ライフサイエンスコーパス: globus

1 older, choking or pill-induced dysphagia or globus caused 37.6% (95% CI, 29.1 to 46.2) of all emerge

2 the thalamus, medial prefrontal cortex, and globus pallidum compared to euglycemia for both PASL-MRI

3 the thalamus, medial prefrontal cortex, and globus pallidum in response to hypoglycemia.

4 orticospinal tract, and reduced thalamic and globus pallidum volumes relate to deficits in cognitive

5 ions (thalamus, medial prefrontal cortex and globus pallidum) were obtained using PASL-MRI and [(15)O

6 thalamus, hypothalamus, caudate and putamen, globus pallidum, anterior cingulate gyrus, and medial te

7 The splenium of the corpus callosum, left globus pallidum, thalamus and hippocampus (P < 0.01) wer

8 , resulting in a heterogeneous distribution (globus pallidus > cortical regions > cerebellum) consist

9 bstantia nigra (SN; +46%; p<0.02) and in the globus pallidus (+9%; p=0.06) and ventral pallidum (+11%

10 intensity increase per injection, P < .001), globus pallidus (0.23% increase, P = .009), posterior th

11 he nucleus accumbens (10 +/- 7%), and in the globus pallidus (13 +/- 15%) measured with [(11)C]PHNO,

12 telangiectasia also had higher metabolism in globus pallidus (16%, P = 0.05), which correlated negati

13 ording single-unit spiking activity from the globus pallidus (232 neurons) while the animals complete

14 spite loss of kinematic tuning, cells in the globus pallidus (63%, 10/16 cells) and VLo (84%, 46/55 c

15 joint position, velocity, or acceleration in globus pallidus (75%, 12/16 cells) and in the pallidal r

16 In the globus pallidus (GP in rodents, homolog of the primate G

17 iprocally connected GABAergic neurons of the globus pallidus (GP) and glutamatergic neurons of the su

18 -related increases in iron concentrations in globus pallidus (GP) and substantia nigra (SN), although

19 -related increases in iron concentrations in globus pallidus (GP) and substantia nigra (SN), although

20 mGluR1 and mGluR5, regulate activity in the globus pallidus (GP) and subthalamic nucleus (STN).

21 The firing patterns of neurons in the globus pallidus (GP) are affected by two major sources o

22 The globus pallidus (GP) consists of two neuron populations,

23 halamus before, during, and after DBS in the globus pallidus (GP) GP-DBS resulted in: (1) stimulus-en

24 SI) increase in the dentate nucleus (DN) and globus pallidus (GP) in relation to the middle cerebella

25 The globus pallidus (GP) is a critical component of the basa

26 leus (STN) neurons and two types of external globus pallidus (GP) neuron inappropriately synchronise

27 Synchronization of globus pallidus (GP) neurons and cortically entrained os

28 Globus pallidus (GP) neurons fire rhythmically in the ab

29 halamic nucleus (STN) and GABAergic external globus pallidus (GP) neurons normally exhibit weakly cor

30 Globus pallidus (GP) neurons recorded in brain slices sh

31 provide a prominent source of excitation to globus pallidus (GP) neurons, despite their scarce numbe

32 MSNs and FSIs together with motor cortex and globus pallidus (GP) neurons, in rats performing a simpl

33 onsidered similar, if not homologous, to the globus pallidus (GP) of mammals.

34 levodopa responses was seen in the striatum/globus pallidus (GP) of the lesioned hemisphere.

35 The globus pallidus (GP) predominantly contains GABAergic pr

36 In comparison, the globus pallidus (GP), a main striatal output nucleus, ha

37 triatum and their subsequent growth into the globus pallidus (GP), an important (intermediate) target

38 The signal intensities (SIs) in the globus pallidus (GP), thalamus (T), dentate nucleus (DN)

39 ons are unknown but activity in the external globus pallidus (GP), which forms a candidate pacemaker

40 ential firing and pattern of activity in the globus pallidus (GP)-subthalamic nucleus (STN) network a

41 with those of the lateral striatum (LSt) and globus pallidus (GP).

