ライフサイエンスコーパス: globus pallidus

1 ance in the striatum and axon entry into the globus pallidus).

2 tabolic activity in the central thalamus and globus pallidus.

3 ssue, most notably in the dentate nuclei and globus pallidus.

4 no changes being detected in the putamen or globus pallidus.

5 ange projections to the substantia nigra and globus pallidus.

6 d prefrontal areas, and between amygdala and globus pallidus.

7 t brain pineal gland, nucleus accumbens, and globus pallidus.

8 mice after local infusion into the external globus pallidus.

9 isorder and prominent iron deposition in the globus pallidus.

10 ar peduncle, colliculi, dentate nucleus, and globus pallidus.

11 llular component (Nkx2-1(-);Npas1(+)) of the globus pallidus.

12 lar nucleus/substantia nigra and to external globus pallidus.

13 ulate cortex and increased metabolism in the globus pallidus.

14 e situation was reversed in the striatum and globus pallidus.

15 d striatum, with associated increases in the globus pallidus.

16 in or substance P and extended fibers to the globus pallidus.

17 resembles neurons recorded in the mammalian globus pallidus.

18 terminalis, and to a remarkable extent, the globus pallidus.

19 ages, including the sensorimotor putamen and globus pallidus.

20 t MRI abnormality is increased signal in the globus pallidus.

21 ortex shifts slightly caudally in the caudal globus pallidus.

22 d as disinhibited downstream activity in the globus pallidus.

23 pulate the cortex, hippocampus striatum, and globus pallidus.

24 ls were recorded throughout the sensorimotor globus pallidus.

25 intensity increase per injection, P < .001), globus pallidus (0.23% increase, P = .009), posterior th

26 esponses to joint articulation (3/4 cells in globus pallidus, 11/33 cells in VLo).

27 he nucleus accumbens (10 +/- 7%), and in the globus pallidus (13 +/- 15%) measured with [(11)C]PHNO,

28 modulation in firing patterns (2/12 cells in globus pallidus; 13/23 cells in VLo), and regularized fi

29 telangiectasia also had higher metabolism in globus pallidus (16%, P = 0.05), which correlated negati

30 n: (1) stimulus-entrained firing patterns in globus pallidus, (2) a monophasic stimulus-entrained fir

31 ording single-unit spiking activity from the globus pallidus (232 neurons) while the animals complete

32 spite loss of kinematic tuning, cells in the globus pallidus (63%, 10/16 cells) and VLo (84%, 46/55 c

33 joint position, velocity, or acceleration in globus pallidus (75%, 12/16 cells) and in the pallidal r

34 bstantia nigra (SN; +46%; p<0.02) and in the globus pallidus (+9%; p=0.06) and ventral pallidum (+11%

35 -fold more exon 10+ MAPT mRNA than H2 in the globus pallidus, a brain region highly affected by tauop

36 et in apparent conflict with this model, the globus pallidus, a GABAergic basal ganglia output struct

37 hod, we were able to map a homunculus in the globus pallidus, a key target area for deep brain stimul

38 reduce central thalamus activity and release globus pallidus activity in DOCs.

39 e alignments between subthalamic nucleus and globus pallidus amplified local neural synchrony in the

40 s well as the internal capsule, putamen, and globus pallidus and basal forebrain extending to the ant

41 The reversal of globus pallidus and central thalamus profiles across BIs

42 erior limb of the internal capsule, putamen, globus pallidus and cerebellum in the postmortem brain t

43 Volumes of the caudate nucleus, putamen, and globus pallidus and gray and white matter volumes of the

44 sed that area X is a mixture of striatum and globus pallidus and has the same functional organization

45 y in the internal capsule extending into the globus pallidus and in the right prefrontal region.

46 ing single-unit activity in the sensorimotor globus pallidus and motor thalamus before, during, and a

47 y specific brain areas such as the thalamus, globus pallidus and orbitofrontal regions of the right h

48 ssociated with a lower T1R (P = 0.01) in the globus pallidus and putamen but were not associated with

49 as recognized as homologous to the mammalian globus pallidus and renamed as such.

