ライフサイエンスコーパス: glomeruli

1 ed glomerular injury when MPO was planted in glomeruli.

2 nto TCs, but not MCs, that project to nearby glomeruli.

3 are subepithelial deposits were found in the glomeruli.

4 local interneurons within antennal lobe (AL) glomeruli.

5 and in particular to the false activation of glomeruli.

6 ient analysis of podocyte depletion in whole glomeruli.

7 ete cortical modules known as olfactory bulb glomeruli.

8 l before they form odorant receptor-specific glomeruli.

9 ey form spherical neuropil structures called glomeruli.

10 ial activity patterns of olfactory bulb (OB) glomeruli.

11 fied both from cultured podocytes and native glomeruli.

12 of odorant receptors (ORs) and antennal lobe glomeruli.

13 stinct central brain structures called optic glomeruli.

14 ts, and second the formation of anteriorized glomeruli.

15 rojection neurons and their associated optic glomeruli.

16 s was not spatially correlated with atubular glomeruli.

17 renal inflammatory milieu, localizing to the glomeruli.

18 and the loss of podocyte markers in isolated glomeruli.

19 he OB nerve layer and ultimately coalesce in glomeruli.

20 n infiltrating inflammatory cells and in the glomeruli.

21 ind the independent evolution of specialized glomeruli.

22 erperfusion and hypertrophy of the remaining glomeruli.

23 dney fibrosis and pro-angiogenic mediator in glomeruli.

24 ife, along with enlargement of the remaining glomeruli.

25 ing long lasting depolarization of olfactory glomeruli.

26 we made measurements of the positions of 352 glomeruli.

27 so in the number and volume of the olfactory glomeruli.

28 ndent and concludes as OSN axons coalesce in glomeruli.

29 uclear cell infiltrates, and reduced size of glomeruli.

30 rulus recruits GABAergic interneurons in all glomeruli.

31 lcholine receptors (mAChRs) are expressed in glomeruli.

32 eability function of isolated diabetic human glomeruli.

33 nduce combinatorial activity from several AL glomeruli.

34 a antigen, IgG, and C3b deposition in VL dog glomeruli.

35 ons and the formation of olfactory bulb (OB) glomeruli.

36 pulated with large and geometrically complex glomeruli.

37 determined in serial sections of individual glomeruli.

38 bular atrophy leads to formation of atubular glomeruli.

39 form immune complexes that accumulate in the glomeruli.

40 pressing endothelial cells (ECs) in affected glomeruli.

41 ) as the fourth most abundant protein in FGN glomeruli.

42 lly, manifests as crescent formation in most glomeruli.

43 ptic input from both temperature and dry-air glomeruli.

44 lized with a podocyte-specific marker in rat glomeruli.

45 egulating the position of T cells within the glomeruli.

46 ce synchronized activity of MCs at different glomeruli.

47 group (66.7% vs 49.8%, P = .001), with more glomeruli (23 vs 16, P = .014).

48 tubule samples (105 replicates in total) and glomeruli (5 replicates).

49 promising candidate miRNAs in microdissected glomeruli a confirmation set of 20 human transplant biop

50 metalloproteinase-12 started manifesting in glomeruli affected by early-stage lesions, whereas AT1 r

51 nse polarity mapped uniformly to discrete OB glomeruli, allowing us to analyze how inhibition shapes

52 h adjacent "hot," "cold," "dry," and "humid" glomeruli-an organization that allows for both unique an

53 29 years old had a mean 990,661 nonsclerotic glomeruli and 16,614 globally sclerotic glomeruli per ki

54 h electron microscopy showed deposits in the glomeruli and along tubular basement membranes.

55 anied by fewer deposits of fibrin within the glomeruli and by decreased peritubular inflammation.

56 In rat glomeruli and cultured human podocytes, KANK2 interacted

57 ation and activity by sulfenylation in mouse glomeruli and cultured podocytes.

58 ant C57BL/6J and DKD-susceptible DBA/2J (D2) glomeruli and demonstrated a significant downregulation

59 rophy and reabsorption of globally sclerotic glomeruli and hypertrophy of remaining nephrons.

