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1 antibody-like reagent against mycosis, wheat germ agglutinin (WGA) was linked to the effector Fc regi
2                               However, wheat germ agglutinin-detectable N-acetyl-glucosamine (GlcNAc)
3          Fumonisins were concentrated in the germ and pericarp at a rate of 322% and 188% (lot 1) and
4 ionship was found between calcium content in germ and steeping time used during nixtamalization proce
5 es of this enzyme, we used a cell-free wheat germ-based expression system in which mRNA encoding FERO
6 ays a novel and important role in primordial germ cell (PGC) development and that ephrinB1 (efnb1) is
7 nheritance correlates with higher primordial germ cell (PGC) survival probability.
8 hway links the environment and proliferative germ cell accumulation.
9 sms of MRLs and their physiological roles in germ cell and neural development, oncogenic functions in
10              Autoimmune responses to meiotic germ cell antigens (MGCA) that are expressed on sperm an
11 g miR-471-5p in Sertoli cells show increased germ cell apoptosis and compromised male fertility.
12 the human fetal testis and alteration of the germ cell biology.
13  routine follow-up, 98% in the International Germ Cell Cancer Collaborative Group good prognosis grou
14 gulators that are enriched at the primordial germ cell cytoplasmic bridge to ensure its stability dur
15                                              Germ cell death occurs in many species [1-3] and has bee
16 ous spermatogonial death and reduces overall germ cell death.
17                                Vasa is a key germ cell determinant in many animal species and is posi
18 tic germ cells by Sertoli cells is vital for germ cell development and differentiation.
19 ion by NRF1 and epigenetic modulation during germ cell development and unequivocally demonstrate a no
20                                              Germ cell development involves major reprogramming of th
21 pecific endosiRNAs present during primordial germ cell development.
22 hese de novo mutations occurred during early germ cell development.
23 e small non-coding RNAs essential for animal germ cell development.
24 for embryonic development and regulates male germ cell development.
25 ryonic cell transcription factor 1) in mouse germ cell development.
26 namic, changing size at precise times during germ cell development.
27 ptibility, consistent with failed primordial germ cell differentiation as an initiating step in oncog
28                                     However, germ cell differentiation resumed after ceasing the abla
29 otic arrest complex, to transcripts for male germ cell differentiation.
30 onal TOPBP1 deletion during pachynema causes germ cell elimination associated with defective X chromo
31 ucing apical ES degeneration, which leads to germ cell exfoliation from the seminiferous epithelium.
32 resistance to infection through the receptor germ cell expressed (gce).
33   These findings suggest that Nanos promotes germ cell fate by downregulating maternal RNAs and prote
34 st development for competency for primordial germ cell fate.
35 imal species and is posited to control avian germ cell formation.
36 XY iPSCs can be differentiated into the male germ cell lineage and functional sperm that can be used
37  the asymmetric division that segregates the germ cell lineage.
38 erm cells by actively excising and digesting germ cell lobes.
39 itiate expression of mesoderm and primordial germ cell markers asymmetrically on the embryonic and ex
40 nables distinct spatiotemporal regulation of germ cell migration.
41 roven testicular tumors (benign or malignant germ cell or stromal tumors).
42 ptor, Trapped in endoderm 1 (Tre1), mediates germ cell polarization at the onset of active migration
43  Transcriptomic analyses of these successive germ cell subtypes reveals dynamic transcription of over
44 s known about how the choice is made between germ cell survival and death.
45 re, we focus on the mechanisms that regulate germ cell survival during embryonic development in Droso
46 s who were 17 years or older, diagnosed with germ cell testicular cancer, and previously treated with
47 adverse drug reaction in adult patients with germ cell testicular cancer.
48                                              Germ cell transcripts POU5F1, TFAP2C, LIN28A, ALPP and K
49    Purpose Patients with relapsed metastatic germ cell tumor (GCT) can be cured with second-line and
50 enome-wide association studies of testicular germ cell tumor (TGCT; 3,558 cases and 13,970 controls)
51 scents with intermediate-risk (IR) malignant germ cell tumors (MGCT) if the administration of cisplat
52 nderstanding of susceptibility to testicular germ cell tumors (TGCTs), but much of the heritability r
53 icancer drug for the treatment of testicular germ cell tumors (TGCTs).
