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1  the synthesis of the ubiquitously expressed glucose transporter 1.
2 of resulting glycoconjugates, where GLUT1 is glucose transporter 1.
3 ls of vascular endothelial growth factor and glucose transporter 1.
4 ch as for VEGF, phosphoglycerate kinase, and glucose transporter-1.
5 ed upregulation of glycolytic pathway genes, glucose transporter 1-4 (Glut1-4), phosphoglycerate kina
6         Microarray experiments revealed that glucose transporters 1, 8, and 10 were expressed in MCF-
7                Immunohistochemical Ki-67 and glucose transporter 1 analysis was used to evaluate tumo
8  cell lines leads to increased expression of glucose transporter 1 and consequent increased cellular
9 imens, mRNA levels and protein expression of glucose transporter 1 and glucose transporter 3 (GLUT-1
10          Furthermore, the expression of both glucose transporter 1 and hexokinase 2, the first enzyme
11 on of glucose transporters (sodium-dependent glucose transporter-1 and glucose transporter-2) and swe
12 rom the small intestine via sodium-dependent glucose transporter-1 and glucose transporter-2, which m
13         Low serum glucose, downregulation of glucose transporter-1 and glucose-6-phosphatase mRNA, an
14  of other hypoxia-inducible genes, including glucose transporter-1 and insulin-like growth factor-bin
15                                              Glucose transporter-1 and Ki-67 were negative in the end
16 unohistochemical staining revealed increased glucose transporter-1 and norepinephrine transporter exp
17 tilization accompanies the downregulation of glucose transporter-1 and poly (ADP-ribose) polymerase c
18 at cells in the invasive edges expressed the glucose transporter-1 and the sodium-hydrogen exchanger-
19              Surprisingly, the expression of glucose transporters 1 and 4 and insulin receptor substr
20 lts in more abundant membrane association of glucose transporters 1 and 4, which enhances glucose upt
21  endothelial markers (von Willebrand factor, glucose transporter-1) and robust expression of tight an
22 ecreased vascular endothelial growth factor, glucose transporter 1, and erythropoietin expression.
23 encoding vascular endothelial growth factor, glucose transporter 1, and erythropoietin were up-regula
24 ith upregulation of the glucose transporter, glucose transporter 1, and glycolytic genes, hk1 and pdk
25 he expression of the HIF-1alpha-target gene, glucose transporter 1, and report that HIF-1alpha promot
26 expression of the HIF-1alpha targets VEGF-A, glucose transporter-1, and lactate dehydrogenase A.
27    The localization of uncoupling protein-1, glucose transporter-1, and norepinephrine transporter wa
28 s vascular endothelial growth factor (VEGF), glucose transporter-1, and pyruvate dehydrogenase kinase
29 E-19 cells were labeled with MCT1, MCT4, and glucose transporter-1 antibodies.
30 le proteomics approaches, we have identified glucose transporter-1 as the receptor for dematin and ad
31 ines by HIV-1 led to increased expression of glucose transporter 1, associated with increased transpo
32                 Profibrotic up-regulation of glucose transporter 1 by TGF-beta involves activation of
33                                     Although glucose transporter 1, claudin-3, and plasmalemma vesicu
34 n CD46-costimulated T cells and identify the glucose transporter 1 encoding transcript SLC2A1 as a ta
35  Because homologues of dematin, adducin, and glucose transporter-1 exist in many non-erythroid cells,
36 y regulators in energy substrate metabolism (glucose transporter 1, glucose transporter 4, pyruvate d
37 nd expression of intestinal sodium-dependent glucose transporter-1, glucose transporter-2, and sweet
38 atients, duodenal levels of sodium-dependent glucose transporter-1, glucose transporter-2, and T1R2 t
39  ligation and puncture mice sodium-dependent glucose transporter-1, glucose transporter-2, and T1R2 t
40 man) and relative levels of sodium-dependent glucose transporter-1, glucose transporter-2, and taste
41 nterference (RNAi), to determine the role of glucose transporter 1 (GLUT-1) and GLUT-3 in L-14C-DHA t
42  searched until July 2012 by using the terms glucose transporter 1 (GLUT-1) deficiency syndrome, gluc
43 U binding studies revealed that, for HTLV-1, glucose transporter 1 (GLUT-1) functions at a postbindin
44       The observation that overexpression of glucose transporter 1 (GLUT-1) in mesangial cells could
45 ascular endothelial growth factor (VEGF) and glucose transporter 1 (Glut-1) is markedly reduced.
46 or-activated receptor gamma (PPAR-gamma) and glucose transporter 1 (GLUT-1) levels in human brain mic
47 has a bona fide candidate cellular receptor, glucose transporter 1 (glut-1), been identified.
48 s in placental homogenates and expression of glucose transporter 1 (GLUT-1), taurine transporter (TAU
49  smooth muscle actin (SMA), D2-40, CD34, and glucose transporter 1 (GLUT-1).
50                                              Glucose transporter-1 (Glut-1) and hexokinase II (HKII)
51  synthesized and covalently attached to anti-glucose transporter-1 (GLUT-1) antibodies via carbodiimi
52 es using adherent cell lines have shown that glucose transporter-1 (GLUT-1) can function as a recepto
53 nd to relate this to physiologic hypoxia and glucose transporter-1 (GLUT-1) expression.