60 RNA-sequencing (RNA-seq) analysis of kidney glomeruli and identified Tug1 as a differentially expres

61 ubstantial regenerative potential of injured glomeruli and identifying the oligomerization cycle of d

62 oreover, transmission electron microscopy of glomeruli and immunofluorescent staining of glomerular e

63 Both glomeruli and interstitium of patients with SVV strongly

64 racterized by predominant C3 deposits in the glomeruli and is commonly the result of acquired or gene

65 Laser microdissection of glomeruli and mass spectrometry of extracted peptides sh

66 amyloidosis with predominant involvement of glomeruli and medullary interstitium.

67 mperature stimulation can be observed in the glomeruli and mitral cells of the olfactory bulb, using

68 iched with H3K4me1 and H3K9/14ac in diabetic glomeruli and podocytes, which remained elevated despite

69 syndrome), then the disease rapidly affects glomeruli and progresses towards end stage renal failure

70 used laser-capture microdissection to obtain glomeruli and proximal tubules from 98 human needle kidn

71 s bound to native LAMP-2 purified from human glomeruli and recombinant hLAMP-2 expressed in ldlD cell

72 the number of T cells positioned inside the glomeruli and reduced inflammation.

73 essed proinflammatory cytokine production in glomeruli and reduced macrophage recruitment to the kidn

74 tion of the OB, reduced numbers of olfactory glomeruli and reduced OB-size without alterations in SVZ

75 posure increases the volume of the activated glomeruli and show that exposure increases M/TC number b

76 ot uniform, but rather heterogeneous, across glomeruli and stereotyped from animal to animal.

77 yed substantial preservation of podocytes in glomeruli and that TWEAK signaling directly damaged barr

78 omalous exponent depend on the size of model glomeruli and the degree of their wrapping.

79 Furthermore, the percentage of inflamed glomeruli and the number of intraglomerular monocytes sh

80 These glomeruli and the postsynaptic targets of OSNs, includin

81 asets were segmented to identify the labeled glomeruli and to assess glomerular positional variabilit

82 Glomeruli and triadic arrangements, characteristic featu

83 ecially NR1, NR2A, and NR2C, was observed in glomeruli and tubules of Akita mice.

84 n of autophagy and inflammasome genes within glomeruli and tubules.

85 correlation of HIF-target genes with eGFR in glomeruli and tubulointerstitium.

86 TrpM5-driven GFP marked overlapping sets of glomeruli and whether expression of these markers was co

87 The number of cores, glomeruli, and complications were reviewed in 431 CT-gui

88 NA antibodies targeting exposed chromatin in glomeruli, and iii) the impact of relative antibody avid

89 NK2, and KANK4 all localized to podocytes in glomeruli, and KANK1 partially colocalized with synaptop

90 including cystic and collapsing/degenerating glomeruli, and marked disruptions in podocyte arrangemen

91 ed 338 genes altered in diabetes-induced DKD glomeruli, and PLK2 exhibited the most dramatic change.

92 ibited dilated proximal tubules and immature glomeruli, and the renal proximal tubular epithelia lack

93 r cellularity, crescent formation, sclerotic glomeruli, and tubulointerstitial injury were significan

94 ge, the number of WT-1-positive cells in the glomeruli, and WT-1 gene expression.

95 performance using just a small subset of the glomeruli ( approximately 15).

96 Glomeruli are highly sophisticated filters and glomerula

97 ivated by odors, and certain combinations of glomeruli are over-represented.

98 Olfactory bulb glomeruli are regions of neuropil that contain input and

99 Glomeruli are richly interconnected by short axon cells

100 In the olfactory bulb (OB), glomeruli are the functional units for odor information

101 The positions of OR-specific glomeruli are traditionally described as stereotyped.

102 (ORN) axons that target a left-right pair of glomeruli, as well as all the projection neurons (PNs) p

103 d adequate if the biopsy yielded at least 10 glomeruli at light microscopy, one glomerulus at immunof

104 dendrites of mitral and tufted cells inside glomeruli at the first stage of olfactory processing.

105 In these glomeruli, at least two of the postsynaptic dendrites or

106 Mer is constitutively expressed in the glomeruli; Axl expression is inducible in glomeruli unde

107 lated with lower PFKFB2 expression in kidney glomeruli (beta = 0.87, P = 0.03).

108 table dislocation and a distortion of the AL glomeruli between the in vitro and in vivo brains.

109 dult mice, we detected significantly smaller glomeruli, but it did not affect glomerular permselectiv

110 We verified the identity of 63% of AL glomeruli by mapping the projections of 34 GAL4-lines of

111 ere clearance preserved the functionality of glomeruli, cardio-protective KATP channels and adipocyte

112 osition relative to four identified landmark glomeruli, close to the entrance of the antennal nerve.

