1 A
genetic screen for A. tumefaciens mutants deficient for
2 Genetic screening for a germline mutation at the RET gen
3 arcoded mutants unlocks the power of reverse
genetic screening for a malaria parasite and will enable
4 In a
genetic screen for aberrant synaptic growth at the neuro
5 In a
genetic screen for adaptive defective mutants, a nonsens
6 are available to the fly community, forward
genetic screens for adult heart phenotypes have been rar
7 A
genetic screen for allele-specific suppressors of the G(
8 of the SIR complex, we performed a targeted
genetic screen for alleles of SIR3 that dominantly disru
9 In a
genetic screen for altered circadian period length in Ar
10 In a reverse
genetic screen for altered drought tolerance, we identif
11 A forward
genetic screen for altered PD transport identified incre
12 e lhr1 mutant was isolated through a forward
genetic screening for altered expression of the lucifera
13 nents that have been identified by extensive
genetic screens for altered sensitivities to osmotic str
14 ng regulator, was uncovered during a forward-
genetic screen for angiogenesis-deficient mutants in the
15 .1 mutant, which was identified in a forward
genetic screen for Arabidopsis (Arabidopsis thaliana) mu
16 A
genetic screen for Arabidopsis mutants affecting tempera
17 for this feedback regulation, we performed a
genetic screen for Arabidopsis mutants that exhibit enha
18 A
genetic screen for Arabidopsis thaliana mutants compromi
19 t protein fluorescence imaging-based forward
genetic screen for Arabidopsis thaliana mutants that sho
20 test this hypothesis, we employed a forward
genetic screen for Arabidopsis thaliana mutants tolerati
21 function in auxin signaling, we performed a
genetic screen for Arabidopsis thaliana mutants with an
22 Through forward
genetic screens for Arabidopsis mutants showing DNA hype
23 and bio2 auxotrophs identified using forward
genetic screens for arrested embryos rescued on enriched
24 novel gene identified in a yeast two hybrid
genetic screen for binding to the MZF1B SCAN box.
25 s have been extended by the development of a
genetic screen for BLIP function in Escherichia coli.
26 The
genetic screen for BLIP function was used to sort the li
27 breast cancer are often counseled to receive
genetic screening for BRCA1 and BRCA2 mutations, the str
28 addition, ENA21 was identified in a forward
genetic screen for C. albicans genomic sequences that in
29 From a forward
genetic screen for C. elegans genes required for RNAi, w
30 In a
genetic screen for C. elegans mutants exhibiting partial
31 To seek such genes, we performed a
genetic screen for C. elegans mutants that age premature
32 In a
genetic screen for C. elegans mutants with defects in do
33 A
genetic screen for C. elegans mutants with PKD-2 ciliary
34 In a previous
genetic screen for Caenorhabditis elegans mutants that s
35 A
genetic screen for Caenorhabditis elegans mutants with b
36 A
genetic screen for Caenorhabditis elegans mutants with e
37 We describe here the results of
genetic screens for Caenorhabditis elegans mutants in wh
38 Two genes, originally identified in
genetic screens for Caenorhabditis elegans mutants that
39 g Beauty is a new tool for unbiased, forward
genetic screens for cancer genes in vivo.
40 A conditional
genetic screen for CckA mislocalization mutants, using a
41 tors, are likely to alter fundamentally both
genetic screening for celiac disease and its therapy.
42 A
genetic screen for cell division cycle mutants of Caulob
43 In a new
genetic screen for cell shape mutants, we identified Rod
44 A
genetic screen for cells that become permeabilized at no
45 ion motif-containing protein, from a forward
genetic screen for cellular antisilencing factors in Ara
46 In a
genetic screen for cellular factors involved in preventi
47 s a binding partner for TopBP1 and also in a
genetic screen for checkpoint regulators in zebrafish.
48 d in zinc metabolism, we conducted a forward
genetic screen for chemically induced mutations that cau
49 In a forward
genetic screen for chemotaxis mutants in Dictyostelium d
50 A
genetic screen for Chlamydomonas reinhardtii mutants wit
51 Based on the success of
genetic screens for circadian mutants in Drosophila mela
52 will enable large-scale forward and reverse
genetic screens for complex behaviors.
