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1 thesis of cell wall polysaccharides, such as glucomannan.
2 lulose, xyloglucan, mixed-linkage glucan and glucomannan.
3 actoglucomannan (GGM) rather than unbranched glucomannan.
4 for cellulose but does not bind to mannan or glucomannan.
5 eta-1,4-linked glucose-mannose heteropolymer glucomannan.
6 y up-regulated during growth on galacto- and glucomannans.
7 on consisted of arabinogalactans, xylans and glucomannans.
8 ellulose acetate, mixed-linkage beta-glucan, glucomannan and arabinoxylan).
9 saccharides were confirmed as low abundance (glucomannan and callose) or undetectable (pectin) in the
10 s as the difference between the mean for the glucomannan and control groups.
11                                              Glucomannans and esterified uronic acids were more abund
12 arides, with 20% (1,3;1,4)-beta-d-glucan, 7% glucomannan, and 4% cellulose.
13 nds to the soluble polysaccharides lichenin, glucomannan, and barley beta-glucan, which are substrate
14   csla9 mutants showed substantially reduced glucomannan, and triple csla2csla3csla9 mutants lacked d
15                                              Glucomannan appears to beneficially affect total cholest
16 an, acetylated glucuronoxylan and acetylated glucomannan as major hemicellulose components, respectiv
17  hydrolase (GH) families 5 and 26, hydrolyze glucomannan by cleaving the glycosidic bond of mannoside
18 sion of CSLA2, CSLA7 and CSLA9 increased the glucomannan content in stems.
19  (GSL), cellulose (CESA), pectin (GAUT), and glucomannan (CSLA) synthesis were also abundant in starc
20       The level of mannosyl residues in stem glucomannans decreased by approximately 40% for Arabidop
21 earlier heterologous expression studies, the glucomannan deficiency observed in csla mutant plants de
22                                   The use of glucomannan did not appear to significantly alter any ot
23                                          The glucomannan enriched surimi-diet induced hypocholesterol
24 ved for 7weeks the squid-surimi control (C), glucomannan-enriched squid-surimi (G) and glucomannan-sp
25 colinate, Ephedra sinica, Garcinia cambogia, glucomannan, guar gum, hydroxy-methylbutyrate, plantago
26    The same proteins can produce beta-linked glucomannan heteropolymers when supplied both GDP-mannos
27 ponsible for the synthesis of all detectable glucomannan in Arabidopsis stems, and that CSLA7 synthes
28 rabidopsis stems, and that CSLA7 synthesises glucomannan in embryos.
29 le csla2csla3csla9 mutants lacked detectable glucomannan in stems.
30 are inconsistent with a substantial role for glucomannan in wall strength in Arabidopsis stems, but i
31           Thus, BaMan5A is able to hydrolyze glucomannan in which the sequence of glucose and mannose
32 ll walls, any role for xylan is unclear, and glucomannan is thought to be the important cellulose-bin
33 f low degrees of arabinosyl substitution and glucomannans, is tightly associated around microfibrils.
34 that supplemented with soluble fibre (konjac glucomannan, KGM; inulin), or insoluble fibre (cellulose
35 ls (RCTs) suggests the consumption of konjac glucomannan (KJM), a viscous soluble fiber, for improvin
36 ngth in Arabidopsis stems, but indicate that glucomannan levels affect embryogenesis.
37 nical trials have investigated the impact of glucomannan on plasma lipids, body weight, fasting blood
38 fect of expansin on uniaxial extensions with glucomannan or galactomannan.
39 n those carried out on composites containing glucomannan or galactomannan.
40 t of high-fat squid-surimi diets enriched in glucomannan or glucomannan-spirulina on lipemia, liver g
41 ogether with smaller amounts of xyloglucans, glucomannans, pectins, and a network of polyphenolic sub
42 overexpression of CSLA9, suggesting that the glucomannan products are similar.
43 uted with arabinosyl residues and additional glucomannan provides an interstitial domain that interco
44 However, gonst1 mutants have no reduction in glucomannan quantity and show no detectable alterations
45                                   The use of glucomannan significantly lowered total cholesterol [wei
46 ), glucomannan-enriched squid-surimi (G) and glucomannan-spirulina enriched squid-surimi (GS).
47 quid-surimi diets enriched in glucomannan or glucomannan-spirulina on lipemia, liver glutathione stat
48 lus trichocarpa catalyze beta-1,4-mannan and glucomannan synthase reactions in vitro.
49 , and each CslA protein catalyzed mannan and glucomannan synthase reactions in vitro.
50 ene family from diverse plant species encode glucomannan synthases and support the hypothesis that ma
51 ts demonstrates that the CSLA family encodes glucomannan synthases.
52 t is not known whether all CslA proteins are glucomannan synthases.
53                                    Increased glucomannan synthesis also caused defective embryogenesi
54 hat CSLA9 is responsible for the majority of glucomannan synthesis in both primary and secondary cell
55 hether the CSLA proteins are responsible for glucomannan synthesis in vivo, we characterised insertio
56                      MUCI10 likely decorates glucomannan, synthesized by CELLULOSE SYNTHASE-LIKE A2,
57 -analysis of randomized controlled trials of glucomannan to better characterize its impact on plasma
58 ired glucose metabolism did not benefit from glucomannan to the same degree.
59 vided by the plant structural polysaccharide glucomannan, which comprises a backbone of beta-1,4-link
60 -D-glucans, together with smaller amounts of glucomannans, xyloglucans, pectins, and a network of pol

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