ライフサイエンスコーパス: gene frequency

1 ons exist at this locus at a relatively high gene frequency.

2 ins and losses were determined by changes in gene frequencies.

3 ially for human populations from blood-group gene frequencies.

4 as and have discovered latitudinal clines in gene frequencies.

5 to be the primary force shaping chloroplast gene frequencies.

6 ngular spikes (Dirac delta functions) at the gene frequencies 0 and 1.

7 Methods for comparing gene frequencies across large, epidemiologically defined

8 duction scenario, we profiled hop-resistance gene frequencies and bacterial and fungal communities in

9 ism, sustained by random processes acting on gene frequencies and population size.

10 rtant complex traits; and to determine their gene frequencies and their homozygous, heterozygous, epi

11 ges in gene expression as well as changes in gene frequency and genetic isolation.

12 Although dependent on marker gene frequency and other factors, efficiency for hyperno

13 ponses in population dynamics, life history, gene frequencies, and morphology in a number of species.

14 t different reproductive modes do not affect gene frequency at mutation-selection equilibrium if muta

15 The understanding that gene frequencies change at random by genetic drift, even

16 new insight into the factors contributing to gene frequency change in this species, and it serves to

17 Historical datasets documenting changes to gene frequency clines are extremely rare but provide a p

18 ctive population size from temporally spaced gene frequency data.

19 ve great promise for capturing signatures of gene frequency difference between human subpopulations,

20 particular, F(ST) is an appropriate index of gene-frequency differentiation if and only if the geneti

21 We investigate the gene frequency distribution of a population at mutation-

22 Analysis at the level of individual loci and gene frequency distributions has had relatively little i

23 f multilocus quantitative genetic models and gene frequency distributions, focusing on the potential

24 ding on the meaning of inbreeding and random gene frequency drift.

25 that a property of the deterministic part of gene frequency dynamics determines when fixation and los

26 The forward diffusion equation for gene frequency dynamics is solved subject to the conditi

27 n-specific protection tends to stabilize the gene frequency dynamics of specific defense.

28 The gene frequency for the inactivating polymorphism (0.31)

29 expression of the Neurospora circadian clock gene frequency (frq), can trigger singularity behavior i

30 ves transcription of the circadian pacemaker gene frequency (frq), whose gene product, FRQ, as a part

31 of codons in the Neurospora circadian clock gene frequency (frq).

32 at activates expression of the central clock gene frequency (frq); FRQ protein is hypothesized to fee

33 hic L1 elements in the human population with gene frequencies greater than 0.05 is between 3000 and 1

34 rty of nucleotide sequences independently of gene frequency, i.e. the 'success' in the gene pool that

35 rol studies, we compared HLA class I and KIR gene frequencies in 250 classic (non-AIDS) KS cases, 280

36 Comparisons of gene frequencies in ALL case and control patients showed

37 Gene frequencies in AML patients and healthy controls we

38 online repository for the storage of immune gene frequencies in different populations across the wor

39 ted red blood cell (RBC) disorders with high gene frequencies in malaria-endemic regions, the distrib

40 The equilibrium distribution of gene frequencies in structured populations is known sinc

41 modes of microbial transmission and spoilage-gene frequency in a commercial food-production scenario,

42 g-1, which was allelic and present at a high gene frequency in gorillas but absent from other primate

43 s have been suggested for changes in melanic gene frequency in the peppered moth Biston betularia and

44 ing the ivory genotype to a geographic-based gene frequency map, developed separately.

45 arent occurrence is explained by statistical gene frequency models of kin selection.

46 Within specific mutated genes, frequency, mutation hotspot residues, in silico p

47 rd diffusion equation is thus solved for all gene frequencies, namely the absorbing frequencies of 0

48 trated that the sites are present in overall gene frequencies of .39 for HindIII, .04 for BamHI, and

49 sequence and deletion analyses gave disease-gene frequencies of 40% for CCM1, 38% for CCM2, 6% for C

50 s each coding region, we have determined the gene frequencies of each allele in a random donor popula

51 The gene frequencies of each of the gene polymorphisms asses

52 Viability selection will change gene frequencies of loci controlling fitness.

53 ermination of significant differences in the gene frequencies of LSRa, LSRg, VNGa, and VNGg among Cau

54 Gene frequencies of the polymorphic sites were also stud

55 Gene frequencies of these alleles in a normal population

56 The gene frequency of -52T in a random Caucasian population

57 of 192 control chromosomes, for an estimated gene frequency of .01+/-.007.

58 K program run at 90% penetrance and a myopia gene frequency of 0.0133.

59 The G(-)92 sequence has a gene frequency of 0.15 in a typical Caucasian population

60 We used changes of gene frequency of 2,326 single-feature polymorphisms (SF

61 among the white population worldwide, with a gene frequency of about 10% and a frequency of homozygos

62 A gene frequency of approximately 10% was found for delta

63 ratic Republic of the Congo have the highest gene frequency of glycophorin B-null in the world, raisi

64 The gene frequency of IXp in male blood donors is 0.25.

65 No estimates of the gene frequency of nonclassical 21-hydroxylase deficiency

66 Based on the gene frequency of the 1529A mutation in the white popula

67 We determined the gene frequency of the polymorphic variant of TNFalpha in

68 The gene frequency of the TNFalpha -308A polymorphism was hi

69 eption of the RASSF1A promoter of the RASSF1 gene, frequencies of aberrant methylation were significa

70 f 0 and 1 along with the continuous range of gene frequencies on the interval (0,1) that excludes the

71 odel of the same heritability h(2) = 0.5 and gene frequency (p = 0.1).

72 re relatively sensitive to assumptions about gene frequency, particularly when gene frequency was low

73 demiological and clinical studies that these gene frequencies reflect selection by, and protection fr

74 This high gene frequency suggests that these mutations confer a se

75 ta has lower genetic diversity and different gene frequencies than the monogyne form, suggesting that

76 and differential equations) used to describe gene frequency trajectories with the mathematics of opti

77 RQH because they do not result in cycling of gene frequencies, unlike a matching allele mechanism.

78 difference between cases and controls in KIR gene frequencies was a trend toward fewer activating KIR

79 The Delta 32 gene frequency was 0.026 for HIV-1-seropositive women an

80 In Caucasians, Db gene frequency was 14%, similar to Db protein from parot

81 ions about gene frequency, particularly when gene frequency was low.

82 zing differentiation from inferred ancestral gene frequencies, we obtained results that are fully con

83 In white participants, the gene frequencies were 0.063 for the C282Y mutation, 0.15

84 In that study, changes in algal gene frequencies were inferred from their effects on pop

85 Gene frequencies were lower in other ethnic groups.

86 ethylation index, a reflection of all of the gene frequencies, with the presence of SV40 large T-anti

87 ns for a complete dynamical treatment of all gene frequencies within a diffusion approximation framew