ライフサイエンスコーパス: glucose uptake

1 ors using a cell-based fluorescence assay of glucose uptake.

2 esistance by impairing insulin signaling and glucose uptake.

3 active oxygen species (81.6%), and decreased glucose uptake.

4 not be distinguishable by the assessment of glucose uptake.

5 fic AMPK-dependent increase in TBC1D1-driven glucose uptake.

6 lyceride accumulation and insulin-stimulated glucose uptake.

7 turbing the cAMP circuit or by inhibition of glucose uptake.

8 ption of barrier integrity and inhibition of glucose uptake.

9 This was associated with increased glucose uptake.

10 n of hBVR released inhibition and stimulated glucose uptake.

11 results in a decrease in HIF1alpha-sensitive glucose uptake.

12 nsporter Glut1 and hexokinase 2, and reduced glucose uptake.

13 by increasing skeletal muscle GLUT4-mediated glucose uptake.

14 T-dependent signaling and insulin-stimulated glucose uptake.

15 triction partially restored the impaired BAT glucose uptake.

16 r endothelial cells (BECs) and reduces brain glucose uptake.

17 g insulin-stimulated AKT phosphorylation and glucose uptake.

18 ther than a significant inhibition in muscle glucose uptake.

19 nd adipose (Adipo-IR) insulin resistance and glucose uptake.

20 n of EGP, and insulin-induced stimulation of glucose uptake.

21 ed GBE expression in C2C12 myotubes promoted glucose uptake.

22 sulin secretion (GSIS) and peripheral tissue glucose uptake.

23 ipolysis does not inhibit insulin-stimulated glucose uptake.

24 ation, connecting miR-150 with modulation of glucose uptake.

25 -I on beta-cell insulin secretion and muscle glucose uptake.

26 insulin action, leading to more rapid muscle glucose uptake.

27 ulated cells and enhances insulin-stimulated glucose uptake.

28 ction (IDIF) desirable for quantification of glucose uptake.

29 which is coordinated with the stimulation of glucose uptake.

30 ein, TUG, to promote GLUT4 translocation and glucose uptake.

31 el pharmacologic interventions that modulate glucose uptake.

32 expression of TXNIP, coinciding with reduced glucose uptake.

33 lines broadly correlates with the amount of glucose uptake.

34 ether GLUT4 is required for overload-induced glucose uptake.

35 re suggested to impact on insulin-stimulated glucose uptake.

36 issue, capable of beta-adrenergic-responsive glucose uptake.

37 sses glucose production without an effect on glucose uptake.

38 e that 2-AG improves insulin sensitivity and glucose uptake.

39 proinflammatory markers and improves IR and glucose uptake.

40 d insulin-stimulated Akt phosphorylation and glucose uptake.

41 in relative TSCR were associated with raised glucose uptake.

42 ctivated kinase (AMPK), leading to increased glucose uptake.

43 from certain nonmalignant tissues with high glucose uptake.

44 sphorylation towards glycolysis and enhanced glucose uptake.

45 pendent roles of the HM domain in regulating glucose uptake.

46 programs related to Glut4 and Glut1-mediated glucose uptake.

47 act HM domain is required for Glut4-mediated glucose uptake.

48 ssion tomography showed increased myocardial glucose uptake.

49 merization, GLUT4 vesicle translocation, and glucose uptake.

50 LUT6, and/or GLUT10 mediate overload-induced glucose uptake.

51 nes such as (R)-(+)-limonene stimulated both glucose uptake (17.4%) and lipolysis (17.7%); the mRNA e

52 The rates of hepatic glucose uptake (5.8 +/- 0.8 vs. 3.2 +/- 0.3 mg kg(-1) mi

53 ystemic glucose metabolism via regulation of glucose uptake across the BBB.

