1          Apnea patients demonstrated greater genioglossal (27.4 +/- 4.0 versus 10.7 +/- 2.1%) and ten

2  local negative pressure reflex in modifying genioglossal activation across inspiration during wakefu

3 stive loading led to significantly increased genioglossal activation, 3.91 +/- 0.77% to 9.64 +/- 1.96

4  highly correlated with within-breath phasic genioglossal activation, probably representing a robust

5 li during NREM sleep would lead to increased genioglossal activation.

6 y afterload exerted by negative UAP and that genioglossal active shortening may be limited if the mus

7 custom intraoral surface electrode to record genioglossal activity (genioglossal electromyography [EM

8 essure, we examined the relationship between genioglossal activity and epiglottic pressure in patient

9   This mechanism could mediate the increased genioglossal activity observed in patients with obstruct

10 ficantly larger than controls) decrements in genioglossal activity.

11 sic activity of airway dilators, we assessed genioglossal electromyogram (GG EMG: rectified with movi

12           Although no important physiologic (genioglossal electromyogram, airflow resistance) differe

13 SRI), paroxetine hydrochloride, may increase genioglossal electromyographic (EMG) activity (EMGgg) in

14 e electrode to record genioglossal activity (genioglossal electromyography [EMGgg]), normalized with

15                                          The genioglossal (GG) muscle is the main protruder and depre

16                         We hypothesized that genioglossal length (Lgg) is dynamically influenced by t

17                             We conclude that genioglossal length is substantially influenced by after

18  methods for inducing long-term increases in genioglossal motoneuronal excitability to AMPA-mediated

19                                              Genioglossal muscle (GG) activity is modulated by both c

20 rying intrapharyngeal negative pressures and genioglossal muscle activation (GGEMG) during wakefulnes