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1 rimates (chimpanzee, gorilla, orangutan, and gibbon).
2 ha2-globin-containing unit in both human and gibbon.
3 he absence of its putative ligand, MHC-G, in gibbons.
4 tains Saitohin in chimpanzees, gorillas, and gibbons.
6 or recognition site in the Alu repeat of the gibbon and a G-->A substitution in the last position of
7 e duplication and dispersion taking place in gibbon and involving loci corresponding to human chromos
9 suggesting that KoRV predates GALV and that gibbons and koalas acquired the virus at different times
11 11 bonobos, 48 gorillas, 37 orangutans and 2 gibbons and observed undescribed variation in great apes
12 ), Nomascus nastusus and Hylobates pileatus (gibbons) and from the New World monkey, Lagothrix lagotr
14 compare the additive model, due to Mesterton-Gibbons, and the multiplicative model, due to Parker, of
15 trovirus (binds Pit-2 receptor) but not with gibbon ape leukemia retrovirus (binds Pit-1 receptor), i
17 y (CHO) cells are resistant to infections by gibbon ape leukemia virus (GALV) and amphotropic murine
20 press distinct but related receptors for the gibbon ape leukemia virus (GALV) and the amphotropic mur
22 kemia virus (MuLV), the related protein from gibbon ape leukemia virus (GaLV) does not form functiona
23 der the direction of MoMSV LTR and using the gibbon ape leukemia virus (GALV) Env for internalization
24 virus (F-MLV) Env, but not with the related gibbon ape leukemia virus (GaLV) Env or with a chimeric
25 ant retroviruses pseudotyped with either the gibbon ape leukemia virus (GaLV) envelope or the vesicul
26 structed functional immunologically reactive gibbon ape leukemia virus (GALV) envelope proteins, tagg
27 g an optimized transduction protocol using a gibbon ape leukemia virus (GaLV) envelope-containing pac
28 e genes from each of the five members of the gibbon ape leukemia virus (GALV) family of type C retrov
30 has remained the only sequence implicated in gibbon ape leukemia virus (GALV) infection, and an acidi
31 ic murine retrovirus-related virus (XMRV) or gibbon ape leukemia virus (GALV) infection, even when th
32 were significantly higher than the level of gibbon ape leukemia virus (GaLV) receptor mRNA in cells
33 s targeting the amphotropic receptor and the gibbon ape leukemia virus (GALV) receptor Pit-1 were use
34 virus preparations of murine leukemia virus, gibbon ape leukemia virus (GALV), and simian sarcoma-ass
35 ity with the exogenous and highly infectious gibbon ape leukemia virus (GALV), the infectivity of KoR
36 y::Fur)] and the fusogenic glycoprotein from gibbon ape leukemia virus (GALV), which it was hoped wou
37 4-enriched marrow cells were cocultivated on gibbon ape leukemia virus (GALV)-based retrovirus vector
38 aging cells FLYRD (LgGLSN and LNX) or by the gibbon ape leukemia virus (GALV)-pseudotype packaging ce
39 nto baboon marrow repopulating cells using a gibbon ape leukemia virus (GALV)-pseudotype retroviral v
47 photropic murine leukemia virus (A-MuLV) and gibbon ape leukemia virus (GALV); E36 cells are highly s
48 e greatest nucleic acid sequence identity to gibbon ape leukemia virus and murine leukemia virus.
51 n packaging cell lines containing either the gibbon ape leukemia virus envelope (PG13 cells), the mur
53 ectofusin-1 variants to promote the modified gibbon ape leukemia virus glycoprotein-pseudotyped lenti
56 Here we show that MDEV is also not in the gibbon ape leukemia virus or RD114 virus interference gr
58 generated a human packaging cell line with a gibbon ape leukemia virus pseudotype (Phoenix-GALV), and
59 ll surface phosphate transport proteins, the gibbon ape leukemia virus receptor Glvr-1 (Pit-1) or the
60 B feline leukemia viruses (FeLV-Bs) use the gibbon ape leukemia virus receptor, Pit1, as a receptor
61 sions and was demonstrated with amphotropic, gibbon ape leukemia virus, and vesicular stomatitis viru
62 trast, the slightly different sequences from Gibbon ape leukemia virus, Moloney leukemia virus, PSAPP
64 For numerous gammaretroviruses, such as the gibbon ape leukemia virus, woolly monkey virus, feline l
70 iruses (MuLVs), feline leukemia viruses, and gibbon-ape leukemia virus, encode an alternate, glycosyl
71 lication of somatic gene therapy by use of a gibbon-ape-leukaemia-virus pseudotyped gammaretroviral v
72 EATO (GALV-S), were originally isolated from gibbon apes, whereas the fifth member of this family, si
74 ad that genetic variation data in humans and gibbons as well as in Old World monkeys are inconsistent
76 A substantial number (11/24) of human-NLE gibbon breakpoints showed new insertions of gibbon-speci
78 neuron number in the LP-pulvinar complex in gibbon, chimpanzee, and gorilla compared to humans, howe
81 In other species, such as the orangutan and gibbon, FISH signals were only identified at the distal
86 HBV variants, including evidence for a novel gibbon/genotype C recombinant among HBV variants from Vi
87 are 1, 4, 13, 13, 17, and 17 repeats in the gibbon, gorilla, orangutan, bonobo, neanderthal, and hum
89 ough diverse and irregular in structure, the gibbon haplotypes are unusually small, containing only t
93 elevant to the cross-species transmission to gibbons in Southeast Asia and broadens the known distrib
94 ) consisting of the mature coding portion of gibbon interleukin-3 (IL-3) and full-length FAC in Esche
96 Phylogenetic analysis indicated that the gibbon isolates lie within the human HBV family, indicat
97 hominoid KIR haplotype indicates that modern gibbon KIR haplotypes were formed by a series of deletio
99 ces shaping the size and organization of the gibbon KIR locus differed from those acting upon the KIR
100 noids lived in an environment that favored a gibbon-like size, but a series of selective regime shift
103 pecies were added in the last year including gibbon (Nomascus leucogenys) and Tasmanian devil (Sarcop
105 teny breakpoint regions in the white-cheeked gibbon (Nomascus leucogenys, NLE) in the form of high-qu
106 plays performed by four adult female cao vit gibbons (Nomascus nasutus) residing in four polygynous g
109 t the non-human HBV clades in orangutans and gibbons resulted from cross-species transmission events
110 es from the human and from the orangutan and gibbon revealed wide overlap of elements across species,
112 s and the great apes but not in lesser apes (gibbon, siamang) or lower-order primates (e.g., old or n
113 gibbon breakpoints showed new insertions of gibbon-specific repeats and mosaic structures formed fro
118 between members of genotypes A, D, F/H, and gibbon variants but not in B, C, or the Asian B/C recomb
119 n of an ancestral retrovirus into koalas and gibbons via one or more intermediate as-yet-unknown host
121 s of four KIR haplotypes from two species of gibbon, we find that the smaller apes do not conform to
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