42 ), respectively, in hippocampus and striatum/globus pallidus (GP).

43 ion of striatal channels at the level of the globus pallidus (GP).

44 substantia nigra (SN), pulvinar thalami, and globus pallidus (GP).

45 c inhibition from the reciprocally connected globus pallidus (GP).

46 th the external and internal segments of the globus pallidus (GPe and GPi, respectively).

47 he reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

48 he reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

49 Neurons in the external segment of the globus pallidus (GPe) are autonomous pacemakers that are

50 ine-depleted rats indicate that the external globus pallidus (GPe) contains two main types of GABAerg

51 Neurons in the external globus pallidus (GPe) generate pacemaker activity that c

52 tal dopamine, the striatum, and the external globus pallidus (GPe) in regulating RLS-like movements,

53 cal excitation and indirect pathway external globus pallidus (GPe) inhibition of the STN are critical

54 The external globus pallidus (GPe) is a key contributor to motor supp

55 The external globus pallidus (GPe) is a key nucleus within basal gang

56 The external segment of the globus pallidus (GPe) is one of the core nuclei of the b

57 We show how external globus pallidus (GPe) neuron is sensitive to the phase o

58 analogous division of labor in the external globus pallidus (GPe) of Parkinsonian rats, showing that

59 the constituent neurons within the external globus pallidus (GPe) was not fully appreciated.

60 ated activity of the external segment of the globus pallidus (GPe), a downstream structure whose comp

61 s that pathological activity of the external globus pallidus (GPe), a nucleus in the basal ganglia, c

62 In the external segment of the globus pallidus (GPe), neuronal populations have been de

63 Focusing on the mouse external globus pallidus (GPe), we demonstrate that the potential

64 ural circuits in the external segment of the globus pallidus (GPe).

65 highest levels in the striatum and external globus pallidus (GPe).

66 ng in neurons of the external segment of the globus pallidus (GPe).

67 ic nucleus (STN) and the external segment of globus pallidus (GPe).

68 , and neurons in the external segment of the globus pallidus (GPe)], and one neuromodulator group [st

69 show that neurons in the habenula-projecting globus pallidus (GPh) in mice are essential for evaluati

70 uit, which regulates the habenula-projecting globus pallidus (GPh) neurons, exists within the basal g

71 n the subthalamic nucleus (STN) and internal globus pallidus (GPi) (P < 0.001), as well as in the dor

72 of the subthalamic nucleus (STN) or internal globus pallidus (GPi) reduces dyskinesias remain largely

73 ive visualization scores of STN and internal globus pallidus (GPi) were recorded by two neuroradiolog

74 drove network activity through the internal globus pallidus (GPi), external globus pallidus, motor c

75 deep brain stimulation (DBS) at the internal globus pallidus (GPi).

76 onal activity in the internal segment of the globus pallidus (GPi).

77 ebellum and from the internal segment of the globus pallidus (GPi).

78 s reticulata (direct pathway) or the lateral globus pallidus (indirect pathway).

79 for post-GBCA group [P < .001]) but not the globus pallidus (mean SI ratio for two-group comparison:

80 shortening in the dentate nucleus (n = 13), globus pallidus (n = 13), substantia nigra (n = 13), pos

81 ation of the subthalamic nucleus (n = 60) or globus pallidus (n = 61).

82 e gray matter of the whole brain (P < .001), globus pallidus (P = .002), dentate nucleus (P = .046),

83 ight caudate nucleus and ventral putamen and globus pallidus (P = .003) compared with that in control

84 years in the substantia nigra (P < .001) and globus pallidus (P = .035), which are both predictors of

85 caudate (P < 0.001), putamen (P < 0.001) and globus pallidus (P = 0.025) in patients with Parkinson's

86 ept for the caudate putamen (P=0.09) and the globus pallidus (P=0.12).