54 veness to suppress EGP and stimulate hepatic glucose uptake; activation of glucokinase was restored a

55 n as cancer stem cells, hijack high-affinity glucose uptake active normally in neurons to maintain en

56 A new platform to visualize glucose uptake activity in live cells and tissues is pre

57 Whereas, Glut1-mediated glucose uptake also requires mTORC2 phosphorylation of t

58 air lymphatic contractile status by reducing glucose uptake, altering cellular metabolic pathways, an

59 id cycle gene signature, alongside increased glucose uptake and activated aerobic glycolysis.

60 ly, the pH shift caused by C5a increased the glucose uptake and activated glycolytic flux in neutroph

61 the heterozygous deletion of Pax5 increased glucose uptake and ATP levels by more than 25-fold.

62 Changes in Glut1 and Mcl-1 levels, glucose uptake and B cell number in the absence of TRAF3

63 Impairment of brain-glucose uptake and brain-copper regulation occurs in Alz

64 insulin signaling, leading to reduced muscle glucose uptake and decreased hepatic glycogen synthesis.

65 The findings suggest that information on glucose uptake and diffusion coefficient carries complem

66 ne (Q282A) doubled the Km(app) for 2-deoxy-d-glucose uptake and eliminated cis-allostery (stimulation

67 ays involved in energy metabolism, including glucose uptake and fermentation, and regulation of mitoc

68 velopment of insulin resistance, lower brain glucose uptake and glucose transporters, alterations in

69 we demonstrate that both insulin-stimulated glucose uptake and GLUT4 translocation to the plasma mem

70 Insulin signaling and effect on glucose uptake and Glut4 translocation were decreased, a

71 of mouse tibialis anterior muscle decreased glucose uptake and glycogen content in vivo, concomitant

72 induced robust, durable, insulin-independent glucose uptake and glycogen synthesis, with resultant im

73 at IL-10 inhibits lipopolysaccharide-induced glucose uptake and glycolysis and promotes oxidative pho

74 onstrate the occurrence and functionality of glucose uptake and glycolysis in the cilia.

75 e vast majority of cancers exhibit increased glucose uptake and glycolysis regardless of oxygen avail

76 Interestingly, glucose uptake and glycolysis were reduced in the bone-d

77 1 cells had low ATP levels due to diminished glucose uptake and glycolysis which was rescued by Vitam

78 he core autophagy machinery also facilitates glucose uptake and glycolytic flux by promoting cell sur

79 derived monocytes and macrophages, increased glucose uptake and glycolytic flux fuel the generation o

80 ose transporter in lung SqCC, which augments glucose uptake and glycolytic flux.

81 glutamate release enhances oligodendroglial glucose uptake and glycolytic support of fast spiking ax

82 Overload-induced muscle glucose uptake and hypertrophic growth were not impaired

83 lipid accumulation associated with increased glucose uptake and impaired endocytosis of GLUT4.

84 xpression specifically in BECs reduces brain glucose uptake and increases VEGF serum concentrations i

85 acts directly upon adipose tissue to improve glucose uptake and indirectly via insulin signaling.

86 Several studies have linked impaired glucose uptake and insulin resistance (IR) to functional

87 Twelve weeks after surgery, glucose uptake and insulin sensitivity were measured usi

88 emonstrate that miR-29a and miR-29c regulate glucose uptake and insulin-stimulated glucose metabolism

89 Net hindlimb glucose uptake and lactate output rates were similar bet

90 Hindlimb glucose uptake and lactate output rates were similar bet

91 how that FILNC1 deficiency leads to enhanced glucose uptake and lactate production through upregulati

92 noma cells overexpressing TIGAR have reduced glucose uptake and lactate production.

93 ICAR-induced AMPK activation and potentiated glucose uptake and lipid oxidation.

94 Tumors exhibit increased glucose uptake and lipogenesis, but the mechanisms contr

95 nd alpha-glucosidase activity and stimulated glucose uptake and lipolysis.