54 scular endothelial growth factor (VEGF), and glucose transporter-1 (Glut-1) is mediated in part by a
55                            The expression of glucose transporter-1 (Glut-1), hexokinase-1 and -2 (Hk-
56 ascular endothelial growth factor (VEGF) and glucose transporter-1 (Glut-1).
57  (DHA), enters mitochondria via facilitative glucose transporter 1 (Glut1) and accumulates mitochondr
58 hypoglycemia-induced gene expression of both glucose transporter 1 (Glut1) and inhibitor of kappa bet
59 n-responsive vesicular pool of intracellular glucose transporter 1 (GLUT1) and transferrin receptors
60 e factor 1 alpha and its downstream targets, glucose transporter 1 (GLUT1) and vascular endothelial g
61 esis was demonstrated by immunostaining with glucose transporter 1 (GLUT1) antibody, in viable vessel
62 rs-2 does regulate the surface expression of glucose transporter 1 (Glut1) as assessed by flow cytome
63                          We examined whether glucose transporter 1 (GLUT1) deficiency causes common i
64        To test this hypothesis, we evaluated glucose transporter 1 (Glut1) expression and glucose upt
65  TATA sequence, whereas the promoter for the glucose transporter 1 (Glut1) gene contains a TATA eleme
66 ingle-nucleotide polymorphisms (SNPs) in the glucose transporter 1 (GLUT1) gene to the uptake of (18)
67                                              Glucose transporter 1 (GLUT1) immunohistochemistry was p
68  we show that expression of the facilitative glucose transporter 1 (GLUT1) is induced by TGF-beta in
69                A significant increase in the glucose transporter 1 (GLUT1) level, as well as in the g
70 tant increases in glucose import and surface glucose transporter 1 (GLUT1) levels, leading to elevate
71                                          The glucose transporter 1 (GLUT1) protein is underexpressed
72 tas of HF-fed animals, protein expression of glucose transporter 1 (GLUT1) was increased 5-fold, and
73 nd lipolysis (17.7%); the mRNA expression of glucose transporter 1 (GLUT1) was upregulated but glucos
74  depleted LICs by reducing the expression of glucose transporter 1 (Glut1), compromising glucose flux
75 parameters were compared with expressions of glucose transporter-1 (GLUT1) and hexokinase-2 measured
76            A structure has been proposed for glucose transporter-1 (GLUT1) based upon homology modeli
77 glycemia of maternal diabetes suppresses the glucose transporter-1 (GLUT1) facilitative glucose trans
78 r of NF-kappaB-kinase beta (IKKbeta) induced glucose transporter-1 (GLUT1) membrane trafficking in bo
79 e and paucity of its translated product, the glucose transporter-1 (Glut1) protein, disrupt brain fun
80 a proliferative arteriopathy associated with glucose transporter-1 (Glut1) up-regulation and a glycol
81 d glucose uptake through the upregulation of glucose transporter-1 (Glut1), lactate secretion and ind
82 ntial proteome analysis, we identify SLC2A1 (glucose transporter 1, GLUT1) as a downstream target of
83 lization of HIF-1alpha and an enhancement in glucose transporter-1, hexokinase-2, and monocarboxylate
84 energy, leading to increased levels of human glucose transporter 1 (hGLUT1).
85 Da protein-interacting protein 3) and GLUT1 (glucose transporter 1); (ii) secretion of pre-formed IL-
86  to meet its increased bioenergetic demands; glucose transporter-1 is up-regulated, basolateral gluco
87 s correlated by flow cytometric detection of glucose transporter 1 levels in mucosal CD4(+) T lymphoc
88 was compared with histologic scores and with glucose transporter 1 levels in mucosal immune cells by
89 ally included perturbed vascular endothelial glucose transporter-1 localization.
90 ecent structural studies suggest that GLUT1 (glucose transporter 1)-mediated sugar transport is media
91 ivity were markedly increased as a result of glucose transporter 1-mediated glucose influx that drive
92 RC2 to drive glycolysis and lipogenesis, and glucose transporter 1-mediated glucose metabolism promot
93                             Furthermore, the glucose transporter-1 mRNA, which is also stabilized by
94 etabolism and increases the transcription of Glucose-transporter-1 mRNA, and of Hexokinase and Pyruva
95 2 and the cerebrovascular-selective proteins glucose transporter 1, permeability-glycoprotein, and la
96 ze, non-signet ring cell carcinoma type, and glucose transporter 1-positive expression on immunohisto
97 absorbed into intestine cells via the sodium glucose transporter 1 (SGLT-1) and glucose transporter 2
98 tration (20%) of the nonmetabolizable sodium-glucose transporter 1 (SGLT1) substrate, methyl-alpha-D-
99 hypoxia-inducible factor 1alpha (HIF1alpha), glucose transporter 1 (SLC2A1; also known as GLUT1), and
100 annexin V-depleted neutrophils, we show that glucose transporter-1 translocation is impaired in neutr
101 VHL function, we found that high basal VEGF, glucose transporter-1, urokinase-type plasminogen activa
102 or BRAF genes, we found that GLUT1, encoding glucose transporter-1, was one of three genes consistent

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