87 I's in the bilateral caudate (C) and putamen/globus pallidus (PGp) were studied based upon previous f

88 0.005), putamen (r = -0.51; P = 0.025), and globus pallidus (r = -0.47; P = 0.030).

89 0.011), putamen (r = -0.48; P = 0.022), and globus pallidus (r = -0.70; P < 0.001).

90 reduce central thalamus activity and release globus pallidus activity in DOCs.

91 e alignments between subthalamic nucleus and globus pallidus amplified local neural synchrony in the

92 s well as the internal capsule, putamen, and globus pallidus and basal forebrain extending to the ant

93 The reversal of globus pallidus and central thalamus profiles across BIs

94 erior limb of the internal capsule, putamen, globus pallidus and cerebellum in the postmortem brain t

95 sed that area X is a mixture of striatum and globus pallidus and has the same functional organization

96 y in the internal capsule extending into the globus pallidus and in the right prefrontal region.

97 ing single-unit activity in the sensorimotor globus pallidus and motor thalamus before, during, and a

98 y specific brain areas such as the thalamus, globus pallidus and orbitofrontal regions of the right h

99 ssociated with a lower T1R (P = 0.01) in the globus pallidus and putamen but were not associated with

100 as recognized as homologous to the mammalian globus pallidus and renamed as such.

101 release than the male subjects in the right globus pallidus and right inferior frontal gyrus.

102 y between external and internal parts of the globus pallidus and saliency of the sensory input, a low

103 t of the parvalbumin-positive neurons in the globus pallidus and striatum of the Ldb1 mutants.

104 put from the caudal BF, including the caudal globus pallidus and substantia innominata and moderate i

105 ar whether abnormal activity measured in the globus pallidus and substantia nigra pars reticulata is

106 the usual striatal target areas such as the globus pallidus and substantia nigra, raising the possib

107 frequently encountered, particularly in the globus pallidus and substantia nigra, where they account

108 xceptions were found, most noticeably in the globus pallidus and substantia nigra, which contained a

109 in addition to many axonal spheroids in the globus pallidus and substantia nigra.

110 Activity in the internal segment of the globus pallidus and subthalamic nucleus correlated stron

111 but also bilateral caudate and left putamen/globus pallidus and subthalamic nucleus.

112 ined the surface morphology of the striatum, globus pallidus and thalamus, and thickness of the cereb

113 the parieto-occipital regions, left putamen/globus pallidus and thalamus; and in white matter of the

114 al neurons in rodents), which project to the globus pallidus and the substantia nigra, and the locall

115 in the striatum and their projections to the globus pallidus and the substantia nigra.

116 eceptor-mediated inhibition arising from the globus pallidus and thus promote single-spike activity r

117 indicating spontaneous beta oscillations in globus pallidus are not pathognomonic.

118 d nucleus of the stria terminalis (BNST) and globus pallidus at 60 and 120 min following KA and in th

119 Globus pallidus binding best distinguished PSP patients

120 ared with NT mice, significantly increase in globus pallidus but decrease in entopeduncular nucleus/s

121 in the dorsal caudate nucleus, putamen, and globus pallidus but the observed variation did not assoc

122 nd caudate nucleus, red nucleus, putamen and globus pallidus by T2* MRI at baseline and after 3 and 6

123 lamic nucleus or the internal segment of the globus pallidus can substantially reduce bradykinesia, r

124 al neurons, and magnocellular neurons in the globus pallidus did not demonstrate a similar extent of

125 No significant changes were seen in the globus pallidus during anticipation of exercise.

126 uantification of single-unit activity in the globus pallidus externa (GPe) and substantia nigra retic

127 a from the globus pallidus interna (GPi) and globus pallidus externa (GPe) in children undergoing dee

128 strong GABAergic inhibitory inputs from the globus pallidus externa (GPe) that is reciprocally conne

129 strates that functional connections from the globus pallidus externa (GPe) to striatum are substantia

130 cortex, the globus pallidus interna, and the globus pallidus externa compared with patients with trem

131 o the homolog of the globus pallidus interna/globus pallidus externa express D1 or D2 receptors.

