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1 ences would allow precise means for specific genetic intervention.
2 es canonically would allow precise means for genetic intervention.
3 tive models and the possibility of precision genetic interventions.
4 K, or NF-kappaB using pharmacological and/or genetic interventions.
5 llular vectors, as well as pharmacologic and genetic interventions.
6 d CD4 T cells could potentially be used as a genetic intervention against both R5- and X4-tropic HIV-
7 d CD4 T cells could potentially be used as a genetic intervention against both R5- and X4-tropic HIV-
8 transcriptional regulatory-network analyses, genetic interventions and chromatin immunoprecipitation
9 elds in various species will require renewed genetic interventions and dramatic improvement of agricu
10 igrostriatal neurons was examined by using a genetic intervention approach.
11 dustrialized societies unless novel means of genetic intervention are developed.
12 nly in developing and mastering endovascular genetic interventions but also in assessing the success
13  human study designed to determine whether a genetic intervention can prolong the survival of T cells
14                              Muscle-specific genetic interventions can induce systemic effects indire
15 ially reversible, and both environmental and genetic interventions can result in the rejuvenation of
16 tivity ratio by multiple pharmacological and genetic interventions confirms that high ERK/p38 ratio f
17                                              Genetic intervention during mitosis prevented both appre
18  diseases has made the design of therapeutic genetic interventions feasible in these clinical entitie
19                                              Genetic intervention for the therapy of human disease ha
20 ve Cas9 (dCas9) and offers an alternative to genetic interventions for studying pervasive antisense t
21 ansiently modify lymphocytes, without direct genetic intervention, for adoptive transfer.
22 at mitochondrial disease, both metabolic and genetic interventions have been attempted.
23 e 1 (HSV-1) plasmid vectors have promise for genetic intervention in the brain, but several problems
24                                The future of genetic interventions in humans critically depends on th
25 s to develop adenovirus vectors suitable for genetic interventions in humans have identified three ma
26                        In our previous work, genetic interventions in the Lama2(Dy-w) mouse model for
27  testing the efficacy of pharmacological and genetic interventions in vivo.
28                                              Genetic intervention inducing over-enlargement of myofib
29 be used to screen pharmacological as well as genetic interventions more rapidly for positive effects
30 d fluid flow, as well as pharmacological and genetic interventions of specific proteins.
31                  We found that both of these genetic interventions produced a several-fold increase i
32                                              Genetic interventions promoting longevity are usually qu
33 y toxic NIR light, labeling without need for genetic intervention, rapid kinetics, remote subsurface
34  either by Brefeldin A (BFA) treatment or by genetic intervention results in increased intracellular
35                          Pharmacological and genetic interventions revealed that insulin regulates GL
36 mbers by pharmacological (with dithizone) or genetic intervention (SOX9 flox/flox Villin cre+/- mice)
37 bitor-resistant PR was blocked, showing that genetic intervention strategies based on td PRs can be e
38                                       Hence, genetic intervention strategies based on trans-dominant
39                           Herein, we show by genetic intervention that prostaglandin E(2) in the spin
40                                            A genetic intervention that specifically decreases NADH le
41                                              Genetic interventions that accelerate or retard aging in
42 essary for membrane and cellular repair, and genetic interventions that restore MLRs to normal cellul
43 n beta(s) mice was blunted by immunologic or genetic interventions that target tissue factor, endothe
44 ts provide the ability to tailor the mode of genetic intervention to specific aspects of a disease st
45 x (PFC) neurons and used pharmacological and genetic interventions to block connexin-mediated hemicha
46 advanced techniques -- ranging from targeted genetic interventions to brain imaging -- that are rapid
47                            We designed three genetic interventions to manipulate the actions of gluco
48            The potential of such a molecular genetic intervention was examined by using the Cre-loxP
49 viduals and potentially serve as a molecular genetic intervention which can contribute to the treatme
50 ithfully recapitulate the effect of multiple genetic interventions would be transformative in our abi

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