ライフサイエンスコーパス: gastrointestinal motility

1 egulation of appetite, immune responses, and gastrointestinal motility.

2 It is an important physiologic regulator of gastrointestinal motility.

3 cells compromises epithelial maintenance or gastrointestinal motility.

4 urmeric-interacts with gut bacteria to alter gastrointestinal motility.

5 acrophages and enteric neurons that controls gastrointestinal motility.

6 to increase survival of enteric neurons and gastrointestinal motility.

7 this effect of H2S modulates CNS control of gastrointestinal motility.

8 system (ENS), but profound abnormalities in gastrointestinal motility.

9 ntribute to enteric inhibitory regulation of gastrointestinal motility.

10 aling in neurons might be developed to alter gastrointestinal motility.

11 uired for body weight homeostasis and normal gastrointestinal motility.

12 s nerve as a key component underlying normal gastrointestinal motility.

13 signaling in survival of enteric neurons and gastrointestinal motility.

14 ptide initially discovered as a regulator of gastrointestinal motility.

15 ide-expressing neurons and are essential for gastrointestinal motility.

16 t of increased beta-cell response or reduced gastrointestinal motility.

17 cells of Cajal (ICC) are required for normal gastrointestinal motility.

18 e Y (NPY) is an important peptide regulating gastrointestinal motility.

19 al physiological functions of these cells in gastrointestinal motility.

20 sting that female steroid hormones influence gastrointestinal motility.

21 utic agents for disorders involving impaired gastrointestinal motility.

22 r interstitial cells of Cajal in controlling gastrointestinal motility.

23 dentified two novel endogenous regulators of gastrointestinal motility.

24 ticipate in the Ca2+-dependent regulation of gastrointestinal motility.

25 iological effects and involves in regulating gastrointestinal motility.

26 teric plexus innervation density and reduced gastrointestinal motility.

27 y be of fundamental importance in regulating gastrointestinal motility.

28 to treat gastrointestinal bleeding, affects gastrointestinal motility.

29 the neuroendocrine factors controlling upper gastrointestinal motility, (2) noninvasive techniques to

30 that leukocyte-derived inducible NO inhibits gastrointestinal motility after manipulation and plays a

31 showing that CO is an integral regulator of gastrointestinal motility and an important factor in the

32 ptamine receptor (5-HT(4)R) agonists promote gastrointestinal motility and attenuate visceral pain, b

33 el with high strength to withstand long-term gastrointestinal motility and can be triggered to dissol

34 Although other methods exist for monitoring gastrointestinal motility and contractility, this study

35 Other acute effects include decreased gastrointestinal motility and food intake and increased

36 on between hunger, satiation, and integrated gastrointestinal motility and hormonal responses in morb

37 Intraduodenal lipid modulates gastrointestinal motility and hormone release and suppre

38 agastrically to assess its effect on fasting gastrointestinal motility and hunger ratings, motilin an

39 Tachykinins contribute to the control of gastrointestinal motility and modulation of somatic and

40 There was a significant delay in gastrointestinal motility and reduced numbers of nitrerg

41 esis that GAL-R1 mediates galanin actions on gastrointestinal motility and secretion by modulating th

42 e multiple physiological responses including gastrointestinal motility and secretion, glucose homeost

43 fects of food, meal intake and microbiota on gastrointestinal motility and sensation are discussed.

44 Altered gastrointestinal motility and sensation, changed activit

45 c ileus characterized by total inhibition of gastrointestinal motility and were excluded from further

46 sensitization, contributing to vasodilation, gastrointestinal motility, and airway resistance.

47 ceral perception, cardioautonomic responses, gastrointestinal motility, and brain activation patterns

48 se and xylose appearance after oral loading, gastrointestinal motility, and glucose transepithelial t

49 , airway mucus secretion, dysphagia, altered gastrointestinal motility, and itchy swollen skin.

50 les that act on enteric neurons to influence gastrointestinal motility, and metabolites that stimulat

51 late diverse biological processes, including gastrointestinal motility, angiogenesis, and circadian r

52 t have been shown to play important roles in gastrointestinal motility, angiogenesis, circadian rhyth

53 ation of enteric neurons, but its effects on gastrointestinal motility are not well characterized.

