ライフサイエンスコーパス: glia cell

1 cells generate non-dopaminergic neurons and glia cells.

2 enhanced Hippo pathway-driven overgrowth in glia cells.

3 teractions between photoreceptors and Muller glia cells.

4 ession and enhanced ATP release from enteric glia cells.

5 olonged cell cycle and maintenance of radial glia cells.

6 control excitability from either neurons or glia cells.

7 ation development of all retinal neurons and glia cells.

8 n the nervous system as a potent mitogen for glia cells.

9 tested in cultures of rat hippocampal neuron glia cells.

10 eurons, disinhibition in the dorsal horn and glia cell activation.

11 ptic contacts between CFs and NG2-expressing glia cells, adding to the intrigue of neuro-glial intera

12 in-associated glycoprotein (MAG), and neuron-glia cell adhesion molecule (L1).

13 cell adhesion molecules (L1-CAMs) [L1/neuron-glia cell adhesion molecule (L1/NgCAM) and neurofascin (

14 tion of the cell adhesion molecule L1/neuron-glia cell adhesion molecule (NgCAM) depends on endocytos

15 The adhesion molecule L1/neuron-glia cell adhesion molecule (NgCAM) travels to axons via

16 In contrast, the neuron-glia cell adhesion molecule (NgCAM), which binds heterop

17 the axonal cell adhesion molecule L1/neuron-glia cell adhesion molecule (NgCAM).

18 lysine and either laminin (LN) or the neuron-glia cell adhesion molecule (NgCAM).

19 he axonal adhesion molecule L1/NgCAM (neuron-glia cell adhesion molecule) and, if so, in what compart

20 containing the axonal protein NgCAM (neuron-glia cell adhesion molecule) were transported into both

21 , tenascin-C, thrombospondin, and the neuron-glia cell adhesion molecule, Ng-CAM.

22 m low-density lipoprotein receptor or neuron-glia cell-adhesion molecule to polarize expression to ei

23 hondroitin sulfate proteoglycans produced by glia cells and containing Alzheimer amyloid precursor pr

24 use brains prolongs the cell cycle of radial glia cells and extends cortical neurogenesis into postna

25 onstrated to promote the formation of radial glia cells and Notch signaling is one such signal.

26 Both GLYT1 and HMGN3 are highly expressed in glia cells and the eye, and we show that both proteins a

27 LRP1) in the metabolism of Abeta in neurons, glia cells, and along the brain vasculatures.

28 In the retina, sEH was localized in Muller glia cells, and Muller cell-specific sEH deletion reprod

29 plexiform vascular endothelial cells, Muller glia cells, and the basolateral side of the retinal pigm

30 ors expressed by Purkinje cells and Bergmann glia cells are activated predominantly by synaptic and e

31 lts demonstrate that astrocytes and Bergmann glia cells are the first cells of the brain parenchyma i

32 Glia cells are uniquely positioned at synapses, contacti

33 r than being composed of specialized midline glia cells as in many other species, the embryonic midli

34 rogenitors, resulting in an excess of radial glia cells at the expense of intermediate progenitors an

35 B1, which interacts with ephrin-B2 on radial glia cells at the optic chiasm to repulse VT axons away

36 ate, suggesting that the appican-transfected glia cells attach more avidly to their substrate than do

37 In the cerebellum, Bergmann glia cells (BGs) are intimately associated with PF synap

38 Radial glia cells both generate neurons and physically guide na

39 646329 is a lncRNA enriched in single radial glia cells but is detected at low abundance in tissues.

40 H1 hinders viral control in B7-H1 expressing glia cells, but does not mediate resistance to CD8 T cel

41 ically active Fic on capitate projections of glia cells, but not neurons, supporting a role in the re

42 s seen in both WT (CD-1) and S-100B KO (-/-) glia cells, but S-100B KO (-/-) GFAP-IR cells appeared l

43 Reducing GAD1 in metastatic cells by primary glia cell coculture abolished the capacity of metastatic

44 nsheath the neuroepithelium, suggesting that glia cells communicate with the neuroepithelium.