50 release than the male subjects in the right globus pallidus and right inferior frontal gyrus.

51 y between external and internal parts of the globus pallidus and saliency of the sensory input, a low

52 t of the parvalbumin-positive neurons in the globus pallidus and striatum of the Ldb1 mutants.

53 put from the caudal BF, including the caudal globus pallidus and substantia innominata and moderate i

54 ar whether abnormal activity measured in the globus pallidus and substantia nigra pars reticulata is

55 the usual striatal target areas such as the globus pallidus and substantia nigra, raising the possib

56 frequently encountered, particularly in the globus pallidus and substantia nigra, where they account

57 xceptions were found, most noticeably in the globus pallidus and substantia nigra, which contained a

58 in addition to many axonal spheroids in the globus pallidus and substantia nigra.

59 Activity in the internal segment of the globus pallidus and subthalamic nucleus correlated stron

60 but also bilateral caudate and left putamen/globus pallidus and subthalamic nucleus.

61 ined the surface morphology of the striatum, globus pallidus and thalamus, and thickness of the cereb

62 the parieto-occipital regions, left putamen/globus pallidus and thalamus; and in white matter of the

63 al neurons in rodents), which project to the globus pallidus and the substantia nigra, and the locall

64 in the striatum and their projections to the globus pallidus and the substantia nigra.

65 eceptor-mediated inhibition arising from the globus pallidus and thus promote single-spike activity r

66 l striatum with the dorsal striatum (putamen/globus pallidus), and lower connectivity between these r

67 tosensory cortex, contralateral amygdala and globus pallidus, and bilateral periaqueductal gray.

68 ume reductions in the hippocampus, thalamus, globus pallidus, and caudate nucleus compared with 26 co

69 in contralateral amygdala, ventral pallidum, globus pallidus, and hippocampus, as well as decreases i

70 -A5 selectively binds areas of the striatum, globus pallidus, and substantia nigra containing EphA7+

71 itization was found in hypothalamus, cortex, globus pallidus, and substantia nigra of betaarr2-KO com

72 alamus, caudate, putamen, nucleus accumbens, globus pallidus, and substantia nigra).

73 nd distinct firing patterns in the striatum, globus pallidus, and subthalamic nucleus related to sens

74 ronal tissues from the dentate nuclei, pons, globus pallidus, and thalamus of these 23 deceased patie

75 ronal tissues from the dentate nuclei, pons, globus pallidus, and thalamus were harvested and analyze

76 ons, including deep cerebellar nuclei (DCN), globus pallidus, and thalamus.

77 ively express NuIP such as striatum, septum, globus pallidus, and the reticular thalamic nucleus.

78 thalamic nucleus (STN), rostromedial lateral globus pallidus, and ventral pallidum.

79 indicating spontaneous beta oscillations in globus pallidus are not pathognomonic.

80 -rich caudate/putamen, nucleus accumbens and globus pallidus, as well as the medial and orbitofrontal

81 d nucleus of the stria terminalis (BNST) and globus pallidus at 60 and 120 min following KA and in th

82 Globus pallidus binding best distinguished PSP patients

83 ared with NT mice, significantly increase in globus pallidus but decrease in entopeduncular nucleus/s

84 in the dorsal caudate nucleus, putamen, and globus pallidus but the observed variation did not assoc

85 nd caudate nucleus, red nucleus, putamen and globus pallidus by T2* MRI at baseline and after 3 and 6

86 lamic nucleus or the internal segment of the globus pallidus can substantially reduce bradykinesia, r

87 tex, corpus callosum, hippocampus, thalamus, globus pallidus, caudoputamen and whole brain.

88 ral, associative, and sensorimotor striatum; globus pallidus; central thalamus and noncentral thalamu

89 ygdala) and sub-cortical (putamen, thalamus, globus pallidus, cerebellum) regions.

