1 riuresis accompanied by blockade of proximal
glomerulotubular balance and TGF characterizes a new cla
2 response by changes in FSS may contribute to
glomerulotubular balance in vivo.
3 That is, the
glomerulotubular balance response in the loop of Henle i
4 e, but fractional reabsorption and therefore
glomerulotubular balance were not distinguishable.
5 This "
glomerulotubular balance" derives from flow dependence o
6 The tubules respond to glomeruli with
glomerulotubular balance, whereas glomeruli respond to t
7 glomerular filtration rate (GFR) to maintain
glomerulotubular balance.
8 mal reabsorption (Jprox) from changes due to
glomerulotubular balance.
9 on are directly coupled, a phenomenon called
glomerulotubular balance.
10 abetic rats too great to be accounted for by
glomerulotubular balance.
11 ular fluid shear stress, serving to maintain
glomerulotubular balance.
12 n morphologic recognition, it is likely that
glomerulotubular disconnection is an underappreciated me
13 ge we term the "uncapped glomerulus lesion."
Glomerulotubular disconnection was confirmed, but connec
14 ular junction are described as precursors of
glomerulotubular disconnection.
15 al nephrons can be lost through a process of
glomerulotubular disconnection.
16 ngiotensin II levels, renal hypertrophy, and
glomerulotubular fibrosis in Akita hnRNP F-Tg mice.
17 Thus, glomerular adhesions and
glomerulotubular junction abnormalities help to explain
18 Thus, AG and
glomerulotubular junction abnormalities may be important
19 Glomerulotubular junction abnormalities were examined in
20 Glomerulotubular junction abnormalities were found in 2%
21 Glomerulotubular junction abnormalities, frequent in pro
22 % of glomeruli from proteinuric patients had
glomerulotubular junction abnormalities, including five
23 er understanding of the vulnerability of the
glomerulotubular junction and its protection from injury
24 Integrity of the
glomerulotubular junction and proximal tubular mass were
25 At the nephron level, lesions started at the
glomerulotubular junction and then extended distally.
26 Narrowing or other abnormalities of the
glomerulotubular junction are described as precursors of
27 omerular diseases, ascribed to injury to the
glomerulotubular junction as a result of proteinuria; ho
28 atrophic and 25% of glomeruli were atubular;
glomerulotubular junction integrity decreased with incre
29 glomeruli, suggesting susceptibility of the
glomerulotubular junction to toxic injury.
30 se linear regression models with glomerular,
glomerulotubular junction, and interstitial parameters a
31 ramatic example of progressive injury to the
glomerulotubular junction, with formation of the "swan-n
32 uction of proximal convoluted tubules at the
glomerulotubular junction.
33 Glomerulotubular junctions were examined on serial secti
34 In cpk mice, all
glomerulotubular junctions were normal at 10 days, but b
35 In pcy mice, most
glomerulotubular junctions were normal at 20 weeks, but
36 were serially sectioned to find and classify
glomerulotubular junctions.
37 cal parenchymal zones, with narrowing of the
glomerulotubular "
neck." In summary, targeted deletion o