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1 to control (15%+/-0.6 oleoresin, 6.4%+/-0.4 gingerol).
2 rption to simultaneously release ASA and [6]-gingerol.
3 was largely owing to 10-gingerol, but not 6-gingerol.
4 in the fresh ginger extract, followed by 10-gingerol.
5 t LTA(4)H might be a potential target of [6]-gingerol.
6 ocyanate (ITC), cinnamaldehyde, allicin, and gingerol].
7 ed higher yield of oleoresin (20%+/-0.5) and gingerol (12.2%+/-0.4) compared to control (15%+/-0.6 ol
8 action with ethanol provided higher yield of gingerol (6.2-6.3%) than the control (5.5%) with compara
10 ginger (Zingiber officinale Roscoe) viz. [6]-gingerol, [6]-shogaol and zingerone exhibited quorum sen
13 pared with other ginger compounds, such as 6-gingerol and 6-shogaol, 6-DHSG showed the most potent an
14 related compounds of the ginger family, [6]-gingerol and [6]-paradol, on EGF-induced cell transforma
16 rase increased the yield of [6]-, [8]-, [10]-gingerols and [6]-shogaol by 64.10, 87.8, 62.78 and 32.0
17 Parameters optimized for maximum recovery of gingerols and [6]-shogaol were ammonium sulphate concent
18 Except for zingerone and 6-gingerol, other gingerols and shogaols at a concentration of 20 muM inhi
19 The pungent bioactive principles of ginger, gingerols and six other gingerol-related compounds were
20 These results suggest that phloretin and [6]-gingerol are potential dietary compounds that can allevi
22 thways such as "stilbenoid, diarylheptanoid, gingerol biosynthesis" were enriched in high weight rabb
26 ficacy in cancer, the mechanism by which [6]-gingerol exerts its chemopreventive effects remains elus
27 pothesized that phloretin from apple and [6]-gingerol from ginger inhibit formation of AGEs and suppr
31 activities of fresh ginger extract and seven gingerol-related compounds on the neuro-inflammation wer
32 rinciples of ginger, gingerols and six other gingerol-related compounds were determined and identifie
35 supported our prediction by showing that [6]-gingerol suppresses anchorage-independent cancer cell gr
36 also support the anticancer efficacy of [6]-gingerol targeting of LTA(4)H for the prevention of colo
41 anced the anti-neuroinflammatory capacity of gingerols yet, conversely, attenuated those of shogaols.
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