90 0.59, 0.51, and 0.50, respectively, for the globus pallidus, cingulate cortex, insula, caudate, puta

91 imaging demonstrated T2 hypointensity in the globus pallidus, confluent T2 white matter hyperintensit

92 on, regions of interest were measured in the globus pallidus, dentate nucleus, thalamus, and pons.

93 al neurons, and magnocellular neurons in the globus pallidus did not demonstrate a similar extent of

94 s in CMR and increases in CBF in the putamen/globus pallidus, dorsal midbrain/pons, STN, and ventral

95 No significant changes were seen in the globus pallidus during anticipation of exercise.

96 Grafted cells were also observed in the globus pallidus, entopeduncular nucleus, and substantia

97 Within the globus pallidus, EphA7+ axons terminate primarily within

98 iated by postsynaptic receptors, and that of globus pallidus-evoked inputs is mediated by presynaptic

99 uantification of single-unit activity in the globus pallidus externa (GPe) and substantia nigra retic

100 a from the globus pallidus interna (GPi) and globus pallidus externa (GPe) in children undergoing dee

101 strong GABAergic inhibitory inputs from the globus pallidus externa (GPe) that is reciprocally conne

102 strates that functional connections from the globus pallidus externa (GPe) to striatum are substantia

103 cortex, the globus pallidus interna, and the globus pallidus externa compared with patients with trem

104 o the homolog of the globus pallidus interna/globus pallidus externa express D1 or D2 receptors.

105 cortex, the globus pallidus interna, and the globus pallidus externa.

106 s dopamine D2 receptors (D2+) project to the globus pallidus external and are thought to inhibit move

107 l neurons comprising subthalamic nucleus and globus pallidus external and suggest this approach for d

108 ent striatal (Str) neurons projecting to the globus pallidus external segment (GPe).

109 ectly through the caudal-ventral part of the globus pallidus externus (cvGPe).

110 ere we describe a direct projection from the globus pallidus externus (GP), a central nucleus of the

111 ne increased rabies-labelled inputs from the globus pallidus externus (GPe), a basal ganglia nucleus

112 Gamma oscillations arise in the subthalamic-globus pallidus feedback loop, and occur during movement

113 transcription real-time PCR in the striatum, globus pallidus, frontal cortex and cingulate cortex of

114 In the globus pallidus (GP in rodents, homolog of the primate G

115 iprocally connected GABAergic neurons of the globus pallidus (GP) and glutamatergic neurons of the su

116 -related increases in iron concentrations in globus pallidus (GP) and substantia nigra (SN), although

117 -related increases in iron concentrations in globus pallidus (GP) and substantia nigra (SN), although

118 mGluR1 and mGluR5, regulate activity in the globus pallidus (GP) and subthalamic nucleus (STN).

119 The firing patterns of neurons in the globus pallidus (GP) are affected by two major sources o

120 The globus pallidus (GP) consists of two neuron populations,

121 halamus before, during, and after DBS in the globus pallidus (GP) GP-DBS resulted in: (1) stimulus-en

122 SI) increase in the dentate nucleus (DN) and globus pallidus (GP) in relation to the middle cerebella

123 The globus pallidus (GP) is a critical component of the basa

124 leus (STN) neurons and two types of external globus pallidus (GP) neuron inappropriately synchronise

125 Synchronization of globus pallidus (GP) neurons and cortically entrained os

126 Globus pallidus (GP) neurons fire rhythmically in the ab

127 halamic nucleus (STN) and GABAergic external globus pallidus (GP) neurons normally exhibit weakly cor

128 Globus pallidus (GP) neurons recorded in brain slices sh

129 provide a prominent source of excitation to globus pallidus (GP) neurons, despite their scarce numbe

130 MSNs and FSIs together with motor cortex and globus pallidus (GP) neurons, in rats performing a simpl

131 onsidered similar, if not homologous, to the globus pallidus (GP) of mammals.