54                                              Germ cell tumors of the ovary constitute less than one p
55                                    Malignant germ cell tumors were identified in 2.8% (31 of 1097) of
56  wide spectrum of human neoplasms, including germ cell tumors, high-grade and low-grade carcinomas an
57 5% CI 20.9-27.5]) and lowest in survivors of germ cell tumours (14.0 [11.5-16.6]).
58  cells (PGCs), as well as the suppression of germ cell tumours in mice.
59 platin and carboplatin are used primarily in germ cell, breast and lung malignancies, oxaliplatin is
60 ipt levels in several tumor tissues, such as germ cell, breast, and ovarian tumors, with in the latte
61  (c.1297_8ins353, p.K433Rins31*) in the male germ cell-associated kinase (MAK) gene (39% of families
62 lies like Drosophila, the ancestral receptor germ cell-expressed (gce) gene has duplicated to yield t
63 falciparum proteins prepared using the wheat germ cell-free system (WGCFS).
64        We find that the decision to die is a germ cell-intrinsic process linked to quantitative diffe
65 rating that NHE8's role in spermiogenesis is germ cell-intrinsic.
66 ly reported ChIP-seq datasets for primordial germ cell-like cells and E18.5 small intestine, combined
67 timulated by retinoic acid gene 8 (Stra8), a germ cell-specific gene activated during meiotic commitm
68 (TSTRs) and associated with highly expressed germ cell-specific genes and histone retention in mature
69 ith distinct developmental states, including germ cell-specific genes.
70                                              Germ cell-specific, but not Sertoli cell-specific Nhe8 d
71 ration in triggering meiotic checkpoints and germ-cell death.
72 ghts piRNA deficiency or actually drives the germ-cell demise.
73               Analysis of adult Mov10l1(-/-) germ-cell fractions indicated a stage-specific increase
74 ntinue indefinitely, because of its immortal germ-cell lineage.
75 tem cells (ESCs) and gonad-derived embryonic germ cells (EGCs) represent two classic types of pluripo
76 e dysgenetic areas, with more ectopic SC and germ cells (GC) than DBP-FW treatment; DBP-LW induces no
77                             Human primordial germ cells (hPGCs), the precursors of sperm and eggs, or
78                  The migration of primordial germ cells (PGCs) from their place of origin to the embr
79                       In animals, primordial germ cells (PGCs) give rise to the germ lines, the cell
80                  The migration of primordial germ cells (PGCs) is a useful model for studying this pr
81 ave profiled the transcriptome of primordial germ cells (PGCs) lacking the nanos homologs nos-1 and n
82 omatin modifications occur in the primordial germ cells (PGCs) of emergent starved L1 larvae that cor
83 the translational activity in the primordial germ cells (PGCs) of the sea urchin embryo (Strongylocen
84 1 is required for the survival of primordial germ cells (PGCs), as well as the suppression of germ ce
85 c cells and, strikingly, to early primordial germ cells (PGCs).
86  the DDX4 (vasa) locus in chicken primordial germ cells (PGCs).
87 constructed by transplantation of primordial germ cells (PGCs).
88 o AURK isoforms (AURKA and AURKB), mammalian germ cells also express a third, AURKC.
89 ly manipulated GSC depletion, E(z) knockdown germ cells also fail to replenish lost GSCs.
90 ession pattern that is primary restricted to germ cells and aberrantly reactivated in various cancers
91  (CTCF&BORIS) or BORIS alone (BORIS-only) in germ cells and in BORIS-positive somatic cancer cells.
92 ritable L1 insertions may therefore arise in germ cells and in pluripotent embryonic cells, prior to
93              PLAG1 was sparsely expressed in germ cells and in Sertoli cells.
94 ed to support cell adhesion and transport of germ cells and organelles (e.g., residual bodies, phagos
95 ion into late primordial germ cells, meiotic germ cells and ovarian follicles.
96 condensate (CSC) causes molecular defects in germ cells and phenotypic effects in their offspring.
