ライフサイエンスコーパス: genital wart

1 uman papilloma virus cervical cell lines and genital warts.

2 e 70% of cervical cancers (CxCas) and 90% of genital warts.

3 toses, superficial basal cell carcinoma, and genital warts.

4 revents two HPV types associated with 90% of genital warts.

5 ch account for approximately 90% of external genital warts.

6 n the acquisition of urethral infections and genital warts.

7 lhydrogel was developed for the treatment of genital warts.

8 (6%); gonorrhea, 5 (4%); chlamydia, 5 (4%); genital warts, 2 (1%); and pelvic inflammatory disease (

9 (12%); gonorrhea, 9 (7%); syphilis, 7 (6%); genital warts, 7 (6%); chlamydia, 5 (4%); and PID, 4 (3%

10 t and has been approved for the treatment of genital warts, actinic keratosis, and superficial basal

11 Determining the rate at which men develop genital warts after infection with alpha genus human pap

12 mulative incidence of clinically ascertained genital warts among women with incident HPV-6 or HPV-11

13 n papillomavirus (HPV)-related cutaneous and genital warts and anogenital (pre)cancer.

14 Human papillomaviruses (HPVs) cause genital warts and cancers in men.

15 evention of HPV-6-related and HPV-11-related genital warts and juvenile-onset recurrent respiratory p

16 Siripornsawan review the latest thinking on genital warts and their relation to specific viral etiol

17 of women reported ever being diagnosed with genital warts, and 1.4% of men and 3.1% of women reporte

18 HPVs also cause genital warts, and are the most common sexually transmit

19 iverse HPVs associated with cervical cancer, genital warts, and epidermodysplasia verruciformis sugge

20 ctual perceptions regarding cervical cancer, genital warts, and HPV vaccination collected from parent

21 omaviruses types 6 and 11, the main cause of genital warts, and types 16 and 18, the main cause of ce

22 6/11/16/18 infection, approximately 90% for genital warts, approximately 45% for low-grade cytologic

23 Benign genital warts are caused principally by the alpha10 viru

24 Our results suggest that genital warts are common after HPV-6 or HPV-11 infection

25 Condylomata acuminata, or genital warts, are proliferative lesions of genital epit

26 protection against both cervical cancer- and genital wart-associated types, we produced at high level

27 rofiles and promising efficacy in preventing genital warts, cervical neoplasia, and cervical cancer.

28 clinic-based reports of a marked decline in genital warts diagnoses among young people in Australia

29 More than 90% of genital warts (GW) cases are caused by human papillomavi

30 Diagnoses of genital warts (GW) in genitourinary medicine (GUM) clini

31 the risk of cancer among men and women with genital warts (GW).

32 n this cohort study, we assessed the risk of genital warts (GWs) according to timing and number of do

33 of up to 20.8% in new diagnoses of external genital warts (GWs) among women aged <19 years since the

34 istory of human papillomavirus (HPV)-related genital warts (GWs) in men are sparse.

35 A reduction in the incidence of genital warts (GWs) is one of the first markers of the e

36 e etiological agents of approximately 90% of genital warts (GWs).

37 al candidiasis, pelvic inflammatory disease, genital warts, herpes simplex virus infection, syphilis,

38 ical cancers (HPV 16, 18, 31, and 45) and in genital warts (HPV 6 and 11).

39 heat treatment correlated with regression of genital warts in a subset of patients, including at dist

40 and Human Papillomavirus (HPV) infection and genital warts in men.

41 The 24-month cumulative genital wart incidence was 57.9% (95% confidence interva

42 he infection and with abnormal Pap tests and genital warts; it was negatively associated with marriag

43 eat various infectious skin diseases such as genital warts, keratosis, and basal cell carcinoma.

44 Eligibility criteria included no history of genital warts or HIV infection.

45 Eligibility included no history of genital warts or human immunodeficiency virus.

46 a woman within the past year, no history of genital warts or penile or anal cancer, and no current d

47 no evidence of an association with prevalent genital warts (OR, 0.93, 95% CI, 0.65-1.33; 15 studies).

48 ich may play a major role in its activity in genital wart patients.

49 Twenty-seven subjects with genital warts received 3 immunizations over 4 weeks in a

50 PV) type 6 (HPV-6) and HPV-11, induce benign genital warts that rarely progress to malignancy.

51 nother topical immunomodulator, approved for genital wart treatment in adults, has also been examined

52 s between 1999-2011 involving a diagnosis of genital warts were obtained from a comprehensive nationa

53 onged rupture of membranes, and a history of genital warts were significantly associated with transmi