53 In a
genetic screen for components involved in the early immu
54 Here, using a yeast
genetic screen for components that control nucleoid dist
55 In a Drosophila
genetic screen for components that promote Wingless sign
56 1p-induced DNA lesions, we described a yeast
genetic screen for conditional tah (top1T722A-hypersensi
57 ation (GAIT) of ceruloplasmin (Cp) mRNA by a
genetic screen for Cp 3'-UTR binding proteins.
58 Using a
genetic screen for cVA-insensitive mutants, we have iden
59 network in Arabidopsis, we used a sensitized
genetic screen for deetiolation-defective seedlings.
60 lsy-6 mutants that we retrieved from a
genetic screen for defects in neuronal left/right asymme
61 d trypanosome VSG exclusion-1 (VEX1) using a
genetic screen for defects in telomere-exclusive express
62 A
genetic screen for defects in the organization of interm
63 es cerevisiae SPT4, previously identified in
genetic screens for defects in chromosome transmission f
64 A zebrafish
genetic screen for determinants of susceptibility to Myc
65 s and demonstrates the promise of functional
genetic screens for dissecting therapeutically relevant
66 A
genetic screen for dominant enhancers of the mod(mdg4) p
67 phila fusilli (fus) gene was identified in a
genetic screen for dominant maternal enhancers of an unu
68 gpp is an essential gene identified in a
genetic screen for dominant suppressors of pairing-depen
69 A
genetic screen for dominant suppressors of wing blisters
70 morphogenesis, we performed two large-scale
genetic screens for dominant enhancers of the malformed
71 clarify the function of Rlr1, we performed a
genetic screen for dosage-dependent suppressors of the c
72 The ird5 gene was identified in a
genetic screen for Drosophila immune response mutants.
73 From a
genetic screen for Drosophila melanogaster mutants with
74 We identified moody in a
genetic screen for Drosophila mutants with altered cocai
75 ga Chlamydomonas reinhardtii, we developed a
genetic screen for early components of singlet oxygen si
76 We undertook a forward
genetic screen for effectors of EpiSC reprogramming, emp
77 el activity, as shown by (i) TrIP and (ii) a
genetic screen for effects of the oncogenic suppressors
78 s bearing N-end rule sequences isolated in a
genetic screen for efficient degradation tags.
79 he only other organism for which large-scale
genetic screens for endocytosis mutants have been perfor
80 We identified mutations in Rab8 in a
genetic screen for enhancement of an FTD phenotype assoc
81 In a
genetic screen for enhancers of sic-1, we isolated a los
82 mb pathway in plants, we performed a forward
genetic screen for enhancers of the like heterochromatin
83 In a
genetic screen for enhancers of the tir1-1 auxin respons
84 A
genetic screen for ethanol sedation mutants in Drosophil
85 Using a
genetic screen for exported proteins in GBS, we identifi
86 We identified HTP-3 in a
genetic screen for factors necessary to maintain SCC unt
87 bly pathways, we have performed a systematic
genetic screen for factors required for centromeric hete
88 cherichia coli, CsrC, was discovered using a
genetic screen for factors that regulate glycogen biosyn
89 s for Pro-16, Asp-18, and Asn-19 followed by
genetic screening for functional proteins.
90 We identified WHSC1 in a
genetic screen for genes involved in responding to repli
91 In a forward
genetic screen for genes regulating endodermal organ dev
92 We conducted a forward
genetic screen for genes that are required for salivary
93 negative regulators of MEN, we carried out a
genetic screen for genes that are toxic to cdc5-1 mutant
94 We identified Abl in a
genetic screen for genes that contribute to Netrin-depen
95 In a
genetic screen for genes that control synapse developmen
96 In a gain-of-function
genetic screen for genes that influence fruit developmen
97 ) gene, which were identified in an unbiased
genetic screen for genes that modify parkin phenotypes.
