15 point), obstructive sleep apnea (+1 point), gastroesophageal reflux (+1 point), and depression (+1 p
16 omorbidities were allergic rhinitis (62.4%), gastroesophageal reflux (42.1%), sinusitis (37.9%), nasa
17 dies were selected by using the search terms gastroesophageal reflux, adenocarcinoma, and Barrett's e
21 tissue remodeling is associated with chronic gastroesophageal reflux and constitutes a premalignant l
24 also significant improvements in symptoms of gastroesophageal reflux and rhinitis, bronchial reversib
25 wn a positive correlation between pathologic gastroesophageal reflux and those with hiatal hernia or
26 index greater than 4 on pH monitoring and/or gastroesophageal reflux and/or herniated wrap on upper g
29 855 demonstrated that esophagitis was due to gastroesophageal reflux, and Hirsch in 1900 diagnosed an
30 y causes symptoms similar to those seen with gastroesophageal reflux, and is characterized by increas
32 ralized anxiety disorder, diabetes mellitus, gastroesophageal reflux, bacterial infection, and bone m
33 is used variably to prevent complications of gastroesophageal reflux, but its effectiveness is unprov
34 ment of patients who fail medical therapy of gastroesophageal reflux, but recurrent gastroesophageal
36 trophy (SMA) notes that patients suffer from gastroesophageal reflux, constipation and delayed gastri
37 ies, including sleep apnea, hypoventilation, gastroesophageal reflux, degenerative joint disease, uri
38 p of these patients is quite important since gastroesophageal reflux, developmental delay, chronic lu
43 of diseases such as functional dyspepsia and gastroesophageal reflux disease (e.g. vomiting, disorder
44 gus (n = 1059) with those from subjects with gastroesophageal reflux disease (gastroesophageal reflux
45 .1; 95% confidence interval [CI], 2.9-12.9), gastroesophageal reflux disease (GERD) (RR, 1.9; 95% CI,
46 lin, leptin, and ghrelin are associated with gastroesophageal reflux disease (GERD) and Barrett's eso
47 We examined the incidence and predictors of gastroesophageal reflux disease (GERD) and dyspepsia and
49 might represent an alternative treatment of gastroesophageal reflux disease (GERD) and may provide d
50 has enormous potential for the treatment of gastroesophageal reflux disease (GERD) and other esophag
52 agement of extraesophageal manifestations of gastroesophageal reflux disease (GERD) and to compare th
53 Lung transplant recipients with documented gastroesophageal reflux disease (GERD) are at increased
55 hageal eosinophilia who were thought to have gastroesophageal reflux disease (GERD) but who did not r
57 t clear why only a minority of patients with gastroesophageal reflux disease (GERD) develop Barrett's
58 .4)] were included; 70% had been treated for gastroesophageal reflux disease (GERD) during infancy.
60 The histologic changes associated with acute gastroesophageal reflux disease (GERD) have not been stu
62 RAs) are frequently used in the treatment of gastroesophageal reflux disease (GERD) in children; howe
63 ull-thickness plication for the treatment of gastroesophageal reflux disease (GERD) in comparison wit
64 n (TF) can decrease or eliminate features of gastroesophageal reflux disease (GERD) in some patients
73 illustrated by the Montreal classification, gastroesophageal reflux disease (GERD) is much more than
75 ump inhibitor (PPI) therapy in patients with gastroesophageal reflux disease (GERD) is reported in up
81 f an underlying acid peptic disorder such as gastroesophageal reflux disease (GERD) nor should it pre
82 is name, currently used for the treatment of gastroesophageal reflux disease (GERD) or completing Hel
84 has been developed for use in patients with gastroesophageal reflux disease (GERD) symptoms despite
85 discrepancy reflects a higher prevalence of gastroesophageal reflux disease (GERD) symptoms or a hig
88 phagus (cases) were matched to subjects with gastroesophageal reflux disease (GERD) without Barrett's
89 tailed critique of objective measurements of gastroesophageal reflux disease (GERD) would improve man
92 cluding Crohn disease (CrD), celiac disease, gastroesophageal reflux disease (GERD), and eosinophilic
93 frequency of irritable bowel syndrome (IBS), gastroesophageal reflux disease (GERD), and overactive b
94 e upper airways dysfunction syndrome (RUDS), gastroesophageal reflux disease (GERD), and rare cases o
95 rm use of PPIs for three common indications: gastroesophageal reflux disease (GERD), Barrett's esopha
96 sophagus has been proposed for patients with gastroesophageal reflux disease (GERD), but there is lit
97 n several pathophysiological states, such as gastroesophageal reflux disease (GERD), functional dyspe
101 ients, who had a previous fundoplication for gastroesophageal reflux disease (GERD), underwent reoper
120 cations in adults were dysphagia (70.1%) and gastroesophageal reflux disease (GERD)/heartburn (27.1%)
122 n 2002 and 2005 were matched to persons with gastroesophageal reflux disease (GERD; n = 316) and to p
123 nths after fundoplication for PPI-refractory gastroesophageal reflux disease (n = 14 270 degrees LPF
124 (n = 296 cases) were matched to persons with gastroesophageal reflux disease (n = 308) without Barret
125 ad EoE (n = 17), indeterminate EoE (n = 15), gastroesophageal reflux disease (n = 7), or normal esoph
127 ether patient-identified domains (dysphagia, gastroesophageal reflux disease [GERD], nausea/vomiting,
128 uency, severity, and duration of symptoms of gastroesophageal reflux disease among randomly selected
129 er improvement in quality of life related to gastroesophageal reflux disease and a 50% or greater red
130 rs to result in decreased prevalence of both gastroesophageal reflux disease and adenocarcinoma of th
131 and surgical techniques in the management of gastroesophageal reflux disease and constipation also ma
133 11,945 patients aged 18 years and older with gastroesophageal reflux disease and erosive esophagitis.
