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1 to evaluate for the presence of concomitant gastroesophageal reflux disease.
2 thereby conclusively distinguishing it from gastroesophageal reflux disease.
3 fundoplication is an effective treatment for gastroesophageal reflux disease.
4 increasingly recognized disease that mimics gastroesophageal reflux disease.
5 ders, including irritable bowel syndrome and gastroesophageal reflux disease.
6 ct patients from developing complications of gastroesophageal reflux disease.
7 itis is a new disease commonly confused with gastroesophageal reflux disease.
8 esophageal motor disorders and treatment of gastroesophageal reflux disease.
9 the association between BMI and symptoms of gastroesophageal reflux disease.
10 relation to the occurrence and management of gastroesophageal reflux disease.
11 and obese persons are at increased risk for gastroesophageal reflux disease.
12 actors usually indicative of severe types of gastroesophageal reflux disease.
13 d-mediated event and a reliable indicator of gastroesophageal reflux disease.
14 directions in the research and treatment of gastroesophageal reflux disease.
15 eading to duodenal ulcer, gastric ulcer, and gastroesophageal reflux disease.
16 ithout Barrett's esophagus or other types of gastroesophageal reflux disease.
17 ux surgery is performed for the treatment of gastroesophageal reflux disease.
18 esophagus in patients with mild-to-moderate gastroesophageal reflux disease.
19 ortened esophagus and stricture secondary to gastroesophageal reflux disease.
20 ormal-appearing cardia are manifestations of gastroesophageal reflux disease.
21 agus and adenocarcinoma are complications of gastroesophageal reflux disease.
22 astroesophageal junction and its relation to gastroesophageal reflux disease.
23 hageal junction are histologic indicators of gastroesophageal reflux disease.
24 that can occur in a subset of patients with gastroesophageal reflux disease.
25 c approach to the treatment of patients with gastroesophageal reflux disease.
26 ery effective and long-lasting treatment for gastroesophageal reflux disease.
27 Obesity is associated with a risk of gastroesophageal reflux disease.
28 llow-up after ARS in pediatric patients with gastroesophageal reflux disease.
29 180-degree LAF for the surgical treatment of gastroesophageal reflux disease.
30 st frequently performed surgical therapy for gastroesophageal reflux disease.
31 ved cells is a novel potential treatment for gastroesophageal reflux disease.
32 drome (IBS) frequently overlap with those of gastroesophageal reflux disease.
33 gents widely prescribed for the treatment of gastroesophageal reflux disease.
38 structive sleep apnea may be associated with gastroesophageal reflux disease, a strong risk factor fo
39 allergic rhinitis, chronic rhinitis, asthma, gastroesophageal reflux disease, adenotonsillitis, sleep
41 uency, severity, and duration of symptoms of gastroesophageal reflux disease among randomly selected
42 er improvement in quality of life related to gastroesophageal reflux disease and a 50% or greater red
43 rs to result in decreased prevalence of both gastroesophageal reflux disease and adenocarcinoma of th
44 and surgical techniques in the management of gastroesophageal reflux disease and constipation also ma
46 11,945 patients aged 18 years and older with gastroesophageal reflux disease and erosive esophagitis.
47 oplication was performed in 758 patients for gastroesophageal reflux disease and in 99 for paraesopha
48 allenges to diagnosis, including the role of gastroesophageal reflux disease and proton pump inhibito
49 75-year-old woman with a medical history of gastroesophageal reflux disease and type II diabetes pre
50 g the role of nonacid reflux in cough due to gastroesophageal reflux disease, and 3) developing relia
51 cent had symptoms of dyspnea, 87 percent had gastroesophageal reflux disease, and 54 percent had nasa
52 lization for asthma, mechanical ventilation, gastroesophageal reflux disease, and aspiration or other
53 lation), and prior diagnoses (eg, pneumonia, gastroesophageal reflux disease, and other comorbidities
54 the individual to irritable bowel syndrome, gastroesophageal reflux disease, and peptic ulcer diseas
56 e mechanisms involved in the pathogenesis of gastroesophageal reflux disease are complex and multifac
58 ractical concepts regarding the treatment of gastroesophageal reflux disease are developed, based on
60 pump inhibitors and has focused attention on gastroesophageal reflux disease as a causative factor in
61 treat a variety of gastric disorders such as gastroesophageal reflux disease, autoimmune gastritis, g
62 factors for EAC have been identified-mainly gastroesophageal reflux disease, Barrett's esophagus, ob
63 We prospectively assessed 100 patients with gastroesophageal reflux disease before and after sphinct
64 shown to be a safe and effective therapy for gastroesophageal reflux disease, but its effect on the L
66 iteria consisted of a confirmed diagnosis of gastroesophageal reflux disease by an abnormal esophagea
67 , screening 50-year-old men with symptoms of gastroesophageal reflux disease by Cytosponge is cost ef
68 emerging disease that is distinguished from gastroesophageal reflux disease by the expression of a u
74 search into the use of these technologies in gastroesophageal reflux disease continues to accelerate,
75 95% confidence interval [CI]: 1.04-2.67) or gastroesophageal reflux disease controls (OR = 1.61; 95%
76 bjects with gastroesophageal reflux disease (gastroesophageal reflux disease controls, n = 1332), and