113 n the periphery map precisely onto olfactory glomeruli ("coding channels") in the brain.

114 Here we investigated how glomeruli combine to control behavior in freely walking

115 VB and B6, using RNA microarrays of isolated glomeruli combined with proteomic glomerular ECM analyse

116 In conclusion, large adult glomeruli contained more podocytes than small glomeruli

117 Adult glomeruli contained significantly more cells than glomer

118 icroscopy revealed that approximately 20% of glomeruli contained structures composed of extracellular

119 from DIC-positive kidneys when the extent of glomeruli containing fibrin thrombi is less than 50% and

120 ransplant renal biopsies showing 100% of the glomeruli containing fibrin thrombi.

121 intraglomerular compartment (IGC), with more glomeruli containing RFP(+)CoRL and, within these glomer

122 We find that positional variability of glomeruli correlates with the OR: For instance, the medi

123 ts due to their association with progressive glomeruli damage in disease states.

124 ldest age groups, the number of nonsclerotic glomeruli decreased by 48%, whereas cortical volume decr

125 an increasing proportion of globally scarred glomeruli, decreasing renal function, and exponentially

126 wed that the DNAJB9 protein deposited in FGN glomeruli did not have any major sequence or structural

127 interneurons are recruited nonspecifically, glomeruli differ dramatically in their sensitivity to in

128 Compared with control glomeruli, DN, FSGS, IgAN, and MPGN glomeruli exhibited

129 The increased number of podocytes in large glomeruli does not match the increase in glomerular size

130 ors on the periostin promoter is enriched in glomeruli during experimental GN.

131 Time-course microarray analysis of glomeruli during nephrotoxic serum nephritis revealed si

132 system is divided into processing channels (glomeruli), each receiving input from a different type o

133 es with Young's modulus near that of healthy glomeruli elicit a pro-differentiation and maturation re

134 ach mitral cell receives input from multiple glomeruli, enables integration of chemosensory stimuli o

135 ctivation of sensory afferents within single glomeruli evoked diverse modes of granule cell activity,

136 Histone toxicity on glomeruli ex vivo was Toll-like receptor 2/4 dependent,

137 Compared with amyloidosis glomeruli, FGN glomeruli exhibited a >6-fold overexpression of DNAJB9 p

138 control glomeruli, DN, FSGS, IgAN, and MPGN glomeruli exhibited differential expression of 18, 12, t

139 ompared with control or FSGS glomeruli, IgAN glomeruli exhibited downregulated expression of hsa-miR-

140 Compared with amyloidosis glomeruli, FGN glomeruli exhibited a >6-fold overexpress

141 correlations with the proportion of scarred glomeruli five weeks after induction of injury.

142 are processed within specific antennal lobe glomeruli following a specialized labeled-line system.

143 n vitro processing, and to reliably identify glomeruli for in vivo applications, we generated a trans

144 lfactory bulb neurons in the vicinity of the glomeruli formed by axons of Gucy1b2+ sensory neurons.

145 (PN) classes precisely target to 50 discrete glomeruli, forming parallel information-processing pathw

146 most of the approximately 500 antennal lobe glomeruli found in wild-type ants.

147 83 podocytes per 10(6) microm(3)) than small glomeruli from adults and children (932 podocytes per 10

148 An ultrastructural evaluation of glomeruli from animals with VL identified mesangial cell

149 lomeruli contained more podocytes than small glomeruli from children and adults, raising questions ab

150 Glomeruli from children were small and contained 452 pod

151 ruli contained significantly more cells than glomeruli from children, including 558 podocytes (IQR=43

152 gulation of essential mitochondrial genes in glomeruli from diabetic D2 mice, but not in C57BL/6J, wi

153 ain 3 puncta indicative of autophagosomes in glomeruli from dogs with clinical VL and renal failure.

154 We isolated glomeruli from formalin-fixed and paraffin-embedded biop

155 vel of KLF15 expression in the podocytes and glomeruli from human biopsy specimens correlated with gl

156 Podocytes in glomeruli from humans with focal segmental glomeruloscle

157 rotective factors for DN using proteomics on glomeruli from individuals with extreme duration of diab

158 Moreover, microdissected glomeruli from patients with small vessel vasculitis (SV

159 followed by high-throughput sequencing with glomeruli from wild-type mice.

