ライフサイエンスコーパス: genital ulcer

1 of CD14(+) cells expressing CCR5 within the genital ulcer.

2 ed sexually and can cause recurrent, painful genital ulcers.

3 by Haemophilus ducreyi in the production of genital ulcers.

4 eptive digital-anal contact, anal warts, and genital ulcers.

5 ion of HSV, H. ducreyi, and T. pallidum from genital ulcers.

6 sidered in women presenting with acute-onset genital ulcers.

7 1 acquisition and secondary was incidence of genital ulcers.

8 49 years old worldwide and causing recurrent genital ulcers.

9 5 and CXCR4) by monocytic cells within human genital ulcers.

10 ins, and other bacteria known to superinfect genital ulcers.

11 esting for HSV when evaluating patients with genital ulcers.

12 ficant worldwide pathogen, causing recurrent genital ulcers.

13 tologic lesions included oral ulcers (100%), genital ulcers (62%), erythema nodosum (46%), and papulo

14 is shed episodically, leading to occasional genital ulcers and efficient transmission.

15 The strong association between genital ulcers and HIV infection in this population high

16 syndromic management will improve healing of genital ulcers and may potentially reduce HIV transmissi

17 How Haemophilus ducreyi, which causes genital ulcers and regional lymphadenitis, interacts wit

18 disease characterized by recurrent oral and genital ulcers and significant organ involvement.

19 To determine the etiology of genital ulcers and to assess the prevalence of human imm

20 ariety of serum specimens from patients with genital ulcers and urethritis and from healthy blood don

21 The annual incidences of genital ulcers and urethritis were 4.2% and 15.5%, respe

22 djustments for other host factors (age, sex, genital ulcer, and index partner's virus load) known to

23 smission (male-to-female or female-to-male), genital ulcers, and carriage of the putative ligand (HLA

24 sexually transmitted infections resulting in genital ulcers, and endemic infectious diseases (e.g., m

25 Syphilitic genital ulcers are cofactors for the bidirectional trans

26 identified unprotected sex with a CSW and a genital ulcer as independent risk factors associated wit

27 The presence of an active genital ulcer at the time of screening was found in 46 (

28 ve risk 0.53 [0.46-0.62]) and HSV-2 positive genital ulcers by 63% (0.37 [0.31-0.45]) in the aciclovi

29 <0.001) and the occurrence of HSV-2-positive genital ulcers by 73% (risk ratio, 0.27; 95% CI, 0.20 to

30 IV-1 virions can consistently be detected in genital ulcers caused by HSV-2, which suggests that geni

31 We evaluated genital ulcer disease (GUD) and HSV-2-associated GUD at

32 higher virus set point, and the presence of genital ulcer disease (GUD) during the early phase of HI

33 es simplex virus type 2 (HSV-2) shedding and genital ulcer disease (GUD) has not been evaluated.

34 dard laboratory methods for the diagnosis of genital ulcer disease (GUD) in 105 patients; 36% were hu

35 d with cervicovaginal HSV-2 DNA shedding and genital ulcer disease (GUD) in a cohort of women living

36 Viremia was increased with genital ulcer disease (GUD) in both subjects with incide

37 Monocytes recruited to genital ulcer disease (GUD) sites express increased leve

38 ltaneously detects the three major causes of genital ulcer disease (GUD), Haemophilus ducreyi, Trepon

39 uman immunodeficiency virus (HIV) infection, genital ulcer disease (GUD), penile epithelial trauma, m

40 tive men with urethritis and with or without genital ulcer disease (GUD).

41 ; 95% confidence interval [CI], 1.9-8.3) and genital ulcer disease (HRR=2.5; 95% CI, 1.1-5.3).

42 pecimens obtained from 163 patients (96 with genital ulcer disease [GUD]).

43 ates for genital-tract infections (syphilis, genital ulcer disease [GUD], Neisseria gonorrhoeae infec

44 asma HIV-1 RNA levels among, and presence of genital ulcer disease among HIV-1-infected partners and

45 irus type 2, human papillomavirus (HPV), and genital ulcer disease among men, and it reduces HPV, gen

46 HSV-2 infection is a common cause of genital ulcer disease and a significant public health co

47 uld have additional benefits beyond reducing genital ulcer disease and HSV-associated HIV transmissio

48 herpes simplex virus (HSV) type 2-associated genital ulcer disease and lesional HSV shedding.

49 Among 40 consecutive patients with genital ulcer disease and with sufficient sample for bot

50 vir had a smaller effect on the frequency of genital ulcer disease as well as a smaller effect on the

51 chancroid, a sexually transmitted cutaneous genital ulcer disease associated with increased heterose

52 Measuring incidence density in the genital ulcer disease cases directly gave the highest es

53 reening), and a cohort study of seronegative genital ulcer disease cases were compared.

54 us lesions of the human sexually transmitted genital ulcer disease chancroid are characterized by the

55 i, a Gram-negative bacterium that causes the genital ulcer disease chancroid, activates inflammasomes

56 philus ducreyi, the etiological agent of the genital ulcer disease chancroid, binds extracellular mat

57 etiologic agent of the sexually transmitted genital ulcer disease chancroid, has been shown to assoc

58 Haemophilus ducreyi, which causes the genital ulcer disease chancroid, requires high basal lev

59 i is a hemin-requiring bacterium causing the genital ulcer disease chancroid.