98 From a
genetic screen for genes that modulate biofilm formation
99 nscription-coupled processes, we performed a
genetic screen for genes that suppress the petite phenot
100 Here we report an RNAi-based
genetic screen for genes that suppress transformation of
101 s initially discovered in budding yeast in a
genetic screen for genes whose deletion confers defects
102 In a
genetic screen for germ-line proliferation-defective mut
103 pies" may greatly improve the reliability of
genetic screens for HD and may provide further insight i
104 Research addressing
genetic screening for hereditary hemochromatosis remains
105 te the benefit from, widespread or high-risk
genetic screening for hereditary hemochromatosis.
106 Limitations: This review considered
genetic screening for HFE-related hereditary hemochromat
107 ast gene BFR1 was originally isolated from a
genetic screen for high-copy suppressors of brefeldin A-
108 In a forward
genetic screen for homeostatic plasticity genes, we iden
109 roup (PcG) proteins were first identified in
genetic screens for homeotic transformations in Drosophi
110 In a forward
genetic screen for host ERAD components hijacked by US11
111 We identified POS5 in a S.cerevisiae
genetic screen for hyperoxia-sensitive mutants, or cells
112 ation sites in murine tumors to be used as a
genetic screen for identification of large numbers of ca
113 A
genetic screen for identifying additional genes that, wh
114 pathways in Arabidopsis, we describe here a
genetic screen for identifying mutants that display a ch
115 To investigate this, we used a
genetic screen for impaired development to isolate four
116 We performed a
genetic screen for impaired walking in Drosophila and is
117 ditis elegans homolog of rictor in a forward
genetic screen for increased body fat.
118 mutation, repro5, was isolated in a forward
genetic screen for infertility mutations induced by ENU
119 In a forward
genetic screen for interaction with mitochondrial iron c
120 A yeast
genetic screen for IpaJ substrates identified ADP-ribosy
121 In a
genetic screen for Kinesin heavy chain (Khc)-interacting
122 In addition, in a
genetic screen for klarsicht-interacting genes, we ident
123 Recently, we reported a
genetic screen for lethal mutations in gene 2.5 that we
124 We also carried out a large-scale
genetic screen for lethal mutations in the region.
125 During a forward
genetic screen for liver and pancreas mutants, we identi
126 cidate LON2 functions, we executed a forward-
genetic screen for lon2 suppressors, which revealed mult
127 A forward
genetic screen for lysozyme-sensitive mutants led to the
128 se of hematopoietic stem cells to facilitate
genetic screening for malaria host factors.
129 A
genetic screen for markedly enhanced fat storage in tub-
130 Here we show, by using a
genetic screen for maternal factors that contribute in a
131 regulators of Dpp signaling, we conducted a
genetic screen for maternal-effect suppressors of dpp ha
132 n previously identified in nearly saturating
genetic screens for maternal effect and zygotic lethals
133 udy, we identified the NIMA kinase Nek4 in a
genetic screen for mediators of the response to Taxol, a
134 A forward
genetic screen for mice with defects in thalamocortical
135 We performed a
genetic screen for micro-RNAs that are differentially ex
136 utant, cassiopeia (csp), was identified by a
genetic screen for mitotic mutant.
137 Using a
genetic screen for modifiers of Drosophila Myc (dMyc)-in
138 In a
genetic screen for modifiers of wingless signaling in th
139 dfxr function in the testes paves the way to
genetic screens for modifiers of dfxr-induced male steri
140 Genetic screening for molecules involved in Shiga toxin
141 Caenorhabditis elegans, which emerged from a
genetic screen for molting-defective mutants sensitized
142 CDC55 was identified in a
genetic screen for monopolins performed by isolating sup
143 In a
genetic screen for mutants affecting endosomal trafficki
144 A
genetic screen for mutants defective for GAL gene memory
145 In a
genetic screen for mutants defective in this noxious hea
146 In an unbiased
genetic screen for mutants exhibiting neurodegeneration
147 idopsis mutant Atcand1-1 that emerged from a
genetic screen for mutants insensitive to sirtinol.