160 role of Ang II/TRPC6 axis in the control of glomeruli function, which is likely important for the de

161 Crosstalk between co-active glomeruli has been proposed to perform a variety of comp

162 glomerular cells from magnetic bead-purified glomeruli have identified glomerulus-infiltrating leukoc

163 Specifically, compared with control or FSGS glomeruli, IgAN glomeruli exhibited downregulated expres

164 , altered serum reactivity to DWEYS, reduced glomeruli IgG deposition, preserved kidney histology, an

165 arning, we modelled local computation within glomeruli in antennal lobes with axons of projection neu

166 bably decaying from a larger pool of smaller glomeruli in early adult life, along with enlargement of

167 Here, we counted all individual glomeruli in murine kidneys and sized the capillary tuft

168 We analyzed the proteomic content of glomeruli in patient biopsy specimens and detected DnaJ

169 de novo expression of connexin 43 in damaged glomeruli in patients with glomerular diseases as well a

170 med quantitative ultrastructural analyses of glomeruli in rat olfactory bulb under conditions in whic

171 Glomeruli in small brains are, however, smaller in both

172 ing microscopy to compare size and number of glomeruli in the antennal lobe in the brain, and scannin

173 nae is tightly correlated with the number of glomeruli in the antennal lobe region innervated by odor

174 nd large wasps also have a similar number of glomeruli in the antennal lobe.

175 esponding expansion of a specific cluster of glomeruli in the antennal lobe.

176 projecting from the lobula to discrete optic glomeruli in the central brain group these sets of figur

177 precisely organized and sorted out onto 1800 glomeruli in the OB, from which the olfactory informatio

178 Odors elicit distributed activation of glomeruli in the olfactory bulb (OB).

179 scence of OSN axons into approximately 3,600 glomeruli in the olfactory bulb.

180 eceptor (OR) gene coalesce into one or a few glomeruli in the olfactory bulb.

181 ty required for their assembly into distinct glomeruli in the olfactory bulb.

182 voked OSN synaptic output into olfactory bub glomeruli in unmanipulated (gonad-intact) adult mice fro

183 characterization of these antigens in human glomeruli in vivo remains inconsistent.

184 patterns of suppression in only a subset of glomeruli in which such suppression could be detected, a

185 ina antennal lobe contains approximately 265 glomeruli (in females), grouped in nine conspicuous clus

186 Cav2.2 was localized in glomeruli including podocytes and in distal tubular cell

187 l changes, but also ameliorated podocyte and glomeruli injury in diabetic mice, which were associated

188 n 'non-sister' MCs affiliated with different glomeruli (interglomerular synchrony).

189 The total number of glomeruli is a fundamental parameter of kidney function

190 hat activation of the Notch3 receptor in the glomeruli is a turning point inducing phenotypic changes

191 e level of interneuron activity in different glomeruli is largely odor invariant.

192 d increased expression of wild-type TRPC6 in glomeruli is observed in several human acquired proteinu

193 dor information coding, but inhibition among glomeruli is poorly characterized.

194 eutrophil localization, deposited MPO within glomeruli is recognized by autoreactive T cells that con

195 estion of whether lateral inhibition between glomeruli is specific or nonspecific.

196 rphological subunits of the AL-the olfactory glomeruli-is usually done using in vitro AL atlases.

197 Podocyte outgrowth from glomeruli isolated from wild-type mice during the early

198 l three-dimensional tissue models containing glomeruli-like structures.

199 d gamma), OSN axons fail to converge to form glomeruli, likely owing to contact-mediated repulsion be

200 In a subset of these glomeruli, mass podocyte detachment events occurred in a

201 sclerosis and/or collapse of juxtamedullary glomeruli, microcystic tubular dilation, and tubulointer

202 Lateral circuit processing among activated glomeruli modulates olfactory signal transformation befo

203 ruli containing RFP(+)CoRL and, within these glomeruli, more RFP(+)CoRL.

204 To estimate the number of functioning glomeruli (NFG), we divided the whole-kidney ultrafiltra

205 nd that the protocerebral bridge contains 18 glomeruli, not 16, as previously believed.

