ライフサイエンスコーパス: girdle

1 verify high expression of IL-22 in chorionic girdle.

2 eurocranium, pharyngeal arches, and pectoral girdle.

3 progressive weakness of the hip and shoulder girdle.

4 pically mild and located around the shoulder girdle.

5 ed microfibrils that we termed the fibrillar girdle.

6 posture within a girdle and between the two girdles.

7 elongate dorsal vertebrae, and modified limb girdles.

8 Odontochelys in various features of the limb girdles.

9 ed below 0.5 MPa and was inhibited by phloem girdling.

10 transpiring flowers were unaffected by bark girdling.

11 The pelvic girdle and appendage of tetrapods is dramatically larger

12 extent, the coordination of posture within a girdle and between the two girdles.

13 The limb girdle and congenital muscular dystrophies (LGMD and CMD

14 Becker muscular dystrophy and forms of limb-girdle and congenital muscular dystrophies, and 21 patie

15 Its shoulder girdle and limbs show fossorial features similar to thos

16 rs characterized by weakness in the shoulder-girdle and peroneal muscles.

17 ep length through the rotation of the pelvic girdle and retraction of the femur.

18 Hip girdle and shoulder girdle weakness appeared later in th

19 s severely atrophic and weak in his shoulder girdle and the entire paraspinal musculature.

20 basal and intermediate snakes retain pelvic girdles and small rudiments of the femur.

21 ransfer of recently assimilated C using stem girdling and root trenching and measured soil CO2 efflux

22 Patient 1 had cervical, limb girdle, and respiratory muscle weakness and died of resp

23 tening injuries, stabilization of the pelvic girdle, and timely insinuation of pelvic angiography and

24 spine and thoracic wall, pelvic and shoulder girdles, and peripheral entheses and joints.

25 isease that affects the shoulder, the pelvic girdles, and the neck, usually in individuals older than

26 duced belowground C supply to soils via tree girdling, and contrasted responses in control and girdle

27 ar sling or loosely attached to the pectoral girdle anteriorly.

28 cluded by four cytoplasmic loops that form a girdle around the central pore (G-loop).

29 progressive weakness of the hip and shoulder girdles, as well as a distinctive dysarthric pattern of

30 e basal layer of the frustule as well as the girdle band and show that these components display simil

31 sely resemble characteristic features of the girdle band silica nanopatterns.

32 microrings are specifically associated with girdle bands, which constitute a substantial part of dia

33 hat IL-22 cytokine produced by the chorionic girdle binds IL-22R1 on endometrium, serving as a mechan

34 pulative investigations (e.g. starvation via girdling) combined with Delta(14) C measurements of this

35 GFPT1) cause the neuromuscular disorder limb-girdle congenital myasthenic syndrome (LG-CMS).

36 ished by rotation of the pectoral and pelvic girdles creating a standing wave of the axial body.

37 In ECM-dominated plots, girdling decreased the activity of enzymes that break do

38 trast with the known requirement for Tbx5 in girdle development in forelimbs.

39 Islet1 and Pitx1 function in parallel during girdle development in hindlimbs, which is in contrast wi

40 f the Hand2-Shh pathway and contributions to girdle development.

41 genes has suggested their potential roles in girdle development.

42 ineate novel regulatory networks in shoulder girdle development.

43 nce skeletal abnormalities, including pelvic girdle dysgenesis, skull bone fusions and polydactyly.

44 ing congenital muscular dystrophies and limb-girdle dystrophies), strokes and stroke-like episodes, e

45 r proximal myopathies characteristic of limb-girdle dystrophy and more severe disorders reminiscent o

46 e more than one myopathy phenotype (MM, limb girdle dystrophy, distal myopathy with anterior tibial o

47 n one patient with an undefined form of limb-girdle dystrophy.

48 The girdling effects of a conditioned muscle wrap alone have

49 and autopod) and the less prominent proximal girdle elements (scapula and clavicle) that brace the li

50 AR-domain superfamily proteins are touted to girdle eukaryotic phospholipid bilayers into narrow tubu

51 Stem-girdling experiments, which leave xylem vessels intact b

52 low spinose sclerites, and a smooth, ventral girdle flanks an extensive mantle cavity.