132 cortex, the globus pallidus interna, and the globus pallidus externa.

133 s dopamine D2 receptors (D2+) project to the globus pallidus external and are thought to inhibit move

134 l neurons comprising subthalamic nucleus and globus pallidus external and suggest this approach for d

135 ent striatal (Str) neurons projecting to the globus pallidus external segment (GPe).

136 ectly through the caudal-ventral part of the globus pallidus externus (cvGPe).

137 ere we describe a direct projection from the globus pallidus externus (GP), a central nucleus of the

138 ne increased rabies-labelled inputs from the globus pallidus externus (GPe), a basal ganglia nucleus

139 Gamma oscillations arise in the subthalamic-globus pallidus feedback loop, and occur during movement

140 the basal ganglia, the caudate, putamen and globus pallidus in brown capuchin monkeys (Cebus apella)

141 nucleus accumbens and frontal cortex but not globus pallidus in the l-DOPA group.

142 n metabolic rates including hyperactivity in globus pallidus indicative of basal ganglia involvement.

143 ance imaging of the brain revealed bilateral globus pallidus injury in the setting of dyskinetic CP.

144 We report microelectrode data from the globus pallidus interna (GPi) and globus pallidus extern

145 ndomised to subthalamic nucleus (STN) versus globus pallidus interna (GPi) DBS surgery.

146 ood effects of subthalamic nucleus (STN) vs. globus pallidus interna (GPi) deep brain stimulation (DB

147 ut a previous case suggested that DBS of the globus pallidus interna (GPi) might.

148 ither the subthalamic nucleus (STN, n=84) or globus pallidus interna (GPi, n=80), using standardised

149 o had undergone bilateral stimulation of the globus pallidus interna (pallidal stimulation) or subtha

150 The basal ganglia, including the striatum, globus pallidus interna and externa (GPe), subthalamic n

151 The globus pallidus interna and the subthalamic nucleus are

152 n of Medtronic quadripolar electrodes in the globus pallidus interna bilaterally.

153 The results suggest anteromedial globus pallidus interna DBS for Tourette's syndrome is a

154 the effectiveness of DBS of the anteromedial globus pallidus interna on tic severity and common comor

155 ating that stimulation of an area called the globus pallidus interna partially reverses deficits in v

156 viously ignored structure in data describing globus pallidus interna responses to cortical stimulatio

157 ilateral dorsolateral prefrontal cortex, the globus pallidus interna, and the globus pallidus externa

158 ilateral dorsolateral prefrontal cortex, the globus pallidus interna, and the globus pallidus externa

159 from the dorsal pallidum, the homolog of the globus pallidus interna.

160 tal neurons projecting to the homolog of the globus pallidus interna/globus pallidus externa express

161 on in the dorsolateral prefrontal cortex and globus pallidus internal part in the picture sequencing

162 rded extracellular single unit discharges of globus pallidus internal segment (GPi) neurons in monkey

163 jor output station of the basal ganglia, the globus pallidus internal segment (GPi) projects to the t

164 ion in the basal ganglia output nucleus, the globus pallidus internal segment.

165 trating that deep brain stimulation (DBS) of globus pallidus internus (GPi DBS) and subthalamic nucle

166 assess the safety and efficacy of bilateral globus pallidus internus (GPi) DBS in patient's with sev

167 ients to either subthalamic nucleus (STN) or globus pallidus internus (GPi) deep brain stimulation (D

168 implicates the subthalamic nucleus (STN) and globus pallidus internus (GPi) in reward and punishment

169 corded the activity of single neurons in the globus pallidus internus (GPi), the primary BG output nu

170 in skeletomotor output region of the BG, the globus pallidus internus (GPi).

