ライフサイエンスコーパス: gingivitis

1 etween health and periodontal disease (i.e., gingivitis).

2 ave a higher prevalence of dental caries and gingivitis.

3 evidence of generalized, moderate-to-severe gingivitis.

4 els of MMP-8 especially when associated with gingivitis.

5 to clarify the relationship between PCOS and gingivitis.

6 isite for the development of both caries and gingivitis.

7 ed with healthy individuals or patients with gingivitis.

8 d dentifrice for the treatment of plaque and gingivitis.

9 either a clinically healthy periodontium or gingivitis.

10 evels had the highest frequency of pregnancy gingivitis.

11 variation was found in cases with health and gingivitis.

12 ation required for microbial development and gingivitis.

13 e and gingival inflammation in patients with gingivitis.

14 ta of WSL, accounting for confounding due to gingivitis.

15 moglobin values in contrast to patients with gingivitis.

16 nd either clinically healthy periodontium or gingivitis.

17 rmation for the therapeutic effects of CT on gingivitis.

18 associated with increased odds of having BGI gingivitis.

19 d as having biofilm-gingival interface (BGI) gingivitis.

20 may play a significant role in patients with gingivitis.

21 givalis increased under conditions emulating gingivitis.

22 severe diarrhoea, and necrotising ulcerative gingivitis.

23 ssified as controls and 164 as children with gingivitis.

24 wever, no such study has been carried out on gingivitis.

25 bile EDS (type III) associated with atypical gingivitis.

26 and genotype frequencies and the severity of gingivitis.

27 chemotherapeutic products for the control of gingivitis.

28 have been shown to be effective in reducing gingivitis.

29 he -1082*A allele could be a risk factor for gingivitis.

30 umber of bleeding sites in a population with gingivitis.

31 clinically demonstrating naturally occurring gingivitis.

32 vely whitens teeth and significantly reduces gingivitis.

33 served clinical improvement in patients with gingivitis.

34 ential therapeutic agent in the treatment of gingivitis.

35 ased chances of having poor oral hygiene and gingivitis.

36 althy controls in the presence or absence of gingivitis.

37 ease chances of having poor oral hygiene and gingivitis.

38 ong sex, digit sucking, OHS, and presence of gingivitis.

39 a (TRP) mouthwash in reduction of plaque and gingivitis.

40 with thalassemia major (TM) with or without gingivitis.

41 rs in a possible association between GDM and gingivitis.

42 of 351 sites, including periodontitis (109), gingivitis (115), and healthy (127) sites.

43 significantly elevated in subjects with BGI gingivitis (136.2 +/- 112.9 ng/ml and 277.2 +/- 187.2 ng

44 l group had statistically significantly less gingivitis (25.8%) and statistically significantly less

45 plotypes were more frequent in children with gingivitis (27% and 23% versus 19% and 21% in controls).

46 e frequent in controls than in children with gingivitis (36% versus 23%) (P=0.036).

47 Eighty participants (40 with gingivitis, 40 healthy) provided saliva at baseline and

48 s significantly more common in children with gingivitis; 49% versus 37% in controls (P = 0.01).

49 evere periodontitis (88%) and the lowest for gingivitis (55%).

50 e frequent in controls than in children with gingivitis (60% versus 50%).

51 riodontal disease (periodontitis = 85.2% and gingivitis = 60.2%).

52 <3 mm and bleeding on probing (BOP) <10%; 2) gingivitis, all PD <3 mm and BOP >/=10%; 3) periodontiti

53 aggressive periodontitis than in those with gingivitis, although not significantly higher than in he

54 cking habits, oral hygiene status (OHS), and gingivitis among a group of Nigerian children.

55 level of serum IL-6, is associated with BGI gingivitis among non-smoking patients with diabetes.

56 Average baseline gingivitis and bleeding scores were similar for the two

57 ration of healthy patients and patients with gingivitis and chronic periodontitis (CP) and correlates

58 However, the full relationship between gingivitis and COHRQoL has been assessed by only a small

59 Differences between subjects with gingivitis and controls in the frequency of haplotypes w

60 s were significantly higher in patients with gingivitis and CP compared with healthy controls (P <0.0

61 cytokines may suggest an interaction between gingivitis and GDM.