148 In a
genetic screen for mutants lacking this regulation, we i
149 solated a novel allele of hid-1 in a forward
genetic screen for mutants mislocalizing RBF-1 rabphilin
150 an inhibitor of cardioblast development in a
genetic screen for mutants that affect heart development
151 Using a forward
genetic screen for mutants that can sustain hyphal elong
152 mitochondrial protein, was identified with a
genetic screen for mutants that diminish RDD.
153 involved in oxygen toxicity, we conducted a
genetic screen for mutants that display altered survival
154 We isolated HTL1 through a
genetic screen for mutants that displayed additive growt
155 By performing an unbiased
genetic screen for mutants that impair the somnogenic ef
156 In a
genetic screen for mutants that lack AvrB-dependent chlo
157 In a
genetic screen for mutants that mislocalize the dendriti
158 We present an exhaustive
genetic screen for mutants that no longer recognize avrR
159 Using a chemical-
genetic screen for mutants that specifically require the
160 To address this issue, we conducted a
genetic screen for mutants that suppress a partial loss
161 A
genetic screen for mutants that suppress acd6-1-conferre
162 CTN-1 was identified in a
genetic screen for mutants that suppressed a lethargic p
163 ants of the mitochondrial Hsp70, Ssq1p, in a
genetic screen for mutants with altered iron homeostasis
164 In a
genetic screen for mutants with defective neuromuscular
165 A Drosophila forward
genetic screen for mutants with defective synaptic devel
166 C. elegans eri-1 was identified in a
genetic screen for mutants with enhanced sensitivity to
167 was previously identified in four different
genetic screens for mutants affecting chromosome transmi
168 tified the C. elegans pat-4 gene in previous
genetic screens for mutants unable to assemble integrin-
169 In
genetic screens for mutants with disruptions in myelinat
170 From a
genetic screen for mutations able to suppress the bloate
171 We isolated mutations in Liprin-alpha in a
genetic screen for mutations affecting the pattern of sy
172 tion in neural crest, we performed a forward
genetic screen for mutations causing DRG deficiencies in
173 in the optic tectum, we undertook a forward
genetic screen for mutations disrupting visual responses
174 We conducted a
genetic screen for mutations in myospheroid, the gene en
175 We performed a
genetic screen for mutations in the catalytic subunit th
176 A
genetic screen for mutations in thioredoxin that render
177 ts of the cell death pathway, we performed a
genetic screen for mutations that abolish the death of t
178 In a
genetic screen for mutations that affect Drosophila eye
179 the mutant motionless (mot), identified in a
genetic screen for mutations that affect neuronal develo
180 In a
genetic screen for mutations that affect the biosynthesi
181 During a
genetic screen for mutations that affect trichome shape,
182 In a forward
genetic screen for mutations that alter intracellular No
183 in tau neurotoxicity, we conducted a forward
genetic screen for mutations that ameliorate tau-induced
184 In a forward
genetic screen for mutations that block PHP we identifie
185 e chromatin or transcription, we performed a
genetic screen for mutations that cause lethality in the
186 Thus, we set up a
genetic screen for mutations that conferred a conditiona
187 In a forward
genetic screen for mutations that destabilize the neurom
188 To address this question, we conducted a
genetic screen for mutations that differentially affecte
189 ontrol genomic DNA methylation patterning, a
genetic screen for mutations that disrupt methylation-co
190 RNA pathway, Pasha and Dicer-1, in a forward
genetic screen for mutations that disrupt wiring specifi
191 A
genetic screen for mutations that dominantly suppress or
192 regulators of Ras signaling, we conducted a
genetic screen for mutations that enhance the excretory
193 migration, we conducted a nonbiased forward
genetic screen for mutations that enhanced the nuclear m
194 ing and tissue repair, we have carried out a
genetic screen for mutations that impair regeneration in
195 ed DAM1-765, a dominant allele of DAM1, in a
genetic screen for mutations that increase stress on the
196 We performed an unbiased
genetic screen for mutations that permit the survival of
197 In a
genetic screen for mutations that restrict cell growth a
198 In a forward
genetic screen for mutations that result in loss of habe
199 nsduction pathway(s), we conducted a forward
genetic screen for mutations that suppressed edr1-mediat
200 Our
genetic screening for mutations that resist CLE peptide
201 a valuable resource for forward and reverse
genetic screens for mutations affecting a wide array of
202 To identify these proteins, we carried out
genetic screens for mutations affecting Drosophila melan
203 Unbiased forward
genetic screens for mutations causing increased gross ch
204 ndant functions of LIN-35 were identified in
genetic screens for mutations that display synthetic phe
205 Forward
genetic screens for mutations that rescue the paralysis
206 Genetic screens for mutations that result in increased p
207 From a forward
genetic screen for myelination defects in zebrafish, we
208 A
genetic screen for negative regulators of olfaction unco
209 proteins, FitA and FitB, was identified in a
genetic screen for Neisseria gonorrhoeae determinants th
210 o understand this process, here we conduct a
genetic screen for nematodes defective in transmitting R
211 approaches have hindered systematic forward
genetic screening for NMD factors in human cells.