171 Deep brain stimulation of the globus pallidus internus alleviates involuntary movement

172 amma synchronization (ERS) recorded from the globus pallidus internus in patients undergoing deep bra

173 a unilateral excitotoxic injection into the globus pallidus leads to a gradual loss of dopaminergic

174 ucleus, and movement-related activity of the globus pallidus leads to transient beta oscillations dur

175 s dopamine cell death at 3 weeks following a globus pallidus lesion, but not thereafter.

176 mean uptake after BI; a relative increase in globus pallidus metabolism was evident in BI subjects wh

177 The subthalamic nucleus-globus pallidus network is a potential source of oscilla

178 We show that most globus pallidus neurons, but very few neocortical intern

179 om the Shh-expressing domain eliminated most globus pallidus neurons, whereas most cortical and stria

180 cortical interneurons; and pattern C, mainly globus pallidus neurons.

181 recordings from the subthalamic nucleus and globus pallidus of five patients with Parkinson's diseas

182 nificantly elevated within the NAc shell and globus pallidus of ko mice, whereas serotonin transporte

183 sed local field in the caudate, putamen, and globus pallidus of patients relative to control subjects

184 activity occurs in the prefrontal cortex and globus pallidus of patients with nontremor-dominant PD c

185 onounced differences in substantia nigra and globus pallidus of PSP compared with control brains.

186 spectively, into the superior colliculus and globus pallidus of Sprague-Dawley rats.

187 tial component compared with inhibition from globus pallidus or substantia nigra pars reticulata.

188 r DA release in the sensorimotor part of the globus pallidus pars interna (GPi) contralateral to the

189 e treated with deep brain stimulation of the globus pallidus pars interna during the period 1999-2010

190 rm efficacy of deep brain stimulation of the globus pallidus pars interna in the treatment of general

191 r predicting a better or poorer outcome from globus pallidus pars interna stimulation in this series

192 ed that supplementary motor area and putamen/globus pallidus regions showed training-related decrease

193 Projections to the globus pallidus showed solely D2-mediated presynaptic in

194 are distributed more caudally in the caudal globus pallidus than noncholinergic projection neurons.

195 density of direct pathway collaterals in the globus pallidus that bridge the direct pathway with the

196 ential values (from approximately 0.5 in the globus pallidus to approximately 3.5 in the insula) for

197 ly distinct projections through striatum and globus pallidus to EP targets within epithalamus and tha

198 discovered feedback projection from external globus pallidus to striatum is crucial for inhibitory co

199 These findings provide evidence of smaller globus pallidus volume in patients with OCD without the

200 l functional connectivity within striatum or globus pallidus was relatively weak.

201 he subthalamic nucleus, substantia nigra and globus pallidus were consistently the regions most sever

202 alamus and thalamic subnuclei, striatum, and globus pallidus were segmented using a fully automated m

203 Protein components of the globus pallidus were studied including glial fibrillary

204 ith diabetes and MDD showed lower MTR in the globus pallidus when compared with the group with MDD.

205 he amygdala, BNST, cingulate cortex, PVN and globus pallidus within 3 h after KA treatment and in the

206 basal ganglia nuclei (caudate, putamen, and globus pallidus) in children with ADHD.

207 " pathways that regulate the pallidal (e.g., globus pallidus) output nuclei involved in the control o

208 l striatum with the dorsal striatum (putamen/globus pallidus), and lower connectivity between these r

209 ance in the striatum and axon entry into the globus pallidus).

210 n: (1) stimulus-entrained firing patterns in globus pallidus, (2) a monophasic stimulus-entrained fir

211 esponses to joint articulation (3/4 cells in globus pallidus, 11/33 cells in VLo).

212 -fold more exon 10+ MAPT mRNA than H2 in the globus pallidus, a brain region highly affected by tauop

213 et in apparent conflict with this model, the globus pallidus, a GABAergic basal ganglia output struct

214 hod, we were able to map a homunculus in the globus pallidus, a key target area for deep brain stimul

215 tosensory cortex, contralateral amygdala and globus pallidus, and bilateral periaqueductal gray.

