ライフサイエンスコーパス: glutamate receptor

1 em Xc- antiporter to activate a metabotropic glutamate receptor.

2 able AMPA receptors and group I metabotropic glutamate receptors.

3 elease by presynaptic Group III metabotropic glutamate receptors.

4 and dephosphorylation of synaptic AMPA-type glutamate receptors.

5 depressed by the activation of metabotropic glutamate receptors.

6 mPFC blockade of AMPA-type but not NMDA-type glutamate receptors.

7 which SHANK2 may interact with post-synaptic glutamate receptors.

8 a positive allosteric modulator of AMPA-type glutamate receptors.

9 internalization of both NMDA- and AMPA-type glutamate receptors.

10 tamate release, and presynaptic metabotropic glutamate receptors.

11 tly mediated by signaling through microglial glutamate receptors.

12 roxy-5-methyl-4-isoxazolepropionic acid-type glutamate receptors.

13 n via the activation of group I metabotropic glutamate receptors.

14 c or glutamatergic neurons) and postsynaptic glutamate receptors.

15 titive/fast off-rate antagonist of NMDA-type glutamate receptors.

16 f homeostatic plasticity, up-scaling of AMPA-glutamate receptors.

17 atergic neurons and regulate the activity of glutamate receptors.

18 Metabotropic glutamate receptor 1 (mGluR1) function in Purkinje neuro

19 PET radioligand for quantifying metabotropic glutamate receptor 1 (mGluR1) in monkey brain.

20 The metabotropic glutamate receptor 1 (mGluR1) is abundantly expressed in

21 mergence of glutamate-activated metabotropic glutamate receptor 1 (mGluR1) signaling, are critical to

22 psin-1, postsynaptic density protein 95, and glutamate receptor 1 and medial prefrontal cortex spine

23 29, glutamate decarboxylase 1, metabotropic glutamate receptor 1, and excitatory amino acid transpor

24 were female-specific changes in metabotropic glutamate receptor 1, NMDA receptor 2A, alpha-amino-3-hy

25 tive antibody antagonist of the metabotropic glutamate receptor-1 (BBB-mGluR1), a widely abundant CNS

26 rin-R, cell adhesion molecules, metabotropic glutamate receptor-1 (mGluR1), voltage-gated sodium chan

27 Phrenic motoneuron AMPA glutamate receptor 2 (GluR2) subunit mRNA expression dec

28 allosteric modulators (PAM) of metabotropic glutamate receptor 2 (mGluR2) are a potential therapy fo

29 Here, we find that metabotropic glutamate receptor 2 (mGluR2) signaling, which acts on v

30 s observed biophysically on the metabotropic glutamate receptor 2 homodimer.

31 The presynaptic inhibitory metabotropic glutamate receptors 2 and 3 (mGluR2/3) are key autorecep

32 iveness (leptin receptors Lepr, metabotropic glutamate receptors-2 mGlu2, neuropeptide-Y NPY, and min

33 The metabotropic glutamate receptor 4 (mGluR4) is an emerging target for

34 synaptic protein synthesis, the metabotropic glutamate receptor 5 (mGlu5) protein, is significantly r

35 vel role of GluD1 in regulating metabotropic glutamate receptor 5 (mGlu5) signaling in the hippocampu

36 modulators (NAMs) targeting the metabotropic glutamate receptor 5 (mGlu5) subtype are currently in cl

37 Metabotropic glutamate receptor 5 (mGlu5)-positive allosteric modulat

38 uding disorders associated with metabotropic glutamate receptor 5 (mGluR5) and dopaminergic dysfuncti

39 ithout prior treatment with the metabotropic glutamate receptor 5 (mGluR5) antagonist 2-methyl-6-(phe

40 advance the novel concept that metabotropic glutamate receptor 5 (mGluR5) fails to engage endocannab