132 levodopa responses was seen in the striatum/globus pallidus (GP) of the lesioned hemisphere.

133 The globus pallidus (GP) predominantly contains GABAergic pr

134 In comparison, the globus pallidus (GP), a main striatal output nucleus, ha

135 triatum and their subsequent growth into the globus pallidus (GP), an important (intermediate) target

136 The signal intensities (SIs) in the globus pallidus (GP), thalamus (T), dentate nucleus (DN)

137 ons are unknown but activity in the external globus pallidus (GP), which forms a candidate pacemaker

138 ential firing and pattern of activity in the globus pallidus (GP)-subthalamic nucleus (STN) network a

139 with those of the lateral striatum (LSt) and globus pallidus (GP).

140 ), respectively, in hippocampus and striatum/globus pallidus (GP).

141 ion of striatal channels at the level of the globus pallidus (GP).

142 c inhibition from the reciprocally connected globus pallidus (GP).

143 substantia nigra (SN), pulvinar thalami, and globus pallidus (GP).

144 th the external and internal segments of the globus pallidus (GPe and GPi, respectively).

145 he reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

146 he reciprocally connected GABAergic external globus pallidus (GPe) and glutamatergic subthalamic nucl

147 Neurons in the external segment of the globus pallidus (GPe) are autonomous pacemakers that are

148 ine-depleted rats indicate that the external globus pallidus (GPe) contains two main types of GABAerg

149 Neurons in the external globus pallidus (GPe) generate pacemaker activity that c

150 tal dopamine, the striatum, and the external globus pallidus (GPe) in regulating RLS-like movements,

151 cal excitation and indirect pathway external globus pallidus (GPe) inhibition of the STN are critical

152 The external globus pallidus (GPe) is a key contributor to motor supp

153 The external globus pallidus (GPe) is a key nucleus within basal gang

154 The external segment of the globus pallidus (GPe) is one of the core nuclei of the b

155 We show how external globus pallidus (GPe) neuron is sensitive to the phase o

156 analogous division of labor in the external globus pallidus (GPe) of Parkinsonian rats, showing that

157 the constituent neurons within the external globus pallidus (GPe) was not fully appreciated.

158 ated activity of the external segment of the globus pallidus (GPe), a downstream structure whose comp

159 s that pathological activity of the external globus pallidus (GPe), a nucleus in the basal ganglia, c

160 In the external segment of the globus pallidus (GPe), neuronal populations have been de

161 Focusing on the mouse external globus pallidus (GPe), we demonstrate that the potential

162 ural circuits in the external segment of the globus pallidus (GPe).

163 highest levels in the striatum and external globus pallidus (GPe).

164 ng in neurons of the external segment of the globus pallidus (GPe).

165 ic nucleus (STN) and the external segment of globus pallidus (GPe).

166 , and neurons in the external segment of the globus pallidus (GPe)], and one neuromodulator group [st

167 show that neurons in the habenula-projecting globus pallidus (GPh) in mice are essential for evaluati

168 uit, which regulates the habenula-projecting globus pallidus (GPh) neurons, exists within the basal g

169 n the subthalamic nucleus (STN) and internal globus pallidus (GPi) (P < 0.001), as well as in the dor

170 of the subthalamic nucleus (STN) or internal globus pallidus (GPi) reduces dyskinesias remain largely

171 ive visualization scores of STN and internal globus pallidus (GPi) were recorded by two neuroradiolog

172 drove network activity through the internal globus pallidus (GPi), external globus pallidus, motor c

173 deep brain stimulation (DBS) at the internal globus pallidus (GPi).

174 onal activity in the internal segment of the globus pallidus (GPi).

175 ebellum and from the internal segment of the globus pallidus (GPi).

