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1 g mice carrying altered copies of a targeted gene ('knockout mice').
2 ood pressure (shared by some but not all BBS gene knockout mice).
3 r mice, an adoptive cell transfer model, and gene knockout mice.
4 ble for IL-10 upregulation was determined in gene knockout mice.
5 on RI-stimulated mast cells derived from btk gene knockout mice.
6 ere markedly reduced in TNF receptor 1 and 2 gene knockout mice.
7 d -0.6 kb of upstream sequence into beta3-AR gene knockout mice.
8 g the generation and analysis of conditional gene knockout mice.
9 evel of glutathione peroxidase using GSHPx-1 gene knockout mice.
10 ng-term potentiation between control and PrP gene knockout mice.
11 s can predict the lethal phenotype of single-gene knockout mice.
12 P4A mRNAs was also blocked in the PPAR alpha gene knockout mice.
13 where possible, testing of islet tissue from gene knockout mice.
14 out mice and sphingomyelin synthase 2 (Sms2) gene knockout mice.
15 rkedly reduced in tamoxifen-inducible Nkx2-5 gene knockout mice.
16 n responses, studies were performed in COX-2 gene knockout mice.
17 nt with neutralizing antibodies and cytokine gene knockout mice.
18 through use of the appropriate transgenic or gene knockout mice.
19 ing into question negative results in single-gene knockout mice.
20 against this parasite has been studied using gene knockout mice.
21 ological inhibitors of iNOS, as well as iNOS gene knockout mice.
22 rmed with the pharmacological inhibition and gene knockout mice.
23 ions in glucose metabolism in transgenic and gene knockout mice.
24 uli infection was further evaluated by using gene-knockout mice.
25 on using pharmacological inhibitors and iNOS gene-knockout mice.
26 neutrophils obtained from A(2A)AR and A(3)AR gene "knockout" mice.
29 /6 major histocompatibility complex class II gene knockout mice and CD4 cell-depleted C3H mice (i.e.,
31 d lipolysis was observed in both adiponectin gene knockout mice and primary adipocytes from these mic
33 ll as in plasma samples from six cholestatic gene knockout mice and six age- and gender-matched wild-
34 almitoyltransferase (SPT) subunit 2 (Sptlc2) gene knockout mice and sphingomyelin synthase 2 (Sms2) g
36 antibody-deficient muMT mice (immunoglobulin-gene knockout mice), and CD4(+) spleen T cells from immu
39 tivator of transcription 4 (STAT4) and STAT6 gene knockout (-/-) mice as recipients of fully mismatch
40 ntribution to its etiology, including single-gene knockout mice associated with diaphragmatic defects
43 feration were compared in wild-type and Zeb1 gene knockout mice by immunostaining, real-time PCR, and
45 ed, major histocompatibility complex class I gene knockout mice compared with no deaths for wild-type
49 e production in Schistosoma mansoni-infected gene knockout mice deficient in either CD8 lymphocytes o
51 % compared with only 49% in control beta3-AR gene knockout mice, demonstrating that the human beta3-A
52 indicated that IFN-gamma-/- and IL-12 p40-/- gene knockout mice developed CTL responses equivalent to
53 n an unexpected result, lenses in the alphaB gene knockout mice developed normally and were remarkabl
54 -1(-/-)) and heterozygous (Flt-1(+/-)) Flt-1 gene knockout mice display increased endothelial cell pr
56 ut mice into R. australis-infected IFN-gamma gene knockout mice dramatically reduced the infectious r
58 , fetal liver-derived macrophages from SHIP2 gene knockout mice enhanced activation of Akt in respons
59 al intracerebral hemorrhage, whereas AbetaPP gene knockout mice exhibited reduced hemorrhage size.