41 PrP(C), laminin, and metabotropic glutamate receptor 5 (mGluR5) form a protein complex on

42 allosteric modulators (NAM) of metabotropic glutamate receptor 5 (mGluR5) have been implicated as a

43 Drugs targeting metabotropic glutamate receptor 5 (mGluR5) have therapeutic potential

44 For example, the metabotropic glutamate receptor 5 (mGluR5) is concentrated at the inn

45 tic glutamate, which stimulates metabotropic glutamate receptor 5 (mGluR5) on a small population of i

46 s release glutamate to activate metabotropic glutamate receptor 5 (mGluR5) on astrocytes, evoking Ca(

47 suggest that inhibition of the metabotropic glutamate receptor 5 (mGluR5) receptor might hold therap

48 Specifically, the metabotropic glutamate receptor 5 (mGluR5) represents a promising tre

49 uces a re-emergence of immature metabotropic glutamate receptor 5 (mGluR5) signaling in S1 astroglia,

50 utamate spillover, initiating a metabotropic glutamate receptor 5 (mGluR5)-dependent increase in nitr

51 ular prion protein (PrP(C)) and metabotropic glutamate receptor 5 (mGluR5).

52 protein synthesis downstream of metabotropic glutamate receptor 5 (mGluR5).

53 the cell-autonomous role of the metabotropic glutamate receptor 5 (mGluR5).

54 -born neurons and rescued by an metabotropic glutamate receptor 5 antagonist.

55 in associates via transmembrane metabotropic glutamate receptor 5 with the intracellular protein medi

56 ABA cells that was dependent on metabotropic glutamate receptor 5, and cannabinoid receptor 1 (CB1).

57 R1, and GABABR2 levels, whereas metabotropic glutamate receptor 5, NMDA receptor 2B, GluR2, and GABAA

58 taining protein 12, mitofusin2, metabotropic glutamate receptor 5, p21-activated kinase 7, and Ras-re

59 ither cellular prion protein or metabotropic glutamate receptor 5.

60 glutamatergic transmission, the metabotropic glutamate receptor 6 and voltage-dependent calcium chann

61 inally, we show that sustained activation of glutamate receptors, a condition occurring in brain isch

62 ular transport machinery, and down-regulates Glutamate receptor A1 trafficking and neurotransmitter r

63 tive allosteric modulators of the ionotropic glutamate receptor A2 (GluA2) are promising lead compoun

64 w that JhI-21 suppresses postsynaptic muscle glutamate receptor abundance, and that JhI-21 expression

65 outon enlargement, and increase postsynaptic glutamate receptor abundance.

66 Ionotropic glutamate receptors activate cell signaling in response

67 Using an in vivo model, we show that glutamate receptor activation can evoke endogenous d-ser

68 ctivity of neurons in the hypothalamus or on glutamate receptor activation in rRPa.

69 on through a process that requires NMDA-type glutamate receptor activation.

70 tic levels of the GluA1 subunit of AMPA-type glutamate receptors after 48 h silencing with the Na(+)

71 somata that release dopamine when exposed to glutamate receptor agonists.

72 urface expression of NMDA-type and AMPA-type glutamate receptors, along with prominent functional imp

73 rologous expression of excitatory ionotropic glutamate receptor AMPA subunits in Xenopus oocytes, we

74 Positive allosteric modulators of AMPA-type glutamate receptors (ampakines) have been shown to rescu

75 Regulation of AMPA-type glutamate receptor (AMPAR) number at synapses is a major

76 regulated addition and removal of AMPA-type glutamate receptors (AMPARs) at excitatory synapses.

77 AMPA-type glutamate receptors (AMPARs) lacking an edited GluA2 sub

78 AMPA-type glutamate receptors (AMPARs) mediate fast excitatory neu

79 KEY POINTS: The AMPA-type ionotropic glutamate receptors (AMPARs) mediate the majority of exc

80 sity protein-95 (PSD-95) localizes AMPA-type glutamate receptors (AMPARs) to postsynaptic sites of gl

81 The regulated transport of AMPA-type glutamate receptors (AMPARs) to the synaptic membrane is

82 AMPA-type glutamate receptors (AMPARs), which are central mediator

83 naptic transmission is mediated by AMPA-type glutamate receptors (AMPARs).

