1 s, and during the development of dietary and genetic obesity.

2 s, and during the development of dietary and genetic obesity.

3  and metabolic abnormalities associated with genetic obesity.

4 e 2 in vitro and in vivo in diet-induced and genetic obesity.

5 evidence for a role of AT LPL in one type of genetic obesity.

6 abnormally activated in mice with dietary or genetic obesity.

7 ents and rhesus monkeys with diet-induced or genetic obesity and diabetes exerts strong antihyperglyc

8 lowing partial hepatectomy (PH) in mice with genetic obesity and hepatic steatosis and also in wild-t

9 or the ob protein leptin in rodent models of genetic obesity, and insulin has been reported to direct

10                                      We used genetic obesity as a tool to uncover latent differences

11                                  Dietary and genetic obesity features marked activation of PKR in adi

12 onal status in a context of diet-induced and genetic obesity; food deprivation increased Deptor mRNA

13 al muscle in the setting of diet-induced and genetic obesity, inflammation, and hyperlipidemia.

14 ents, or reverses, high-fat diet-induced and genetic obesity, insulin resistance and hepatic steatosi

15    We now demonstrate that either dietary or genetic obesity is a potent bona fide liver tumor promot

16                                              Genetic obesity susceptibility appears to promote a norm

17 dy size and body composition associated with genetic obesity susceptibility inform the mechanisms tha

18 es), being at greatest risk for exacerbating genetic obesity susceptibility.

19          Here we review in a rodent model of genetic obesity, the fa/fa Zucker diabetic fatty (ZDF) r

20 uscle of the ob/ob mouse, an animal model of genetic obesity with extreme insulin resistance, exhibit