62 he GO+ and GO- groups compared with both the gingivitis and healthy groups (P <0.008).

63 gP, than those in the group of patients with gingivitis and in the group that was healthy (P <0.001).

64 at are significantly associated with health, gingivitis and mild periodontitis (<25% attachment loss)

65 ollected from 223 dogs with healthy gingiva, gingivitis and mild periodontitis with 72 to 77 samples

66 scenarios ranging from periodontal health to gingivitis and mild, moderate, and severe periodontitis.

67 features, however, were also identified for gingivitis and mucositis.

68 No significant association was found between gingivitis and obesity.

69 insult and inflammatory responses leading to gingivitis and peri-implant mucositis.

70 nas gingivalis results in the development of gingivitis and periapical bone loss, which apparently ar

71 ly for periodontitis, and five included both gingivitis and periodontal measures.

72 uggest that, at least in the studied sample, gingivitis and periodontitis operate as antagonistic mod

73 ean amounts of GCF TGF-beta1 were greater in gingivitis and periodontitis sites than in healthy sites

74 tissues, comparing samples from experimental gingivitis and periodontitis subjects to those from heal

75 Mouse models of acute gingivitis and periodontitis were used to assess the ear

76 Periodontal diseases, such as gingivitis and periodontitis, are characterized by bacte

77 plaque, a highly complex biofilm that causes gingivitis and periodontitis, requires specific adherenc

78 depth were used as measures of the extent of gingivitis and periodontitis, respectively.

79 studies, the two major periodontal diseases, gingivitis and periodontitis, were combined and consider

80 h and without HIV who presented with chronic gingivitis and periodontitis.

81 elated negatively to IL-34, in patients with gingivitis and periodontitis.

82 maintaining chronic inflammation, including gingivitis and periodontitis.

83 limit caries, as well as halt progression to gingivitis and periodontitis.

84 Predictors of presence of gingivitis and poor oral hygiene were determined using m

85 n the same direction after onset of clinical gingivitis and returned to baseline levels after resolut

86 paste is effective in controlling plaque and gingivitis and slowing progression of periodontitis; how

87 /IgA pemphigoid as the cause of desquamative gingivitis and the other mucosal findings in this patien

88 ydroxyapatite) of oral pathogens involved in gingivitis and tooth decay.

89 idated hydrogen peroxide-based mouthrinse on gingivitis and tooth whitening in a two-phase study.

90 385) and 11% (n = 80) of the sample had BGI gingivitis and type 2 diabetes, respectively.

91 the anteromandibular sextant with or without gingivitis and with or without periodontitis at the lowe

92 the 80 subjects was clinically diagnosed as gingivitis and/or mild periodontitis.

93 .31 years), 15 with generalized AgP, 15 with gingivitis, and 10 who were periodontally healthy.

94 females with PCOS, 30 females with PCOS and gingivitis, and 12 systemically and periodontally health

95 althy periodontium, 30 females with PCOS and gingivitis, and 12 systemically and periodontally health

96 zed aggressive periodontitis (GAgP), 20 with gingivitis, and 20 classified as healthy (H).

97 ithout GO [GO-; n = 20], 20 individuals with gingivitis, and 20 healthy participants).

98 his study: 31 periodontally healthy, 27 with gingivitis, and 31 with CP.

99 nts: 8 periodontally healthy, 9 experimental gingivitis, and 9 periodontitis subjects.

100 y determines prevalence of digit sucking and gingivitis, and association among age, sex, socioeconomi

101 us (attachment level, probing depth, plaque, gingivitis, and bleeding on probing scores) and signific

102 ations in periodontal assessment for health, gingivitis, and mild, moderate, and severe periodontitis

103 evicular fluid (GCF) collected from healthy, gingivitis, and periodontitis sites in humans, by liquid

104 were performed to compare data from healthy, gingivitis, and periodontitis sites.

105 val crevicular fluid collected from healthy, gingivitis, and periodontitis sites.