77 sing and treating chest pain associated with gastroesophageal reflux disease, correlates abnormal amb
78 terms: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
79 lowing: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
80 esophageal mucosal injury, the diagnosis of gastroesophageal reflux disease currently rests on 24-ho
82 Mild-to-moderate chronic tissue injury in gastroesophageal reflux disease differentially affects m
83 es for detailed drugs (lipid-lowering drugs, gastroesophageal reflux disease drugs, antihypertensive
84 ugs in 8 drug classes (lipid-lowering drugs, gastroesophageal reflux disease drugs, diabetes drugs, a
85 of diseases such as functional dyspepsia and gastroesophageal reflux disease (e.g. vomiting, disorder
86 ecome available in the last 2 years for many gastroesophageal reflux disease endotherapies, providing
87 l gastrointestinal complications of obesity: gastroesophageal reflux disease, erosive esophagitis, Ba
89 This issue provides a clinical overview of gastroesophageal reflux disease, focusing on diagnosis,
90 gus (n = 1059) with those from subjects with gastroesophageal reflux disease (gastroesophageal reflux
91 .1; 95% confidence interval [CI], 2.9-12.9), gastroesophageal reflux disease (GERD) (RR, 1.9; 95% CI,
92 lin, leptin, and ghrelin are associated with gastroesophageal reflux disease (GERD) and Barrett's eso
93 We examined the incidence and predictors of gastroesophageal reflux disease (GERD) and dyspepsia and
96 might represent an alternative treatment of gastroesophageal reflux disease (GERD) and may provide d
97 has enormous potential for the treatment of gastroesophageal reflux disease (GERD) and other esophag
99 agement of extraesophageal manifestations of gastroesophageal reflux disease (GERD) and to compare th
100 Whether inflammation of the cardia indicates gastroesophageal reflux disease (GERD) and/or is a manif
101 Lung transplant recipients with documented gastroesophageal reflux disease (GERD) are at increased
103 hageal eosinophilia who were thought to have gastroesophageal reflux disease (GERD) but who did not r
105 t clear why only a minority of patients with gastroesophageal reflux disease (GERD) develop Barrett's
106 .4)] were included; 70% had been treated for gastroesophageal reflux disease (GERD) during infancy.
108 The histologic changes associated with acute gastroesophageal reflux disease (GERD) have not been stu
110 RAs) are frequently used in the treatment of gastroesophageal reflux disease (GERD) in children; howe
111 ull-thickness plication for the treatment of gastroesophageal reflux disease (GERD) in comparison wit
112 n (TF) can decrease or eliminate features of gastroesophageal reflux disease (GERD) in some patients
122 hageal mucosal injury across the spectrum of gastroesophageal reflux disease (GERD) is controversial.
124 illustrated by the Montreal classification, gastroesophageal reflux disease (GERD) is much more than
126 ump inhibitor (PPI) therapy in patients with gastroesophageal reflux disease (GERD) is reported in up
133 f an underlying acid peptic disorder such as gastroesophageal reflux disease (GERD) nor should it pre
134 is name, currently used for the treatment of gastroesophageal reflux disease (GERD) or completing Hel
137 has been developed for use in patients with gastroesophageal reflux disease (GERD) symptoms despite
138 discrepancy reflects a higher prevalence of gastroesophageal reflux disease (GERD) symptoms or a hig
141 phagus (cases) were matched to subjects with gastroesophageal reflux disease (GERD) without Barrett's
142 tailed critique of objective measurements of gastroesophageal reflux disease (GERD) would improve man
145 cluding Crohn disease (CrD), celiac disease, gastroesophageal reflux disease (GERD), and eosinophilic
146 frequency of irritable bowel syndrome (IBS), gastroesophageal reflux disease (GERD), and overactive b
147 e upper airways dysfunction syndrome (RUDS), gastroesophageal reflux disease (GERD), and rare cases o
148 rm use of PPIs for three common indications: gastroesophageal reflux disease (GERD), Barrett's esopha
149 sophagus has been proposed for patients with gastroesophageal reflux disease (GERD), but there is lit
150 eneral U.S. population and for patients with gastroesophageal reflux disease (GERD), diabetes mellitu
151 n several pathophysiological states, such as gastroesophageal reflux disease (GERD), functional dyspe
155 ients, who had a previous fundoplication for gastroesophageal reflux disease (GERD), underwent reoper
177 cations in adults were dysphagia (70.1%) and gastroesophageal reflux disease (GERD)/heartburn (27.1%)
178 and safety in the treatment of patients with gastroesophageal reflux disease (GERD); similarly, lapar
180 n 2002 and 2005 were matched to persons with gastroesophageal reflux disease (GERD; n = 316) and to p
181 ether patient-identified domains (dysphagia, gastroesophageal reflux disease [GERD], nausea/vomiting,
182 For the gastric cardia, the contributions of gastroesophageal reflux disease, H. pylori infection, an
183 rm outcome of ARS in pediatric patients with gastroesophageal reflux disease have shown good to excel
184 months improved in patients with normal GE (Gastroesophageal Reflux Disease Health-Related Quality o
185 is a laryngeal symptom that can be caused by gastroesophageal reflux disease; however, treatment outc
186 atients included degenerative joint disease, gastroesophageal reflux disease, hypertension, urinary s
187 imary laparoscopic antireflux surgery due to gastroesophageal reflux disease in adults (>18 years).
