ライフサイエンスコーパス: gel retardation assay

1 e yegM promoter was demonstrated in vitro by gel retardation assay.

2 This result is further supported by gel retardation assay.

3 and DNA binding ability of Cbfa1 based on a gel retardation assay.

4 igrating specific DNA-protein complexes in a gel retardation assay.

5 ance the DNA binding activity of EmBP-1 in a gel retardation assay.

6 sensitive and specific liquid hybridization gel retardation assay.

7 d abasic site was supershifted by XRCC1 in a gel retardation assay.

8 ed by the operator variants, and in vitro by gel-retardation assay.

9 ity DNA binding sequences were identified by gel retardation assays.

10 rms that include these exons fail to bind in gel retardation assays.

11 inability of each protein to bind to DNA in gel retardation assays.

12 anscription, using cotransfection assays and gel retardation assays.

13 electron microscopy and competitive assembly gel retardation assays.

14 rom each of these cell lines are examined by gel retardation assays.

15 site binding studies to be undertaken using gel retardation assays.

16 Moreover, in gel retardation assays, a 38-bp oligomer in the shortest

17 However, data derived from gel retardation assays, a modified ligation-mediated pol

18 Gel retardation assays also demonstrated that staurospor

19 In gel retardation assays, an RCS cell-specific protein and

20 the effects of mutation on competition in a gel retardation assay and on promoter activity were stud

21 ts with single and multiple PRH sites, using gel retardation assays and DNase I and chemical footprin

22 s on Fur binding was examined using in vitro gel retardation assays and DNase I footprinting experime

23 Gel retardation assays and DNase I footprinting studies

24 rate and product analogs was evaluated using gel retardation assays and footprinting experiments.

25 d by chromatin immunoprecipitation, in vitro gel retardation assays and gene expression analyses.

26 Gel retardation assays and mutational analysis demonstra

27 nd to cellular SSDB factors by use of common gel-retardation assays and PCR.

28 ational analyses using reporter gene assays, gel retardation assays, and immunostaining experiments,

29 d E7 genes by in vitro DNase I footprinting, gel retardation assays, and transfection studies, we als

30 As estimated by a gel retardation assay, binding saturates at a stoichiome

31 In a gel retardation assay both the phosphatidic acid-depende

32 Quantitative gel retardation assays confirm this observation and show

33 ounds of random oligonucleotide selection in gel retardation assays, coupled with PCR amplification o

34 Gel retardation assay demonstrated that H2O2 (hydrogen p

35 Gel retardation assays demonstrated that an increase in

36 Gel retardation assays demonstrated that in vitro transl

37 Gel retardation assays demonstrated the binding of homog

38 Gel-retardation assays demonstrated binding of IHF to th

39 A gel retardation assay detected two sites within the pali

40 Gel retardation assays employing T.brucei mRNAs demonstr

41 Gel retardation assay experiments showed that DNA fragme

42 pearance of an NF-kappaB complex as shown in gel retardation assays; however, induction of the HSR by

43 An RNA gel retardation assay identified a 37-nucleotide cognate

44 irected mutagenesis experiments and in vitro gel retardation assays identified Ets and GATA elements

45 Gel retardation assays identified Sp1, Sp3 and DeltaSp3

46 lex RT/DNA/dNTP could be readily detected by gel retardation assays if the DNA had a template overhan

47 Gel retardation assays indicated that the Ap1/NF-E2 moti

48 Gel retardation assays indicated that the master negativ

49 esults from P. aeruginosa mutant studies and gel retardation assays indicated that this regulation wa

50 Gel retardation assays involving the purified QscR have

51 and monitoring changes to RepB binding in a gel-retardation assay, it was shown that the central, GC

52 In gel retardation assays, Mot21 and MotA bind DNA containi

53 Using DNA competition and gel retardation assays, NaeI-L43K showed reduced affinit

54 Here, circular permutation gel retardation assays provide evidence that the polymer

55 Consistent with this observation, in gel retardation assays, purified TraM abolished the DNA

56 Gel retardation assays reveal ribonucleoprotein complexe

57 Gel retardation assays revealed that another islet-enric

58 Gel retardation assays revealed that full-length GTF3, a

59 Gel retardation assays revealed that the reason for this

60 acting factors, in vitro transcriptional and gel retardation assays revealed that the RpoN-recognized

61 Gel retardation assays revealed that the three proteins

62 Gel retardation assays revealed time-dependent, hypoxia-

63 As assessed by gel retardation assays RpoAAt, but not RpoAEc, formed a

64 rease the binding of E2 to non-specific DNA, gel retardation assays show that these ions have no effe

65 ia-mediated activation of C/EBPbeta-NF-IL-6, gel retardation assays showed no change in the intensity

66 Gel retardation assays showed PPARgamma bound hexamers 3

67 Gel retardation assays showed that dephosphorylation of

68 Gel retardation assays showed that IL-4 induced the form

69 Gel retardation assays showed that nuclear proteins from

70 Gel retardation assays showed that the F-F-x-R architect

71 Gel retardation assays showed that while the mutant MITF

72 n gene transfection and proteolytic clipping gel retardation assays suggest that the AP-1 flanking se

73 In UV cross-linking, filter binding, and gel retardation assays, the MSH2-msh6-F337A complex disp

74 In gel retardation assays, the specific binding seen with e

75 ogen receptor-specific antisera and shown by gel retardation assay to bind oligonucleotides containin

76 erized tomato homeobox protein, was shown by gel retardation assay to interact with the promoter of L

77 Lastly, we used gel retardation assays to show that alterations in trans

78 We identified this complex by gel retardation assay using short, linear fragments and

79 Gel retardation assays using a probe containing the repe

80 Gel retardation assays using extracts from both Jurkat T

81 Gel retardation assays using the Hol75 DNA confirm these

82 To understand this phenomenon better, a gel retardation assay was used to measure the equilibriu

83 A gel retardation assay was used to purify a potential reg

84 y the mechanistic details of this process, a gel-retardation assay was used to measure the dissociati

85 oupling novel immunoblotting techniques to a gel retardation assay, we observed that nucleosome cores

86 Using a gel retardation assay, we show here that nuclear extract

87 Furthermore, using gel retardation assay, we show that 12-O-tetradecanoylph

88 Using gel retardation assays, we mapped an IE2-binding site wi

89 Using gel retardation assays, we now demonstrate that the bind

90 Using gel retardation assays, we show that the hierarchy of E2

91 nce, circular dichroism, and electrophoretic gel retardation assays were carried out on recombinant 8

92 wo-dimensional (2D) and one-dimensional (1D) gel retardation assays were used to analyze occludin pho

93 Gel retardation assays were used to investigate the pair

94 or the AC-rich region was also shown using a gel retardation assay with an ACBF recombinant protein e

95 extracts from these strains were inactive in gel retardation assays with a 158-bp fragment of the DNA

96 ter with increased methylation level, and in gel retardation assays with an oligonucleotide containin

97 Gel retardation assays with crude cell extracts confirme

98 essing the recombinant proteins were used in gel retardation assays with double stranded oligonucleot

99 Gel retardation assays with ER+ and ER- cell lines ident

100 Gel retardation assays with extracts from the cells grow

101 Gel retardation assays with extracts from the complement

102 Gel retardation assays with mutant MotA boxes are consis

103 Electrophoretic gel retardation assays with purified M.RSR:I were perfor

104 rmined by RNAII and RNAIII transcription and gel retardation assays with the P2 and P3 promoters of a