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1 with placebo after the resection of primary gastrointestinal stromal tumour.
2 y be performed in all patients with advanced gastrointestinal stromal tumour.
3 ective in first-line treatment of metastatic gastrointestinal stromal tumour.
4 lacebo after resection of localised, primary gastrointestinal stromal tumour.
5 d with sunitinib in patients with metastatic gastrointestinal stromal tumours.
6 transformation of ICC and the development of gastrointestinal stromal tumours.
7 pared with placebo in patients with advanced gastrointestinal stromal tumour after failure and discon
8 in the treatment of recurrent or metastatic gastrointestinal stromal tumours and is now being tested
11 ts had complete gross resection of a primary gastrointestinal stromal tumour at least 3 cm in size an
12 nts with metastatic renal-cell carcinoma and gastrointestinal stromal tumours, but concerns have aris
14 approved as third-line therapy for advanced Gastrointestinal Stromal Tumour (GIST) at a starting dos
16 ave proven clinical benefit in patients with gastrointestinal stromal tumours (GIST), but almost all
18 initiating events, fostering hyperplasia in gastrointestinal stromal tumours (GISTs), and additional
21 on of isolated metastatic colorectal cancer, gastrointestinal stromal tumours, neuroendocrine cancers
22 apparent antitumour activity was noted in a gastrointestinal stromal tumour, papillary thyroid cance
24 e intracellular Bcr-Abl tyrosine kinase, and gastrointestinal stromal tumours, where it targets eithe
25 kinase inhibitor, in patients with advanced gastrointestinal stromal tumour who were resistant to or
26 atients with imatinib-resistant, metastatic, gastrointestinal stromal tumours who had been enrolled i
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