60 ontrol, perforin gene-knockout, and granzyme gene-knockout mice exposed by the realistic pulmonary ro
61 le wild-type mice and in the original single gene knockout mice for ERalpha (ERalphaKO(Chapel Hill) [
62 promoter and interbreeding of transgenic and gene knockout mice for generating a mouse strain that ex
66 pared with the wild-type controls, the B(2)R gene knockout mice had a higher baseline BP (109.7+/-1.1
67 nt in E. faecalis infection, since IFN-gamma gene knockout mice had reduced mortality and massive coa
68 ce, interleukin-4, but not interferon-gamma, gene knockout mice had significantly less S. aureus bind
69 Recently, the use of transgenic mice and gene-knockout mice has allowed investigators to evaluate
71 revious in vitro studies with transgenic and gene-knockout mice have shown that lenses with elevated
75 llicular pathway is significantly delayed in gene knockout mice in which both the endothelial and neu
76 similar deficit has been reported in Hoxa-1 gene knockout mice in which pattern formation of the hin
78 and susceptible mice as well as in cytokine gene knockout mice infected with Trypanosoma brucei rhod
79 of wild type BALB/C mice or interferon-gamma gene knockout mice, interleukin-4, but not interferon-ga
80 r of immune CD8 T lymphocytes from IFN-gamma gene knockout mice into R. australis-infected IFN-gamma
81 subset depletion studies and the analysis of gene knockout mice, it is evident that CD8(+) T cells co
86 ouse embryonic fibroblast cells derived from gene knockout mice lacking the gene for RNase L(-/-) or
87 lyceride metabolism in lysosomal acid lipase gene knockout mice (lal-/-) results in severe pathogenic
88 Based on studies with LPS-nonresponder and gene-knockout mice, LPS-induced proliferation of CD44(hi
91 d a significant increase in opacification in gene-knockout mice relative to control animals of the sa
92 ice were adoptively transferred into various gene-knockout mice rendered T cell-deficient by subletha
93 contrast, deletion of PN2/AbetaPP in AbetaPP gene knockout mice resulted in a significant increase in
96 e organ of Corti of homozygous Cx26 and Cx30 gene knockout mice show that cochlear hair cells degener
97 ing to echocardiography, we found that Efnb3 gene knockout mice showed enhanced constriction in the c
98 Accordingly, cultured T-cells from Spry1 gene knockout mice showed increased proliferation in res
99 -deficient Tc2 cells generated from perforin gene knockout mice showed no differences in therapeutic
100 onsisted of a single population spike in PrP gene knockout mice similar to that recorded from control
101 manganese superoxide anion dismutase (SOD2) gene-knockout mice (SOD2+/-), in which SOD2 activity is
102 ol mice only until day 8 p.i., in all of the gene knockout mice studied except those lacking C3 and C
106 eased after pulmonary infection of IFN-gamma gene knockout mice, suggesting a protective role for IFN
107 otective effect of A(1)AR activation in iNOS gene-knockout mice suggests a direct cause-and-effect re
108 ponse to pathogens, it was noted that in CD4 gene knockout mice, the CD8 population made significant
109 With the use of three distinctive strains of gene knockout mice, the current study has provided the f
111 nimal model for DMD, were crossed with Flt-1 gene knockout mice to create a model with increased vasc
116 om hippocampal slices of prion protein (PrP) gene knockout mice to investigate whether the loss of th
117 d a streptozotocin-induced DKD model in FHL2 gene-knockout mice to determine the possible role of FHL
119 he central corneal epithelium of C57BL/6 and gene knockout mice was abraded, and 1 x 10(7) S. marcesc
122 r mammalian-cell viability in vivo: calnexin gene knockout mice were carried to full term, although 5
125 that inducible nitric oxide synthase (iNOS) gene knockout mice were resistant to endotoxin-induced b
127 in systemic autoimmunity, IL-4 and IFN-gamma gene knockout mice were studied for susceptibility to th
130 by targeted disruption of the 5-lipoxygenase gene (knockout mice) were studied following intratrachea
131 % of CD4+ T cells, or from MHC-class II I-Ab gene knockout mice, where they constitute 42% of CD4+ T
133 g the estrogen-synthesizing enzyme aromatase gene knockout mice with APP23 transgenic mice, a mouse m
134 of lens in vivo by comparing lens changes of gene-knockout mice with age-matched control animals.
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