96 hysiology and pathophysiologically increased glucose uptake and may have the potential to provide inf

97 We engineered mutants in genes affecting glucose uptake and metabolism (oprB, gltK, gtrS and glk)

98 eparate mechanisms by which genes regulating glucose uptake and metabolism are involved in the hypoxi

99 ted in animal models suggest that intestinal glucose uptake and metabolism are upregulated after Roux

100 ppressed the expression of genes involved in glucose uptake and metabolism in skeletal muscle by inhi

101 ed mice, including insulin resistance, brain glucose uptake and metabolism, and synaptic function, co

102 imaging can potentially detect and visualize glucose uptake and metabolism, without the need for radi

103 nce--including cell enlargement, and greater glucose uptake and mitochondrial activity--which promote

104 vivo high-resolution simultaneous imaging of glucose uptake and mitochondrial metabolism within a dyn

105 Thus, glucose uptake and mitochondrial pyruvate import prevent

106 yocytes reproduced the effects of SBI-477 on glucose uptake and myocyte lipid accumulation.

107 in terms of (a) insulin signaling, (b) brain glucose uptake and neuronal- and astrocytic metabolism,

108 significant improvements in liver function, glucose uptake and pancreatic beta-cell function indepen

109 increases cellular energy potential even as glucose uptake and phosphoprotein signaling is repressed

110 provide evidence that SGLT2 inhibitors block glucose uptake and reduce tumor growth and survival in a

111 metabolic gene program that enhances muscle glucose uptake and storage as glycogen.

112 in cytosolic zinc levels are concurrent with glucose uptake and suppression of glycogen synthesis.

113 amino]-2-deoxy-D-glucose (2-NBDG) reports on glucose uptake and Tetramethylrhodamine ethyl ester (TMR

114 young hearts (P < 0.05); the AMPK downstream glucose uptake and the rate of glucose oxidation were si

115 carbon starvation, where it plays a role in glucose uptake and the recovery of hyphal growth.

116 ays for intracellular metabolites, including glucose uptake and three other species, are designed as

117 of T cell bioenergetics involved restricted glucose uptake and use, despite persisting mechanistic t

118 eral tissue activator of insulin-independent glucose uptake and verify skeletal muscle as an apoA-I t

119 key glycolytic enzymes and transporters for glucose uptake, and downregulation of enzymes participat

120 P antagonist angiotensin IV (AngIV)-mediated glucose uptake, and enhanced the levels of the AngIV-deg

121 SIRT6, glucose transporter Glut1 expression, glucose uptake, and glycolysis.

122 ssion (P < .05, FDR < 0.20), cell migration, glucose uptake, and hexokinase activity (paired t test,

123 ed membranous GLUT1 translocation, elevating glucose uptake, and increased acetyl-CoA levels, leading

124 d Wnt-signaling, enhanced insulin-stimulated glucose uptake, and inhibited the proliferation of DLD-1

125 n mice eliciting, in HFD, DNA demethylation, glucose uptake, and insulin response.

126 hanisms involving endocannabinoid signaling, glucose uptake, and IR in cardiomyocytes are understudie

127 el) mice impairs BBB-GLUT1 expression, brain glucose uptake, and memory formation in obese, but not i

128 -AG abates inflammatory responses, increases glucose uptake, and overcomes IR in an AMPK-dependent ma

129 e-body glucose turnover, and skeletal muscle glucose uptake ( approximately 60%; P < 0.05), as compar

130 increased insulin-stimulated skeletal muscle glucose uptake as measured by a hyperinsulinemic-euglyce

131 its agonist activity in both lipogenesis and glucose uptake assays.

