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2 d; moreover, expression of the ErbB3 ligand, glial growth factor 2 (GGF2) escalated rapidly after its
4 (BMP2) promotes neuronal differentiation and glial growth factor 2 (GGF2) promotes glial differentiat
5 y, we use three distinct assays to show that glial growth factor 2 (GGF2), a secreted neuregulin, exe
6 xamined the effect of the NRG-1beta isoform, glial growth factor 2 (GGF2), in rats with myocardial in
7 ast growth factor (FGF) or of the neuregulin glial growth factor 2 (GGF2), two mitogens that are norm
11 ability of a soluble NRG1 (recombinant human glial growth factor 2) to promote proliferation, surviva
13 ddition of a soluble NRG1, recombinant human glial growth factor 2, resulted in rapid (2-minute) tran
18 ing cerebral cortex, we examined the role of glial growth factor (a soluble form of neuregulin), in n
21 alled neu differentiation factor, heregulin, glial growth factor, and acetylcholine receptor-inducing
23 ludes heregulin, neu differentiation factor, glial growth factor, and the acetylcholine receptor-indu
24 sgenic mice overexpressing the NRG-1 isoform glial growth factor beta3 (GGFbeta3) in myelinating Schw
25 ith neurons, and proliferated in response to glial growth factor, confirming their identity as Schwan
28 A selective induction of mRNAs encoding the glial growth factor (GGF) subfamily of NRGs occurs in ne
29 dents, Schwann cell survival is regulated by glial growth factor (GGF), a member of the neuregulin fa
31 ) are strongly activated in Schwann cells by glial growth factor (GGF), a soluble neuregulin, and by
32 n for ciliary neurotrophic factor (CNTF) and glial growth factor (GGF2) mRNA were used to compare the
34 application of a soluble neuregulin isoform, glial growth factor II (GGF2), to developing rat muscles
35 actor and brain-derived neurotrophic factor, glial growth factor receptors such as ErbB-2; and PASK,
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