176 , resulting in a heterogeneous distribution (globus pallidus > cortical regions > cerebellum) consist

177 ucleus accumbens, amygdala, caudate nucleus, globus pallidus, hippocampus, putamen, thalamus, and bra

178 the basal ganglia, the caudate, putamen and globus pallidus in brown capuchin monkeys (Cebus apella)

179 nucleus accumbens and frontal cortex but not globus pallidus in the l-DOPA group.

180 basal ganglia nuclei (caudate, putamen, and globus pallidus) in children with ADHD.

181 n metabolic rates including hyperactivity in globus pallidus indicative of basal ganglia involvement.

182 s reticulata (direct pathway) or the lateral globus pallidus (indirect pathway).

183 ance imaging of the brain revealed bilateral globus pallidus injury in the setting of dyskinetic CP.

184 re reward circuit regions including putamen, globus pallidus, insula and thalamus.

185 We report microelectrode data from the globus pallidus interna (GPi) and globus pallidus extern

186 ndomised to subthalamic nucleus (STN) versus globus pallidus interna (GPi) DBS surgery.

187 ood effects of subthalamic nucleus (STN) vs. globus pallidus interna (GPi) deep brain stimulation (DB

188 ut a previous case suggested that DBS of the globus pallidus interna (GPi) might.

189 ither the subthalamic nucleus (STN, n=84) or globus pallidus interna (GPi, n=80), using standardised

190 o had undergone bilateral stimulation of the globus pallidus interna (pallidal stimulation) or subtha

191 The basal ganglia, including the striatum, globus pallidus interna and externa (GPe), subthalamic n

192 The globus pallidus interna and the subthalamic nucleus are

193 n of Medtronic quadripolar electrodes in the globus pallidus interna bilaterally.

194 The results suggest anteromedial globus pallidus interna DBS for Tourette's syndrome is a

195 the effectiveness of DBS of the anteromedial globus pallidus interna on tic severity and common comor

196 ating that stimulation of an area called the globus pallidus interna partially reverses deficits in v

197 viously ignored structure in data describing globus pallidus interna responses to cortical stimulatio

198 ilateral dorsolateral prefrontal cortex, the globus pallidus interna, and the globus pallidus externa

199 ilateral dorsolateral prefrontal cortex, the globus pallidus interna, and the globus pallidus externa

200 from the dorsal pallidum, the homolog of the globus pallidus interna.

201 tal neurons projecting to the homolog of the globus pallidus interna/globus pallidus externa express

202 on in the dorsolateral prefrontal cortex and globus pallidus internal part in the picture sequencing

203 rded extracellular single unit discharges of globus pallidus internal segment (GPi) neurons in monkey

204 jor output station of the basal ganglia, the globus pallidus internal segment (GPi) projects to the t

205 ion in the basal ganglia output nucleus, the globus pallidus internal segment.

206 trating that deep brain stimulation (DBS) of globus pallidus internus (GPi DBS) and subthalamic nucle

207 assess the safety and efficacy of bilateral globus pallidus internus (GPi) DBS in patient's with sev

208 ients to either subthalamic nucleus (STN) or globus pallidus internus (GPi) deep brain stimulation (D

209 implicates the subthalamic nucleus (STN) and globus pallidus internus (GPi) in reward and punishment

210 corded the activity of single neurons in the globus pallidus internus (GPi), the primary BG output nu

211 in skeletomotor output region of the BG, the globus pallidus internus (GPi).

212 Deep brain stimulation of the globus pallidus internus alleviates involuntary movement

213 amma synchronization (ERS) recorded from the globus pallidus internus in patients undergoing deep bra

214 av1.8 expression in the amygdala, brainstem, globus pallidus, lateral and paraventricular hypothalamu

215 a unilateral excitotoxic injection into the globus pallidus leads to a gradual loss of dopaminergic

216 ucleus, and movement-related activity of the globus pallidus leads to transient beta oscillations dur

217 s dopamine cell death at 3 weeks following a globus pallidus lesion, but not thereafter.