53 f the M2 helices that disrupts a hydrophobic girdle formed by 9' and 13' residues.

54 crease in IL-22 mRNA expression in chorionic girdle from days 32 to 35 and an absence of IL-22 expres

55 In AM-dominated plots, girdling had little effect on SOM-degrading enzymes, but

56 By contrast, JA treatments, branching and girdling had little or no impact on (1)(5)N import.

57 ements projecting anteriorly from the pelvic girdle into the abdominal region in a variety of Mesozoi

58 Our discovery of IL-22 in the chorionic girdle is a novel finding, as this cytokine has been pre

59 Unlike terrestrial theropods, the pelvic girdle is downsized, the hindlimbs are short, and all of

60 ggesting the possibility of an intracellular girdle just below the mouth of the ENaC pore.

61 ors covered include vein cutting, trenching, girdling, leaf clipping, and application of fluids from

62 rp residues are essential for gating and the girdle-like structure that lines the intracellular mouth

63 mb, as well as input from limbs of the other girdle made a much smaller contribution to the PTN modul

64 g different experimental methods (trenching, girdling, microcosms), as well as considering different

65 netic mechanism that reoriented the shoulder girdle more laterally.

66 ) involved in the control of head, trunk and girdle movements, while the other, lateral set of fibres

67 plexus MR imaging, brachial plexus and limb-girdle muscle abnormalities were evaluated in 23 patient

68 Nerve root and limb-girdle muscle abnormalities were visually evaluated by t

69 Limb-girdle muscle alterations (ie, T2 signal intensity alter

70 dependent proteinase that is mutated in Limb Girdle Muscle Dystrophy type 2A.

71 us diseases including Alstrom syndrome, limb-girdle muscle dystrophy, and Miyoshi myopathy.

72 f the peripheral nervous system and the limb-girdle muscle may be useful for monitoring the evolution

73 In contrast, the deeper girdle muscles (e.g. rhomboideus profundus and serratus

74 The more superficial girdle muscles (pectoral and latissimus dorsi) develop b

75 us proteins known to be involved in the limb-girdle muscular dystrophies (alpha, beta, gamma, delta s

76 Limb-girdle muscular dystrophies (LGMD) are a heterogeneous g

77 y which has long been recognized in the limb-girdle muscular dystrophies (LGMD) has been shown to rel

78 The limb-girdle muscular dystrophies (LGMD) have recently undergo

79 nts with sarcoglycanopathies, which are limb-girdle muscular dystrophies (LGMD2C-2F) caused by mutati

80 nother group of MD's referred to as the limb-girdle muscular dystrophies (LGMDs) can affect boys or g

81 ate analysis of current knowledge about limb-girdle muscular dystrophies (LGMDs).

82 ypes of muscular dystrophies, including limb-girdle muscular dystrophies (LGMDs).

83 Autosomal recessive limb girdle muscular dystrophies 2C-2F represent a family of

84 Limb-girdle muscular dystrophies 2C-F represent a family of a

85 e pathogenesis of sarcoglycan deficient limb-girdle muscular dystrophies and for the development of t

86 Limb-girdle muscular dystrophies are a genetically diverse gr

87 The limb-girdle muscular dystrophies are a group of disorders wit

88 ecialist Commissioning Team service for limb-girdle muscular dystrophies at Newcastle (UK).

89 lycan causes an array of congenital and limb girdle muscular dystrophies known as dystroglycanopathie

90 muscular dystrophy or various forms of limb-girdle muscular dystrophies respectively.

91 of the cardiac involvement in forms of limb girdle muscular dystrophies secondary to abnormal glycos

92 ar dystrophies ranging from adult onset limb girdle muscular dystrophies to severe congenital forms w

93 ar dystrophies ranging from adult onset limb girdle muscular dystrophies to severe congenital forms w

94 in any of the sarcoglycan genes lead to limb-girdle muscular dystrophies type 2C-2F.