132 roplets exhibited impaired insulin-dependent glucose uptake, associated with defects in GLUT4 traffic

133 , together with a strong impairment of brain glucose uptake at the time of attempted memory retrieval

134 lucose production and stimulating peripheral glucose uptake, brain insulin may improve glucose metabo

135 lin- and contraction-induced skeletal muscle glucose uptake, but its role in chronic muscle loading (

136 artly regulate contraction-stimulated muscle glucose uptake, but whether those two signaling pathways

137 ChREBPbeta expression in part by controlling glucose uptake, but without impairing pan-AKT signalling

138 ce remained insulin sensitive, had increased glucose uptake by adipose cells and skeletal muscle in v

139 Spontaneous glucose uptake by brown adipose tissue (BAT) is lower in

140 l production were reduced cell viability and glucose uptake by D5A and not loss of enzyme activity or

141 onance spectroscopy, and determined systemic glucose uptake by euglycemic-hyperinsulinemic glucose cl

142 in resistance, we measured insulin-dependent glucose uptake by hAT from nondiabetic and T2DM subjects

143 Ala PIMT mutant abrogated insulin stimulated glucose uptake by L6 myotubes and neonatal rat skeletal

144 Insulin resistance results in inhibition of glucose uptake by liver and other peripheral tissues, pr

145 in the kidney, without affecting intestinal glucose uptake by SGLT1.

146 lin secreted into the circulation stimulated glucose uptake by the liver spheroids, while the latter,

147 tolerance tests on different days, promoted glucose uptake by the liver spheroids.

148 Cells with low glucose uptake capacity and low protein synthesis rates

149 tivation was due to impaired skeletal muscle glucose uptake caused by attenuated insulin delivery, an

150 We demonstrate that during glucose uptake, cell surface abundance of zinc transport

151 which transporters mediate overload-induced glucose uptake, chemical inhibitors were used.

152 ce of any cytokine), and had higher rates of glucose uptake compared with CD56(dim) cells.

153 y fructose intake is elevated and myocardial glucose uptake compromised by insulin resistance, increa

154 und impairments in insulin-stimulated muscle glucose uptake, creating an increased reliance on fatty

155 Adipose tissue triacylglyceride (TAG) and glucose uptake decreased, and the free fatty acid/glycer

156 Although total uterine glucose uptake determined by Fick's principle was unaffe

157 ntified using ELISA or radioimmunoassay, and glucose uptake determined through 2-deoxy glucose 6 phos

158 Regional glucose uptake determined using FDG-PET and neuropsychol

159 infusion, glucose disappearance, and muscle glucose uptake during a hyperinsulinemic-euglycemic clam

160 We show that A549 cells increase glucose uptake during EMT, but instead of increasing the

161 Rac1, but not alpha2 AMPK, regulates muscle glucose uptake during submaximal exercise in vivo.

162 EPO production was associated with increased glucose uptake, enhanced glycolysis, reduced mitochondri

163 colytic capacity accompanied by an increased glucose uptake, expression of glucose transporter, and g

164 ding vasoregulation, mitochondrial function, glucose uptake, fatigue and excitation-contraction coupl

165 Glucose uptake favors osteoblast differentiation by supp

166 Net glucose uptake from the CVVH circuit was 54 +/- 5 mg/min

167 We show that despite increased glucose uptake, GLN addicted cells do not metabolize glu

168 th and metabolism through dynamic control of glucose uptake, global protein translation and transcrip

169 lation into the brain, reduced in vivo brain glucose uptake, GLUT4 expression, and spatial memory.

170 the influence of a brief (two weeks) HFD on glucose uptake (GU) +/- insulin in single fibers that we

171 variant (p.P50T/AKT2) on insulin-stimulated glucose uptake (GU) in the whole body and in different t

172 is and only 8% worsening of insulin-mediated glucose uptake (IMGU).

173 targets of PI3K-Akt signaling that regulate glucose uptake in adipocytes as potential therapeutic ta

174 ure experiments showed that ApoA-IV improved glucose uptake in adipocytes in the absence of insulin b

175 e mice, the partial inhibition of stimulated glucose uptake in adipocytes induces insulin resistance

176 sponsive compartment, and insulin-stimulated glucose uptake in adipocytes is suppressed.

