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1 echin after enzymatic release of sulfate and glucuronide conjugates.
2 over-the-counter drugs as their sulfate and glucuronide conjugates.
3 a significant factor in renal elimination of glucuronide conjugates.
5 ty is expected to reduce the reactivation of glucuronide-conjugated drugs in the intestine, thereby r
8 ent glucuronidation to form a novel O-linked glucuronide conjugate, O-(beta-D-glucosidurony1)-2-hydro
9 es and the urinary excretion level of the N2-glucuronide conjugate of 2-hydroxyamino-MeIQx--N2-(beta-
11 tographic (HPLC) analysis, and found to be a glucuronide conjugate of a known DCF metabolite, 4'-hydr
13 y either bacteria-mediated hydrolysis of the glucuronide conjugate of the active metabolite 7-ethyl-1
14 e metabolites were identified as sulfate and glucuronide conjugates of a ring hydroxy-substituted met
20 se activity was stimulated by bile acids and glucuronide conjugates, reaching 170 +/- 28 nmol min-1 m
21 und decrease in the biliary clearance of the glucuronide conjugates was observed in Bcrp-deficient mo
24 roid conjugates in all sample types, whereas glucuronide conjugates were hydrolyzed in the presence o
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