84 synaptic scaffolding protein PSD-95 and AMPA glutamate receptors (AMPARs).

85 ssion is mediated by AMPA-subtype ionotropic glutamate receptors (AMPARs).

86 des GluA3, a subunit of AMPA-type ionotropic glutamate receptors (AMPARs).

87 ed interactions with AMPA-type and NMDA-type glutamate receptors (AMPARs/NMDARs).

88 f annotated genes demonstrated enrichment in glutamate receptor and adipogenesis pathways.

89 Our experiments show metabotropic glutamate receptor and endocannabinoid 2-arachidonoylgly

90 We show the coexistence of time-locked, glutamate receptor and GABA receptor-mediated mono synap

91 vivo imaging of N-methyl-d-aspartate (NMDA) glutamate receptor and gamma-aminobutyric acid (GABA)-A

92 some of these spines are immunopositive for glutamate receptor and postsynaptic density proteins (vi

93 ein uniquely affects the expression of other glutamate receptor and transporter proteins.

94 Vasoconstriction required metabotropic glutamate receptors and CYP omega-hydroxylase, the enzym

95 ed tau from infiltrating spines, dislocating glutamate receptors and impairing synaptic function in c

96 2+) in MNTB neurons, which is independent of glutamate receptors and is absent in neurons from ASIC1a

97 D95), a major synaptic protein that clusters glutamate receptors and is critical for plasticity.

98 These results identified ionotropic glutamate receptors and NMDA-Rs, specifically, as potent

99 c densities and increased synaptic levels of glutamate receptors and PSD-95.

100 ide insight into the activation mechanism of glutamate receptors and the complex conformational space

101 ethyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors and the function of synapses in the

102 nding gating across the family of ionotropic glutamate receptors and the role of AMPA receptors in ex

103 by modulating the clustering of postsynaptic glutamate receptors and thereby regulating the strength

104 fers basal glutamate activation of AMPA-type glutamate receptors and therefore decreases baseline exc

105 en shown to directly inhibit AMPA receptors (glutamate receptors), and to change cell energetics thro

106 peroxide dismutase, ornithine decarboxylase, glutamate receptor, and ammonia transporter.

107 ons with VLEs, requires group I metabotropic glutamate receptors, and has a presynaptic mechanism.

108 rgeting multiple ionotropic and metabotropic glutamate receptors, and intracellular cascades involved

109 ctivity study of the broad-acting ionotropic glutamate receptor antagonist 1a.

110 H2 )5 [D-Tyr(2) ,Thr(4) ]OVT, the ionotropic glutamate receptor antagonist kynurenate or the GABAA an

111 Ketamine, an N-methyl-d-aspartate glutamate receptor antagonist, has demonstrated a rapid-

112 effects of ketamine, an N-methyl-D-aspartate glutamate receptor antagonist, have not been fully eluci

113 ion of either a D1 dopamine receptor or NMDA glutamate receptor antagonist.

114 tic standpoint because numerous metabotropic glutamate receptor antagonists are available, many of wh

115 Ionotropic glutamate receptor antagonists are valuable tool compoun

116 GABA or glutamate receptor antagonists did not block the ethanol

117 ne, and/or the N-methyl-d-aspartate class of glutamate receptor antagonists have been shown to be eff

118 cularly those involving N-methyl-D-aspartate glutamate receptor antagonists, to illustrate principles

119 5-HT because it was abolished by ionotropic glutamate receptor antagonists.