97 ialized region of the oocyte that prefigures germ cells and specifies the germline of descendants in
98 ombinant human GDF9 and BMP15, these meiotic germ cells are further induced to form ovarian FLCs, inc
99 f two modes: a likely ancestral mode wherein germ cells are induced during embryogenesis by cell-cell
100  oocyte to embryo, genetic programs in mouse germ cells are reshaped by chromatin remodeling to orche
101 ich the fittest, strongest, or least damaged germ cells are selected for transmission to the next gen
102 g (induction) or a derived mechanism whereby germ cells are specified by using germ plasm-that is, ma
103                              Male and female germ cells are usually produced during spermatogenesis a
104 impaired LAP-mediated clearance of apoptotic germ cells as miR-471-5p transgenic mice show lower leve
105 rational inheritance by invading presumptive germ cells before they are formed.
106  cells interact with and regulate primordial germ cells by actively excising and digesting germ cell
107 P.Although phagocytic clearance of apoptotic germ cells by Sertoli cells is essential for spermatogen
108            Phagocytic clearance of apoptotic germ cells by Sertoli cells is vital for germ cell devel
109 sential for efficient clearance of apoptotic germ cells by Sertoli cells using LAP.Although phagocyti
110                         Our data showed that germ cells cultured with 5H-purin-6-amine could maintain
111                                              Germ cells develop as a cyst of interconnected sibling c
112                 The beta-TrCP-deficient male germ cells did not enter meiosis, but instead underwent
113 enesis, a process through which haploid male germ cells differentiate into spermatozoa, represents an
114  events that characterize the development of germ cells during fetal life as they commit to, and prep
115 ent, Arf-null mice are blind, and their male germ cells exhibit defects in meiotic maturation and spe
116 piRNA-deficient mice, L1-overexpressing male germ cells exhibit excessive DNA damage and meiotic defe
117                 We show that mouse and human germ cells exhibit non-canonical X dosage states that di
118                         We hypothesized that germ cells express negative regulators of nucleic acid s
119          Somatic gonadal precursor cells and germ cells fail to proliferate fully and complete their
120 sophila oocyte defines where the abdomen and germ cells form in the embryo.
121 ly, DPY-21 also associates with autosomes of germ cells in a DCC-independent manner to enrich H4K20me
122 trans-epithelial migration of the primordial germ cells in early embryos.
123         Two broadly known characteristics of germ cells in many organisms are their development as a
124                                              Germ cells in most animals are connected by intercellula
125  developmental accumulation of proliferative germ cells in the C. elegans hermaphrodite is sensitive
126 verexpression rescues clearance of apoptotic germ cells in the testes of Mertk (-/-) mice it fails to
127 adotropes of the pituitary and in Leydig and germ cells in the testes, but at very low levels in Sert
128      Whether X dosage compensation occurs in germ cells is unclear.
129 ine (Aub) maintains genome integrity in late germ cells of the Drosophila ovary through Piwi-associat
130                        In mammalian females, germ cells remain arrested as primordial follicles.
131 unctions, and speculates as to why mammalian germ cells require expression of three AURKs instead of
132                 Inactivation of PRC1 in male germ cells results in the gradual loss of a stem cell po
133 M was more strongly expressed in human fetal germ cells than in somatic cells.
134        Bombyx mori BmN4 cells are culturable germ cells that equip the piRNA pathway.
135 could increase true SSC concentration within germ cells to 1.96-fold.
136 data suggest that E(z) acts intrinsically in germ cells to activate dedifferentiation and thus replen
137 tivates the GLP-1/Notch receptor on adjacent germ cells to maintain germline stem cell fate.
138 roalgal life cycles - from the production of germ cells to the growth and fertility of the adult orga
139 rgeted and reversible ablation of premeiotic germ cells undergoing differentiation into oocytes in tr
140 er proteins regulates clearance of apoptotic germ cells via LC3-associated phagocytosis (LAP).
141                      To find the components, germ cells were cultured with comprehensive natural plan
142 d its expression promotes spermatogenesis in germ cells when they are present in a male soma.
143                                During aging, germ cells with reduced E(z) activity cannot meet that r
144 at SMG-1 mainly acts in mitotically dividing germ cells, and during late embryonic and larval develop
145 on in the dermal papilla, the secondary hair germ cells, and the epidermis.
146  are required for normal piRNA biogenesis in germ cells, are dispensable.
147 A-binding proteins specifically expressed in germ cells, DAZL and BOULE, regulate the exit from pluri
148 7 exhibits male-specific expression in early germ cells, germline stem cells and spermatogonia in ins
149 em cell differentiation into late primordial germ cells, meiotic germ cells and ovarian follicles.