212 Fireworks) that enables CRISPR-based forward
genetic screening for NMD pathway defects in human cells
213 ion in the Sgs1-Top3 pathway, we undertook a
genetic screen for non-sgs1 suppressors of top3 defects.
214 ri1 allele, bri1-5, in an activation-tagging
genetic screen for novel brassinosteroid (BR) signal tra
215 Using a murine forward
genetic screen for novel determinants of axon guidance,
216 We further performed an unbiased
genetic screen for novel modifiers of instability.
217 FAM83B was recently discovered in a forward
genetic screen for novel oncogenes that drive human mamm
218 In a
genetic screen for novel TOR interactors in Drosophila m
219 Recent
genetic screens for novel components of brassinosteroid
220 8-1) mutant was identified in an independent
genetic screen for NPC assembly (npa) mutants.
221 Patients underwent
genetic screening for NPM1, FLT3-ITD, FLT3-D835, and CEB
222 In this study, in a genome-wide
genetic screen for other ion channel subunits required f
223 tumour suppressor gene by using a mammalian
genetic screen for p53-dependent genes involved in tumor
224 2delta-2), were also identified in a forward
genetic screen for pain genes (alpha2delta-3).
225 r during muscle degeneration and performed a
genetic screen for parkin modifiers.
226 Using a
genetic screen for PCL defect, we identified a mutation
227 From a forward
genetic screen for phagocytosis mutants in Drosophila me
228 atopoietic stem cells to carry out a forward
genetic screen for Plasmodium falciparum host determinan
229 This observation instigated further
genetic screening for prostacyclin receptor variants on
230 A
genetic screen for proteins that can block the spread of
231 e the molecular basis of NKX-3.1 function, a
genetic screen for proteins that interact with NKX-3.1 w
232 brain 4.1 proteins, in yeast two-hybrid and
genetic screens for proteins that interact with and loca
233 is thaliana mutant smd1b was identified in a
genetic screen for PTGS deficiency, revealing the involv
234 i named PEANUT1-5 (PNT) were identified in a
genetic screen for radially swollen embryo mutants.
235 d in axon navigation, we conducted a forward
genetic screen for recessive alleles affecting motor neu
236 Genetic screens for recessive mutations continue to prov
237 In a forward
genetic screen for regulators of C-REPEAT BINDING FACTOR
238 In a forward
genetic screen for regulators of C. elegans PKD-2 ciliar
239 A
genetic screen for regulators of EGFR signaling has led
240 n minus-end directed microtubule motor, in a
genetic screen for regulators of EGFR signaling.
241 A previous
genetic screen for regulators of inv identified RovA, wh
242 In a
genetic screen for regulators of muscle development in D
243 C. elegans cdf-1 was identified in a
genetic screen for regulators of Ras-mediated signaling.
244 ore this facet of myogenesis, we performed a
genetic screen for regulators of somatic muscle morpholo
245 In a
genetic screen for regulators of synaptic morphology, we
246 d (ptc), an inhibitor of Hh signalling, in a
genetic screen for regulators of the Retinoblastoma (Rb)
247 uction have already been defined, continuous
genetic screening for regulators of innate immunity may
248 The par genes were discovered in
genetic screens for regulators of cytoplasmic partitioni
249 We carried out two forward
genetic screens for regulators of endodermal organ devel
250 A
genetic screen for relevant mutations in Drosophila gene
251 A
genetic screen for resistance to ethanol intoxication in
252 entified EDM2 (enhanced downy mildew 2) in a
genetic screen for RPP7 suppressors.
