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1 dation, and plasma membrane translocation of glucose transporter 4.
2 4 and increased cardiomyocyte expression of glucose transporter 4.
3 tty acid synthase, uncoupling protein-1, and glucose transporter 4.
4 ntration-dependent manner, promoted membrane glucose transporter-4 accumulation, and enhanced [(3)H]-
5 d cells results in reduction of HCMV-induced glucose transporter 4 and glucose transporter 2 expressi
6 g NFkappaB p65 attenuated CLA suppression of glucose transporter 4 and peroxisome proliferator-activa
7 -binding protein alpha, fatty acid synthase, glucose transporter 4, and the transcription factor sign
8 mediate the effects of GIP, we analyzed Akt, glucose transporter-4, and glucose uptake, all of which
9 whereas the amount of the insulin-sensitive glucose transporter 4 either remained unchanged or decre
10 levels in the TB of insulin receptor (InsR), glucose transporter-4 (GLUT-4) and type 1 insulin-like g
11 differentiated C2C12 myotubes by stimulating glucose transporter-4 (GLUT-4) membraned translocation.
12 termined by key adipocyte markers, including glucose transporter 4 (GLUT4) and adiponectin expression
13 , Akt, and AS160, to promote the net gain of glucose transporter 4 (GLUT4) at the plasma membrane of
14 is regulated by changing the distribution of glucose transporter 4 (GLUT4) between the cell interior
16 ensitive to the levels of insulin-responsive glucose transporter 4 (GLUT4) expression in adipocytes.
17 ls are associated with reduced expression of glucose transporter 4 (GLUT4) in adipocytes, an early pa
18 ly increased plasma membrane localization of glucose transporter 4 (GLUT4) in skeletal muscle and adi
19 as a distinct role from CHC17 in trafficking glucose transporter 4 (GLUT4) in skeletal muscle and fat
23 in cardiac PPARalpha-transgenic mice, heart glucose transporter 4 (GLUT4) mRNA expression and glucos
24 e by stimulating the movement of sequestered glucose transporter 4 (GLUT4) proteins from intracellula
26 the translocation of the insulin-responsive glucose transporter 4 (GLUT4) to the plasma membrane in
31 ulin receptor (IR) and the redistribution of glucose transporter 4 (GLUT4) to the plasma membrane.
32 uscle and adipose tissue by translocation of glucose transporter 4 (GLUT4) to the plasma membrane.
33 into skeletal muscle through recruitment of glucose transporter 4 (GLUT4) to the plasma membrane.
34 ing augments glucose transport by regulating glucose transporter 4 (GLUT4) trafficking from specializ
35 resulted from a Nef-dependent inhibition of glucose transporter 4 (GLUT4) trafficking, as assessed b
36 lin A disrupts all actin filaments, inhibits glucose transporter 4 (GLUT4) translocation, and causes
37 to define the mechanism of insulin-regulated glucose transporter 4 (Glut4) translocation, we have dev
38 duction of insulin-induced hexose uptake and glucose transporter 4 (GLUT4) translocation, whereas Akt
39 reased insulin-stimulated insulin-responsive glucose transporter 4 (GLUT4) translocation, while adeno
41 se transporter 1 (GLUT1) was upregulated but glucose transporter 4 (GLUT4) was unaffected, and adipos
42 ng enhanced green fluorescent protein-tagged glucose transporter 4 (GLUT4) within a zone about 100 nm
44 h as insulin receptor substrate 2 (IRS2) and glucose transporter 4 (GLUT4), are present in the basal
45 is factor-alpha (TNF-alpha) mRNA and induced glucose transporter 4 (GLUT4), muscle carnitine palmitoy
46 stimulated glucose uptake is mediated by the glucose transporter 4 (GLUT4), which is expressed mainly
50 rated by genetic disruption of one allele of glucose transporter 4 (GLUT4+/-), the insulin-responsive
51 ment of the facilitative glucose transporter glucose transporter-4 (Glut4) from an intracellular comp
52 im of this study was to evaluate the role of glucose transporter-4 (GLUT4) in the anti-diabetic effec
54 lar glucose uptake by changing the amount of glucose transporter-4 (GLUT4) in the plasma membrane thr
56 tilization at the mRNA and protein level and glucose transporter-4 (GLUT4) localization in skeletal m
58 ), activation of protein kinase B (Akt), and glucose transporter-4 (GLUT4) translocation to the plasm
59 ved in glucose and fat metabolism, including glucose transporter-4, hexokinase-2, muscle-pyruvate kin
60 ng protein that regulates the trafficking of glucose transporter 4 in response to insulin and muscle
62 pression of insulin receptor substrate 1 and glucose transporter 4 in the skeletal muscle, but thiazo
63 ride lipase enzymes, leptin, adiponectin and glucose transporter-4 in 3T3-L1 cells which may have con
64 ha (hypoxia-inducible factor 3a), and GLUT4 (glucose transporter 4) in male placentas but not females
65 at the effects of AMPK on gene expression of glucose transporter 4, mitochondrial genes, and PGC-1alp
66 n more markedly attenuated insulin action on glucose transporter 4 movements, hexose transport activi
67 pomyosin, intercellular adhesion molecule-4, glucose transporter-4, Na-K-ATPase, sodium/hydrogen exch
69 mulated IRbeta tyrosine phosphorylation, and glucose transporter-4 protein level were each lower in b
70 rotein, IRbeta tyrosine phosphorylation, and glucose transporter-4 protein) in the epitrochlearis mus
72 substrate metabolism (glucose transporter 1, glucose transporter 4, pyruvate dehydrogenase kinase 4,
74 t through the translocation of intracellular glucose transporter 4 to the plasma membrane in muscle a
76 binds its receptor on adipocytes or muscle, glucose transporter-4 vesicles fuse with the cell membra
77 reduction of glucose transporter I, but not glucose transporter 4, was restored in HIF transgenic mo
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