106 nts (29 patients with periodontitis, 12 with gingivitis, and seven healthy patients) and related to t

107 evicular fluid samples from six healthy, six gingivitis, and three periodontitis sites were collected

108 e interval [CI]: 0.20 to 0.35; P <0.001) and gingivitis (AOR: 0.21; 95% CI: 0.14 to 0.31; P <0.001) w

109 Desquamative gingivitis associated with IgG/IgA pemphigoid can be cha

110 (OR = 0.87, 95% CI = 0.10 to 7.84), nor was gingivitis associated with the ESS (OR = 1.25, 95% CI =

111 n age, 23.3 +/- 5.0 [SD] years) with minimal gingivitis at baseline.

112 periodontitis (BGI-P1), health (BGI-H), and gingivitis (BGI-G) (P = 0.005).

113 uped as BGI-healthy (14.3% of sample) or BGI-gingivitis (BGI-G, 15.1%).

114 Plaque, gingivitis, bleeding on probing, suppuration, probing de

115 bohydrate metabolism network in experimental gingivitis but not in periodontitis.

116 h certain microbial taxa not associated with gingivitis by a previous study or the current one.

117 via wiggsiae (p = 0.04) Taxa associated with gingivitis by microarray included: Gemella sanguinis (p

118 ed with both healthy patients and those with gingivitis, by 2.60-fold and 2.77-fold, respectively (P

119 Plaque-induced gingivitis can be alleviated by various treatment regime

120 GI), and plaque index (PI) were monitored in gingivitis cases among systemically healthy patients (n

121 ted the ED with a primary diagnosis of acute gingivitis, chronic gingivitis, gingival recession, aggr

122 ons following SIV infection in macaques with gingivitis compared to controls.

123 serum IL-6 was elevated in subjects with BGI gingivitis compared to subjects with gingival health onl

124 ium hexametaphosphate dentifrice in reducing gingivitis compared to the triclosan/copolymer control i

125 res brought an additional risk of developing gingivitis compared with a high VPI score alone.

126 igher in the systemically healthy women with gingivitis compared with periodontally healthy women wit

127 PO levels were higher in women with PCOS and gingivitis compared with periodontally healthy women wit

128 ger than those in the group of patients with gingivitis, consistent with significantly greater ADAM8

129 the increased volume of GCF associated with gingivitis could potentially dilute macrolides.

130 atients with periodontal diseases, including gingivitis, CP, and AgP, in comparison to control partic

131 subject at baseline (day 0), at the peak of gingivitis (day 28), and at resolution (day 35) and proc

132 Participants with gingivitis demonstrated significantly higher bleeding on

133 Bacterial levels, gingivitis, dental plaque, and caries experience were as

134 Desquamative gingivitis (DG) is a common clinical manifestation of or

135 described in the literature as desquamative gingivitis (DG).

136 The presence of periodontitis and gingivitis did not increase CHD risk among these at-risk

137 However, gingivitis does not affect the underlying supporting str

138 It is concluded that periodontitis or gingivitis does not elevate CHD risk among individuals w

139 ective of this study was to compare the anti-gingivitis efficacy of a 0.454% stannous fluoride/sodium

140 ntitis] and 39 natural teeth [19 healthy, 12 gingivitis, eight periodontitis] in 15 systemically heal

141 Subject age, plaque, and measures of gingivitis exhibited associations with attachment loss a

142 included self-reported measures specific to gingivitis, four included measures only for periodontiti

143 Salivary PGE2 and MIP-1alpha discriminate gingivitis from health, and patients with gingivitis who

144 ation with MIP-1alpha, readily discriminated gingivitis from health.

145 ve periodontitis (GAgP), 18 individuals with gingivitis (G), and 19 periodontally healthy (H) partici

146 20 healthy (H) individuals; 20 patients with gingivitis (G); 20 CsA-medicated patients with GO (CsA G

147 25), chronic periodontitis (CP, n = 14), and gingivitis (G, n = 18) were tested for the presence of a

148 Seventy-one women with GDM and gingivitis (Gg), 30 women with GDM and healthy periodont

149 imary diagnosis of acute gingivitis, chronic gingivitis, gingival recession, aggressive or acute peri

150 s were lower in the mucositis group than the gingivitis group (P <0.05).