95 Limb Girdle Muscular Dystrophies type 2I (LGMD2I), a recessiv

96 facioscapulohumeral muscular dystrophy, limb-girdle muscular dystrophies types 2A and 2B, Miyoshi myo

97 spectrum of GMPPB mutations to include limb-girdle muscular dystrophies with adult onset with or wit

98 ions are responsible for congenital and limb-girdle muscular dystrophies with hypoglycosylation of al

99 s and intellectual delay to adult-onset limb-girdle muscular dystrophies without mental retardation.

100 ying the molecular mechanism of several limb-girdle muscular dystrophies, particularly those where in

101 te promising therapeutic strategies for limb-girdle muscular dystrophies.

102 hasis on animal models for Duchenne and limb-girdle muscular dystrophies.

103 in the encoding genes cause congenital/limb-girdle muscular dystrophies.

104 absence is common to both Duchenne and limb girdle muscular dystrophies.

105 rcoglycan result in autosomal recessive limb girdle muscular dystrophy (AR-LGMD).

106 Dysferlin, the gene product of the limb girdle muscular dystrophy (LGMD) 2B locus, encodes a mem

107 Duchenne muscular dystrophy (DMD) and limb girdle muscular dystrophy (LGMD) 2C-F result from the lo

108 Limb girdle muscular dystrophy (LGMD) 2F is caused by mutatio

109 ncluding Walker-Warburg syndrome (WWS), limb girdle muscular dystrophy (LGMD) 2I and congenital muscu

110 Four types of limb-girdle muscular dystrophy (LGMD) are known to be caused

111 re of four types of autosomal recessive limb girdle muscular dystrophy (LGMD) is that mutations in a

112 POMT2 have also been linked to a milder limb-girdle muscular dystrophy (LGMD) phenotype, named LGMD t

113 PTP dysregulation is found in limb girdle muscular dystrophy (LGMD) type 2B myoblasts but n

114 tary inclusion body myopathy (hIBM) and limb-girdle muscular dystrophy (LGMD), are a genetically hete

115 lvement, to relatively mild adult onset limb girdle muscular dystrophy (LGMD).

116 family of 4 with cardiac arrhythmia and limb-girdle muscular dystrophy (LGMD).

117 Autosomal dominant limb-girdle muscular dystrophy (LGMD-1C) in humans is due to

118 Autosomal dominant limb girdle muscular dystrophy (LGMD-1C) in humans is due to

119 fied a novel autosomal dominant form of limb-girdle muscular dystrophy (LGMD-1C) in humans that is du

120 regulated in patients with this form of limb girdle muscular dystrophy (LGMD-1C).

121 n of a new locus for autosomal dominant limb-girdle muscular dystrophy (LGMD1) on 7q.

122 in the diagnosis of autosomal dominant limb-girdle muscular dystrophy (LGMD1A), it is not always pos

123 xpressing an autosomal dominant form of limb girdle muscular dystrophy (LGMD1A).

124 including Emery-Dreifuss (EDMD2/3) and Limb-Girdle muscular dystrophy (LGMD1B).

125 The other 18 had limb girdle muscular dystrophy (LGMD2I).

126 Limb-girdle muscular dystrophy 1A (LGMD1A [MIM 159000]) is an

127 1 (DYX1) and TMOD4 a candidate gene for limb girdle muscular dystrophy 1B (LGMD1B).

128 Of these, Emery-Dreifuss and Limb-Girdle muscular dystrophy 1B mainly affect striated musc

129 Limb girdle muscular dystrophy 1D/1E (OMIM nomenclature LGMD1

130 Limb girdle muscular dystrophy 2A is due to loss-of-function

131 amily member calpain 3 (CAPN3) underlie limb-girdle muscular dystrophy 2A.

132 Mutations in the dysferlin gene cause limb girdle muscular dystrophy 2B (LGMD2B) and Miyoshi myopat

133 Deficiency of dysferlin leads to limb-girdle muscular dystrophy 2B (LGMD2B) and Miyoshi myopat

134 y in a mouse model of the human disease limb girdle muscular dystrophy 2B (LGMD2B), caused by a mutat

135 gnaling offers a therapeutic avenue for limb girdle muscular dystrophy 2B and Miyoshi myopathy patien

136 Limb girdle muscular dystrophy 2B and Miyoshi myopathy were r

137 Dysferlinopathies, most commonly limb girdle muscular dystrophy 2B and Miyoshi myopathy, are d

138 INTS: Dysferlin, the protein missing in limb girdle muscular dystrophy 2B and Miyoshi myopathy, conce

139 ve developed a new diagnostic assay for limb girdle muscular dystrophy 2B and Miyoshi myopathy, which

140 ations in dysferlin are responsible for limb girdle muscular dystrophy 2B and Miyoshi myopathy.

