ライフサイエンスコーパス: glomus

1 os fated to form its vascular structure, the glomus.

2 main protein, is expressed in the pronephric glomus.

3 phros, the tubules and the duct, but not the glomus.

4 ching of arbuscular mycorrhizal fungi of the Glomus and Gigaspora spp., and they promote rhizobial sy

5 the size-exclusion barrier of the glomerulus/glomus and recruit mesangial and endothelial cells to fo

6 -anterior pronephros anlagen, permitting the glomus and tubules to develop in isolation.

7 mour-1 gene, xWT1, a marker specific for the glomus at the stages analysed, together with other mesod

8 Unlike the pronephric tubules, the glomus can also be induced by FGF and RA.

9 metabolic model postulates that the rise in glomus cell [Ca2+]i, the initiating reaction in the sign

10 ntified acetate (which is known to affect CB glomus cell activity) as an agonist for the most highly

11 physiologically or pharmacologically induced glomus cell depolarization or hyperpolarization may not

12 cell RNA-Seq results characterized novel CB glomus cell genes, including members of the G protein-co

13 a2+]i seemed to play a significant a role in glomus cell intercellular communication.

14 7BL/6J) strain measuring the ventilatory and glomus cell intracellular calcium ([Ca(2+)](i)) response

15 s fully consistent with release of Ca2+ from glomus cell intracellular stores according to metabolic

16 in expression (Kv3.4 versus Kv4.3) in the CB glomus cell may contribute to the suppression of IK and

17 Accordingly, K(+) channel inhibition of the glomus cell membrane is expected to be followed by excit

18 s Ca2+-activated K+ (K+(Ca)) currents in the glomus cell of neonatal rat carotid body.

19 h bilateral carotid body resections (BR) for glomus cell tumours.

20 gs and glomus cells was more complex, When a glomus cell was stimulated, current spread to the nerve

21 elease of an excitatory transmitter from the glomus cell, which is a secretory cell that is presynapt

22 forming next-generation sequencing on single glomus cell-derived cDNAs to eliminate contamination of

23 essed in glomus cells, which contained novel glomus cell-specific genes.

24 and resting potential (Em) of cultured mouse glomus cells (clustered and isolated) were simultaneousl

25 In the case of glomus cells (GC/GC coupling), it was mostly resistive a

26 osed of the neurotransmitter (NT)-containing glomus cells (GCs) and the sensory afferent fibers synap

27 ammasome and interleukin-1beta expression in glomus cells (p < 0.01).

28 e excitability of afferent nerve endings and glomus cells (putative chemoreceptor cells).

29 Short-term cultures of glomus cells (up to seven days), were employed to study

30 Release of transmitter from glomus cells activates the sensory afferent fibers to tr

31 Carotid body glomus cells also expressed IL-1 receptor and responded

32 Isolated carotid body glomus cells also sense glucose, and animal studies have

33 taneous stimulation and recording of coupled glomus cells and carotid nerve endings.

34 Glomus cells and carotid sinus afferents are anatomicall

35 uring Na2S2O4 occur by direct effects on the glomus cells and feedback action through released ACh an

36 -like immunoreactivity was localized to many glomus cells and nerve fibers and the concentration of S

37 stablished, the presence of TRPC channels in glomus cells and sensory nerves of the carotid body sugg

38 Glomus cells and sheath cells were immunocytochemically

39 rk, we quantify functional differences among glomus cells and show reciprocal sensitivity to acidosis

40 ly connected, and the chemical events in the glomus cells are expected to be conveyed reflexly as aff

41 Glomus cells are present in normal numbers and appear st

42 Carotid body glomus cells are the primary sites of chemotransduction

43 not affect the basal [Ca(2+) ]i in isolated glomus cells bathed in 5 mm KClo , but elicited transien

44 hus, hypoxia may suppress the K+ currents in glomus cells but K+ current suppression of itself does n

45 ate that this selective chemotransduction of glomus cells by either stimulus may result in the activa

46 und K(+) channels that mediate activation of glomus cells by hypoxia.

47 changes in arterial O(2) with activation of glomus cells by inhibition of unidentified background K(

48 est the redox inhibition of K(+) channels of glomus cells by reduced glutathione (GSH), dithiothreito

49 our results show that BK/Kv are activated as glomus cells depolarize in response to hypoxia, which th

50 from intracellular store in the carotid body glomus cells during hypoxia, we stimultaneously measured

51 lux both contribute to the depolarization of glomus cells during moderate to severe hypoxia.

