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   1 ences would allow precise means for specific genetic intervention.                                   
     2 es canonically would allow precise means for genetic intervention.                                   
     3 tive models and the possibility of precision genetic interventions.                                  
     4 K, or NF-kappaB using pharmacological and/or genetic interventions.                                  
     5 llular vectors, as well as pharmacologic and genetic interventions.                                  
     6 d CD4 T cells could potentially be used as a genetic intervention against both R5- and X4-tropic HIV-
     7 d CD4 T cells could potentially be used as a genetic intervention against both R5- and X4-tropic HIV-
     8 transcriptional regulatory-network analyses, genetic interventions and chromatin immunoprecipitation 
     9 elds in various species will require renewed genetic interventions and dramatic improvement of agricu
  
  
    12 nly in developing and mastering endovascular genetic interventions but also in assessing the success 
    13  human study designed to determine whether a genetic intervention can prolong the survival of T cells
  
    15 ially reversible, and both environmental and genetic interventions can result in the rejuvenation of 
    16 tivity ratio by multiple pharmacological and genetic interventions confirms that high ERK/p38 ratio f
  
    18  diseases has made the design of therapeutic genetic interventions feasible in these clinical entitie
  
    20 ve Cas9 (dCas9) and offers an alternative to genetic interventions for studying pervasive antisense t
  
  
    23 e 1 (HSV-1) plasmid vectors have promise for genetic intervention in the brain, but several problems 
  
    25 s to develop adenovirus vectors suitable for genetic interventions in humans have identified three ma
  
  
  
    29 be used to screen pharmacological as well as genetic interventions more rapidly for positive effects 
  
  
  
    33 y toxic NIR light, labeling without need for genetic intervention, rapid kinetics, remote subsurface 
    34  either by Brefeldin A (BFA) treatment or by genetic intervention results in increased intracellular 
  
    36 mbers by pharmacological (with dithizone) or genetic intervention (SOX9 flox/flox Villin cre+/- mice)
    37 bitor-resistant PR was blocked, showing that genetic intervention strategies based on td PRs can be e
  
  
  
  
    42 essary for membrane and cellular repair, and genetic interventions that restore MLRs to normal cellul
    43 n beta(s) mice was blunted by immunologic or genetic interventions that target tissue factor, endothe
    44 ts provide the ability to tailor the mode of genetic intervention to specific aspects of a disease st
    45 x (PFC) neurons and used pharmacological and genetic interventions to block connexin-mediated hemicha
    46 advanced techniques -- ranging from targeted genetic interventions to brain imaging -- that are rapid
  
  
    49 viduals and potentially serve as a molecular genetic intervention which can contribute to the treatme
    50 ithfully recapitulate the effect of multiple genetic interventions would be transformative in our abi
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