60 etiologic agent of the sexually transmitted genital ulcer disease chancroid.

61 etiologic agent of the sexually transmitted genital ulcer disease chancroid.

62 tive obligate human pathogen that causes the genital ulcer disease chancroid.

63 etiologic agent of the sexually transmitted genital ulcer disease chancroid.

64 Genital ulcer disease has been epidemiologically linked

65 eyi causes chancroid, a sexually transmitted genital ulcer disease implicated in increased heterosexu

66 on an increasingly important role in causing genital ulcer disease in addition to being the primary n

67 ed diseases and are the most common cause of genital ulcer disease in the United States.

68 Haemophilus ducreyi causes chancroid, a genital ulcer disease that facilitates the transmission

69 c agent of chancroid, a sexually transmitted genital ulcer disease that facilitates the transmission

70 es simplex virus is the most common cause of genital ulcer disease worldwide.

71 on, and only at the STI clinic were marital, genital ulcer disease, and HIV-infection status associat

72 Sexually transmitted diseases, genital ulcer disease, and progesterone therapy increase

73 h and without sexually transmitted diseases, genital ulcer disease, and progesterone-predominant cond

74 d with increased risk of HIV-1 infection and genital ulcer disease, and these effects remained after

75 s who had an AIDS-defining illness or active genital ulcer disease, and those that were taking antire

76 - and late-stage infection, higher HIV load, genital ulcer disease, and younger age of the index part

77 ulcer disease among men, and it reduces HPV, genital ulcer disease, bacterial vaginosis, and trichomo

78 to assess the possible benefits of treating genital ulcer disease, chorioamnionitis, mastitis, and m

79 ducreyi, the etiologic agent of chancroid, a genital ulcer disease, produces a cell-associated hemoly

80 Transmission of HIV was associated with genital ulcer disease, syphilis, and vaginal or penile d

81 In multivariate models, vulvitis, genital ulcer disease, vaginal discharge, and Candida va

82 Haemophilus ducreyi causes chancroid, a genital ulcer disease.

83 Haemophilus ducreyi causes chancroid, a genital ulcer disease.

84 uction contributing to the pathogenesis of a genital ulcer disease.

85 HSV-2) infection is the most common cause of genital ulcer disease.

86 creyi is the etiologic agent of chancroid, a genital ulcer disease.

87 c agent of chancroid, a sexually transmitted genital ulcer disease.

88 mples from patients with chancroid and other genital ulcer diseases and from normal subjects containe

89 ubjects and patients with chancroid or other genital ulcer diseases contained antibodies to purified

90 emophilus ducreyi, is one of the most common genital ulcer diseases in developing countries.

91 ter and a 73% reduction in the occurrence of genital ulcers due to HSV-2.

92 or seroconversion during follow-up, reported genital ulcer, history of STD, and number of sex partner

93 l vaginosis (HR, 2.1), and the occurrence of genital ulcers (HR, 2.7).

94 In addition to recurrent genital ulcers, HSV-2 causes neonatal herpes, and it is

95 eased the incidence of extrapulmonary TB and genital ulcers in HIV-negative patients.

96 used PCR assays to determine the etiology of genital ulcers in patients presenting to a sexually tran

97 3 times daily) was conducted among men with genital ulcers in South Africa.

98 ulation highlights the urgency of preventing genital ulcers in the southern United States.

99 easured the microbial community structure of genital ulcers in women.

100 mplicating interpretation of the role of the genital ulcer itself in the infectiousness of HIV.

101 ood of early HIV infection the most included genital ulcers (LR, 5.4; 95% CI, 2.5-12), weight loss (L

102 V-2 infection are needed, particularly since genital ulcers may facilitate the transmission of the hu

103 virus 2 (HSV-2) is the most common cause of genital ulcers, no study has systematically evaluated th

104 Incidence of genital ulcers on examination was reduced by 47% (relati

105 Genital ulcer or whole blood specimens from patients wit

106 V disease stage, antiretroviral regimen, and genital ulcers or cervical tenderness.

107 scores indicating worse pain), the number of genital ulcers, overall disease activity, and quality of

108 iral load correlates with the development of genital ulcers, shedding also commonly occurs even when

109 lent in some US cities and that persons with genital ulcers should be a focus of HIV prevention activ

110 In Madagascar, primary care of genital ulcers should include syndromic treatment for sy

111 virus (HSV) type 2, the most common cause of genital ulcers, should be evaluated as a strategy for HI

112 We tested 112 genital ulcer specimens by the polA PCR, obtaining a sen

113 V DNA were detected in 56, 15, and 13% of 39 genital ulcer specimens, respectively, and H. ducreyi DN

114 oligonucleotides in a microwell format, 298 genital ulcer swab specimens collected in New Orleans du

115 lso had reduced risks of HSV-2 infection and genital ulcer syndrome in the past 12 months compared wi

116 eria and viruses, including those that cause genital ulcers, tested negative.

117 partners, herpes simplex virus 2 infection, genital ulcers, Trichomonas vaginalis, vaginitis or cerv

118 In 1994, an apparent outbreak of atypical genital ulcers was noted by clinicians at the sexually t

119 ana and the Central African Republic who had genital ulcers were enrolled in a randomized, double-bli

120 Genital herpes is the main cause of genital ulcers worldwide; the prevalence of herpes simpl