206 A morphometric analysis of glomeruli obtained during nephrectomy was performed in 1

207 levels are also significantly reduced in the glomeruli of albuminuric BTBR ob/ob mice, indicating its

208 al growth factor (VEGF) signaling within the glomeruli of Alport mice is strongly elevated early on i

209 nd SREBP-1-dependent effects were induced in glomeruli of angiotensin II-infused mice, and administra

210 uction of MCP-1 and IL-6 was observed in the glomeruli of C/EBP-alpha knockout mice and was associate

211 stress and reduced NAPDH oxidase 4 (NOX4) in glomeruli of diabetic eNOS(-/-) mice.

212 were also significantly up-regulated in the glomeruli of diabetic patients and mice, suggesting indu

213 lomeruli of Hmox1(-/-) rats and augmented in glomeruli of GEC(HO-1) rats.

214 lomeruli of Hmox1(-/-) rats and augmented in glomeruli of GEC(HO-1) rats.

215 Hyaluronan staining in the glomeruli of heparin-treated diabetic rats was very high

216 onstitutively expressed DAF was decreased in glomeruli of Hmox1(-/-) rats and augmented in glomeruli

217 This effect was attenuated in glomeruli of Hmox1(-/-) rats and augmented in glomeruli

218 We compared KMO abundance in the glomeruli of mice and humans under normal and diabetic c

219 f the disease was higher than outgrowth from glomeruli of mice lacking Notch3.

220 (lnc-MGC) are coordinately increased in the glomeruli of mouse models of DN, and mesangial cells tre

221 o observed that CDK2 is downregulated in the glomeruli of obese Zucker rats before the onset of prote

222 MIF, CD74, and CD44 were upregulated in the glomeruli of patients and mice with proliferative glomer

223 n of KIBRA and phosphorylated YAP protein in glomeruli of patients with biopsy-proven focal segmental

224 ellular traps (NETs) have been documented in glomeruli of patients with glomerulonephritis.

225 t miR-146a expression levels decrease in the glomeruli of patients with type 2 diabetes (T2D), which

226 pecifically blocks TRPC5 channel activity in glomeruli of proteinuric rats.

227 as increased VWF deposition in C1q-positive glomeruli of SLE patients compared with control nephropa

228 ed expression and activation of Stat3 in the glomeruli of Stat1-KO host mice but not WT mice with cGV

229 l degradation of HIPK2, was decreased in the glomeruli of streptozotocin injected diabetic mice.

230 Renal glomeruli of the G3YR mice had significantly reduced amo

231 ly bound to structures within the developing glomeruli of the kidney, which express CS-E.

232 rs that establish excitatory synapses within glomeruli of the olfactory bulb.

233 ession and phosphorylation were decreased in glomeruli of type 1 diabetic Akita mice (Ins2(+/C96Y)) c

234 In glomeruli of wild-type rats, the natural Hmox substrate,

235 of cleaved caspase 3 and Bcl2 expression in glomeruli of WT diabetic mice.

236 16% and the proportion of globally sclerotic glomeruli on biopsy increased by only 15%.

237 in all patients with FGN but not in healthy glomeruli or in 19 types of non-FGN glomerular diseases.

238 dneys from alpha7(-/-) mice displayed severe glomeruli (P < 0.0001) and tubulointerstitial lesions (P

239 in FVB/N Cd151 (-/-) compared to Cd151 (+/+) glomeruli (p < 0.05).

240 gressively decreased to 520,410 nonsclerotic glomeruli per kidney and increased to 141,714 globally s

241 and increased to 141,714 globally sclerotic glomeruli per kidney in donors 70-75 years old.

242 otic glomeruli and 16,614 globally sclerotic glomeruli per kidney, which progressively decreased to 5

243 average loss of 8834 (P=0.03, sex adjusted) glomeruli per single kidney over the first 38 years of a

244 ctin, C1q, IgM, and C5b-9 were scored in the glomeruli, peritubular capillaries, and arterioles.

245 In normal glomeruli, podocyte survival is mediated via nephrin-dep

246 in situ hybridization detected APOL1 mRNA in glomeruli (podocytes and endothelial cells) and tubules,

247 Although uninflamed glomeruli rarely contained DCs, injury with nephrotoxic

248 al connections that mediate cross talk among glomeruli, releasing GABA and DA onto sensory nerve term

249 Ubiquitylomic analysis of mouse glomeruli revealed that podocin is ubiquitylated at two

250 rformed on podocytes of the freshly isolated glomeruli showed enhanced basal TRPC channel activity in

251 However, large adult glomeruli showed markedly lower podocyte density (183 po

252 the tubule compartment and 34 transcripts in glomeruli showed statistically significant correlation w

253 tricular injected AFSC that homed within the glomeruli showed strong modulation of the VEGF activity,

254 Analysis of the murine glomeruli showed that LPS administration reduced the lev

255 Studies in isolated glomeruli showed that treatment reduced inflammation and

256 phin-3 (NT-3) project to defined clusters of glomeruli situated ventrally in the main olfactory bulb.