253 Here, we describe two independent
genetic screens for rsc suppressors that yielded mutatio
254 In a
genetic screen for Saccharomyces cerevisiae mutants hype
255 challenges faced by families as a result of
genetic screening for SADS to enable equitable access to
256 To identify salt tolerance determinants, a
genetic screen for salt overly sensitive (sos) mutants w
257 of the MVA pathway in plants, we performed a
genetic screen for second-site suppressor mutations of t
258 From a
genetic screen for second-site suppressors of the DNA de
259 mechanisms we performed a GFP-based forward
genetic screen for seedling-lethal biosynthetic membrane
260 In independent
genetic screens, for shade-avoidance response and cytoki
261 required for silencing in S. bayanus using a
genetic screen for silencing-defective mutants.
262 A
genetic screen for similar lumbar maintenance mutants re
263 ated gene disruption procedure and performed
genetic screening for single P-element insertion mutatio
264 In a chemical
genetic screen for small molecules that dampened the inh
265 A chemical
genetic screen for small molecules that suppress growth
266 al genes involved in body size regulation, a
genetic screen for small mutants was previously performe
267 We performed a
genetic screen for spoIIE mutants that were impaired in
268 These elements were identified in a
genetic screen for spontaneous mutations that caused col
269 lated paf1 and leo1 mutations in an unbiased
genetic screen for suppressors of a cold-sensitive spt5
270 In a
genetic screen for suppressors of a lethargic phenotype
271 Here we describe a
genetic screen for suppressors of a postrecruitment-defe
272 on (lf) mutants of hrpu-2 were isolated in a
genetic screen for suppressors of a sluggish phenotype c
273 scent tryptophan metabolites, we conducted a
genetic screen for suppressors of blue fluorescence in t
274 A
genetic screen for suppressors of BS A-to-G mutants, whi
275 By deploying a
genetic screen for suppressors of cell death triggered b
276 rt machinery component Tic40, we performed a
genetic screen for suppressors of chlorotic tic40 knocko
277 A
genetic screen for suppressors of loss of yeast CTD kina
278 In a
genetic screen for suppressors of reduced neurotransmitt
279 Here, we report the results of a forward
genetic screen for suppressors of ref4-3.
280 In a
genetic screen for suppressors of sni1, we discovered th
281 A
genetic screen for suppressors of the Arabidopsis bri1-9
282 y and chromosome segregation, we performed a
genetic screen for suppressors of the increase-in-ploidy
283 In a
genetic screen for suppressors of the SIN mutant sid2-25
284 Genetic screens for suppressors of SWI6 mutants have bee
285 ) component vha100-1 in flies in an unbiased
genetic screen for synaptic malfunction.
286 In a Drosophila
genetic screen for synaptogenesis mutants, we identified
287 Genetic screens for synaptogenesis mutants have been per
288 A
genetic screen for synergistic increase in fat storage o
289 In
genetic screens for temperature-sensitive maternal effec
290 Here, we report a
genetic screen for the enhancement of maize inflorescenc
291 In this study, we developed a forward
genetic screen for the identification of host factors re
292 Using a confocal microscopy forward
genetics screen for the identification of Arabidopsis th
293 Currently, there is no effective therapy or
genetic screens for these diseases; however, nuclear gen
294 is important for basic and medical research;
genetic screening for those genes in Caenorhabditis eleg
295 )) has also been identified in C. elegans in
genetic screens for touch insensitivity (MEC-2(P134S)).
296 In a
genetic screen for transcription factors regulating sene
297 This work provides a sensitive
genetic screen for uncovering auxin-resistant mutants in
298 e results of a large-scale, microscopy-based
genetic screen for Vibrio cholerae mutants that are defe
299 In a
genetic screen for yeast mutants with elevated microsate
300 t influence liver lipid mass, we performed a
genetic screen for zebrafish mutants with hepatic steato