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1 testinal epithelium, host immune system, and gastrointestinal microbiota.
2 hat affect the size and composition of their gastrointestinal microbiota.
3 e of inflammation, lipid metabolism, and the gastrointestinal microbiota.
4 n supraorganismal system, which includes the gastrointestinal microbiota.
5 g infant and instead targeted to its cognate gastrointestinal microbiota.
6 ffin and mast cells, enteric nerves, and the gastrointestinal microbiota.
7 ch of which involves an interaction with the gastrointestinal microbiota.
8 ew its application to the study of the human gastrointestinal microbiota.
9 esource to facilitate investigation of human gastrointestinal microbiota.
10 e effective through their impact on the host gastrointestinal microbiota and promotion of mucosal imm
11 nflammatory bowel disease (IBD) involves the gastrointestinal microbiota and some evidence that the m
12 e complex and bidirectional networks between gastrointestinal microbiota and their host, highlighting
13 in the gut generally show alterations in the gastrointestinal microbiota, and changes during weight l
14 itigation of colitis, maintenance of healthy gastrointestinal microbiota, and protected gut mucosal b
15  microbiome, potential interactions with the gastrointestinal microbiota, and the possibilities for m
16 and essential micronutrients, as well as the gastrointestinal microbiota, can change the balance betw
17 ration.These results indicate that the human gastrointestinal microbiota composition and function var
18    Studies in adults have indicated that the gastrointestinal microbiota could be involved in IBS.
19    Here we show that secretory products from gastrointestinal microbiota derived from a human donor s
20 ntify previously unidentified alterations in gastrointestinal microbiota-derived short-chain fatty ac
21 eveloped a mouse model of antibiotic-induced gastrointestinal microbiota disruption that is character
22      Preclinical research has shown that the gastrointestinal microbiota exhibits circadian rhythms a
23  and continues to evolve as knowledge of the gastrointestinal microbiota (GiMb) develops.
24                           Alterations in the gastrointestinal microbiota have been implicated in obes
25                                          The gastrointestinal microbiota have been shown to impact in
26           Dynamic balances exist between the gastrointestinal microbiota, host physiology, and diet t
27 s growing appreciation for the importance of gastrointestinal microbiota in many physiological and pa
28 ion may result in deleterious effects on the gastrointestinal microbiota, including reductions in bif
29  set the groundwork for interpreting how the gastrointestinal microbiota influence the health of the
30 ess incidence, how these products impact the gastrointestinal microbiota is not completely understood
31                       The composition of the gastrointestinal microbiota is strongly influenced by fa
32 dings highlighting the interplay between the gastrointestinal microbiota, its host and bacterial path
33 letion, and reestablishment of a physiologic gastrointestinal microbiota might be beneficial for this
34 ins elusive; however, it's hypothesized that gastrointestinal microbiota might play an important role
35 en correlated with large-scale imbalances in gastrointestinal microbiota, or 'dysbiosis'.
36 imed to identify microbial signatures in the gastrointestinal microbiota preceding NEC diagnosis in p
37 ition that may be associated with an altered gastrointestinal microbiota that promotes an immune envi
38  what is known to date with respect to human gastrointestinal microbiota - the niche which has been m
39 cted profoundly by the ability to modify the gastrointestinal microbiota through the rational deploym
40 iceptive influences; the contribution by the gastrointestinal microbiota to this balance has received
41          Vertebrates typically harbor a rich gastrointestinal microbiota, which has coevolved with th

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