216 ume reductions in the hippocampus, thalamus, globus pallidus, and caudate nucleus compared with 26 co

217 in contralateral amygdala, ventral pallidum, globus pallidus, and hippocampus, as well as decreases i

218 -A5 selectively binds areas of the striatum, globus pallidus, and substantia nigra containing EphA7+

219 itization was found in hypothalamus, cortex, globus pallidus, and substantia nigra of betaarr2-KO com

220 alamus, caudate, putamen, nucleus accumbens, globus pallidus, and substantia nigra).

221 nd distinct firing patterns in the striatum, globus pallidus, and subthalamic nucleus related to sens

222 ronal tissues from the dentate nuclei, pons, globus pallidus, and thalamus of these 23 deceased patie

223 ronal tissues from the dentate nuclei, pons, globus pallidus, and thalamus were harvested and analyze

224 ons, including deep cerebellar nuclei (DCN), globus pallidus, and thalamus.

225 ively express NuIP such as striatum, septum, globus pallidus, and the reticular thalamic nucleus.

226 thalamic nucleus (STN), rostromedial lateral globus pallidus, and ventral pallidum.

227 -rich caudate/putamen, nucleus accumbens and globus pallidus, as well as the medial and orbitofrontal

228 tex, corpus callosum, hippocampus, thalamus, globus pallidus, caudoputamen and whole brain.

229 ygdala) and sub-cortical (putamen, thalamus, globus pallidus, cerebellum) regions.

230 0.59, 0.51, and 0.50, respectively, for the globus pallidus, cingulate cortex, insula, caudate, puta

231 imaging demonstrated T2 hypointensity in the globus pallidus, confluent T2 white matter hyperintensit

232 on, regions of interest were measured in the globus pallidus, dentate nucleus, thalamus, and pons.

233 s in CMR and increases in CBF in the putamen/globus pallidus, dorsal midbrain/pons, STN, and ventral

234 Within the globus pallidus, EphA7+ axons terminate primarily within

235 transcription real-time PCR in the striatum, globus pallidus, frontal cortex and cingulate cortex of

236 ucleus accumbens, amygdala, caudate nucleus, globus pallidus, hippocampus, putamen, thalamus, and bra

237 re reward circuit regions including putamen, globus pallidus, insula and thalamus.

238 av1.8 expression in the amygdala, brainstem, globus pallidus, lateral and paraventricular hypothalamu

239 In the globus pallidus, medium spiny neurons connected strongly

240 the internal globus pallidus (GPi), external globus pallidus, motor cortex, thalamus, or cerebellum.

241 s by stimulation of afferents from striatum, globus pallidus, or pars reticulata have been shown to b

242 0A is upregulated in striatal projections to globus pallidus, preferentially expressing D2 receptors

243 e group differences in the substantia nigra, globus pallidus, pulvinar thalamus, thalamus, and caudat

244 and magnetic field correlation (MFC) in the globus pallidus, putamen, caudate nucleus, and thalamus

245 ional hedonic (orbitofrontal cortex, insula, globus pallidus, putamen, hippocampus, and amygdala) and

246 rally elevated (18) F-flortaucipir uptake in globus pallidus, putamen, subthalamic nucleus, midbrain,

247 s accumbens, amygdala, caudate, hippocampus, globus pallidus, putamen, thalamus, lateral ventricles.

248 f GPi from the internal capsule and external globus pallidus, respectively.

249 < 0.001) metabolic reductions in the putamen/globus pallidus, sensorimotor cortex and cerebellar verm

250 cortex, amygdala, olfactory bulb, striatum, globus pallidus, septum, and nucleus basalis.

251 ei, including the subthalamic nucleus (STN), globus pallidus, striatum, and substantia nigra.

252 ding severe atrophy and neuronal loss in the globus pallidus, substantia nigra and subthalamic nucleu

253 erebral cortex, cerebellum, caudate/putamen, globus pallidus, substantia nigra, and hippocampus.