151 GDM with gingivitis group than non-GDM with gingivitis group (P = 0.044).

152 bjects were equally recruited into a healthy/gingivitis group or a periodontitis population.

153 The PCOS + gingivitis group revealed significantly higher GCF, sali

154 The PCOS + gingivitis group revealed significantly higher insulin c

155 ANKL (P <0.0001) were higher in the GDM with gingivitis group than GDM without gingivitis group.

156 Saliva IL-6 level was higher in the GDM with gingivitis group than non-GDM with gingivitis group (P =

157 were significantly higher (2.8 times) in the gingivitis group than the healthy group (P </=0.02).

158 index was significantly higher in the PCOS + gingivitis group than the PCOS + healthy group (P = 0.03

159 decrease significantly from baseline in the gingivitis group, although concentrations of IL-1beta, I

160 infected with multiple viral variants in the gingivitis group.

161 e GDM with gingivitis group than GDM without gingivitis group.

162 higher in the CP group than the healthy and gingivitis groups (P <0.001).

163 th chronic periodontitis (CP), patients with gingivitis (GV), and individuals with no periodontal dis

164 the CPQ11-14; those with extended levels of gingivitis had a 1.20 times higher mean score than those

165 (Hh), and 37 systemically healthy women with gingivitis (Hg) were evaluated.

166 Gingivitis, however, showed a stronger positive correlat

167 alivary glucose levels on the development of gingivitis in a prospective longitudinal study of Kuwait

168 ompounds, on the development of experimental gingivitis in beagle dogs.

169 d have an active role in the pathogenesis of gingivitis in Caucasian children.

170 wed that allele A remained a risk factor for gingivitis in children (P = 0.03) regardless of plaque o

171 es the association of parenting practices on gingivitis in children using path analysis to evaluate i

172 y demographic factors that could account for gingivitis in children, with a focus on the mediational

173 the induction and resolution of experimental gingivitis in humans were not previously explored using

174 during onset and resolution of experimental gingivitis in smokers.

175 may exacerbate preconditions associated with gingivitis in susceptible individuals.

176 tered, may significantly reduce experimental gingivitis in the beagle dog.

177 ould exist between children with and without gingivitis in the distribution of IL-10 alleles and hapl

178 between Caucasian children with and without gingivitis in the distribution of IL-10 alleles at posit

179 s, only a few investigations have focused on gingivitis in this at-risk population.

180 e association between the -1082*A allele and gingivitis in white Caucasian children.

181 being selectively modulated in experimental gingivitis, including neural processes, epithelial defen

182 Gingivitis increased over time in children who had short

183 oral SIV infection rates were similar in the gingivitis-induced and control groups (5 infections foll

184 giene practices ceased for 21 days to induce gingivitis (induction), after which home care was reinst

185 pants were enrolled in a 21-day experimental gingivitis investigation and grouped according to clinic

186 Ligneous gingivitis is a rare periodontal disorder closely associ

187 Although pregnancy gingivitis is widely believed to result from elevated ho

188 membranous periodontal disease, or ligneous gingivitis, is a rare condition involving nodular gingiv

189 L were minimally changed with adjustment for gingivitis level.

190 ically significant differences in plaque and gingivitis levels would help to determine whether oral h

191 matory immune modulators in the SIV-infected gingivitis macaques could also be observed in blood plas

192 Gingivitis may be a useful clinical model to evaluate th

193 he validity of self-reported periodontal and gingivitis measures against clinical gold standards.

194 ate that the presence of extensive levels of gingivitis might be negatively associated with how child

195 m accumulation, and GCF exudation in a human gingivitis model were examined.

196 ccumulation over 21 days in the experimental gingivitis model would elicit systemic inflammatory resp

197 s during a 28-day stent-induced experimental gingivitis model, followed by treatment, and resolution

198 ing a prospective, split-mouth, experimental gingivitis model, pre-menopausal women either taking (n

199 healthy individuals (n = 15), patients with gingivitis (n = 15), and patients with severe chronic pe

200 ically healthy, non-smoking individuals with gingivitis (n = 20) or chronic periodontitis (CP) (n = 2

201 25); and 4) systemically healthy women with gingivitis (n = 20).