141 sulting in dysferlin-deficiency lead to limb-girdle muscular dystrophy 2B and Myoshi myopathy in huma

142 ysferlin, underlie Miyoshi myopathy and limb-girdle muscular dystrophy 2B in humans and produce a slo

143 In our analysis of 12 patients with limb girdle muscular dystrophy 2B or Miyoshi myopathy, the fi

144 muscular dystrophies Miyoshi myopathy, limb girdle muscular dystrophy 2B, and distal anterior compar

145 Limb-girdle muscular dystrophy 2B, Miyoshi myopathy, and dist

146 s mutations in patients presenting with limb-girdle muscular dystrophy 2B.

147 6 and dysferlin, the protein mutated in limb girdle muscular dystrophy 2B.

148 Limb girdle muscular dystrophy 2C is caused by mutations in t

149 a sarcoglycan (Sgca), a mouse model for limb girdle muscular dystrophy 2D.

150 , congenital muscular dystrophy 1A, and limb girdle muscular dystrophy 2D.

151 s unclear why mutations in Tcap lead to limb-girdle muscular dystrophy 2G (LGMD2G) in human patients.

152 TRIM32) are responsible for the disease limb-girdle muscular dystrophy 2H (LGMD2H).

153 Limb girdle muscular dystrophy 2H is caused by mutations in t

154 tal muscular dystrophy type 1C (MDC1C), limb girdle muscular dystrophy 2I (LGMD2I) and congenital mus

155 Limb-girdle muscular dystrophy 2I (LGMD2I) is caused by mutat

156 e a wide range of pathologies from mild limb girdle muscular dystrophy 2I (LGMD2I), severe congenital

157 448L-mutant mouse representing moderate limb-girdle muscular dystrophy 2I.

158 e clinical phenotypes, most commonly as limb-girdle muscular dystrophy 2I.

159 y in FKRP P448Lneo(-) mice, a model for limb girdle muscular dystrophy 2I.

160 Human tibial muscular dystrophy and limb-girdle muscular dystrophy 2J are caused by mutations in

161 of two previously identified disorders, limb-girdle muscular dystrophy 2L and non-dysferlin Miyoshi m

162 we have identified an important case of limb girdle muscular dystrophy and cardiomyopathy with normal

163 wild type (WT) Cav3 and associates with limb-girdle muscular dystrophy and cardiomyopathy.

164 mutation (Thr192-->Met) in a woman with limb-girdle muscular dystrophy and cognitive impairment.

165 trate that FLN2 protein localization in limb-girdle muscular dystrophy and Duchenne muscular dystroph

166 g from congenital muscular dystrophy to limb-girdle muscular dystrophy and identified allelic ISPD va

167 l vectors have recently started, one in limb-girdle muscular dystrophy and one in Duchenne muscular d

168 s dramatically reduced in patients with limb-girdle muscular dystrophy arising from the loss of one o

169 ster is a naturally occurring model for limb-girdle muscular dystrophy caused by a primary deficiency

170 ar dystrophy type 2A is the most common limb girdle muscular dystrophy form worldwide.

171 rmalities were not found, we identified limb-girdle muscular dystrophy gene mutations affecting DYSF,

172 genetically distinct subset within the limb-girdle muscular dystrophy group.

173 rstanding the molecular pathogenesis of limb-girdle muscular dystrophy has been gained.

174 Some patients with autosomal recessive limb-girdle muscular dystrophy have mutations in the genes co

175 pairs) with a predominantly late onset limb-girdle muscular dystrophy in whom an absence of merosin

176 Limb-girdle muscular dystrophy is a class of disorders encomp

177 n that may be a good candidate gene for limb-girdle muscular dystrophy or other neuromuscular disorde

178 ep in the diagnostic algorithm of adult limb-girdle muscular dystrophy patients.