150 lean switch from mitosis to meiosis in mouse germ cells, revealing a conserved role for YTHDC2 in thi
151  compartment, which surrounds the nucleus of germ cells, suggesting that sequestration of RNA to this
152                                Surprisingly, germ cells, which do not arise from the CE, were also af
153 genetic features of closed chromatin in male germ cells, which suggests that CNVs may repress recombi
154 the mitosis-meiosis transition in mouse male germ cells.
155 matin upon sex differentiation in developing germ cells.
156 ites (BORIS) are simultaneously expressed in germ cells.
157  ovaries can result in further rapid loss of germ cells.
158 autophagy member proteins to clear apoptotic germ cells.
159 of gamete arise from the same precursor, the germ cells.
160  and in adult post-mitotic and proliferative germ cells.
161 ist of two cell lineages - somatic cells and germ cells.
162 e-encoding mature mRNA isoform in Drosophila germ cells.
163  associated with hypoadenylated mRNAs (e.g., germ cells/neurons) and/or limiting cytoplasmic PABP (e.
164 ooth germs while their maxillary molar tooth germs completed morphogenesis.
165   This research studied the influence of the germ components on the physicochemical properties of coo
166 ents an intermediate between short- and long-germ development, ideal for comparative study of PRGs.
167 res ancestral to arthropods, including short germ embryogenesis.
168 st blocks to both Escherichia coli and wheat germ extract translation systems, whereas N2-methylguano
169                In this article, we show that germ-free (GF) and Toll-like receptor-2 (Tlr2)-deficient
170 tributes to tumor distribution, we generated germ-free (GF) Apc(Min/+) and Apc(Min/+) ;Il10(-/-) mice
171 crobiota in specific pathogen-free (SPF) and germ-free (GF) mice given more than 40 unique diets; we
172 typically, and functionally compared between germ-free (GF), specific pathogen-free, and GF mice reco
173                     Here we demonstrate that germ-free adult pregnant mice inoculated with a gut micr
174 Oral tolerance was induced by DNFB gavage in germ-free and mice deficient in several TLRs.
175 ective role was observed in conventional and germ-free animal facilities, indicating that it does not
176 ystem, with for example the observation that germ-free animals harbor a poorly developed intestinal i
177 endent manner, as assessed by antibiotic and germ-free approaches.
178 intestinal microbiota by oral antibiotics or germ-free condition can prevent arthritis in mice.
179 ated with certain antibiotics or raised in a germ-free environment.
180 st variation in colonization when individual germ-free flies were fed their own natural commensals (i
181 on tissue sections from immunocompromised or germ-free hosts, chronically infected hosts where the ti
182                                      We used germ-free mice (GF) to assess visceral sensitivity, spin
183                  Restoring the microbiota of germ-free mice abrogated this protection.
184 llus reuteri This species induced DP IELs in germ-free mice and conventionally-raised mice lacking th
185 duced mechanical hyperalgesia was reduced in germ-free mice and in mice pretreated with antibiotics.
186 rance induced by DNFB gavage was impaired in germ-free mice and TLR4-deficient mice.
187                                              Germ-free mice are protected from CCM formation, and a s
188                       A higher proportion of germ-free mice live to 600 days than their conventional
189 nterparts, and macrophages derived from aged germ-free mice maintain anti-microbial activity.
190 ith a favorable gut microbiome as well as in germ-free mice receiving fecal transplants from respondi
191 er, in vivo administration of NOD1 ligand to germ-free mice restored the numbers of hematopoietic ste
192 s or mice (IBD, metabolic syndrome, etc.) to germ-free mice was found to be sufficient to transfer so
193       Young adult specific-pathogen-free and germ-free mice were used to delineate the commensal micr
194                                   Co-housing germ-free mice with old, but not young, conventionally r
195                          By colonizing adult germ-free mice with the cecal contents of neonatal and a
196 hted by anatomical and functional changes in germ-free mice, affecting the gut epithelium, immune sys
197 diabetic mice; when transferred to recipient germ-free mice, oral microbiota from IL-17-treated donor
198 ve effects were lost in both Ifnar1(-/-) and germ-free mice, revealing essential roles for type I int
199 al bacterial composition and, by transfer to germ-free mice, that the oral microbiota of diabetic mic
200 ensal colonization in antibiotic-treated and germ-free mice, using cultured commensals from the Actin
201                        DP IELs are absent in germ-free mice, which suggests that their differentiatio
202 sed mice, but provide no growth advantage in germ-free mice.