202 n additional separate group of patients with gingivitis (n = 21) and some of the patients with period

203 n with PCOS (n = 45); 2) women with PCOS and gingivitis (n = 35); 3) systemically and periodontally h

204 nically manifested as necrotizing ulcerative gingivitis (NUG).

205 use was associated with an increased risk of gingivitis (odds ratio [OR] =1.7; 95% confidence interva

206 ve children were at increased odds of having gingivitis of 1.8 (95% confidence interval [CI]: 1.05 to

207 Concerning the chronic gingivitis of the individuals with HIV, both strong and

208 the relationship between sleep duration and gingivitis on 3 levels: within schools, among children,

209 ge: 46.0 +/- 8.8 years) and 16 patients with gingivitis only (mean age: 31.5 +/- 7.5 years) were inve

210 e results with serum levels of patients with gingivitis only.

211 patients with generalized AgP and those with gingivitis or a healthy periodontium.

212 aggressive periodontitis than in those with gingivitis or in healthy persons.

213 t were significantly associated with health, gingivitis or mild periodontitis.

214 patients having the basic clinical signs of gingivitis or periodontal disease).

215 e possible association between the extent of gingivitis or periodontitis and an index of gingival mic

216 C use is associated with increased levels of gingivitis or periodontitis and suggests an important re

217 , respectively, the clinical predominance of gingivitis or periodontitis in such a site.

218 hout HIV, both groups presented with chronic gingivitis or periodontitis, and no statistically signif

219 All individuals presented gingivitis or periodontitis.

220 ipants were examined and those with clinical gingivitis or slight chronic periodontitis were included

221 ring inflammatory (cardiovascular disease or gingivitis) or autoimmune (psoriasis, arthritis, or mult

222 ho had more decayed or filled teeth had more gingivitis ( P < 0.05).

223 alivary glucose levels >1.13 mg/dL predicted gingivitis ( P < 0.05).

224 n TM gingivitis than in systemically healthy gingivitis (P <0.001).

225 patients with CP compared with patients with gingivitis (P <0.01).

226 ontally healthy women or women with PCOS and gingivitis (P <0.05).

227 ts with GCP when compared with patients with gingivitis (P = 0.007, P = 0.004, P = 0.033, and P = 0.0

228 An additional sample was collected from gingivitis participants 10 to 30 days after dental proph

229 nd -1beta in a sample of naturally occurring gingivitis patients.

230 have an affect on BOP in naturally occurring gingivitis patients.

231 ants presenting with either clinical health, gingivitis/peri-implant mucositis, or chronic periodonti

232 us of individuals affected by plaque-induced gingivitis (pGI).

233 st phase (28 days) included the experimental gingivitis phase; the second phase (5 months) was the or

234 Clinical measures examined included gingivitis, plaque, alveolar bone height, age, gender, e

235 ntal problems, such as gingival enlargement, gingivitis, poor oral hygiene, dental hypoplasia, and ca

236 soft-bristle manual brush in a non-flossing gingivitis population (n = 157).

237 wing gums or mouthwashes for both caries and gingivitis prevention.

238 flammation for a prolonged period, pregnancy gingivitis rarely progresses to periodontitis and usuall

239 derate gingivitis was designed with two anti-gingivitis regimens: the brush-alone treatment and the b

240 al signatures of the three treatments during gingivitis relieve indicate distinct mechanisms of actio

241 1 to 5 years had poor oral hygiene and mild gingivitis, respectively.

242 6 to 12 years had poor oral hygiene and mild gingivitis, respectively.

243 The periodontitis sample but not the gingivitis sample revealed HCMV mRNA of major capsid pro

244 regnancy histories, risk factors, plaque and gingivitis scores, and current pregnancy outcomes.

245 In this case report we describe desquamative gingivitis secondary to IgG/IgA pemphigoid and the manag

246 ntally healthy individuals and patients with gingivitis showed similar HBD-2 levels, the CP group dis

247 (GI >0; PD > or =5 mm; AL > or =3 mm), three gingivitis sites (GI >0; PD < or =3 mm; AL = 0), and two

248 mycin concentrations are similar in GCF from gingivitis sites and healthy sites, suggesting that the

249 ine, lysine, putrescine, and xanthine at the gingivitis sites as early as week 1.

