ライフサイエンスコーパス: glomerular capillary

1 ration barrier and cover the outer aspect of glomerular capillaries.

2 igitating secondary extensions to enwrap the glomerular capillaries.

3 and the deposition of fibrin thrombi in the glomerular capillaries.

4 isruption under the high shear conditions in glomerular capillaries.

5 of these podocytes mimicked the formation of glomerular capillaries.

6 teoglycans, coats the luminal surface of the glomerular capillaries.

7 wing more and slow RAP fluctuations to reach glomerular capillaries.

8 g and detachment of endothelial cells of the glomerular capillaries.

9 he treated group, as was thrombosis of renal glomerular capillaries.

10 utrophils, which were similar in diameter to glomerular capillaries, abruptly arrested following anti

11 ant) rats showed splitting and thickening of glomerular capillaries and had a longer survival rate, c

12 in neutrophil recruitment and dwell time in glomerular capillaries and in reactive oxygen species (R

13 levels of all inflammatory markers, restored glomerular capillaries and podocyte structure, and arres

14 ctor (vWF), were identified predominantly in glomerular capillaries and rarely in arterioles, but not

15 fferentiated endothelial cells in developing glomerular capillaries and reduced lumen formation in vi

16 The kidney glomerular capillaries are frequent sites of immune comp

17 Cells of renin lineage residing alongside glomerular capillaries are reported to have progenitor c

18 e interstitial space and within the walls of glomerular capillaries as well as the cellular processes

19 results from immune complex precipitation in glomerular capillaries, as in some cryoglobulinaemic hum

20 merular volume, fractional mesangial volume, glomerular capillary basement membrane width, and urinar

21 uring formation of the renal glomerulus, the glomerular capillary becomes enveloped by highly special

22 The glomerular capillary bed seems to contribute to all subt

23 dothelium, and mesangium associated with the glomerular capillary bed to maintain filtration barrier

24 correlation was observed between retinal and glomerular capillary BM thickness (r=0.79, P=0.0001), be

25 chanism whereby mesangial cells organize the glomerular capillaries by adhering to the G domain of la

26 endothelial cells are cleared from immature glomerular capillaries by apoptosis, a process regulated

27 molecule in glomerular endothelial cells and glomerular capillary circumference.

28 lecule-1 in glomerular endothelial cells and glomerular capillary circumference.

29 In animals with renal failure, glomerular capillary collapse and tubular necrosis were

30 Human, but not murine, renal peritubular and glomerular capillaries constitutively express class II m

31 Physiologic permeability of the glomerular capillary depends on the normal structure of

32 e expression of growth factors that regulate glomerular capillary development and that abnormal capil

33 genesis of the kidney vasculature, including glomerular capillary development, arterial mural cell co

34 lium adjacent to the GBM, but convolution of glomerular capillaries did not occur.

35 r pressure, the latter eventually leading to glomerular capillary dropout (rarefaction) and further i

36 patterning, excess endothelial cells within glomerular capillaries, effaced podocytes with extremely

37 tients, viral inclusions were present in the glomerular capillary endothelia without any associated i

38 ne methyl ester (NMMA) increased TGF-beta in glomerular capillary endothelial cells (GCECs) and stimu

39 nchymal cells and on putative progenitors of glomerular capillary endothelial cells early in their re

40 Hg and led to severe loss of fenestration of glomerular capillary endothelial cells in both eNOS-defi

41 Tie2 was demonstrated on glomerular capillary endothelial cells, particularly on

42 ECRTP/DEP-1 is expressed in anticipation of glomerular capillary endothelial recruitment during deve

43 tic eNOS(-/-) mice, even though it inhibited glomerular capillary enlargement in both.

44 Normal glomerular capillaries filter plasma through a basement

45 y a pathway of neutrophil recruitment within glomerular capillaries following IgG deposition that may

46 (pod-Cre), which express cre at the time of glomerular capillary formation.

47 Detailed assessment of glomerular capillaries from diabetic D2 mice demonstrate

48 The glomerular capillaries function as the filtration barrie

49 dified serum proteins adversely affect renal glomerular capillary function, structure, and metabolism

50 elae of endothelial dysfunction in diabetes: glomerular capillary growth and effects on neighboring p

51 t protocol demonstrated greater reduction of glomerular capillary hydraulic pressure with OMA than wi

52 tionship between systemic blood pressure and glomerular capillary hydrostatic pressure and by decreas

53 Glomerular capillary hydrostatic pressure and hydrostati

54 iltration coefficient and an increase in the glomerular capillary hydrostatic pressure gradient.