203 bial origin since no formate was detected in germ-free mice.
204 or after the introduction of microbiota into germ-free models.
205                                     Further, germ-free neonates had reduced skin Tregs indicating tha
206 ed in specific-pathogen-free conditions into germ-free Nlrp12-deficient mice showed that NLRP12 and t
207 m cancer patients who responded to ICIs into germ-free or antibiotic-treated mice ameliorated the ant
208 gnificant decrease in serum triglycerides in germ-free rats fed a high sugar diet compared to convent
209  that critically ill humans never exist in a germ-free state.
210        Investigations included an intestinal germ-free study and a C15:0/C17:0 diet dose response stu
211 ferences in the number of secretory cells in germ-free zebrafish and their conventional counterparts,
212 erved a broad convergence of interactions of germ granule and P body mRNP components on AIN-1/GW182 a
213 he CCR4-NOT complex, decapping and decay, or germ granule proteins.
214 ly disordered N-terminal 236 residues of the germ-granule protein Ddx4.
215  and identify a functional continuum between germ granules and P bodies in the C. elegans embryo.
216 se the proteins involved in the formation of germ granules to coalesce into liquid droplets.
217 tomesenchymal cells that surrounds the tooth germ in early stages of tooth development.
218  degree of mineralization of permanent tooth germs in dental age assessment has been an area of inter
219 ptome of small groups of cells from a single germ layer and to retain spatial information, dorsal and
220                    Our results indicate that germ layer induction in the zebrafish tailbud is not a s
221 ormation and ingression, known mechanisms of germ layer internalisation.
222 e critical molecular signaling inputs during germ layer specification in bilaterian metazoans, but th
223 sensory placode specification, ciliogenesis, germ layer specification).
224 des evidence that the endoderm was the first germ layer to evolve.
225                                              Germ-layer formation during gastrulation is both a funda
226 b-group proteins, which coordinate embryonic germ-layer formation in response to extraembryonic cues.
227    Overall, these results lead to a model of germ-layer formation in which, upon N-cadherin expressio
228 avoidance as an unexplored mechanism driving germ-layer formation.
229 cation, leading to the formation of distinct germ layers and specialized tissues.
230 n, the relative mRNA expression in the three germ layers and the trophoblast was abnormal in the EBs
231 y-active somatic cells derived from separate germ layers can be interconverted.
232 rived organoids with components of all three germ layers have been generated, resulting in the establ
233 iotemporal pattern of gene expression across germ layers provides evidence that the endoderm was the
234 eposited as pillars between widely separated germ layers, namely the somitic mesoderm and the endoder
235 tency and commit to multiple lineages in all germ-layers.
236 CH1/FBXW7 (N/F) mutations and RAS/PTEN (R/P) germ line (GL) were classified as oncogenetic low risk (
237 l IgE production was quantified with epsilon germ line (IGHE) transcripts and correlated with serum I
238          Mice with reduced VGF levels in the germ line (Vgf+/-) or in dHc (AAV-Cre-injected floxed mi
239  are the most recent entrants into the human germ line and are transcriptionally active.
240 activity is important in the nervous system, germ line and intestine.
241              Patients underwent searches for germ line and somatic mutations using next-generation se
242 ble analyses of the role of Cdk5 in GVHD, as germ line Cdk5 gene deletion is embryonically lethal.
243 e MAGE gene family that is expressed in male germ line cells and placenta under normal physiological
244 rprisingly, dramatically affected by the non germ line encoded portions of CDR3 of the T cell recepto
245 te the antiquity of some, and possibly most, germ line HHV-6 integrations, the majority of ciHHV-6B (
246 ere able to assign a causal or likely causal germ line mutation in 86 patients (48.0%), involving a t
247                                  Conversely, germ line mutations in GATA2 are associated with GATA2 d
248 or ETAA1 in this process by surveying random germ line mutations in mice using exome sequencing and b
249                                              Germ line mutations of the gene encoding the tricarboxyl
250  NOD2 mutations, apart from 1 patient with a germ line NLRP3 p.V198M substitution.
251            Genetic analysis revealed neither germ line nor somatic NLRP3, TNFRSF1A, NLRC4, or NOD2 mu
252                                              Germ line Notch2(COIN) inversion phenocopied the Notch2H
253   Here, we show that knockdown of Hop in the germ line nurse cells (GLKD) of Drosophila ovaries leads
254                          As individuals with germ line predisposition to hematologic malignancies are
255 ation as well as disease progression such as germ line predisposition, inflammation, and aging.