250 Clarithromycin levels at control and gingivitis sites were higher than serum by 5.7- and 7.0-

251 tal sites with advanced attachment loss or 3 gingivitis sites with no clinical attachment loss.

252 romycin was compared in GCF from healthy and gingivitis sites.

253 crevicular fluid (GCF) could be increased at gingivitis sites.

254 ind, placebo-controlled, 28-day experimental gingivitis study involving 21 healthy adults, in which s

255 ls participated in an experimentally induced gingivitis study, and gingival biopsies were collected a

256 olism are markedly different in experimental gingivitis subjects compared with healthy controls.

257 ross-sectional study was to evaluate whether gingivitis susceptibility is associated with periodontit

258 suggest that among never and former smokers, gingivitis susceptibility is higher among men with perio

259 d IL-6 and IL-8 (P <0.005) were higher in TM gingivitis than in systemically healthy gingivitis (P <0

260 MP-9 levels were lower in healthy women with gingivitis than systemically and periodontally healthy w

261 centrations were significantly higher in the gingivitis than the mucositis group (P = 0.004).

262 Many taxa showed a stronger association with gingivitis than with WSL.

263 5 for each group: the group of patients with gingivitis, the group with aggressive periodontitis [AgP

264 Gingivitis, the mildest form of periodontal disease, is

265 hange as a consequence of the progression of gingivitis to periodontitis and that both cytokines coul

266 serum YKL-40 and IL-6 levels increased from gingivitis to periodontitis.

267 Response to gingivitis treatment in patients with diabetes can sligh

268 ed clinical trial compared the response to a gingivitis treatment protocol that combined mechanical p

269 To probe the impacts of various anti-gingivitis treatments on plaque microflora, here a doubl

270 enzymes could help to explain why pregnancy gingivitis typically is not characterized by progression

271 the induction and resolution of experimental gingivitis using bioinformatic tools.

272 ildren were assessed for OHS and severity of gingivitis using the simplified oral hygiene index and t

273 The prevalence of gingivitis was 53.9% for females who reported having use

274 odel of supragingival plaque associated with gingivitis was characterized by traditional culture tech

275 Because improvement in gingivitis was comparable with that of CHX mouthwash, TR

276 nical examination of oral hygiene status and gingivitis was conducted using simplified oral hygiene a

277 controlled trial of 91 adults with moderate gingivitis was designed with two anti-gingivitis regimen

278 The level of gingivitis was different among the 6 governorates of Kuw

279 An increased risk of having gingivitis was found in allele A positive children (G/A,

280 ically applied to the gingiva after moderate gingivitis was identified through clinical and immunolog

281 The odds of having gingivitis was increased in children with low socioecono

282 Experimental gingivitis was induced in 15 smokers for 21 days, follow

283 Experimental gingivitis was induced in eight healthy subjects at one

284 Experimental gingivitis was induced in one maxillary posterior sextan

285 In conclusion, while moderate gingivitis was not associated with increased susceptibil

286 Onset of clinical gingivitis was preceded by significant changes in the ma

287 Gingivitis was present in 67% of the children examined.

288 ctive association between current OC use and gingivitis was suggestive but not significant in both NH

289 Experimental gingivitis was then induced, with cessation of plaque co

290 those with healthy periodontal tissues/mild gingivitis were included.

291 Ninety individuals with chronic generalized gingivitis were randomly assigned to three groups: 1) gr

292 patients diagnosed with chronic generalized gingivitis were selected and randomly divided into three

293 Clinical features of health and gingivitis were stable at both baseline visits.

294 l caries, dentate status, root remnants, and gingivitis, were examined, and an asymptotic dental scor

295 r resolution phase of experimentally induced gingivitis, which represented a reversible periodontal l

296 IL-17E were lowest in females with PCOS and gingivitis who also had the highest FGS.

297 te gingivitis from health, and patients with gingivitis who return to clinical health continue to pro

298 were reported using 10 different indices and gingivitis with nine indices.

299 regression models fitted the association of gingivitis with overall and domain-specific CPQ11-14 sco

300 estigation is to explore the relationship of gingivitis with salivary biomarkers, periodontal pathoge