45 and of nitric oxide (NO) production in these glia cells contributes to neuroinflammation and neurodeg

46 In embryonic rat midbrain neuron/glia cell cultures exposed to LPS, even delayed administ

47 and rod markers; but coinciding with Muller glia cell cycle withdrawal.

48 Ependymal cells are generated from radial glia cells during embryonic brain development and acquir

49 Enteric glia cells (EGC) play an important role in the maintenan

50 Enteric glia cells (EGCs) form a dense network around myenteric

51 cerebellar cortex the processes of Bergmann glia cells encase synapses between presynaptic climbing

52 e cells express neuregulin (NRG), and radial glia cells express erbB4 in the developing cerebellum an

53 c plexus layer, and cultured primary enteric glia cells expressed IL-6 and the chemokine monocyte che

54 ue of Neuron, Li et al. show that the neuron/glia cell fate switch of cortical progenitors is regulat

55 ify Dmrt5 as a novel regulator of the neuron-glia cell-fate switch in the developing hippocampus.

56 Regulation of the neuron-glia cell-fate switch is a critical step in the developm

57 y regulated network that controls the neuron-glia cell-fate switch.SIGNIFICANCE STATEMENT We identify

58 te transporter-mediated currents in Bergmann glia cells follow the rules of synaptic release more clo

59 Radial glia cells function as guide cells for neuronal migratio

60 of PrP in neurons (NSE-HPrP/mPrP(0/0)) or in glia cells (GFAP-HPrP/mPrP(0/0)).

61 Specific assays show that in Bergmann glia cells Gpr37l1 is associated with primary cilium mem

62 Here, we demonstrated that mouse Muller glia cells have the capacity to be reprogrammed into the

63 Conditionally immortalized mouse Muller glia cells (ImM10) were cultured under nonimmortalizing

64 how that the survival of a subset of midline glia cells in Drosophila depends upon direct suppression

65 In addition, glia cells in the optic lamina that contact photorecepto

66 These results suggest that glia cells in the prefrontal cortex are particularly sen

67 Radial glia cells in the VZ primarily divide with a vertical or

68 Studies in cultured neurons and glia cells indicate that these changes in MAO A and B ar

69 synaptic Schwann cells (PSCs), which are the glia cells juxtaposed to the motor nerve terminal, activ

70 ably, a distinct human-specific outer radial glia cell layer.

71 uch as brain-derived neurotrophic factor and glia cell line-derived neurotrophic factor also reduced

72 h factor, brain-derived neurotrophic factor, glia cell line-derived neurotrophic factor, or ciliary n

73 otent and give rise only to cells within the glia cell lineage, although they are capable of forming

74 ncentration glutamate transients at Bergmann glia cell membranes that are necessary to activate low-a

75 provide a geographical cue to guide Bergmann glia cell membranes to surround active synapses and ensu

76 by the processes of one type of glia, Muller glia cells (MGs).

77 Radial glia cells play an important role in guiding migrating n

78 Panx1 deletion in GFAP-positive glia cells prevented hypersensitivity completely, wherea

79 LncND expression is enriched in radial glia cells (RGCs) in the ventricular and subventricular

80 intrinsic ganglia, comprised of neurons and glia cells that innervate airway smooth muscle, is a rec

81 ina with changes in the morphology of radial glia cells, the aberrant migration of Cajal-Retzius cell

82 hown that morphine modulates the function of glia cells through both opioid receptor dependent and in

83 neurotoxicity and their ability to activate glia cells, two biological activities of amyloid peptide

84 These data are the first to demonstrate glia cell type-dependent B7-H1 regulation in vivo, resul

85 d Cry2 in the hemispherical layer of laminal glia cells underneath the retina.

86 ransgenic mouse that expresses Cre in Muller glia cells was generated.

87 real-time imaging of cohorts of mouse radial glia cells, we show that the radial glial scaffold, upon

88 Glia cells were largely negative.

89 s with a disrupted basement membrane, radial glia cells were retracted from the pial surface, and rad

90 and expressed constitutively in the Bergman glia cells within the molecular layer of the cerebellum.