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1 , many of which were not known previously to gastrulate.
2 s mutants entirely lack mesoderm and fail to gastrulate.
3 form an abnormal egg cylinder which does not gastrulate.
4 en they became growth-arrested and failed to gastrulate.
5 wall when most of the embryos have begun to gastrulate.
6 and fail to grow, to become patterned or to gastrulate.
9 Western analysis, resulted in the failure to gastrulate, absence of the gut and an animalized phenoty
10 cation-checkpoint defective but cellularize, gastrulate and activate high levels of zygotic gene expr
11 nic tissue allows the double null embryos to gastrulate and begin organogenesis, suggesting that extr
12 s of SRF(LacZ/)(+) "knock-in" mice failed to gastrulate and form mesoderm, similar to the findings of
15 nsistent developmental defects, a failure to gastrulate, and embryonic lethality, including changes i
16 Unlike Bmpr1a-null embryos, which fail to gastrulate, Bmpr-MORE embryos initiate gastrulation, but
17 velopment reveals that C5a knockdown embryos gastrulate, but approximately 90% develop a prolapse of
18 end on common actomyosin-based mechanisms to gastrulate, but different cell fate regulators, and, sur
20 , like sea urchins, has an early embryo that gastrulates by invagination, but like vertebrates, has a
21 evelopment involves the aggregation of newly gastrulated cells into epithelial fields, as a prelude t
26 e mesendoderm cells were injected into early gastrulating chick embryos, which revealed that they int
30 ryos homozygous for gammaGCS-HS(tm1) fail to gastrulate, do not form mesoderm, develop distal apoptos
31 gination of the mesodermal primordium in the gastrulating Drosophila embryo, the internalized cells m
33 re we apply particle tracking velocimetry in gastrulating Drosophila embryos to measure the movement
36 ated functions for the AVE in organizing the gastrulating embryo and indicate that visceral endoderm-
39 e derives from a small group of cells in the gastrulating embryo, known as "the organizer" in recogni
44 tiating embryonic stem cells, and in vivo in gastrulating embryos, the lineage specification of early
52 rom eggs depleted of xSmad8(11) mRNA fail to gastrulate; instead, at the time of gastrulation, they i
54 l genetic fate mapping, that Mesp1+ cells in gastrulating mesoderm are rapidly specified into committ
55 y, Axial Protocadherin (AXPC), it subdivides gastrulating mesoderm into paraxial and axial domains.
56 that oriented tissue strain generated by the gastrulating mesoderm plays a major role in determining
57 aused an increase in the population of newly gastrulated mesodermal (NGM) cells that express the tran
58 establish the anterior-posterior axis of the gastrulating mouse embryo and is necessary later for mes
59 is expressed in the primitive streak of the gastrulating mouse embryo and is required for paraxial m
65 the epiblast and nascent Flk1(+) mesoderm of gastrulating mouse embryos using single-cell RNA sequenc
69 heckpoint defective and fail to cellularize, gastrulate or to initiate high-level zygotic transcripti
71 en they became growth arrested and failed to gastrulate, pointing to the early essential role for hep
73 e Wnt and Bmp signaling pathways pattern the gastrulating vertebrate embryo using a network of secret
77 ogs scn5Laa and scn5Lab were detected in the gastrulating zebrafish embryo and subsequently in the em
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