179 trophin-glycoprotein complex, produce a limb-girdle muscular dystrophy phenotype; however, the precis

180 in these genes might also cause milder limb-girdle muscular dystrophy phenotypes.

181 Miyoshi myopathy (MM) and limb-girdle muscular dystrophy subtype 2B (LGMD2B) map to the

182 oad spectrum of disorders, ranging from limb girdle muscular dystrophy to Walker-Warburg syndrome.

183 Limb-girdle muscular dystrophy type 1A (LGMD1A) is an autosom

184 ns have been described in patients with limb-girdle muscular dystrophy type 1A (LGMD1A), myofibrillar

185 fforts for several disorders, including limb-girdle muscular dystrophy type 1A and the genes deleted

186 Limb-girdle muscular dystrophy type 1D (LGMD1D) is caused by

187 One such example recently discovered is limb-girdle muscular dystrophy type 1D (LGMD1D), which is cau

188 e protein dysferlin, the product of the Limb Girdle Muscular Dystrophy type 2 locus, has been shown t

189 pain-3 (CAPN3) the product of the human limb-girdle muscular dystrophy type 2A (LGMD2A) gene.

190 calpain 3 (CAPN3) result in the disease limb girdle muscular dystrophy type 2A (LGMD2A).

191 somal cysteine protease calpain 3 cause limb-girdle muscular dystrophy type 2A (LGMD2A).

192 athological signs in a murine model for limb-girdle muscular dystrophy type 2A after intramuscular an

193 Diagnosis of limb girdle muscular dystrophy type 2A can be complex due to

194 l muscle physiology and in the study of limb-girdle muscular dystrophy type 2A in humans.

195 Limb girdle muscular dystrophy type 2A is the most common lim

196 Blots of muscle from nine limb-girdle muscular dystrophy type 2A patients with defined

197 tic defects in calpain3 (CAPN3) lead to limb-girdle muscular dystrophy type 2A, a disease of the skel

198 s patients deficient in calpain 3 as in limb girdle muscular dystrophy type 2A, albeit in a milder fo

199 s was generally milder than observed in limb girdle muscular dystrophy type 2A, but affected the same

200 esult in an autosomal recessive form of limb girdle muscular dystrophy type 2A.

201 unction, since mutations in CAPN3 cause limb girdle muscular dystrophy type 2A.

202 calpain 3 (C3), the protein mutated in limb girdle muscular dystrophy type 2A.

203 roduct of the gene that is defective in limb-girdle muscular dystrophy type 2A.

204 identified as a novel gene involved in limb-girdle muscular dystrophy type 2B (LGMD2B) and its allel

205 ified which is mutated in patients with limb-girdle muscular dystrophy type 2B (LGMD2B) and with Miyo

206 , a gene which is also mutated in human limb-girdle muscular dystrophy type 2B (LGMD2B).

207 Dysferlin deficiency causes limb-girdle muscular dystrophy type 2B (LGMD2B; proximal weak

208 Limb girdle muscular dystrophy type 2B and Miyoshi myopathy a

209 Defects in dysferlin result in limb-girdle muscular dystrophy type 2B and Miyoshi myopathy i

210 ncy of the dysferlin protein results in limb girdle muscular dystrophy type 2B and Miyoshi myopathy,

211 wo clinically distinct muscle diseases, limb-girdle muscular dystrophy type 2B and Miyoshi myopathy,

212 Mutations in the dysferlin gene cause limb girdle muscular dystrophy type 2B and Miyoshi myopathy.

213 a member of the ferlin family, lead to limb girdle muscular dystrophy type 2B and Myoshi myopathy.

214 The dysferlin gene is mutated in limb-girdle muscular dystrophy type 2B, Miyoshi myopathy, and

215 herapeutically useful for patients with limb girdle muscular dystrophy type 2B.

