ライフサイエンスコーパス: glucoprivation

1 pulations activated by 2-deoxyglucose evoked glucoprivation.

2 rdiorespiratory activity and the response to glucoprivation.

3 o regulate adrenaline release in response to glucoprivation.

4 ry peptide enkephalin in responses evoked by glucoprivation.

5 cholaminergic A1-C1m neurons is activated by glucoprivation.

6 counter-regulatory neurohumoral responses to glucoprivation.

7 mic responses to both systemic and hindbrain glucoprivation.

8 5%) and feeding (47%) responses to focal PFH glucoprivation.

9 -glucose (2DG; 200 mg/kg) was used to induce glucoprivation.

10 in modulate the CRR and feeding responses to glucoprivation.

11 Fos-ir was diminished or unaffected by prior glucoprivation.

12 in the feeding and hyperglycemic response to glucoprivation.

13 tors in mediating the hyperphagic effects of glucoprivation.

14 olamine neurons in mediation of responses to glucoprivation.

15 catecholamine neurons that are activated by glucoprivation.

16 ympathetic nerve activity (ASNA) to systemic glucoprivation.

17 ng) response to focal PFH (69%) and systemic glucoprivation (39%), while increasing PFH 5-HT levels a

18 eprivation (24 h), 2-deoxy-D-glucose-induced glucoprivation (500 mg/kg) or mercaptoacetate-induced li

19 Glucoprivation activates neurons in the perifornical hyp

20 roanatomical sites that are altered by prior glucoprivation and that mediate some of the physiologica

21 , neuroendocrine, and autonomic responses to glucoprivation are impaired after a glucoprivic episode.

22 These neurons are activated by glucoprivation, but unlike the C1 cell group, not by hyp

23 %) projecting to celiac ganglia activated by glucoprivation could direct pancreatic and hepatic or ot

24 Thirdly, in vivo or in vitro glucoprivation did not affect the activity of RVLM adren

25 In these animals, prior glucoprivation did not attenuate 2DG-induced feeding in

26 Consequently, glucose deficit (glucoprivation) elicits a variety of physiological and b

27 Glucocorticoids, which are elevated by glucoprivation, have been implicated in the pathogenesis

28 ke elicited by such regulatory challenges as glucoprivation induced by 2-deoxy-D-glucose (2DG) or foo

29 ce, DD mice fail to eat in response to acute glucoprivation induced by insulin or 2-DG.

30 untary overconsumption of palatable food, or glucoprivation induced by systemic 2-deoxy-D-glucose.

31 n, their hyperglycemic response to hindbrain glucoprivation induced with 5-thio-glucose was impaired,

32 tic preganglionic neurons (SPN) activated by glucoprivation, induced by 2-deoxy-D-glucose (2DG).

33 e signaling in the CPu is not sufficient for glucoprivation-induced feeding, we propose that this fee

34 These results suggest that glucoprivation-induced increases in gastric motility are

35 We investigated the hypothesis that during glucoprivation, lactate regulates neuronal monocarboxyla

36 entially alter feeding responses elicited by glucoprivation, lipoprivation and by different opioid pe

37 Secondly, glucoprivation of neurons in the PeH increased ASNA.

38 fuel attenuates transactivational effects of glucoprivation on PVN and SON AVP neurons.

39 of repeated 2-deoxy-D-glucose (2DG)-induced glucoprivation on subsequent 2DG-induced feeding and hyp

40 cated in mediating the suppressive effect of glucoprivation on the reproductive neuroendocrine axis,

41 Glucoprivation or hypoglycemia induces a range of counte

42 in vivo and 2) whether direct activation by glucoprivation or orexin release in the RVLM modulates t

43 These results indicate that cerebral glucoprivation produced by pharmacological doses of 2DG

44 Prior repeated glucoprivation reduced subsequent feeding and hyperglyce

45 model, we compared the effect of antecedent glucoprivation targeting hindbrain or hypothalamic gluco

46 modulate feeding elicited by deprivation or glucoprivation, there has been no systematic examination

47 trating that glycogen depletion from morning glucoprivation was not responsible for the absence of th

48 the hyperphagic response elicited by central glucoprivation was suppressed by an ODN agonist.

49 o 2-deoxy-D-glucose (2DG; 200 mg/kg)-induced glucoprivation were tested 3-7 d later.

50 In awake, behaving rats, bilateral PFH glucoprivation with 5-thioglucose stimulated adrenal med

51 neuroendocrine, and behavioral responses to glucoprivation, yet the functions of the individual grou