52 o, 4-AP did not evoke any rise in [Ca2+]i in glomus cells either during normoxia and hypoxia, althoug

53 Chemosensitive carotid body glomus cells exhibited toll-like receptor (TLR-2 and TLR

54 Cultured glomus cells expressed immunoreactivity for alpha3, alph

55 morphology and size of these cells resemble glomus cells found in amphibians, mammals, tortoises, an

56 ve O2-sensing cells were similar to those of glomus cells found in other vertebrates.

57 Experiments were performed on glomus cells from adult rat carotid bodies, rat pheochro

58 r normoxic conditions were blunted in the CB glomus cells from CHF rabbits compared with sham rabbits

59 tage-gated K+ (Kv) channels to hypoxia in CB glomus cells from CHF rabbits, and whether endogenous an

60 sensitivity of Kv channels to hypoxia in CB glomus cells from CHF rabbits; (2) high concentrations o

61 e NO donor SNAP (100 microm) increased IK in glomus cells from HF rabbits to a greater extent than th

62 results demonstrate that IK is reduced in CB glomus cells from HF rabbits.

63 Pairs of electrically coupled glomus cells from rat carotid bodies were impaled with m

64 Carotid body (CB) glomus cells from rat express a TASK-like background K+

65 of Ang II (> 1 nM) directly inhibit IK in CB glomus cells from sham and CHF rabbits; (3) changes in K

66 ker iberiotoxin (IbTx, 100 nm) reduced IK in glomus cells from sham rabbits, but had no effect on IK

67 the pH sensitivity of isolated carotid body glomus cells from young spontaneously hypertensive rats

68 Glomus cells had less negative Em and lower Ro.

69 Glomus cells harvested from Wistar rat carotid bodies we

70 Although carotid chemosensitive glomus cells have been the most extensively studied from

71 we compared the outward K+ currents (IK) of glomus cells in sham rabbits with that in HF rabbits and

72 Glomus cells in the carotid body respond to decreases in

73 sible for the oxygen-sensitive properties of glomus cells in the rat carotid body (CB) we used Ba2+,

74 [Ca2+]i in glomus cells increased in response to hypoxia (pO2 = 35

75 cular machinery and signalling pathway in CB glomus cells is still limited.

76 ody pH sensing by recording the responses of glomus cells isolated from rat carotid body to rapid cha

77 Carotid body (CB) glomus cells mediate acute oxygen sensing and the initia

78 ed cells and strings of cells, but clustered glomus cells never responded.

79 550 Torr) on the pHi and [Ca2+]i in cultured glomus cells of adult rat carotid body (CB) as a test of

80 IK was attenuated in glomus cells of HF rabbits, and their resting membrane p

81 g cells of fish (5HT) and those found in the glomus cells of mammals (acetylcholine, adenosine, and c

82 These results suggest that most glomus cells of the adult cat carotid body possess oxyge

83 The chemosensory glomus cells of the carotid body (CB) detect changes in

84 ialized for rapid functional oxygen sensing: glomus cells of the carotid body (peripheral respiratory

85 decrease in P(O2) (hypoxia) oxygen-sensitive glomus cells of the carotid body release ATP to activate

86 nd selectively expressed in oxygen-sensitive glomus cells of the carotid body, a chemosensory organ a

87 ia with a heterogenous pattern in individual glomus cells of the carotid body.

88 nd protein, which was primarily localized to glomus cells of the carotid body.

89 racterize the gap junctions between cultured glomus cells of the rat carotid body and to assess the e

90 enign neuroendocrine tumors derived from the glomus cells of the vegetative nervous system.

91 These results indicate that cultured cat glomus cells possess functional nAChRs, and that their c

92 heath cells and possibly a small fraction of glomus cells possess oxygen-insensitive potassium channe

93 Dual voltage clamping of coupled glomus cells showed a mean macrojunctional conductance (

94 Nicotinic AChRs of glomus cells showed high affinity for ACh.

95 ensitive ion channels have been described in glomus cells that respond directly to extracellular acid

96 acidosis evoked transient inward current in glomus cells that was inhibited by the acid-sensing ion

97 creased outward potassium current (Ik) in CB glomus cells to levels similar to those that were observ

98 Ik was measured in glomus cells using conventional and perforated whole-cel

99 tinic ACh receptors (nAChRs) in cultured cat glomus cells using immunocytochemistry and whole cell pa

100 tracellular calcium ([Ca(2+)](i)) release in glomus cells via ryanodine receptor (RyR) activation by