257 hip between how odors are represented at the glomeruli stage, which corresponds to a compression, and

258 This may render hypertrophic glomeruli susceptible to pathology.

259 , we then determined the total volume of the glomeruli (TGV) formed by coalescence of the fluorescent

260 ifferentiated epithelial cells in the kidney glomeruli that act as a key component of the glomerular

261 from sparse and stereotyped combinations of glomeruli that are coactivated by odors, and certain com

262 ch for assessing podocyte depletion in whole glomeruli that combines immunofluorescence, optical clea

263 ervations in the AL and the SEZ, identifying glomeruli that may respond to human body odours or carbo

264 e promoters in mesangial cells as well as in glomeruli that were purified from type I and type II dia

265 coded by combinatorial patterns of activated glomeruli, the initial signal transformation site of the

266 the basal forebrain innervate olfactory bulb glomeruli, the initial site of synaptic integration in t

267 of both nonsclerotic and globally sclerotic glomeruli; the total number of glomeruli was estimated f

268 be in ORNs or PNs causes overwrapping of the glomeruli their axons or dendrites target.

269 oids that possess renoprotective activity in glomeruli through their interaction with the glucocortic

270 modynamically mediated processes that damage glomeruli to cause progression.

271 s the response intensity range of individual glomeruli to increasing concentration making them more s

272 we imaged responses of mouse olfactory bulb glomeruli to mixtures.

273 ugh panglomerular inhibition, or allows some glomeruli to respond to faint aversive odors in the pres

274 s to a compression, and the connections from glomeruli to third-order neurons (neurons in the olfacto

275 The predicted connectivity rate from glomeruli to third-order neurons can be tested experimen

276 he glomeruli; Axl expression is inducible in glomeruli under inflammatory conditions.

277 onocytes patrol both uninflamed and inflamed glomeruli using beta2 and alpha4 integrins and CX3CR1.

278 his gap, we studied serial sections of three glomeruli using electron microscopy.

279 quantitatively the positions of OR-specific glomeruli using serial two-photon tomography, an automat

280 In the majority of glomeruli, vacuolized parietal epithelial cells attached

281 ed amyloidosis with extensive involvement of glomeruli, vessels, and medulla.

282 ent of proximal tubular atrophy and atubular glomeruli was determined in serial sections of individua

283 lly sclerotic glomeruli; the total number of glomeruli was estimated from cortical volumexglomerular

284 ffness spanning that of healthy and diseased glomeruli, we demonstrate that kidney podocytes show mar

285 d scaled in strength with excitation as more glomeruli were activated.

286 Glomeruli were microdissected from biopsy samples of 20

287 emnants between damaged tubules and atubular glomeruli were observed.

288 pendently associated with fewer nonsclerotic glomeruli were older age, shorter height, family history

289 ould be detected, and excited and suppressed glomeruli were spatially intermingled.

290 sies, Nox5 was identified to be expressed in glomeruli, which appeared to be increased in diabetes.

291 a surprising dorsal shift of the M71 and M72 glomeruli, which often fuse with their contralateral cou

292 ated synaptic clusters related to individual glomeruli, which we call glomerular units.

293 hesion genes was elevated in Mecp2-deficient glomeruli, while acute odor stimulation in control mice

294 asses target dendrites to distinct olfactory glomeruli, while PNs of the same class exhibit indisting

295 In glomeruli with advanced lesions, AT1 receptor expression

296 material accumulated in the Bowman space of glomeruli with low podocyte density.

297 les, the Gsalpha-deficient mice had enlarged glomeruli with mesangial expansion, injury, and FSGS at

298 or-evoked responses were readily recorded in glomeruli with reduced spontaneous afferent activity, al

299 irst time the presence of sexually dimorphic glomeruli within a distinct macroglomerular complex (MGC

300 tion in the main olfactory epithelium and in glomeruli within the olfactory bulb.