134 allenges to diagnosis, including the role of gastroesophageal reflux disease and proton pump inhibito
135 75-year-old woman with a medical history of gastroesophageal reflux disease and type II diabetes pre
136 e mechanisms involved in the pathogenesis of gastroesophageal reflux disease are complex and multifac
138 pump inhibitors and has focused attention on gastroesophageal reflux disease as a causative factor in
139 We prospectively assessed 100 patients with gastroesophageal reflux disease before and after sphinct
140 iteria consisted of a confirmed diagnosis of gastroesophageal reflux disease by an abnormal esophagea
141 , screening 50-year-old men with symptoms of gastroesophageal reflux disease by Cytosponge is cost ef
142 emerging disease that is distinguished from gastroesophageal reflux disease by the expression of a u
147 search into the use of these technologies in gastroesophageal reflux disease continues to accelerate,
148 95% confidence interval [CI]: 1.04-2.67) or gastroesophageal reflux disease controls (OR = 1.61; 95%
149 bjects with gastroesophageal reflux disease (gastroesophageal reflux disease controls, n = 1332), and
151 es for detailed drugs (lipid-lowering drugs, gastroesophageal reflux disease drugs, antihypertensive
152 ugs in 8 drug classes (lipid-lowering drugs, gastroesophageal reflux disease drugs, diabetes drugs, a
153 ecome available in the last 2 years for many gastroesophageal reflux disease endotherapies, providing
155 rm outcome of ARS in pediatric patients with gastroesophageal reflux disease have shown good to excel
156 months improved in patients with normal GE (Gastroesophageal Reflux Disease Health-Related Quality o
157 imary laparoscopic antireflux surgery due to gastroesophageal reflux disease in adults (>18 years).
159 re of the height in meters - and symptoms of gastroesophageal reflux disease in persons of normal wei
160 related to the acidic environment caused by gastroesophageal reflux disease in the gastroesophageal
167 esophageal sphincter (LES) in patients with gastroesophageal reflux disease often has a low resting
168 is (EoE) was historically distinguished from gastroesophageal reflux disease on the basis of histolog
169 ated with EoE (or indeterminate EoE) but not gastroesophageal reflux disease or normal esophagus and
170 intravenous therapy in patients with severe gastroesophageal reflux disease or the Zollinger-Ellison
172 esents a new option for selected symptomatic gastroesophageal reflux disease patients who are intoler
173 5), and more had a >50% improvement in their gastroesophageal reflux disease quality of life score (n
175 ted with a relatively high rate of recurrent gastroesophageal reflux disease requiring treatment, dim
177 uency energy delivery significantly improved gastroesophageal reflux disease symptoms and quality of
178 racticed, its value in patients with chronic gastroesophageal reflux disease symptoms is of unproven
179 ng upper endoscopy for patients with chronic gastroesophageal reflux disease symptoms to assess for B
180 men in the United Kingdom with histories of gastroesophageal reflux disease symptoms, assuming the p
181 inistration recently cleared new endoluminal gastroesophageal reflux disease treatments; however, no
186 structive sleep apnea may be associated with gastroesophageal reflux disease, a strong risk factor fo
187 allergic rhinitis, chronic rhinitis, asthma, gastroesophageal reflux disease, adenotonsillitis, sleep
189 cent had symptoms of dyspnea, 87 percent had gastroesophageal reflux disease, and 54 percent had nasa
190 lization for asthma, mechanical ventilation, gastroesophageal reflux disease, and aspiration or other
191 lation), and prior diagnoses (eg, pneumonia, gastroesophageal reflux disease, and other comorbidities
192 the individual to irritable bowel syndrome, gastroesophageal reflux disease, and peptic ulcer diseas
193 treat a variety of gastric disorders such as gastroesophageal reflux disease, autoimmune gastritis, g
194 factors for EAC have been identified-mainly gastroesophageal reflux disease, Barrett's esophagus, ob
195 shown to be a safe and effective therapy for gastroesophageal reflux disease, but its effect on the L
198 sing and treating chest pain associated with gastroesophageal reflux disease, correlates abnormal amb
199 terms: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
200 lowing: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
201 l gastrointestinal complications of obesity: gastroesophageal reflux disease, erosive esophagitis, Ba
202 This issue provides a clinical overview of gastroesophageal reflux disease, focusing on diagnosis,
203 of the lower esophagus epithelium related to gastroesophageal reflux disease, is the strongest known
204 a diligent search for an esophageal etiology-gastroesophageal reflux disease, motility abnormalities,
205 itant chemical carcinogen treatment leads to gastroesophageal reflux disease, multilayered epithelium
207 on airway disease, including rhinosinusitis, gastroesophageal reflux disease, obesity and dysfunction
208 , including history of peptic ulcer disease, gastroesophageal reflux disease, or gastrointestinal ble
211 premorbid prevalence of anxiety, headaches, gastroesophageal reflux disease, sleep apnea, and infect
212 44 patients (52%) had objective findings of gastroesophageal reflux disease, such as esophagitis.