256 o the accumulation of gamma-H2Av foci in the germ line, indicating increased DNA damage in the ovary.
257 cific AHA-variable regions were mutated from germ line-derived sequences and displayed a high sequenc
258 teracting RNAs (piRNAs) are 26-30-nucleotide germ line-specific small non-coding RNAs that have evolu
259 ous isoform in all tissues (LIG3alpha) and a germ line-specific splicing isoform (LIG3beta) that diff
260 , specifically in the lineage leading to the germ line.
261 e movement of mobile genetic elements in the germ line.
262 eplication of mobile genetic elements in the germ line.
263  sex chromosomes may be specific to the male germ line.
264 lular differentiation and maintenance of the germ line.
265 ing germ plasm-that is, maternally specified germ-line determinants (inheritance).
266 nd mutations during affinity maturation, the germ-line IG loci are also diverse across human populati
267 iments with purified recombinant iPLA2gamma, germ-line iPLA2gamma(-/-) mice, cardiac myocyte-specific
268                                              Germ-line knockouts of CCR2 or treatment with an anti-CC
269                                  Conversely, germ-line loss of E2f1 or E2f3b, but not E2f3a, protecte
270 tic mutations contribute little to the human germ-line mutation rate.
271                                              Germ-line mutations in components of the Ras/MAPK pathwa
272                                 Heterozygous germ-line mutations in the bone morphogenetic protein ty
273                                      Whether germ-line or somatic alterations in these genes or other
274                                              Germ-line pathogenic variants in components of the H2AUb
275 that gene expression remains inactive in the germ-line precursors during adverse conditions.
276 ng with diploid spermatogonia, which include germ-line stem cells, and ending with haploid spermatozo
277 roach that enables us to model the effect of germ-line variation on tissue-specific gene expression i
278 rimordial germ cells (PGCs) give rise to the germ lines, the cell lineages that produce sperm and egg
279 n chronological age and the formation of the germ of the second lower premolar (r=0.67; p<0.001).
280  of non-transgenic corn and their fractions (germ, pericarp, endosperm, cornmeal and grits), collecte
281 that could explain the repeated evolution of germ plasm and propose potential consequences of the inh
282 , whereby Tahre retroelements traffic to the germ plasm by mimicking oskar RNAs and engaging the Stau
283 as been hypothesized that the acquisition of germ plasm confers enhanced evolvability, resulting from
284                        Consistent with this, germ plasm determinants attracted retroelement RNAs even
285                   The causes of the shift to germ plasm for PGC specification in some animal clades r
286  in germ plasm inheritance, such that higher germ plasm inheritance correlates with higher primordial
287 rocess linked to quantitative differences in germ plasm inheritance, such that higher germ plasm inhe
288                Here, we consider reasons why germ plasm might be neither a direct target of selection
289 ey ensure selection of the PGCs with highest germ plasm quantity and least cellular damage.
290         Together, these results suggest that germ plasm targeting represents a fitness strategy adopt
291 e classes of retrotransposons migrate to the germ plasm, a specialized region of the oocyte that pref
292 s, and resulting in high speciation rates in germ plasm-containing lineages (denoted herein as the "P
293 sm whereby germ cells are specified by using germ plasm-that is, maternally specified germ-line deter
294 ed that calcium carbonate is formed into the germ structure to 2.1 w/w of calcium hydroxide and 9h st
295 on wounded hosts, but significantly promoted germ tube elongation and sclerotium production.
296  morphology, characterized by emergence of a germ tube from the yeast cell followed by mold-like grow
297 es pombe Germinating spores develop a single germ tube that emerges from the outer spore wall in a pr
298 e enriched in the spore body relative to the germ tube.
299 pmental arrest of the mandibular molar tooth germs while their maxillary molar tooth germs completed
300 nogaster Whereas Drosophila embryos are long-germ, with segments specified more or less simultaneousl

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