101 Coupling between nerve endings and glomus cells was more complex, When a glomus cell was st

102 us to conclude that the redox modulation of glomus cells was not conveyed to the afferents, and this

103 ypoxic chemotransduction in the carotid body glomus cells was tested by using 4-aminopyridine (4-AP),

104 Glomus cells were identified by catecholamine fluorescen

105 Dissociated rat glomus cells were loaded with Fura-2 AM to study the eff

106 olarizing effect of low pH in SHR versus WKY glomus cells which was caused by overexpression of 2 aci

107 is a major arterial chemoreceptor containing glomus cells whose activities are regulated by changes i

108 of TASK by external acid, depolarization of glomus cells with high external KCl (20 mm) or opening o

109 current clamping after impaling two adjacent glomus cells with microelectrodes, and alternate stimula

110 (glomus cells, sheath cells, and subtypes of glomus cells) and oxygen sensitivity of potassium channe

111 formation between peripheral chemoreceptors (glomus cells) in the carotid body and relay neurons in t

112 In carotid body chemosensitive glomus cells, activation of toll-like receptors increase

113 In carotid body glomus cells, AMPK is thought to link changes in arteria

114 In CHF glomus cells, an AT1 receptor (AT1R) antagonist, L-158 8

115 amniote respiratory reflexes - carotid body glomus cells, and 'pulmonary neuroendocrine cells' (PNEC

116 ar effects on cytosolic calcium ([Ca2+]i) in glomus cells, and if so, whether a heterogenous response

117 n experiments, induces calcium transients in glomus cells, and stimulates carotid sinus nerve activit

118 calcium currents accompany calcium inflow in glomus cells, but calcium influx may not depend exclusiv

119 v) are highly expressed in carotid body (CB) glomus cells, but their role in hypoxia-induced excitati

120 tissues most commonly studied, i.e. carotid glomus cells, central neurons, smooth muscle cells, and

121 lished the first transcriptome profile of CB glomus cells, highlighting genes with potential implicat

122 the most specifically expressed genes in CB glomus cells, highlighting their potential roles in mito

123 toplasmic Ca(2+) concentration in individual glomus cells, isolated in clusters from rat carotid bodi

124 etermine the correlation between cell types (glomus cells, sheath cells, and subtypes of glomus cells

125 Glomus cells, the site of O2 sensing in the carotid body

126 Homology has been proposed between glomus cells, which are neural crest-derived, and the hy

127 ified a set of genes abundantly expressed in glomus cells, which contained novel glomus cell-specific

128 y a drop in intracellular pH of carotid body glomus cells, which inhibits a K+ current.

129 constant normoxic conditions in sham and CHF glomus cells, with threshold concentrations of about 900

130 ssion of Kv3.4 but not Kv4.3 channels in CHF glomus cells.

131 transmembrane Ca2+ influx into carotid body glomus cells.

132 geted to the plasma membrane in carotid body glomus cells.

133 TRPC proteins studied were present in type I glomus cells.

134 ent nerve terminals that encircle individual glomus cells.

135 of [Ca(2+)](i) and intercellular coupling in glomus cells.

136 by ratio fluorometry in isolated cat and rat glomus cells.

137 ings enveloped tyrosine hydroxylase-positive glomus cells.

138 for identifying highly expressed genes in CB glomus cells.

139 g of the suppressant effect on K+ current of glomus cells.

140 tors modulate junctional conductance between glomus cells.

141 d variations in the response pattern between glomus cells.

142 ous response pattern similar to that seen in glomus cells.

143 nt G protein-coupled receptor (Olfr78) in CB glomus cells.

144 s an approximately 20 pS channel in isolated glomus cells.

145 ld by an inhibitor of Kv2, a major Kv in rat glomus cells.

146 tion of hypoxia and acidosis at the level of glomus cells.

147 sensitivity to acidosis and hypoxia in most glomus cells.

148 3 (ASIC3) which we had identified earlier in glomus cells.

149 a did not change the Em of nerve endings and glomus cells.

150 d by hypoxia were greater in CHF versus sham glomus cells.

151 sensitivity of IK and RMP to hypoxia in sham glomus cells.

152 : (a) hypoxia increases cytosolic calcium in glomus cells; (b) response patterns were heterogeneous i

153 le similar to mammalian carotid body Type I (glomus) cells and pulmonary neuroepithelial cells.