214 ic conditions (hypertension, hyperlipidemia, gastroesophageal reflux disease, thyroid disease, diabet
216 for 5.2 million visits annually, followed by gastroesophageal reflux disease, with 4.5 million visits
217 l adenocarcinoma (BEAC) is a complication of gastroesophageal reflux disease, with no effective chemo
218 ministered a modified version of a validated gastroesophageal reflux disease-specific QOL tool to pat
244 the AGA Institute on "Endoscopic Therapy for Gastroesophageal Reflux Disease." It was approved by the
245 is a laryngeal symptom that can be caused by gastroesophageal reflux disease; however, treatment outc
246 cough may have abnormal proximal exposure to gastroesophageal reflux documented by HMII that would ha
247 ers, including nonalcoholic steatohepatitis, gastroesophageal reflux, gallstones, and increased risk
248 oplication on the incidence of postoperative gastroesophageal reflux (GER) after Heller myotomy.
254 f a disease engendered more controversy than gastroesophageal reflux (GER), a highly prevalent condit
257 sts with erosive tooth wear have significant gastroesophageal reflux (GERD), despite minor reflux sym
258 n the temporal association between cough and gastroesophageal reflux (GOR) in patients in whom non-GO
259 laparoscopically but failure with recurrent gastroesophageal reflux has resulted in the development
260 tocompatibility locus antigen antibodies and gastroesophageal reflux have been associated with poor o
261 sphincter (UES) to simulated or spontaneous gastroesophageal reflux have shown conflicting results.
264 rtburn with and without regurgitation due to gastroesophageal reflux in the absence of esophageal muc
265 py of gastroesophageal reflux, but recurrent gastroesophageal reflux is a persistent problem that req
267 he diagnosis of cough, LPR, or asthma due to gastroesophageal reflux is difficult, as no criterion st
269 allograft injury from donor-directed Abs or gastroesophageal reflux led to new ColV and KAT Abs post
270 = 8), leiomyomatosis (n = 5), and refractory gastroesophageal reflux (n = 2) who underwent gastric tr
271 discussed: rhinitis, chronic rhinosinusitis, gastroesophageal reflux, obstructive sleep apnoea, vocal
274 rative complications, sleep difficulties and gastroesophageal reflux progressively worsened during fo
275 in the postoperative period from SSc-related gastroesophageal reflux, renal impairment, and skin fibr
276 ireflux surgery, 17.7% experienced recurrent gastroesophageal reflux requiring long-term medication u
278 , sex, percent predicted FEV1, self-reported gastroesophageal reflux, St. George's Respiratory Questi
279 presenting with extra-esophageal symptoms of gastroesophageal reflux such as chronic cough and asthma
280 based on the modified Rome III criteria) and gastroesophageal reflux symptoms (GERS) in a population-
282 eep apnea and to determine whether nocturnal gastroesophageal reflux symptoms affect the relationship
283 estioned regarding severity of their typical gastroesophageal reflux symptoms and presence of nocturn
284 nal GERD in 1999 (>/=3 episodes of nocturnal gastroesophageal reflux symptoms per week) had an OR of
285 Barrett's esophagus patients with nocturnal gastroesophageal reflux symptoms should be further evalu
286 's esophagus was associated with more severe gastroesophageal reflux symptoms, and nocturnal reflux s
297 pump inhibitors (PPIs) are popular drugs for gastroesophageal reflux, which are now available for lon
298 BACKGROUND & AIMS: Central obesity promotes gastroesophageal reflux, which may be related to increas
299 s have failed to demonstrate that preventing gastroesophageal reflux with antireflux surgery halts th
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