218 for post-GBCA group [P < .001]) but not the globus pallidus (mean SI ratio for two-group comparison:

219 In the globus pallidus, medium spiny neurons connected strongly

220 mean uptake after BI; a relative increase in globus pallidus metabolism was evident in BI subjects wh

221 the internal globus pallidus (GPi), external globus pallidus, motor cortex, thalamus, or cerebellum.

222 shortening in the dentate nucleus (n = 13), globus pallidus (n = 13), substantia nigra (n = 13), pos

223 ation of the subthalamic nucleus (n = 60) or globus pallidus (n = 61).

224 The subthalamic nucleus-globus pallidus network is a potential source of oscilla

225 We show that most globus pallidus neurons, but very few neocortical intern

226 om the Shh-expressing domain eliminated most globus pallidus neurons, whereas most cortical and stria

227 cortical interneurons; and pattern C, mainly globus pallidus neurons.

228 recordings from the subthalamic nucleus and globus pallidus of five patients with Parkinson's diseas

229 nificantly elevated within the NAc shell and globus pallidus of ko mice, whereas serotonin transporte

230 sed local field in the caudate, putamen, and globus pallidus of patients relative to control subjects

231 activity occurs in the prefrontal cortex and globus pallidus of patients with nontremor-dominant PD c

232 onounced differences in substantia nigra and globus pallidus of PSP compared with control brains.

233 spectively, into the superior colliculus and globus pallidus of Sprague-Dawley rats.

234 tial component compared with inhibition from globus pallidus or substantia nigra pars reticulata.

235 s by stimulation of afferents from striatum, globus pallidus, or pars reticulata have been shown to b

236 " pathways that regulate the pallidal (e.g., globus pallidus) output nuclei involved in the control o

237 e gray matter of the whole brain (P < .001), globus pallidus (P = .002), dentate nucleus (P = .046),

238 ight caudate nucleus and ventral putamen and globus pallidus (P = .003) compared with that in control

239 years in the substantia nigra (P < .001) and globus pallidus (P = .035), which are both predictors of

240 caudate (P < 0.001), putamen (P < 0.001) and globus pallidus (P = 0.025) in patients with Parkinson's

241 ept for the caudate putamen (P=0.09) and the globus pallidus (P=0.12).

242 r DA release in the sensorimotor part of the globus pallidus pars interna (GPi) contralateral to the

243 e treated with deep brain stimulation of the globus pallidus pars interna during the period 1999-2010

244 rm efficacy of deep brain stimulation of the globus pallidus pars interna in the treatment of general

245 r predicting a better or poorer outcome from globus pallidus pars interna stimulation in this series

246 I's in the bilateral caudate (C) and putamen/globus pallidus (PGp) were studied based upon previous f

247 0A is upregulated in striatal projections to globus pallidus, preferentially expressing D2 receptors

248 e group differences in the substantia nigra, globus pallidus, pulvinar thalamus, thalamus, and caudat

249 and magnetic field correlation (MFC) in the globus pallidus, putamen, caudate nucleus, and thalamus

250 ional hedonic (orbitofrontal cortex, insula, globus pallidus, putamen, hippocampus, and amygdala) and

251 rally elevated (18) F-flortaucipir uptake in globus pallidus, putamen, subthalamic nucleus, midbrain,

252 s accumbens, amygdala, caudate, hippocampus, globus pallidus, putamen, thalamus, lateral ventricles.

253 y correlated with resting fMRI signal in the globus pallidus-putamen, extending previous findings of

254 0.005), putamen (r = -0.51; P = 0.025), and globus pallidus (r = -0.47; P = 0.030).

255 0.011), putamen (r = -0.48; P = 0.022), and globus pallidus (r = -0.70; P < 0.001).

256 ed that supplementary motor area and putamen/globus pallidus regions showed training-related decrease

257 f GPi from the internal capsule and external globus pallidus, respectively.

258 lly significant dose-dependent relationship (globus pallidus: rho = 0.90, P = .04).

