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1 ap (CAF) has been shown to effectively treat gingival recession.
2 y advanced flap (CAF) has been used to treat gingival recession.
3 e an alternative graft material for treating gingival recession.
4 ationship between occlusal discrepancies and gingival recession.
5 he current treatment of choice for reversing gingival recession.
6 as subepithelial grafts for the treatment of gingival recession.
7  an effective procedure for the treatment of gingival recession.
8 dly broadened clinicians' abilities to treat gingival recession.
9 obing depths, clinical attachment level, and gingival recession.
10 igating the relationship between smoking and gingival recession.
11 erage techniques at reducing CDH in cases of gingival recession.
12 zyme for the modulation of bone turnover and gingival recession.
13 ntal elements to determine probing depth and gingival recession.
14 cting optimally effective protocols to treat gingival recession.
15 omes of clinical treatments in patients with gingival recession.
16 ed for reproducible volumetric evaluation of gingival recession.
17 h, good preservation of the papillae, and no gingival recession.
18 echnique using PCG was effective in reducing gingival recession.
19 connective tissue to treat multiple areas of gingival recession.
20 ough #31 were classified as Miller Class III gingival recession.
21 e tissue grafts performed to cover localized gingival recessions.
22 als in the treatment of Miller Class I or II gingival recessions.
23  advanced flap (CAF) procedures in localized gingival recessions.
24 ical benefits in the treatment of teeth with gingival recessions.
25 of a surgical microscope in the treatment of gingival recessions.
26 (PD; 6.3 +/- 1.1 mm and 6.2 +/- 1.1 mm), and gingival recession (0.1 +/- 1.9 mm and 0.2 +/- 1.4 mm).
27  and was associated with minimal increase in gingival recession (0.4 +/- 0.7 mm).
28  and was associated with minimal increase in gingival recession (-0.1 +/- 0.7 mm).
29 ix patients participated, each providing one gingival recession; 19 received citric acid demineraliza
30  one or more tooth surfaces with > or = 3 mm gingival recession; 53.2 million have gingival bleeding;
31                                              Gingival recession accounted for a significant amount of
32 h severe attachment loss of sudden onset and gingival recession affecting the facial right surfaces o
33 sis of acute gingivitis, chronic gingivitis, gingival recession, aggressive or acute periodontitis, c
34 , generalized early-onset periodontitis, and gingival recession all had similar levels of anti-PC IgG
35 ssue graft (CTG) is a popular means to treat gingival recession and augment keratinized tissue.
36 ly significant difference was only found for gingival recession and bone fill, yielding a more favora
37 cks had the highest prevalence and extent of gingival recession and dental calculus, whereas Mexican
38 n a differential diagnosis in cases of rapid gingival recession and dental erosion of unknown etiolog
39 esponse to treatment of a patient with rapid gingival recession and dental erosion secondary to local
40 crevicular fluid (GCF) samples from sites of gingival recession and saliva; and 2) clinical outcomes
41 ar) and tongue barbell size (stem length) on gingival recession and tooth chipping.
42                                              Gingival recession and white mucosal lesions frequently
43 ex, probing depth, probing attachment level, gingival recession, and bleeding on probing.
44 e index, bleeding on probing, probing depth, gingival recession, and clinical attachment level (CAL).
45 ue measurements included probing depth (PD), gingival recession, and clinical attachment level (CAL).
46 tachment loss and probing depth, > or = 3 mm gingival recession, and dental calculus.
47 , vertical clinical attachment level (VCAL), gingival recession, and horizontal probing depth (HPD).
48 ding index, probing depth, attachment level, gingival recession, and open horizontal and vertical fur
49  presentation were pain, rapidly progressive gingival recession, and significant changes in alveolar
50 ctory solution in the treatment of localized gingival recessions, and 2) citric acid demineralization
51 (PD); 2) clinical attachment level (CAL); 3) gingival recession; and 4) percentage of sites with blee
52 nship; 3) previous orthodontic treatment; 4) gingival recession; and 5) band of keratinized gingiva f
53      Dental calculus, gingival bleeding, and gingival recession are common in the U.S. adult populati
54                                 The areas of gingival recession associated with teeth #18 through #22
55 nt increase in the rate of CAL gain, reduced gingival recession at 3 months post-surgery, and improve
56  higher gingival index and amount of lingual gingival recession at both time periods compared to cont
57  investigators, included CAL, probing depth, gingival recession, bleeding on probing (BOP), visible p
58 inical attachment loss (AL), the presence of gingival recession, bleeding on probing, and full-mouth
59 to limit the post-surgery increase in buccal gingival recession (bREC), effect of a connective tissue
60 inically evaluate the treatment of localized gingival recessions by using gingival unit grafts (palat
61                                     The mean gingival recession changed from +0.1 mm at surgery to 1.
62 ntly different clinical attachment level and gingival recession changes by the end of the maintenance
63 ericans had similar prevalence and extent of gingival recession compared with non-Hispanic whites.
