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1 sappearance of the basement membrane and the glia limitans.
2 had direct contacts with astrocytes near the glia limitans.
3 of glial fibrillary acidic protein-positive glia limitans.
4 ciated with small blood vessels and with the glia limitans.
5 es that terminate on blood vessels or on the glia limitans.
6 d with the ependyma, choroid plexus, and the glia limitans.
7 monocytes facilitate penetration through the glia limitans.
9 action composed primarily of the superficial glia limitans and pial vessels, but trended toward a dec
10 ches in basement membrane and the underlying glia limitans are the key initial events of the cellular
11 ches in basement membrane and the underlying glia limitans are the key initial events of the cellular
13 sappearance of the basement membrane and the glia limitans at the cerebral cortical surface during de
16 -feet that surround capillaries and form the glia limitans; however, the structural organization of A
17 olleagues demonstrate that astrocytes of the glia limitans induce tight junction formation in respons
18 sms regulating their penetration through the glia limitans into the parenchyma are less well studied,
21 y indicate that the inducible barrier of the glia limitans should be further explored as a therapeuti
22 face of PPC with features in common with the glia limitans that is formed by endfeet in other cortica
23 ntinuities in the pial surface basal lamina (glia limitans) that probably underlie the neuronal migra
24 ycan stabilizes the basement membrane of the glia limitans, thereby supporting the cortical infrastru
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