259 < 0.001) metabolic reductions in the putamen/globus pallidus, sensorimotor cortex and cerebellar verm

260 cortex, amygdala, olfactory bulb, striatum, globus pallidus, septum, and nucleus basalis.

261 Projections to the globus pallidus showed solely D2-mediated presynaptic in

262 ei, including the subthalamic nucleus (STN), globus pallidus, striatum, and substantia nigra.

263 ding severe atrophy and neuronal loss in the globus pallidus, substantia nigra and subthalamic nucleu

264 erebral cortex, cerebellum, caudate/putamen, globus pallidus, substantia nigra, and hippocampus.

265 ojecting back to the same cortical areas via globus pallidus, substantia nigra, and thalamus.

266 aled discrete expression in caudate/putamen, globus pallidus, substantia nigra, cerebellar Purkinje c

267 severe degeneration in the cardinal nuclei - globus pallidus, subthalamic nucleus and substantia nigr

268 ped and successfully applied in the internal globus pallidus, subthalamic nucleus, and thalamus for t

269 -Fos(+) neurons in the external and internal globus pallidus, subthalamic nucleus, and ventral motor

270 m and instead receiving more inputs from the globus pallidus, subthalamic nucleus, and zona incerta.

271 ed in the sensorimotor cortex, striatum, and globus pallidus, support the hypothesis of a circuitwide

272 atients bilaterally in the ventral striatum, globus pallidus, thalamus, amygdala and right insula.

273 and right caudate nucleus (washing); putamen/globus pallidus, thalamus, and dorsal cortical areas (ch

274 creased susceptibility of the neurons of the globus pallidus, thalamus, and substantia nigra pars com

275 The SI in the globus pallidus, thalamus, dentate nucleus, and pons was

276 ead of the caudate nucleus (hCaud), putamen, globus pallidus, thalamus-and four cortical regions-rost

277 Globus pallidus-thalamus and dentate nucleus-pons SI rat

278 are distributed more caudally in the caudal globus pallidus than noncholinergic projection neurons.

279 density of direct pathway collaterals in the globus pallidus that bridge the direct pathway with the

280 hin the major striatal efferent targets: the globus pallidus, the entopeduncular nucleus, and the SN

281 in demonstrate the highest Mn content in the globus pallidus, the thalamus, and the substantia nigra

282 e stria terminalis; the caudate-putamen; the globus pallidus; the lateral septum; and the islands of

283 um spiny neurons successfully project to the globus pallidus, their usual developmental target, exten

284 ential values (from approximately 0.5 in the globus pallidus to approximately 3.5 in the insula) for

285 ly distinct projections through striatum and globus pallidus to EP targets within epithalamus and tha

286 discovered feedback projection from external globus pallidus to striatum is crucial for inhibitory co

287 reservation in vicinities of the cerebellum, globus pallidus, visual cortex, and parietal cortex in o

288 These findings provide evidence of smaller globus pallidus volume in patients with OCD without the

289 correlated significantly and positively with globus pallidus volume in the healthy volunteers but not

290 Patients had smaller globus pallidus volumes than healthy volunteers, but the

291 l functional connectivity within striatum or globus pallidus was relatively weak.

292 nervous system, specifically the retina and globus pallidus, we have generated a mouse knock-out of

293 he subthalamic nucleus, substantia nigra and globus pallidus were consistently the regions most sever

294 alamus and thalamic subnuclei, striatum, and globus pallidus were segmented using a fully automated m

295 Protein components of the globus pallidus were studied including glial fibrillary

296 ation, including the dorsal striatum and the globus pallidus, were also activated.

297 ith diabetes and MDD showed lower MTR in the globus pallidus when compared with the group with MDD.

298 ) with glucose metabolism in the putamen and globus pallidus, which receive projections from this str

299 ging showed iron in the substantia nigra and globus pallidus, with a 'halo' of T1 hyperintense signal

300 he amygdala, BNST, cingulate cortex, PVN and globus pallidus within 3 h after KA treatment and in the