55 Experimental studies showed that glomerular capillary hypertension and impaired sieving f

56 esangial cell stretching, mimicking in vitro glomerular capillary hypertension, enhances MCP-1 expres

57 rophy and glomerular hyperfiltration but not glomerular capillary hypertension.

58 abetes BM thickening develops in retinal and glomerular capillaries in a correlated manner.

59 cytes are epithelial cells that surround the glomerular capillaries in the kidney and are necessary f

60 showed accumulation of fibrin/fibrinogen in glomerular capillaries, increased numbers of glomerular

61 tification of key structural proteins in the glomerular capillary loop which may contribute to defect

62 duct, (2) the fleer mutation with distended glomerular capillary loops and cystic tubules, and (3) t

63 ed both histological TMA lesions (thrombi in glomerular capillary loops and small arteries, mesangiol

64 about the molecular changes occurring within glomerular capillary loops during development of disease

65 n mutant kidneys, the extent of branching of glomerular capillary loops was decreased, with capillary

66 around the circumference of human and mouse glomerular capillary loops, it co-localized only partial

67 ly increased APA staining in areas of intact glomerular capillary loops.

68 ction in vasculature density and dilation of glomerular capillary loops.

69 This study explored the question whether glomerular capillary lumen formation in vivo may involve

70 ious finding, that TGF-beta1 blockade blunts glomerular capillary lumen formation in vivo, it is prop

71 subendothelial lucency, platelet thrombi in glomerular capillary lumina).

72 eory analysis to explore the topology of the glomerular capillary network.

73 bloodstream and bind to proteoglycans in the glomerular capillaries of kidneys, where it can react wi

74 between GEC MMP-9 expression and changes in glomerular capillary permeability.

75 Glomerular capillary pressure (P(GC)) was also significa

76 olecular pathways that are activated by high glomerular capillary pressure and hyperglycemia and how

77 tes may be able to respond to changes in the glomerular capillary pressure and modulate the GFR.

78 in the kidney glomerulus that are exposed to glomerular capillary pressure and possible increases in

79 ated with glomerular capillary pressure, and glomerular capillary pressure no longer correlated with

80 lic blood pressure no longer correlated with glomerular capillary pressure, and glomerular capillary

81 ACEI reduced glomerular capillary pressure, increased glomerular ultr

82 ve agents affect systemic blood pressure and glomerular capillary pressure.

83 densa and (b) glomerular filtration rate or glomerular capillary pressure.

84 s due to a significantly increased number of glomerular capillary segments and capillary branch point

85 odocyte injury and depletion and collapse of glomerular capillary segments.

86 se glomerular endothelial cells and maintain glomerular capillary structure by mechanical and poorly

87 Glomerular capillary surface area remained stable, but t

88 se or focal, global or segmental collapse of glomerular capillaries, swelling and hypercellularity of

89 Endothelial cells of glomerular capillaries, the podocytes wrapped around the

90 in eNOS-/- mice, especially with respect to glomerular capillary thrombosis and neutrophil infiltrat

91 iated mesangial cell process invasion of the glomerular capillary tuft as an initiation mechanism of

92 order characterized by focal scarring of the glomerular capillary tuft, podocyte injury, and nephroti

93 duced mesangial cell process invasion of the glomerular capillary tuft.

94 yte catastrophe) and subsequent wrinkling of glomerular capillaries, tuft collapse, and periglomerula

95 Streptozotocin-induced diabetes increased glomerular capillary volume in both C57BL/6 and eNOS(-/-

96 In addition, they had substantial glomerular capillary wall deposits of IgG and C3, which

97 standing to emerge of how the 3 parts of the glomerular capillary wall jointly determine its function

98 circulation causes albuminuria by increasing glomerular capillary wall permeability and intraglomerul

99 glomerulitis showed ultrastructural signs of glomerular capillary wall remodeling.

100 the exact locations of Ang1 and Tie2 in the glomerular capillary wall.

101 e combined properties of the three layers of glomerular capillary wall: glomerular endothelial cells

102 deposits of immune complexes in situ in the glomerular capillary walls.

103 ng showed extensive linear C3 staining along glomerular capillary walls.

104 Odor-evoked functional hyperemia in glomerular capillaries was highly correlated with glutam

105 and positive C4d staining of peritubular or glomerular capillaries were present at the time of diagn

106 lomerulonephritis, NETs are generated in the glomerular capillaries, where they are short lived and m

107 trophils underwent prolonged interactions in glomerular capillaries, with the duration of these inter

108 Platelets accumulated in glomerular capillaries within 4 hours of TGN before evid