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1 echin after enzymatic release of sulfate and glucuronide conjugates.
2  over-the-counter drugs as their sulfate and glucuronide conjugates.
3 a significant factor in renal elimination of glucuronide conjugates.
4 L) at 10 mg/kg was due to the formation of O-glucuronide conjugate by phase 2 metabolism.
5 ty is expected to reduce the reactivation of glucuronide-conjugated drugs in the intestine, thereby r
6                                          The glucuronide conjugates ethinylestradiol-3-glucuronide an
7                                      Several glucuronide-conjugated metabolites of LTB4 were characte
8 ent glucuronidation to form a novel O-linked glucuronide conjugate, O-(beta-D-glucosidurony1)-2-hydro
9 es and the urinary excretion level of the N2-glucuronide conjugate of 2-hydroxyamino-MeIQx--N2-(beta-
10  immune cytopenia caused by sensitivity to a glucuronide conjugate of a drug metabolite.
11 tographic (HPLC) analysis, and found to be a glucuronide conjugate of a known DCF metabolite, 4'-hydr
12 , produced a single detectable metabolite, a glucuronide conjugate of N-hydroxy-PhIP.
13 y either bacteria-mediated hydrolysis of the glucuronide conjugate of the active metabolite 7-ethyl-1
14 e metabolites were identified as sulfate and glucuronide conjugates of a ring hydroxy-substituted met
15 uid chromatography (HPLC) was used to detect glucuronide conjugates of BPD formed in vitro.
16 ences in the pharmacokinetics of sulfate and glucuronide conjugates of isoflavones.
17 rase (UGT) enzymes catalyze the formation of glucuronide conjugates of phase II metabolism.
18                                              Glucuronide conjugates of the estrogen D-ring (100 micro
19                         We hypothesized that glucuronide conjugates of xenobiotics, such as the antic
20 se activity was stimulated by bile acids and glucuronide conjugates, reaching 170 +/- 28 nmol min-1 m
21 und decrease in the biliary clearance of the glucuronide conjugates was observed in Bcrp-deficient mo
22       Because Mrp3 preferentially transports glucuronide conjugates, we investigated the in vivo disp
23            Unlike in rats, where sulfate and glucuronide conjugates were excreted into bile predomina
24 roid conjugates in all sample types, whereas glucuronide conjugates were hydrolyzed in the presence o
25                           Several classes of glucuronide conjugates, which include acyl glucuronides,

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