177 Delta/+) was resistant to insulin-stimulated glucose uptake in adipose tissue and skeletal muscle com

178 DG PET identified intense inter-scapular BAT glucose uptake in all ZL control rats, while no focally

179 ctly investigated the role of Glut1-mediated glucose uptake in apolipoprotein E-deficient (ApoE(-/-))

180 ulin action was improved, stimulating muscle glucose uptake in association with decreased intracellul

181 identified that Glut1 connects the enhanced glucose uptake in atheromatous plaques of ApoE(-/-) mice

182 ing and severely impaired insulin-stimulated glucose uptake in BAT was confirmed in a rat model of ty

183 In addition, cold-induced glucose uptake in BAT was positively related to glucose

184 on period resulted in increased cold-induced glucose uptake in BAT, as assessed by [(18)F]fluorodeoxy

185 both WT and diabetic KKAy mice by increasing glucose uptake in cardiac muscle, white adipose tissue,

186 Metformin, a drug that stimulates glucose uptake in cells, mimicked these effects, with a

187 SF and GM-CSF generated comparable levels of glucose uptake in cultured macrophages and murine athero

188 Shizukaol F increased glucose uptake in differentiated C2C12 myotubes by stimu

189 As the primary route of glucose uptake in E. coli, the PTS plays a key role in r

190 te that silencing MondoA expression improves glucose uptake in EndoC-betaH1 cells.

191 d only partially impaired insulin-stimulated glucose uptake in fat and muscle tissue.

192 glucose CEST (glucoCEST) to image unlabeled glucose uptake in head and neck cancer by using a clinic

193 We measured insulin-stimulated glucose uptake in hormone-sensitive lipase (HSL) knockou

194 Loss of Rab20 impairs insulin-stimulated glucose uptake in human and mouse skeletal muscle by blo

195 glyceride (TAG) synthesis and enhanced basal glucose uptake in human skeletal myocytes.

196 muscle capillary blood volume (CBV) reduces glucose uptake in insulin resistance (IR).

197 esents an important alternative to stimulate glucose uptake in insulin-resistant muscle.

198 ved a dose-dependent increase of [(18)F]-FDG-glucose uptake in interscapular BAT (iBAT) of DIOs upon

199 are in line with previously reported delayed glucose uptake in LA carriers after starch consumption.

200 apelin administration in neonates increased glucose uptake in lung and muscle.

201 ter focal cerebral ischemia showed increased glucose uptake in LysMcreTNF(fl/fl) mice, representing s

202 cible IDIF for Patlak analysis of myocardial glucose uptake in mice.

203 phage chemotaxis, reduced insulin-stimulated glucose uptake in myocytes and 3T3-L1 adipocytes and imp

204 imer's disease to demonstrate that enhancing glucose uptake in neurons has strong neuroprotective eff

205 LUT4 protein levels and in exercise-mediated glucose uptake in nonoxidative muscle fibers.

206 ain energy consumption and impaired systemic glucose uptake in obese compared with normal-weight volu

207 esis which proposes that the reduced adipose glucose uptake in obesity is a physiological down-regula

208 abolic profile of skeletal muscle to benefit glucose uptake in patients with type 2 diabetes.

209 suggests they play a key role in regulating glucose uptake in response to axonal glutamate release,

210 glycolysis, despite an inability to increase glucose uptake in response to IGF-1.

211 cise bypasses insulin resistance to increase glucose uptake in skeletal muscle and therefore represen

212 extent insulin-induced hypoglycemia affects glucose uptake in skeletal muscle and whether hypoglycem

213 An acute bout of exercise increases glucose uptake in skeletal muscle by an insulin-independ

214 sults expand the model of insulin-stimulated glucose uptake in skeletal muscle cells by implicating p

215 itical for insulin- and contractile-mediated glucose uptake in skeletal muscle.

216 g increased insulin sensitivity is increased glucose uptake in skeletal muscle.

217 We investigated the effect of ApoA-IV on glucose uptake in the adipose and muscle tissues of mice

218 hepatic insulin resistance, and had reduced glucose uptake in the brain.