64 the RC, KMW, or CAL of Miller Class I and II gingival recessions compared with the other treatment mo
65 ative, can be successfully used in obtaining gingival recession coverage.
66                           Treatment of human gingival recession defects by means of either GTR or CTG
67 of these two therapies in surgically created gingival recession defects in restoring missing cementum
68 nty-six patients with Miller's Class I or II gingival recession defects of 2.5 mm were recruited for
69                Overall, data from 325 single gingival recession defects revealed a statistically sign
70 etic absorbable devices for the treatment of gingival recession defects should be advised of the poss
71 ) in combination with a CAF in subjects with gingival recession defects using a randomized, controlle
72 ted that regeneration of the periodontium in gingival recession defects was possible through a growth
73         Seventeen patients with 40 bilateral gingival recession defects were compared.
74                                 Standardized gingival recession defects were surgically created on th
75 randomized controlled trial, Miller Class II gingival recession defects were treated with either a co
76 d root coverage has been utilized to correct gingival recession defects with promising results.
77                                Management of gingival recession defects, a common periodontal conditi
78  polylactic acid barrier was used to correct gingival recession defects, were evaluated.
79  coverage can be used successfully to repair gingival recession defects.
80 an be successfully used for the treatment of gingival recession defects.
81 ctive tissue graft (CTG) in the treatment of gingival recession defects.
82 acid-based barrier in the treatment of human gingival recession defects.
83 barrier device in root coverage treatment of gingival recession defects.
84 eatment modalities for clinically correcting gingival recession defects.
85        Clinical parameters, such as vertical gingival recession depth (VRD), clinical attachment leve
86 y prevent the development and progression of gingival recession, especially when restorative margins
87 identified that used GTR approaches to treat gingival recession from January 1990 to October 2001.
88 y was to assess the prevalence and extent of gingival recession, gingival bleeding, and dental calcul
89                                              Gingival recession, gingival bleeding, and dental calcul
90                 Males had significantly more gingival recession, gingival bleeding, subgingival calcu
91 ative caries experience), gingival bleeding, gingival recession, gingival probing depth, and periodon
92 ogous sites (control group), with or without gingival recession (GR) and with attached gingiva, were
93 dy is to evaluate the 2-year term results of gingival recession (GR) associated with non-carious cerv
94 d amount of attached gingiva associated with gingival recession (GR) at baseline were treated with FG
95 inical outcomes after treatment of localized gingival recession (GR) by a coronally advanced flap (CA
96                         Root exposure due to gingival recession (GR) can cause cervical dentin hypers
97 t patients presenting with 21 Miller Class I gingival recession (GR) defects (isolated or adjacent mu
98 s the long-term outcomes of untreated buccal gingival recession (GR) defects and the associated repor
99 gical approaches for the treatment of single gingival recession (GR) defects are documented in the li
100                                              Gingival recession (GR) defects can be treated by variou
101                   The effective treatment of gingival recession (GR) defects is crucial for predictab
102 TG) associated with LLLT in the treatment of gingival recession (GR) defects.
103 ividual, and approximately 10% of teeth with gingival recession (GR) had DH.
104                                              Gingival recession (GR) is one of the most common esthet
105 ncluded clinical attachment levels (CAL) and gingival recession (GR) measured clinically and linear b
106                                              Gingival recession (GR) might be associated with patient
107 , clinical attachment level (CAL), amount of gingival recession (GR), and change in class of clinical
108 depth (PD), clinical attachment level (CAL), gingival recession (GR), and radiographic bone level (BL
109 hip between toothbrushing and development of gingival recession (GR), but relevant GR data for the mu
110 depth (PD), clinical attachment level (CAL), gingival recession (GR), gingival index (GI), bleeding o
111  such as clinical attachment level (CAL) and gingival recession (GR).
112 h [PD], clinical attachment level [CAL], and gingival recession [GR]) and radiographic (defect bone l
113 urface area (AERSA) as a prognostic test for gingival recessions (GRs) and to compare the predictive
114       Fifteen bilateral Miller Class I or II gingival recessions (GRs) were selected.
115 edures performed for the treatment of single gingival recessions (GRs).
116 ntal attachment apparatus when used to treat gingival recessions (GRs).
117 7.0% and 5.8% of teeth per individual showed gingival recession > or = 3 mm and > or = 5 mm, respecti
118 ients with bilateral Miller's Class I or II (gingival recession > or = 3.0 mm) recession defects were
119  either Miller Class I or II description and gingival recession > or =2.5 mm.