54 een used in the study of normal and abnormal gastrointestinal motility, as well as for the characteri

55 a, using immunofluorescence, immunoblot, and gastrointestinal motility assays.

56 rity to withstand the forces associated with gastrointestinal motility, be triggerable to address any

57 Gastrointestinal motility causes movement of food during

58 1(flox/flox); Phox2b-Cre mice have increased gastrointestinal motility compared with controls.

59 They also suggest that increased gastrointestinal motility contributes to elimination of

60 Observations suggest that constitutive gastrointestinal motility depends more on neuronal than

61 : Cell therapy offers the potential to treat gastrointestinal motility disorders caused by diseased o

62 Cell therapy has the potential to treat gastrointestinal motility disorders caused by diseases o

63 s of muscles from patients with a variety of gastrointestinal motility disorders have raised the exci

64 ay be a potential therapeutic target for the gastrointestinal motility disorders related to diabetes.

65 ptions currently available for patients with gastrointestinal motility disorders, especially patients

66 stinal pacemaker cells, underlie a number of gastrointestinal motility disorders, including diabetic

67 tients with Hirschsprung disease suffer from gastrointestinal motility disorders, whereas Waardenburg

68 of the forebrain/midbrain and impairment of gastrointestinal motility due to a reduction in postmito

69 ation between subjective hunger feelings and gastrointestinal motility during the MMC is largely unkn

70 ld enhance effects of intraduodenal lipid on gastrointestinal motility, gut hormones, appetite, and e

71 eart rate, blood pressure, respiratory rate, gastrointestinal motility, hormone release, and body tem

72 The effects of CT on gastrointestinal motility, however, have not been adequa

73 Recovery of fasted gastrointestinal motility in dogs is equally rapid after

74 val of neurons in the intestine, but altered gastrointestinal motility in female, but not male, mice.

75 s and microbes increases neuron survival and gastrointestinal motility in mice.

76 The authors investigate the recovery of gastrointestinal motility in the fed and fasted state af

77 We measured epithelial barrier function and gastrointestinal motility in these mice and littermate c

78 Finally, Giardia infection increased gastrointestinal motility in wild-type mice, but not in

79 Altered gastrointestinal motility is associated with significant

80 A requirement for normal gastrointestinal motility is the tight regulation of ion

81 Gastrointestinal motility is the underlying program that

82 , significance, pitfalls, and guidelines for gastrointestinal motility measurements by scintigraphy,

83 The A-ZIP/F-1 mice also did not show reduced gastrointestinal motility or 24-h food intake during bet

84 ant developments in our approach to studying gastrointestinal motility over the past few years has be

85 actual effect of a laparoscopic approach on gastrointestinal motility, particularly fed-state motili

86 and microcephaly, neurodevelopmental delay, gastrointestinal motility problems, and respiratory comp

87 tion of cannabinoid CB(1) receptors inhibits gastrointestinal motility, propulsion, and transit, wher

88 ce the expression of nNOS, a key molecule in gastrointestinal motility regulation.

89 HT(4) agonist for the potential treatment of gastrointestinal motility-related disorders.

90 s to be designed to therapeutically modulate gastrointestinal motility, secretion, and sensation.

91 by years, and include anosmia, problems with gastrointestinal motility, sleep disturbances, sympathet

92 in tandem, leading to disturbances in infant gastrointestinal motility that manifests clinically as c

93 advent of antidiabetes therapies that alter gastrointestinal motility, the effect of bariatric surge

94 e effects that genetic knockouts may have on gastrointestinal motility; therefore, an understanding o

95 Gastrointestinal motility was assessed by measuring gast

96 the vagus nerve on metabolic homeostasis and gastrointestinal motility, we generated and characterize

97 n of pancreatic enzymes and secretin-induced gastrointestinal motility, which are mediated by vago-va

98 P-1 has effects on glucose metabolism and on gastrointestinal motility years after RYGB.