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1 lacking Nanog fail to mature on reaching the genital ridge.
2 y due to the action of WNT4 within the early genital ridge.
3 erm cells begin to migrate to the developing genital ridge.
4 erm cells shortly after their arrival in the genital ridge.
5 initiates just before translocation into the genital ridge.
6 ation, followed by directed migration to the genital ridges.
7 GCs), resulting in fewer PGCs colonizing the genital ridges.
8 approximately 10.5 dpc as the PGCs enter the genital ridges.
9  in which they migrate out of the gut to the genital ridges.
10 al and migration as these cells colonize the genital ridges.
11 ctionally from the dorsal body wall into the genital ridges.
12 exit the gut, but do not migrate towards the genital ridges.
13  the gut mesentery at E10.5 migrate into the genital ridges.
14 e identify a possible PGC niche in the mouse genital ridges.
15  both around the dorsal aorta and in the uro-genital ridges.
16  in the allantois to the time they enter the genital ridges.
17  expressed in cells located centrally in the genital ridge and then later in cells located at the cra
18  find that chicken Dmrt1 is expressed in the genital ridge and Wolffian duct prior to sexual differen
19 romotes mesonephric cell survival within the genital ridges and that these cells support correct deve
20 nitial specification until they colonize the genital ridges, and suggest the existence of a ;spatio-t
21 reduced survival of PGCs that migrate to the genital ridge around embryonic day 11.5 (E11.5).
22 entifies the most striking difference in the genital ridge at early stages of its development between
23 s within the coelomic epithelium (CE) of the genital ridge, but from cells also able to give rise to
24  reactivation is thought to emanate from the genital ridge, but it is unclear whether it is specific
25  no longer migrate directionally towards the genital ridges, but instead rapidly fragment and disappe
26 to the masculinising environment of the male genital ridge, defining a temporal window during which P
27 ells (PGCs) fail to migrate correctly to the genital ridges early in organogenesis.
28 s CXCR4, whilst the body wall mesenchyme and genital ridges express the ligand SDF1.
29 s migrate normally, but somatic cells of the genital ridge fail to proliferate and a discrete gonad f
30 n, irrespective of the phenotypic sex of the genital ridge from which the EGCs had been derived.
31           The numbers of PGCs populating the genital ridge in TIAR-deficient embryos are severely red
32 ather than ovary from the indifferent gonad (genital ridge) in mammalian embryos.
33 uced to minimal levels in the Lhx9-deficient genital ridge, indicating that Lhx9 may lie upstream of
34   C-met expression is not detectable in male genital ridges isolated from embryos at 11.5 days postco
35                                         Male genital ridges isolated from embryos at 11.5 dpc are mor
36 t of Wt1 in mice results in apoptosis of the genital ridge, it is unknown whether WT1 is required for
37  cells derived after PGC colonisation of the genital ridge, "late" and embryonic stem (ES) cell lines
38             Reduced BMP signaling within the genital ridges led to increased somatic cell death withi
39 ac and the para-aortic splanchnopleura/aorta-genital ridges-mesonephros (P-Sp/AGM) region are the mai
40 tral aspect of the dorsal aorta in the aorta/genital ridge/mesonephros (AGM) region.
41 e Dmrt1 mRNA is expressed exclusively in the genital ridge of early XX and XY embryos.
42  of Sry in the undifferentiated, bipotential genital ridges of mammalian XY fetuses initiates testis
43                                  In a female genital ridge, or in a non-gonadal environment, PGCs dev
44                              Using rat organ genital ridge organ cultures, we found that inhibition o
45 o track through endoderm on their way to the genital ridge, our work raises the possibility that cons
46                                  Once in the genital ridge, PGCs cease dividing and differentiate acc
47 to be expressed exclusively in the mammalian genital ridge prior to sexual differentiation.
48 as deleted before sex determination and most genital ridge somatic cells differentiated into steroido
49 mous, and is not due to any influence of the genital ridge somatic cells upon the PGCs.
50 haracteristics in both sexes at indifferent (genital ridge) stages and that these persist, becoming m
51 stitution of the EGCs or with the sex of the genital ridge (testis versus ovary) from which the PGCs
52       In the mouse Dmrt1 is expressed in the genital ridge (the gonadal primordium) in both sexes and
53 cation in the extraembryonic mesoderm to the genital ridge, the gonadal anlage.
54 e number of PGCs reaching and populating the genital ridges, the molecular identity of only two of th
55 mitive vasculature is identical in XX and XY genital ridges until 11.5 days postcoitum (dpc), by 12.5
56  the developing hindgut, and traverse to the genital ridge where they cluster and ultimately inhabit
57 e through somatic tissues to the presumptive genital ridges, where they proliferate and differentiate

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