216 gamma-sarcoglycan-null mice, a model of limb-girdle muscular dystrophy type 2C, with a Col6a2-deficie

217 Limb-girdle muscular dystrophy type 2D (LGMD 2D) is an autoso

218 enne muscular dystrophy (DMD) patients, limb-girdle muscular dystrophy type 2D (LGMD2D) and their res

219 Limb-girdle muscular dystrophy type 2D (LGMD2D) is caused by

220 in a severe form of muscular dystrophy (limb-girdle muscular dystrophy type 2D [LGMD2D]) without trea

221 Limb-girdle muscular dystrophy type 2E (LGMD 2E) is caused by

222 an (delta sgc), a mouse model for human limb girdle muscular dystrophy type 2F.

223 Autosomal recessive limb-girdle muscular dystrophy type 2G (LGMD2G) is an adult-o

224 igase-ubiquitin ligase mutated in human limb-girdle muscular dystrophy type 2H (LGMD2H) and Bardet-Bi

225 Limb-girdle muscular dystrophy type 2H (LGMD2H) and sarcotubu

226 ) cause two seemingly diverse diseases: limb-girdle muscular dystrophy type 2H (LGMD2H) or sarcotubul

227 veral hereditary disorders that include limb girdle muscular dystrophy type 2H (LGMD2H), sarcotubular

228 two forms of muscular dystrophy called limb girdle muscular dystrophy type 2H and sarcotubular myopa

229 muscle and is orthologous to the human limb-girdle muscular dystrophy type 2H disease gene Trim32.

230 , mutations in the FKRP gene also cause limb-girdle muscular dystrophy type 2I (LGMD2I), a considerab

231 muscular dystrophy type 1C (MDC1C) and limb girdle muscular dystrophy type 2I (LGMD2I).

232 ngenital muscular dystrophy type 1C and limb girdle muscular dystrophy type 2I are allelic, both bein

233 ted with FKRP mutations range from mild limb-girdle muscular dystrophy type 2I with predominantly mus

234 es in biopsies of 6 patients, 3 with an limb-girdle muscular dystrophy type 2J (LGMD2J) phenotype.

235 both Walker-Warburg syndrome (WWS) and limb girdle muscular dystrophy type 2K (LGMD2K).

236 o several clinical phenotypes including limb-girdle muscular dystrophy type 2L and Miyoshi myopathy t

237 Limb-girdle muscular dystrophy types 2E and F are characteriz

238 Here we review each form of limb-girdle muscular dystrophy with attention to molecular ge

239 t severe form of dystroglycanopathy, to limb-girdle muscular dystrophy with cognitive defects.

240 ited to patients with Duchenne-like and limb-girdle muscular dystrophy with normal dystrophin and occ

241 tructural eye defects to a mild form of limb-girdle muscular dystrophy with onset in adult life and n

242 phy, facioscapulohumeral dystrophy, and limb-girdle muscular dystrophy).

243 y (EDMD), dilated cardiomyopathy (DCM), limb-girdle muscular dystrophy, and Hutchison-Gilford progeri

244 s of muscular dystrophy, including DM1, limb-girdle muscular dystrophy, congenital merosin-deficient

245 esented as an adult or adolescent-onset limb-girdle muscular dystrophy, one presented with isolated e

246 Limb-girdle muscular dystrophy, type 2A (LGMD 2A), is an auto

247 ng alpha-sarcoglycan gene expression in limb-girdle muscular dystrophy, type 2D (LGMD2D) subjects med

248 Limb girdle muscular dystrophy, which was described for the f

249 ent with the mild phenotype observed in Limb-girdle muscular dystrophy-1C (LGMD-1C) in humans, charac

250 Cav-3-related muscle diseases, such as limb-girdle muscular dystrophy-1C, distal myopathy, and rippl

251 expression in skeletal muscle resembles limb-girdle muscular dystrophy-1C.

252 ase calpain-3 cause autosomal recessive limb girdle muscular dystrophy.

253 oglycan-null (scgd(-/-)) mouse model of limb-girdle muscular dystrophy.

254 delta-sarcoglycan (scgd-/-), a model of limb-girdle muscular dystrophy.

255 in dysferlin, whose gene is mutated in limb girdle muscular dystrophy.

256 for the development of gene therapy for limb-girdle muscular dystrophy.

