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1 enia, diarrhea, vomiting, and syncope with a generalized seizure.
2 f two hospitals in Boston after a witnessed, generalized seizure.
3 6J mice to flurothyl until they experience a generalized seizure.
4 ell excitability, and is sufficient to cause generalized seizures.
5 of the time with ictal SPECT in secondarily generalized seizures.
6 th fast polyspike activity during convulsive generalized seizures.
7 splayed sustained protection from developing generalized seizures.
8 energy depletion in the brain resulting from generalized seizures.
9 - mice appear ataxic and display spontaneous generalized seizures.
10 ey role of the thalamus in the generation of generalized seizures.
11 epsy, a history of febrile convulsions or of generalized seizures.
12 linergic neurons influence the expression of generalized seizures.
13 neurons may participate in the modulation of generalized seizures.
14 odialysis before and during the induction of generalized seizures.
15 pilepsy of unknown cause, and 66 percent had generalized seizures.
16 associated with waking, slow-wave sleep, and generalized seizures.
17 mal dominant complex partial and secondarily generalized seizures, a greatly reduced threshold for hi
22 for the R1648H mutation exhibit spontaneous generalized seizures and premature death between P16 and
25 nuous partial seizures and even new bouts of generalized seizures, and (3) development of mild sponta
26 mutation in the L-ferritin gene, idiopathic generalized seizures, and atypical restless leg syndrome
27 immunodeficiency, neurodevelopmental delay, generalized seizures, and impaired hearing but with no m
30 s interpretation, ictal SPECT in secondarily generalized seizures can help localize the region of sei
31 -46 mSMEI had myoclonic seizures followed by generalized seizures caused by elevated core body temper
32 e of a more diffuse disorder with additional generalized seizures, cognitive and behavioural disturba
34 frequency bands was highest for secondarily generalized seizures, followed by complex partial seizur
35 yr-old woman experienced several episodes of generalized seizures following 2 wk of immunosuppressive
37 ance of these findings for the triggering of generalized seizures from the deep piriform region are d
38 taA mRNA induction, after the first stage 5 (generalized) seizure, had spread to the contralateral am
40 , duration of epilepsy, history of secondary generalized seizures, history of head injury, handedness
41 ple partial, complex partial and secondarily generalized seizures in 28 patients with temporal lobe s
43 correlated with the frequency of secondarily generalized seizures in the 3-5 days preceding the recor
45 the hippocampus are critical to the onset of generalized seizures, indicating that the hippocampus pl
47 ker of acid-sensing ion channels, suppresses generalized seizures induced by maximal electroshock and
50 modified tau score and history of secondary generalized seizures (likelihood-ratio chi(2), P < 0.05)
51 New insights into these different types of generalized seizures may lead eventually to new therapie
52 e entire brain, indicating that the onset of generalized seizures may originate locally rather than d
54 elop additional extreme hyper-reactivity and generalized seizures not seen in any other genotypes.
56 ork site for mediating analgesia and also in generalized seizures, particularly in genetically epilep
57 rozygous mice exhibit infrequent spontaneous generalized seizures, reduced threshold and accelerated
58 several brain sites, including the PAG, and generalized seizures result in endocannabinoid release.
59 e, with severe nocturnal focal and secondary generalized seizures starting in early childhood with de
60 ple partial, complex partial and secondarily generalized seizures suggest a possible connection betwe
61 nist caused a dose-dependent increase in the generalized seizure threshold (GST) of these seizure sus
62 sulted in a progressive decrease in both the generalized seizure threshold and the latency to the fir
66 hibition and thus increase susceptibility to generalized seizures through a reduction of alphabetagam
68 e-3-carboxylate (beta-CCM)]-induced model of generalized seizures was genetically dissected in mice.
69 extrinsic and intrinsic caspase pathways in generalized seizures, which produce morphologically necr
70 ng intractable complex partial and secondary generalized seizures, whose magnetic resonance imaging s
71 hat are associated with febrile seizures and generalized seizures with febrile seizures plus pedigree
72 with AAV Nrf2 displayed significantly fewer generalized seizures, with profound reduction in microgl
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