ライフサイエンスコーパス: gastrointestinal mucosa

1 mbryo, to turnover in the homeostasis of the gastrointestinal mucosa.

2 nic infection, few have explored Treg in the gastrointestinal mucosa.

3 ortion of intraepithelial lymphocytes in the gastrointestinal mucosa.

4 moting the resolution of inflammation in the gastrointestinal mucosa.

5 rologists to obtain in-vivo histology of the gastrointestinal mucosa.

6 e of inducing growth in normal and malignant gastrointestinal mucosa.

7 ities, including growth-promoting actions on gastrointestinal mucosa.

8 or, and the number of tumors per 1000 mm2 of gastrointestinal mucosa.

9 ecreted CA and exists in both saliva and the gastrointestinal mucosa.

10 ns in terminally differentiated cells in the gastrointestinal mucosa.

11 Aminobiphosphonates can irritate the upper gastrointestinal mucosa.

12 course), which occurs across the oral and/or gastrointestinal mucosa.

13 real-time, microscopic visualization of the gastrointestinal mucosa, allowing an endoscopic approach

14 olvement in lymphocyte recruitment to normal gastrointestinal mucosa and associated lymphoid tissue.

15 A-producing plasma cell populations in human gastrointestinal mucosa and bone marrow and the specific

16 ine and etoposide in the bone marrow and the gastrointestinal mucosa and emphasize the potential for

17 ing of vaccine-elicited T lymphocytes to the gastrointestinal mucosa and for vaccine protective effic

18 6 appears to be most highly expressed in the gastrointestinal mucosa and in kidney and lung.

19 so reduced proviral DNA in peripheral blood, gastrointestinal mucosa and lymph nodes without the deve

20 ressing endings were seen clearly within the gastrointestinal mucosa and myenteric plexus, respective

21 ammadelta T cells are primarily found in the gastrointestinal mucosa and play an important role in th

22 and function in blood, spleen, lymph nodes, gastrointestinal mucosa, and bronchoalveolar lavage of u

23 eptor (EGFR) triggers mitogenic signaling in gastrointestinal mucosa, and its expression is also upre

24 ed levels of proviral DNA in lymph nodes and gastrointestinal mucosa as compared with treatment at la

25 Galpha(t)-(2), also was demonstrated in the gastrointestinal mucosa as well as in STC-1 cells, as re

26 e sites of origin include the oral, lung and gastrointestinal mucosa, as data consistent with this hy

27 Functional analysis of the gastrointestinal mucosa-associated lymphoid tissue revea

28 The organisms penetrate the gastrointestinal mucosa by unknown mechanisms and are ph

29 gnificant accumulation of eosinophils in the gastrointestinal mucosa compared with control mice.

30 The gastrointestinal mucosa contains a complex network of ly

31 The gastrointestinal mucosa contains an intact immune system

32 umor size, the proportion of the area of the gastrointestinal mucosa covered with tumor, and the numb

33 of rhIL-11 to preserve the integrity of the gastrointestinal mucosa during cancer treatment regimens

34 the poor bioavailability of 2-MeOE2, as the gastrointestinal mucosa expresses high levels of 17beta-

35 protein contributes to the protection of the gastrointestinal mucosa from injury by strengthening and

36 The gastrointestinal mucosa harbors the majority of the body

37 The gastrointestinal mucosa has proven to be an interesting

38 ary, we demonstrate that UGTs are located in gastrointestinal mucosa, have vast overlapping activitie

39 glands, lymphoid organs, bone, bone marrow, gastrointestinal mucosa, heart, and kidneys.

40 IV-specific cellular immune responses to the gastrointestinal mucosa in a primate model.

41 The gastrointestinal mucosa is a major lymphoid tissue reser

42 Gastrointestinal mucosa is an early target of HIV and a

43 As the gastrointestinal mucosa is an important site of HIV tran

44 c stem cell transplantation (allo-HSCT), the gastrointestinal mucosa is damaged, and colonizing bacte

45 The human gastrointestinal mucosa is exposed to a diverse normal m

46 The upper gastrointestinal mucosa is exposed to endogenous and exo

47 The gastrointestinal mucosa is exposed to numerous chemical

48 The gastrointestinal mucosa is the primary site where human

49 (HIV) in peripheral blood, but the effect in gastrointestinal mucosa is uncertain.

50 emokine receptor expression in the blood and gastrointestinal mucosa; mucosal cells are predominantly

51 samples of normal pancreas (n = 11), normal gastrointestinal mucosa (n = 22), resected pancreas canc

52 on was also seen on organisms colonizing the gastrointestinal mucosa of mice, indicating that the ant

53 etabolism of SCFAs affected apoptosis in the gastrointestinal mucosa of the mouse, and whether this a

54 Therefore, the metabolism of SCFAs by the gastrointestinal mucosa plays a role in modulating apopt

55 Epithelial and other cells of the gastrointestinal mucosa rely on both luminal and bloodst

56 conclude that, based on these findings, the gastrointestinal mucosa represents a favored target for

57 (BaP) and the MCP in the bone marrow and the gastrointestinal mucosa, respectively.

58 may be a common mechanism by which the upper gastrointestinal mucosa responds to noxious insults.

59 bout the effects of individual NSAIDs on the gastrointestinal mucosa, risk factors for sustaining an

60 e chemokine receptor CCR9 to localize to the gastrointestinal mucosa; their respective ligands, mucos

61 responsible for the unique tolerance of the gastrointestinal mucosa to proinflammatory stimuli.

62 (renal proximal tubular epithelial cells and gastrointestinal mucosa) to damage to renal glomerular t

63 press toxicity to the bone marrow and to the gastrointestinal mucosa when used at therapeutic doses,

64 owever, these neurons are not present in the gastrointestinal mucosa, where 5-HT initiates peristalti

65 the host-toxin response at the level of the gastrointestinal mucosa, where STEC infection begins.

66 that induces potent immune responses in the gastrointestinal mucosa would be highly desirable.