189 re of the height in meters - and symptoms of gastroesophageal reflux disease in persons of normal wei
190 related to the acidic environment caused by gastroesophageal reflux disease in the gastroesophageal
198 of the lower esophagus epithelium related to gastroesophageal reflux disease, is the strongest known
199 the AGA Institute on "Endoscopic Therapy for Gastroesophageal Reflux Disease." It was approved by the
201 ssue injury in the esophagus associated with gastroesophageal reflux disease may result in sensitizat
202 a diligent search for an esophageal etiology-gastroesophageal reflux disease, motility abnormalities,
203 itant chemical carcinogen treatment leads to gastroesophageal reflux disease, multilayered epithelium
204 nths after fundoplication for PPI-refractory gastroesophageal reflux disease (n = 14 270 degrees LPF
205 (n = 296 cases) were matched to persons with gastroesophageal reflux disease (n = 308) without Barret
206 ad EoE (n = 17), indeterminate EoE (n = 15), gastroesophageal reflux disease (n = 7), or normal esoph
208 on airway disease, including rhinosinusitis, gastroesophageal reflux disease, obesity and dysfunction
209 esophageal sphincter (LES) in patients with gastroesophageal reflux disease often has a low resting
210 is (EoE) was historically distinguished from gastroesophageal reflux disease on the basis of histolog
211 ceiving long-term proton pump inhibitors for gastroesophageal reflux disease or in patients with any
212 ated with EoE (or indeterminate EoE) but not gastroesophageal reflux disease or normal esophagus and
213 intravenous therapy in patients with severe gastroesophageal reflux disease or the Zollinger-Ellison
214 , including history of peptic ulcer disease, gastroesophageal reflux disease, or gastrointestinal ble
218 esents a new option for selected symptomatic gastroesophageal reflux disease patients who are intoler
220 5), and more had a >50% improvement in their gastroesophageal reflux disease quality of life score (n
223 ted with a relatively high rate of recurrent gastroesophageal reflux disease requiring treatment, dim
225 premorbid prevalence of anxiety, headaches, gastroesophageal reflux disease, sleep apnea, and infect
226 ministered a modified version of a validated gastroesophageal reflux disease-specific QOL tool to pat
228 44 patients (52%) had objective findings of gastroesophageal reflux disease, such as esophagitis.
230 uency energy delivery significantly improved gastroesophageal reflux disease symptoms and quality of
231 racticed, its value in patients with chronic gastroesophageal reflux disease symptoms is of unproven
232 ng upper endoscopy for patients with chronic gastroesophageal reflux disease symptoms to assess for B
233 men in the United Kingdom with histories of gastroesophageal reflux disease symptoms, assuming the p
234 ost commonly occurs in patients with chronic gastroesophageal reflux disease that involves recurring
236 ic conditions (hypertension, hyperlipidemia, gastroesophageal reflux disease, thyroid disease, diabet
237 inistration recently cleared new endoluminal gastroesophageal reflux disease treatments; however, no
241 atic treatment of nocturnal heartburn due to gastroesophageal reflux disease, was approved by the US
244 nflammation in squamous epithelium caused by gastroesophageal reflux disease, whereas intestinal meta
247 for 5.2 million visits annually, followed by gastroesophageal reflux disease, with 4.5 million visits
248 l adenocarcinoma (BEAC) is a complication of gastroesophageal reflux disease, with no effective chemo
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