219 usion in both legs and abrogated the greater glucose uptake in the exercised compared with the rested

220 nducible factor 1alpha (HIF-1alpha) controls glucose uptake in the hypothalamus and that it is upregu

221 r plasma insulin concentrations, but reduced glucose uptake in the hypothalamus.

222 tomography scan demonstrated a reduction in glucose uptake in the left thalamus and bilateral inferi

223 poE(-/-) bone marrow and resulted in reduced glucose uptake in the spleen and aortic arch of these mi

224 e in visceral white adipose tissue, although glucose uptake in visceral and subcutaneous white adipos

225 cose uptake in BAT was positively related to glucose uptake in visceral white adipose tissue, althoug

226 ucose transport, whereas exercise-stimulated glucose uptake in vivo was only partially reduced by Rac

227 5% in the rested leg (P < 0.05) and that leg glucose uptake increased 50% more (P < 0.05) in the exer

228 -inducible factor) activation with increased glucose uptake, increased 6-phosphofructo-2-kinase/fruct

229 though canagliflozin also inhibited cellular glucose uptake independently of SGLT2, this did not acco

230 t transient, HFD-elicited reduction of brain glucose uptake initiates a compensatory increase of VEGF

231 Insulin controls glucose uptake into adipose and muscle cells by regulati

232 nce through impairment of insulin-stimulated glucose uptake into brown adipose tissue (BAT).

233 Through regulating GLUT1 level, GLD4 affects glucose uptake into cells and lactate levels.

234 ulin, downstream of Akt activation, promotes glucose uptake into fat and muscle cells to lower postpr

235 modulated (14)C-D-glucose and (14)C-deoxy-D-glucose uptake into hepatic HepG2 cells.These data indic

236 ed in UniNx mice, whereas insulin-stimulated glucose uptake into isolated skeletal muscle was similar

237 e broader metabolic consequences of enhanced glucose uptake into muscle.

238 are suggested to regulate exercise-mediated glucose uptake into muscle.

239 inhibition of insulin secretion and enhances glucose uptake into skeletal muscle cells.

240 ich promotes GLUT4 vesicle translocation and glucose uptake into skeletal muscle cells.

241 Insulin promotes glucose uptake into skeletal muscle through recruitment

242 We show that GLUTs are most significant for glucose uptake into the brain and liver, whereas SGLTs a

243 Furthermore, glucose uptake into tissue tended to be higher after nas

244 Impaired GLUT4-dependent glucose uptake is a contributing factor in the developme

245 that muscle insulin sensitivity to stimulate glucose uptake is enhanced several hours after an acute

246 s a direct stimulator of insulin-independent glucose uptake is not known.

247 e that the full effect of insulin on adipose glucose uptake is the integrated effect of Rab10-depende

248 in chronic muscle loading (overload)-induced glucose uptake is unknown.

249 xamate-induced inhibition of LDHA suppressed glucose uptake, lactate secretion, invasion and prolifer

250 olic state is manifested by lowered neuronal glucose uptake, metabolic shift in the astrocytes, and a

251 of insulin resistance significantly impaired glucose uptake, mitochondrial function, oxygen consumpti

252 es (TSCR) from IRT and the metabolic rate of glucose uptake (MR(gluc)) from PET/CT were determined.

253 Glucose uptake on MR(gluc)MIP was found to correlate pos

254 Furthermore, mutants defective in glucose uptake or dependent upon peptides for growth als

255 is not necessary for overload-induced muscle glucose uptake or hypertrophic growth and suggest that G

256 r one day stimulate the contraction-mediated glucose uptake pathway, 3) both acute interruptions to s

257 defective in type 2 diabetes despite reduced glucose uptake per BAT volume and BAT "whitening."