254 ojecting back to the same cortical areas via globus pallidus, substantia nigra, and thalamus.

255 aled discrete expression in caudate/putamen, globus pallidus, substantia nigra, cerebellar Purkinje c

256 severe degeneration in the cardinal nuclei - globus pallidus, subthalamic nucleus and substantia nigr

257 ped and successfully applied in the internal globus pallidus, subthalamic nucleus, and thalamus for t

258 -Fos(+) neurons in the external and internal globus pallidus, subthalamic nucleus, and ventral motor

259 m and instead receiving more inputs from the globus pallidus, subthalamic nucleus, and zona incerta.

260 ed in the sensorimotor cortex, striatum, and globus pallidus, support the hypothesis of a circuitwide

261 atients bilaterally in the ventral striatum, globus pallidus, thalamus, amygdala and right insula.

262 creased susceptibility of the neurons of the globus pallidus, thalamus, and substantia nigra pars com

263 The SI in the globus pallidus, thalamus, dentate nucleus, and pons was

264 ead of the caudate nucleus (hCaud), putamen, globus pallidus, thalamus-and four cortical regions-rost

265 hin the major striatal efferent targets: the globus pallidus, the entopeduncular nucleus, and the SN

266 in demonstrate the highest Mn content in the globus pallidus, the thalamus, and the substantia nigra

267 reservation in vicinities of the cerebellum, globus pallidus, visual cortex, and parietal cortex in o

268 ation, including the dorsal striatum and the globus pallidus, were also activated.

269 ) with glucose metabolism in the putamen and globus pallidus, which receive projections from this str

270 ging showed iron in the substantia nigra and globus pallidus, with a 'halo' of T1 hyperintense signal

271 iated by postsynaptic receptors, and that of globus pallidus-evoked inputs is mediated by presynaptic

272 Globus pallidus-thalamus and dentate nucleus-pons SI rat

273 tabolic activity in the central thalamus and globus pallidus.

274 ssue, most notably in the dentate nuclei and globus pallidus.

275 no changes being detected in the putamen or globus pallidus.

276 ange projections to the substantia nigra and globus pallidus.

277 d prefrontal areas, and between amygdala and globus pallidus.

278 t brain pineal gland, nucleus accumbens, and globus pallidus.

279 mice after local infusion into the external globus pallidus.

280 ar peduncle, colliculi, dentate nucleus, and globus pallidus.

281 isorder and prominent iron deposition in the globus pallidus.

282 lar nucleus/substantia nigra and to external globus pallidus.

283 llular component (Nkx2-1(-);Npas1(+)) of the globus pallidus.

284 ulate cortex and increased metabolism in the globus pallidus.

285 e situation was reversed in the striatum and globus pallidus.

286 d striatum, with associated increases in the globus pallidus.

287 in or substance P and extended fibers to the globus pallidus.

288 resembles neurons recorded in the mammalian globus pallidus.

289 terminalis, and to a remarkable extent, the globus pallidus.

290 ages, including the sensorimotor putamen and globus pallidus.

291 ortex shifts slightly caudally in the caudal globus pallidus.

292 d as disinhibited downstream activity in the globus pallidus.

293 pulate the cortex, hippocampus striatum, and globus pallidus.

294 ls were recorded throughout the sensorimotor globus pallidus.

295 lly significant dose-dependent relationship (globus pallidus: rho = 0.90, P = .04).

296 modulation in firing patterns (2/12 cells in globus pallidus; 13/23 cells in VLo), and regularized fi

297 ral, associative, and sensorimotor striatum; globus pallidus; central thalamus and noncentral thalamu

298 e stria terminalis; the caudate-putamen; the globus pallidus; the lateral septum; and the islands of

299 Although the majority of FGIDs, including globus, rumination syndrome, IBS, bloating, constipation

300 , dysphagia (32.3% vs. 13.1%; p = 0.001) and globus sensation (27.7% vs. 13.8%; p = 0.021) were found