154 ypertensive rats (SHRs) carotid body type I (glomus) cells exhibit hypersensitivity to chemosensory s

155 of O2 sensing by carotid body chemoreceptor (glomus) cells is that hypoxia inhibits the outward K(+)

156 Type I (also called glomus) cells, the site of O2 sensing in the carotid bod

157 Three populations of spores of Glomus claroideum (W2537) and three populations of spore

158 m (W2537) and three populations of spores of Glomus DAOM 225952 (W2538) were analysed using a microsa

159 retic types for G. claroideum, and 15-27 for Glomus DAOM 225952 depending on the population.

160 ands were found for G. claroideum, and 43 in Glomus DAOM 225952.

161 of G. claroideum, and none by populations of Glomus DAOM 225952.

162 tream of wt1 in the gene network controlling glomus differentiation.

163 However, when Glomus etunicatum was chosen as the outgroup, the polari

164 ils of conspecific plants, and feedback with Glomus etunicatum, a dominant mycorrhizal fungus.

165 rphic genetic markers in natural isolates of Glomus etunicatum, coupled with direct amplification of

166 irs of plants with and without the AM fungus Glomus hoi in microcosms that allowed only the fungus ac

167 he bacterial community responded to the AMF, Glomus hoi.

168 alysed the growth factor inducibility of the glomus in the presence or absence of retinoic acid (RA)

169 rmation by the arbuscular mycorrhizal fungus Glomus intraradices (Schenck & Smith) was limited to cor

170 h the symbiotic arbuscular mycorrhiza fungus Glomus intraradices and the rhizobial bacterium Sinorhiz

171 transformed carrot (Daucus carota) roots and Glomus intraradices grown monoxenically on bicompartment

172 pair of test plants, interlinked by a CMN of Glomus intraradices or Glomus mosseae.

173 ary from germinating spores of the AM fungus Glomus intraradices showed strong homology to gene seque

174 involved in the pathway were identified from Glomus intraradices, and for six of them the full-length

175 ired for infection by the mycorrhizal fungus Glomus intraradices, suggesting that LNP plays a role in

176 ated with the degree of root colonization by Glomus intraradices.

177 by which these fluxes occur in the AM fungus Glomus intraradices.

178 We show that the glomus is specified at stage 12.5, the same stage at whi

179 rsity of spores of two indigenous species of Glomus isolated from three soils of a long-term field ex

180 ascular approach to three patients harboring glomus jugulare paragangliomas.

181 ifficult to achieve complete obliteration of glomus jugulare tumors with the use of embolization and

182 t of the neoplasms are glomus tympanicum and glomus jugulare tumors.

183 HNPs, 26 were carotid body tumors (CBTs), 15 glomus jugulare, 3 glomus tympanicum, and 1 laryngeal pa

184 FAME profiles supported this hypothesis when Glomus leptotichum was used as the outgroup.

185 ards a mycorrhizal fungus closely related to Glomus macrocarpum.

186 tion, cobalt exerted a nonspecific effect on glomus membrane channels.

187 colonised and non-colonised by the AM fungus Glomus mosseae and five putative differentially regulate

188 second experiment, hyphae of both G. hoi and Glomus mosseae that exploited an organic material patch

189 terlinked by a CMN of Glomus intraradices or Glomus mosseae.

190 In this study effect of Glomus mosseae/Medicago sativa mycorrhiza on atrazine de

191 gan; each nephron consists of a single large glomus, one set of tubules and a single duct.

192 d lim1 which influences differentiation into glomus or tubule derivatives in vivo.

193 increase podocyte gene expression within the glomus proper.

194 e report a study on the specification of the glomus, the filtration device of the amphibian pronephri

195 er with high concentrations of RA can induce glomus tissue from animal cap ectoderm.

196 ions under which these growth factors induce glomus tissue in animal cap tissue.

197 s responsible for hereditary paragangliomas (glomus tumors, MIM No.

198 ereditary nonchromaffin paragangliomas (PGL; glomus tumors; MIM 168000) are mostly benign, slow-growi

199 Most of the neoplasms are glomus tympanicum and glomus jugulare tumors.

200 id body tumors (CBTs), 15 glomus jugulare, 3 glomus tympanicum, and 1 laryngeal paraganglioma.

201 ring the development of an AM symbiosis with Glomus versiforme and during growth under differing phos

202 onse to infection by the mycorrhizal fungus, Glomus versiforme.

203 esicular-arbuscular mycorrhizal (VAM) fungus Glomus versiforme.

204 eurospora crassa, and the mycorrhizal fungus Glomus versiforme.

205 la roots colonized by the mycorrhizal fungus Glomus versiforme.

206 etion of lmx1b results in the formation of a glomus with reduced size.