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1 t proinflammatory cytokine expression in rat glomerular mesangial cells.
2 up-regulate the expression of lipocalin-2 in glomerular mesangial cells.
3 the ability of collagen IV to interact with glomerular mesangial cells.
4 with proliferation and apoptosis of resident glomerular mesangial cells.
5 COL1A2 gene activation by TGF-beta1 in human glomerular mesangial cells.
6 phospholipase C-dependent, pathway in human glomerular mesangial cells.
7 al growth factor-induced Ca2+ entry in human glomerular mesangial cells.
8 ad signaling in collagen production by human glomerular mesangial cells.
9 cipate in the development and maintenance of glomerular mesangial cells.
10 cells have the ability to differentiate into glomerular mesangial cells.
11 ition and, H3 and H4- acetylation in primary glomerular mesangial cells.
12 ; also called capacitative calcium entry) in glomerular mesangial cells.
13 by platelet-derived growth factor (PDGF) in glomerular mesangial cells.
14 oncomitant release of nitric oxide (NO) from glomerular mesangial cells.
15 a concomitant release of prostaglandins from glomerular mesangial cells.
16 staglandins (PGs) and nitric oxide (NO) from glomerular mesangial cells.
17 on, bradykinin (BK) is mitogenic in cultured glomerular mesangial cells.
18 fic transactivator of MMP-2 transcription by glomerular mesangial cells.
19 duces a biphasic activation of p21ras in rat glomerular mesangial cells.
20 rix accumulation was explored in vitro using glomerular mesangial cells.
21 racellular matrix turnover, was evaluated in glomerular mesangial cells.
24 , we investigated the effects of 9-cis-RA on glomerular mesangial cell activation induced by transfor
25 iposomes were detected in the glomerulus and glomerular mesangial cells after tail vein injection in
26 ctivation and transient proliferation of the glomerular mesangial cells and by a prominent glomerular
27 regulates microRNA-192 (miR-192) in cultured glomerular mesangial cells and in glomeruli from diabeti
28 tubular epithelial cells (HKC-8) but not in glomerular mesangial cells and interstitial fibroblasts.
29 rate that GATA3 is specifically expressed in glomerular mesangial cells and plays a critical role in
30 g pathway is present and functional in human glomerular mesangial cells and plays a role in activatin
31 creases angiotensin II (AngII) levels in rat glomerular mesangial cells and that AngII mediates the i
32 stem cell is capable of differentiating into glomerular mesangial cells and that the process does not
33 to interact physically with NF-kappaB p65 in glomerular mesangial cells and thereby to inhibit NF-kap
35 We find that kidney beta8 is localized to glomerular mesangial cells, and expression is decreased
36 lipopolysaccharide treatment in vitro induce glomerular mesangial cell apoptosis by hitherto incomple
40 quires the removal of proliferating resident glomerular mesangial cells, but excessive loss of glomer
41 Here, we show that TGF-beta activates Akt in glomerular mesangial cells by inducing miR-200b and miR-
42 TNF-restricted M(phi)-directed apoptosis of glomerular mesangial cells can be down-regulated by M(ph
44 oth muscle actin is known to be expressed in glomerular mesangial cells exclusively in pathologic sit
45 itioned by several cell lines and found that glomerular mesangial cells exclusively secrete a factor
46 ERK) pathway was inhibited in HMC and in rat glomerular mesangial cells expressing the dominant-negat
48 hat TGF-beta1 induces autophagy and protects glomerular mesangial cells from undergoing apoptosis dur
52 COX-2 expression also has been described in glomerular mesangial cells (GMC), where COX-2 is not exp
57 ly decreased PEDF secretion in primary human glomerular mesangial cells (HMCs), suggesting that hyper
58 ized by in situ hybridization to vessels and glomerular mesangial cells in allogeneic and syngeneic (
59 scular smooth muscle cells and pericytes and glomerular mesangial cells in the kidney and that Tbx18-
60 tion in approximately 5000 gene promoters in glomerular mesangial cells, including those of Tgfb1, Tg
61 is study we examined the mechanisms by which glomerular mesangial cells ingest apoptotic cells and th
62 hysiologically activated human monocytes and glomerular mesangial cells (MC) by employing a cell cult
71 ) collagen gene (COL1A2) activation in human glomerular mesangial cells, potentially contributing to
77 a hyperglycemic-induced hypocontractility of glomerular mesangial cells that may be associated with d
78 -kappaB selectively sensitizes primary adult glomerular mesangial cells to TNF-induced apoptosis but
79 alpha8 integrin expressed on the surface of glomerular mesangial cells was selected as a target mole
81 might regulate HK activity and expression in glomerular mesangial cells, which constitute the princip
82 the G3YR mice showed degenerative changes in glomerular mesangial cells, which deteriorated progressi
83 f NF-kappaB on cytokine-induced apoptosis of glomerular mesangial cells, which is important in determ
84 eport we demonstrate that conditioning human glomerular mesangial cells with IL-1beta results in the
86 scular pericytes, liver fat-storing cells or glomerular mesangial cells, with IFN-gamma dramatically
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