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1 transcriptional programme similar to that of germ cell tumours.
2 chemotherapy-induced apoptosis in testicular germ cell tumours.
3 elatively frequent loss of heterozygosity in germ cell tumours.
4 miology, genetics, and biology of testicular germ cell tumours.
5 s in the clinical management of intracranial germ-cell tumours.
6 cal management of patients with intracranial germ-cell tumours.
7 progression-free survival for patients with germ-cell tumours.
8 maeric embryos, and is associated with human germ-cell tumours.
9 cryptorchidism, risk factors for testicular germ-cell tumours.
10 34 patients with cancer, 53% had testicular germ-cell tumours.
11 eview the assessment and management of early germ-cell tumours.
12 e of approaches offer high survival in early germ-cell tumours.
13 tility and increases risk for development of germ-cell tumours.
15 ise the consensus on how to treat testicular germ cell tumours and focus on a few controversies and i
17 ntly overexpressed in human non-seminomatous germ cell tumours and transitional carcinoma of the blad
18 ion or death in patients with poor prognosis germ-cell tumours and an unfavourable tumour marker decl
28 the results obtained in treating metastatic germ-cell tumours are superior to those with other solid
29 operitoneal, or mediastinal non-seminomatous germ cell tumours based on histological findings or clin
33 a indicating fewer second primary testicular germ-cell tumours favour carboplatin use, these findings
36 stratification, and treatment approaches for germ-cell tumours have evolved disparately along several
40 of in certain pathological contexts such as germ-cell tumours, melanoma or human immunodeficiency vi
41 Fewer than 70 new cases of malignant ovarian germ cell tumours (MOGCTs) are seen each year in the UK.
42 rs, the malignant cells in 73% of testicular germ-cell tumours (seminomas and teratomas), expressed h
43 Tumours within this group are testicular germ-cell tumours (such as benign teratoma, epidermoid c
44 icipants of the 2013 Third International CNS Germ Cell Tumour Symposium (Cambridge, UK) agreed to und
47 identified multiple risk loci for testicular germ cell tumour (TGCT), revealing a polygenic model of
54 es that control susceptibility to testicular germ-cell tumours (TGCTs), the most common cancer affect
58 tements encompassing aspects of intracranial germ-cell tumour work-up, staging, treatment, and follow
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