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1 and to the chronic development of bilateral glaucomatous optic neuropathy.
2 yes with concomitant epiretinal membrane and glaucomatous optic neuropathy.
3 door allergen exposure in the development of glaucomatous optic neuropathy.
4 nds on the signs of chronic angle damage and glaucomatous optic neuropathy.
5 lar pressure, can lead to the development of glaucomatous optic neuropathy.
6 ay lead to the development or progression of glaucomatous optic neuropathy.
7 ribute to the development and progression of glaucomatous optic neuropathy.
8 h regard to the likelihood of neurologic and glaucomatous optic neuropathy.
9 ft failure and irreversible vision loss from glaucomatous optic neuropathy.
10 pled populations using standard criteria for glaucomatous optic neuropathy.
11 w that oxidative damage contributes early to glaucomatous optic neuropathy.
12 athic epiretinal membranes, with and without glaucomatous optic neuropathy.
13 loss of retinal ganglion cells in a model of glaucomatous optic neuropathy.
14 s are relevant for a better understanding of glaucomatous optic neuropathy.
15 r the pathophysiology and treatment of human glaucomatous optic neuropathy.
16 the clinical interpretation of the state of glaucomatous optic neuropathy.
17 nvestigation of the biological mechanisms of glaucomatous optic neuropathy.
18 nisms of neuroprotection in the treatment of glaucomatous optic neuropathy.
19 ecedes detectable visual field loss in early glaucomatous optic neuropathy.
20 een said to contribute to the development of glaucomatous optic neuropathy.
21 curs during the neurodegenerative process of glaucomatous optic neuropathy.
22 for the immune system in the development of glaucomatous optic neuropathy.
23 rget for neuroprotection in the treatment of glaucomatous optic neuropathy.
24 as an indicator of retinal function in early glaucomatous optic neuropathy.
25 athogenic significance in some patients with glaucomatous optic neuropathy.
26 lary retinal perfusion, which suggests early glaucomatous optic neuropathy.
28 ipants, with 174 eyes of 110 patients having glaucomatous optic neuropathy and 196 eyes of 101 subjec
30 f reactive astrocytes and genes related with glaucomatous optic neuropathy and other neural disorders
31 rs continue to investigate the mechanisms of glaucomatous optic neuropathy and search for IOP-indepen
32 urophthalmological conditions that may mimic glaucomatous optic neuropathy and to determine which mos
33 ere classified as POAG (open drainage angle, glaucomatous optic neuropathy, and glaucomatous field de
34 (HRT-3), HRT's ability to correctly classify glaucomatous optic neuropathy, and HRT's role in monitor
35 teric anterior ischemic optic neuropathy and glaucomatous optic neuropathy are significantly correlat
36 uroprotection as a strategy in therapies for glaucomatous optic neuropathy as a means of preserving r
37 ch test identified a subset of the eyes with glaucomatous optic neuropathy as abnormal: 46% with stan
39 are significant differences in prevalence of glaucomatous optic neuropathy between African Americans
40 promise optic nerve head perfusion and cause glaucomatous optic neuropathy by creating transient hypo
42 ibute to the pathogenesis and development of glaucomatous optic neuropathy continues to accumulate.
43 ucoma was based on clinical examination with glaucomatous optic neuropathy defined by the presence of
44 years, the patient also developed bilateral glaucomatous optic neuropathy despite a well-controlled
45 One hundred four eyes of 104 patients with glaucomatous optic neuropathy detected by optic disc ste
46 rom 22 countries who self-registered for the Glaucomatous Optic Neuropathy Evaluation (GONE) Project
47 ent patterns of visual field loss related to glaucomatous optic neuropathy (GON) along maximally inde
48 ge from one eye of each of 156 patients with glaucomatous optic neuropathy (GON) and 189 eyes of heal
49 nto 3 groups on the basis of the presence of glaucomatous optic neuropathy (GON) and 24-2 visual fiel
51 from 110 patients (57.4 +/- 13.2 years) with glaucomatous optic neuropathy (GON) and 45 eyes of 45 co
52 distinguishing healthy eyes from those with glaucomatous optic neuropathy (GON) can be optimized by
53 , CA) from 189 normal eyes and 156 eyes with glaucomatous optic neuropathy (GON) determined by masked
54 isc photographs of subjects with and without glaucomatous optic neuropathy (GON) followed during the
56 valuation Study (ADAGES) with a diagnosis of glaucomatous optic neuropathy (GON) or ocular hypertensi
57 to determine the feasibility of identifying glaucomatous optic neuropathy (GON) using IOP corrected
60 d, regardless of visual function under test; glaucomatous optic neuropathy identified on stereophotog
61 patient was taking two glaucoma medications, glaucomatous optic neuropathy in both eyes, and visual f
62 t abnormal elastin synthesis in experimental glaucomatous optic neuropathy in the monkey is specific
63 etinal vascular geometry are associated with glaucomatous optic neuropathy independently of vascular
66 be an effective modality in the treatment of glaucomatous optic neuropathy, not all patients respond
68 ng IOP elevation of >/= 10 mmHg and incident glaucomatous optic neuropathy over the first 2 years com
71 demonstrated by several independent methods, glaucomatous optic neuropathy was not associated with a
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