120 with bilateral Miller's Class I or II buccal gingival recessions >/=2.0 mm in canines or premolars we
121 CT), 30 patients with Miller Class II buccal gingival recession, > or = 3 mm deep and > or = 3 mm wid
122   A relationship between occlusal forces and gingival recession has been postulated in the past.
123 eration (GTR) procedures in the treatment of gingival recession has shown promising results and is ga
124 tic surgery techniques for the management of gingival recession have typically used soft tissue graft
125             Data on Class I, II, III, and IV gingival recessions, histologic attachment achieved afte
126     This study describes the epidemiology of gingival recession in a representative, urban Brazilian
127                 There was significantly less gingival recession in the bioactive glass sites (1.29 mm
128 ), missing teeth (r = 0.784; P = 0.001), and gingival recession in the esthetic zone (r = 0.718; P =
129 r = 0.517; P = 0.017); and the more sites of gingival recession in the esthetic zone the subjects had
130 ation of a high prevalence of tooth loss and gingival recession in the oldest age cohorts.
131 inicopathologic case report documents severe gingival recession in the primary dentition of a trisomy
132 diseases may reduce the prevalence of severe gingival recession in this and similar populations.
133                            The high level of gingival recession in this Brazilian population may be p
134      The prevalence, extent, and severity of gingival recession increased with age, as did the preval
135     Prevalence of moderate and severe LA and gingival recession increased with age, while prevalence
136 eding on probing, clinical attachment level, gingival recession, interleukin-1beta, tumor necrosis fa
137 ertained to the unesthetic appearance of the gingival recession involving both maxillary central inci
138                                              Gingival recession is a common manifestation of periodon
139                                              Gingival recession is a frequent clinical finding in the
140           Measurements of gingival bleeding, gingival recession level, periodontal pocket depth, and
141 ws the reproducible volumetric evaluation of gingival recession marks on stone replicas.
142 bing depth, clinical attachment level (CAL), gingival recession, number of missing teeth, and radiogr
143 ange in buccal (bREC) and interdental (iREC) gingival recession observed at 6 months after treatment
144    Twenty-two patients with similar isolated gingival recession of > or = 2 mm on 2 separate teeth we
145                  At 12 months, a mean +/- SE gingival recession of 1.1 +/- 0.3 mm in OF and 0.9 +/- 0
146 hereas lower bicuspid teeth were at risk for gingival recession on buccal surfaces.
147  22) with tongue piercings were examined for gingival recession on the lingual aspect of the 12 anter
148 , despite many advantages, carries a risk of gingival recession, papilla loss, collapse of ridge cont
149 e index (FMPI), full-mouth BOP score (FMBS), gingival recession, PD, and clinical attachment level (C
150               Clinical measurements included gingival recession, PD, clinical attachment level, and m
151 odontal examination including probing depth, gingival recession, plaque index, and bleeding on probin
152  caries, defective restoration presence, and gingival recession presence.
153                                              Gingival recession presents destruction of both soft and
154            The results indicate that greater gingival recession prevalence and extent are associated
155 ll-mouth plaque score), bleeding on probing, gingival recession, probing depth (PD), and vertical (VA
156 achment level (CAL), probing depth (PD), and gingival recession (REC) were assessed immediately befor
157 depth (PD), clinical attachment level (CAL), gingival recession (REC), bleeding on probing (BOP), sup
158  alternative to CTG+CAF for the treatment of gingival recessions (REC), in a prospective randomized,
159                                              Gingival recession remains an important problem in denta
160                                              Gingival recession represents a significant concern for
161  a free gingival graft to restore an area of gingival recession resulting from an oral lesion.
162 reatment of root sensitivity associated with gingival recession stemming from toothbrush abrasion.
163 ignificantly higher prevalence and extent of gingival recession than females.
164 prevalence and extent of attachment loss and gingival recession than non-smokers, suggesting poorer p
165 duals with at least one Miller Class I or II gingival recession underwent a surgical root coverage pr
166 mphigoid in a patient with multiple sites of gingival recession using connective tissue grafting to a
167 tion-based root coverage (GTRC) is to repair gingival recession via new attachment formation.
168                         The determination of gingival recession volume was highly reproducible.
169       The most significant results were that gingival recession was greater for the group treated wit
170                                              Gingival recession was much more prevalent and also more
171                                              Gingival recession was not associated with the thickness
172                           Probing depths and gingival recession were measured at two points (mid-labi
173             Differences in the volume of the gingival recession were recorded with reference to the i
174                            Marks designating gingival recession were scratched into the anterior segm
175 th at least one site of Miller Class I or II gingival recession were treated by a coronally advanced
176                                              Gingival recessions were randomly designated to receive
177 althy adult subjects with multiple bilateral gingival recessions were treated with CTGs and PC combin
178 venteen arches in 15 patients with bilateral gingival recessions were treated with SCTG and PCG cover
179 buting a pair of Miller Class I or II buccal gingival recessions, were treated.
180 titis may have increased attachment loss and gingival recession when compared to their HIV-negative c
181  results suggest that the treatment of human gingival recession with a bioabsorbable membrane with or
182      This case demonstrates the treatment of gingival recession with a novel collagen bilayer membran
183 s of CAL gains, shallow pockets, and minimal gingival recession with the four regenerative approaches

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