258 L KO mice showed a higher insulin-stimulated glucose uptake postexercise compared with wild-type mice

259 m for generating high temporal resolution of glucose uptake profiles, and consequently insulin sensit

260 An improvement in skeletal glucose uptake rate was also observed in obese-stimulate

261 was lower by 34% (P < 0.01) and the hepatic glucose uptake rate was lower by 33% (P < 0.01) in obese

262 Insulin-stimulated adipose tissue glucose uptake rates are lower in obese versus lean indi

263 Finally, lower insulin-stimulated SCAT glucose uptake rates in obese individuals are proportion

264 Lower insulin-stimulated glucose uptake rates in obese versus lean individuals we

265 However, the growth rates and glucose uptake rates of the mutant WX-zwf were decreased

266 insulin-stimulated Akt activation and tissue glucose uptake rates were also determined.

267 ereas their glycolytic flux and 2-deoxy-(3)H-glucose uptake rates were largely unaffected.

268 d cells simulated at different glutamine and glucose uptake rates.

269 6(bright) cells expressed high levels of the glucose uptake receptor, Glut1 (in the absence of any cy

270 show that insulin-stimulated Glut4-mediated glucose uptake requires PDPK1 phosphorylation of the kin

271 The increase in glucose uptake resulting from glucokinase activation in

272 This interplay between 27-OH and glucose uptake revealed the engagement of the insulin-re

273 Contraction- and insulin-mediated glucose uptake signaling pathways as well as changes in

274 tensin converting enzyme I (ACE) inhibition, glucose uptake stimulation and antioxidant properties.

275 Cold-induced skeletal muscle glucose uptake tended to increase upon cold acclimation,

276 ry task normally and displaying normal brain glucose uptake, they display faster forgetting after a l

277 ion of LDHA in a PA cell line (GH3) promoted glucose uptake through the upregulation of glucose trans

278 Growth factors also enhance glucose uptake to fuel an anabolic metabolism required f

279 enced PDAC exhibited a relative reduction in glucose uptake, tumorigenicity, and metastasis.

280 glycogen was increased by augmenting hepatic glucose uptake using hyperglycemia and a low-dose intrap

281 SUV measurements and rate of glucose uptake values were reproducible, with an average

282 by the uteroplacental tissues, so net fetal glucose uptake was 29% lower in cortisol-infused than co

283 Overload-induced [(3)H]-2-deoxy-d-glucose uptake was not inhibited by d-fructose, demonstr

284 nce using (18)F-fluorodeoxyglucose (FDG) for glucose uptake was performed in 21 ST-segment-elevation

285 reduced in FL-N/35 mice and that hepatocyte glucose uptake was perturbed, partly accounting for the

286 th 140 +/- 14 mg/dL; P < 0.05); however, net glucose uptake was similar (46 +/- 8 compared with 61 +/

287 The rate of glucose uptake was similarly elevated depending on the I

288 muscle weights and ex vivo [(3)H]-2-deoxy-d-glucose uptake were assessed.

289 d increases in sarcolemmal GLUT4 content and glucose uptake were lower in the white gastrocnemius of

290 EGP and glucose uptake were measured using the tracer dilution t

291 ee fatty acid receptor 1 (Ffar1), that curbs glucose uptake when fatty acids are available.

292 ured hepatic (HGU) and adipose tissue (ATGU) glucose uptake with [(18) F]2-fluoro-2-deoxy-D-glucose/p

293 bserved that infected Schwann cells increase glucose uptake with a concomitant increase in glucose-6-

294 ta- and omega-oxidation and reduced cellular glucose uptake with consequent diversion of acetyl-CoA i

295 We observed increased glucose uptake with SOD1(G93A) expression in all co-cult

296 tant target tissue for the apoA-I-stimulated glucose uptake, with potential implications in diabetic

297 nfarction patients showed reduced myocardial glucose uptake within the area at risk and closely match

298 areas with the most intense temperature and glucose uptake within the supraclavicular regions were c

299 the Wnt3a ligand induced a large increase in glucose uptake, without changes in the expression or loc

300 thesized that the area of reduced myocardial glucose uptake would closely match the area at risk deli