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   1 re interstitial inflammation, tubulitis, and glomerulitis.                                           
     2 or tubulitis, interstitial inflammation, and glomerulitis.                                           
     3 less numerous CD3+ cells was found in TG and glomerulitis.                                           
     4 ar to but not higher than cases of g2 and g3 glomerulitis.                                           
     5 trophils in PTC, 65% versus 9%; neutrophilic glomerulitis, 55% versus 4%; neutrophilic tubulitis, 55%
     6  associated with capillaritis in the biopsy (glomerulitis, 6.1% vs. 32%, P=0.003; peritubular capilla
     7 at progressed to rejection had more frequent glomerulitis (7 of 18 versus 3 of 47, P = 0.003) and Ban
     8 lls, predominantly macrophages manifested as glomerulitis and capillaritis.Throughout the course of A
  
  
    11 circulation inflammation was prevalent, with glomerulitis and peritubular capillaritis found in 60.0%
    12  to assess the reproducibility of transplant glomerulitis and to prospectively investigate the pathog
  
    14 ) infiltrate, tubulitis, endothelialitis and glomerulitis, and anti-donor CTL reactivity in vitro.   
    15 transplant glomerulopathy lesions, and lower glomerulitis, but similar levels of peritubular capillar
    16 Inter-observer reproducibility of transplant glomerulitis can be improved by using more stringent his
  
    18 inflammation (MI; defined by the addition of glomerulitis (g) and peritubular capillaritis (ptc) scor
  
    20 nt in mean scores for acute Banff components glomerulitis (g), C4d, g+ peritubular capillaritis (ptc)
    21 tudied, comparing one group with significant glomerulitis (G, n=28) with those with no glomerulitis (
  
  
  
  
  
  
    28  Microcirculation inflammation, particularly glomerulitis, irrespective of C4d, is associated with a 
    29  Microcirculation inflammation, particularly glomerulitis, irrespective of C4d, is associated with a 
  
  
  
    33 and chronic antibody-mediated rejection with glomerulitis, microthrombosis, microaneurysms, glomerula
  
    35 flamed (borderline changes or above) without glomerulitis (n=21), and transplant glomerulitis (n=18).
  
  
  
    39 teritis (OR=0.5, 95% CI=0.2-1.2, P=0.11) and glomerulitis (OR=0.9, 95% CI=0.4-2.1, P=0.8) were not.  
    40 ity, as well as microvascular injury scores (glomerulitis + peritubular capillaritis), were less in t
    41 rrent ABMR criteria, including capillaritis, glomerulitis, peritubular capillary C4d deposition, and 
    42 y-mediated changes with significantly higher glomerulitis scores and numerically higher C4d staining 
  
  
    45 ction (OR=4.9, 95% CI=1.1-20.8, P=0.03), and glomerulitis was associated with the development of tran
  
  
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