257 olemma and produce one of four types of limb-girdle muscular dystrophy.

258 can have been identified in humans with limb-girdle muscular dystrophy.

259 d delta) have been found in humans with limb-girdle muscular dystrophy.

260 ad to four forms of autosomal recessive limb-girdle muscular dystrophy.

261 ystrophy resembling what is observed in limb girdle muscular dystrophy.

262 syndrome, to mild forms of adult-onset limb-girdle muscular dystrophy.

263 revalence among patients with proximal (limb-girdle) muscular dystrophy with a later onset was 6 perc

264 minant involvement of the pelvic or shoulder girdle musculature.

265 n, and DOK7 mutations underlie familial limb-girdle myasthenia (DOK7 myasthenia), a neuromuscular dis

266 cting primarily proximal limb muscles ['limb-girdle myasthenia' (LGM)].

267 he phenotype can be distinguished from 'limb-girdle' myasthenia associated with tubular aggregates, w

268 In contrast, the pelvic girdle of Cryptotora is a large, broad puboischiadic pla

269 smonate (JA) treatment, branching and phloem girdling on (1)(3)C and (1)(5)N import, invertase activi

270 competing branches, treated with JA or not, girdled or not, had no impact on carbohydrate (CHO) impo

271 Soil CO2 efflux was not influenced by girdling or trenching through the 14-month observation p

272 henic syndrome (CMS) characterized by a limb-girdle pattern of muscle weakness.

273 group of individuals with an inherited limb-girdle pattern of myasthenic weakness.

274 ng defect in patients with an inherited limb-girdle pattern of myasthenic weakness.

275 S patients have a recessively inherited limb-girdle pattern of weakness caused by mutations in DOK7.

276 oup of patients shows a characteristic "limb girdle" pattern of muscle weakness, in which the muscles

277 ore affected than distal, leading to a 'limb-girdle' pattern of weakness; although ptosis was often p

278 the avian furculum, and they retain shoulder girdle plesiomorphies of mammaliaforms and monotremes.

279 ing, and contrasted responses in control and girdled plots for three consecutive growing seasons.

280 efects progress from preaxial polydactyly to girdle reduction combined with hypoplasia, aplasia or mi

281 th and involving the neck, shoulders, or hip girdle regions; and an erythrocyte sedimentation rate (E

282 RNA encoding the cytokine IL-22 in chorionic girdle relative to noninvasive chorion.

283 Comparative analysis of the upper girdle scan in 181 of these subjects was also performed.

284 eurocranium, pharyngeal arches, and pectoral girdle similar to humans with campomelic dysplasia.

285 loss of Dok-7 activity cause a distinct limb-girdle subtype of the inherited NMJ disorder congenital

286 equence gait and has evolved a robust pelvic girdle that shares morphological features associated wit

287 dimeric apoA-I(Milano) on 78 A diameter rHDL girdles the edge of a phospholipid disk assuming a "belt

288 In the shoulder girdle, the type of resection and soft-tissue reconstruc

289 o stabilize three ligands in the metallocene girdle to form saturated (C(5)H(5))(2)ML(3) species, in

290 xcised panicle stems subjected to a +/- stem-girdling treatment during grain fill.

291 abolism on photosynthesis in cassava, a heat-girdling treatment was applied to petioles of cassava le

292 er muscular dystrophy (BMD, n = 14) and limb-girdle type 2I muscular dystrophy (LGMD2I, n = 11), befo

293 ects affecting the palatal shelves, shoulder girdle, vertebrae, and sternum.

294 The fibrillar girdle was located directly above the transition zone, a

295 Patients have prominent limb girdle weakness and minimal craniobulbar symptoms.

296 Hip girdle and shoulder girdle weakness appeared later in the disease course.

297 inical spectrum varied from mild static limb-girdle weakness to severe generalized progressive diseas

298 All patients had prominent limb-girdle weakness with minimal or absent craniobulbar mani

299 cioscapulohumeral; oculopharyngeal; and limb-girdle which is the most heterogeneous group.

300 ead onto the anterior lining of the shoulder girdle, while a Hox-gene-controlled mesoderm links trunk