120 Ionotropic glutamate receptors are a family of tetrameric ion chann

121 However, glutamate receptors are also expressed at the NMJ.

122 Ionotropic glutamate receptors are important for estradiol feedback

123 NMDA-type glutamate receptors are ligand-gated ion channels that c

124 Glutamate receptors are ligand-gated tetrameric ion chan

125 Ionotropic glutamate receptors are postsynaptic tetrameric ligand-g

126 Ionotropic glutamate receptors are thought to play an essential rol

127 Glutamate receptors are well characterized channels that

128 ARs), one of three subfamilies of ionotropic glutamate receptors, as well as the putative KAR auxilia

129 mily of synaptic scaffolding proteins anchor glutamate receptors at CNS synapses.

130 Internalization of glutamate receptors at the postsynaptic membrane via cla

131 um channels to a trans-synaptic complex with glutamate receptors at the visual system's first synapse

132 ner drugs (DREADDs) or by the treatment with glutamate receptor blockers attenuated misfolded tau acc

133 mature animals require MAGUKs for anchoring glutamate receptors, but are much more stable.

134 aptic cell to prevent block of NMDA-specific glutamate receptors by Mg(2+) The ratio of postsynaptic

135 Excessive activation of glutamate receptors causes excitotoxicity and delayed ce

136 reticulum, the postsynaptic density, and the glutamate receptor cluster.

137 We examined the AMPA and kainate glutamate receptor composition in each bipolar cell type

138 d glutamate receptor field size, and altered glutamate receptor composition.

139 The delta family of ionotropic glutamate receptors consists of glutamate delta-1 (GluD1

140 y recruitment of calcium-permeable AMPA-type glutamate receptors (CP-AMPARs) after drug withdrawal re

141 RP in neurons abolishes group 1 metabotropic glutamate receptor-dependent DGK activity combined with

142 Evidence suggests that AMPA glutamate-receptor-dependent synaptic plasticity within

143 en 1 (EEA1), a protein involved in AMPA-type glutamate receptor endocytosis.

144 le subanesthetic dose of ketamine normalized glutamate receptor expression and synaptic function of g

145 We used RT-PCR to profile ionotropic glutamate receptor expression in cultured SCs.

146 Indeed glutamate receptor expression profiles are largely predi

147 ated CaMKII and Rac1 activity, reduced GluN1 glutamate receptor expression, and impaired synaptic pla

148 c compartment of mutant NMJs include reduced glutamate receptor field size, and altered glutamate rec

149 ng in an increased fraction of post-synaptic glutamate receptor fields that lack the active zone scaf

150 rved phenotypes are likely due to defects in glutamate receptor function.

151 possibly via the modulation of postsynaptic glutamate receptor functionality.

152 esponsiveness and the link between PMCA2 and glutamate receptors, GABA receptors (GABARs), and glutam

153 ated nonsynonymous SNP, rs34144324, was in a glutamate receptor gene (GRID2, P = 8.65 x 10(-6) [OR 3.

154 acent to the analogous residue in the Grid2 (glutamate receptor) gene, which is mutated in the mouse

155 AMPK targets both the AMPA-type glutamate receptor GLR-1 and the metabotropic glutamate

156 f Mint orthologue LIN-10, internalization of glutamate receptor GLR-1, and depression of GLR-1-mediat

157 Following ER exit, the AMPA-type glutamate receptor GluA1 and neuroligin 1 undergo spatia

158 resynaptic Cav1.3 clusters with postsynaptic glutamate receptor GluA4 and PSD-95 clusters was signifi

159 age dependent increase in the levels of the glutamate receptor, GluN2B in aged MHCI knockout mice, w

160 ys a role on the recycling and clustering of glutamate receptors (GLUR) at the postsynaptic density.

161 The formin Delphilin binds the glutamate receptor, GluRdelta2, in dendritic spines of P

162 a*ss* receptors) as well as the two types of glutamate receptors (GluRs) (AMPARs and NMDARs).

163 s underlying gating, numerous structures for glutamate receptors have been solved in complexes with a

164 NMDA glutamate receptors have key roles in brain development,

165 manner requiring intact group 1 metabotropic glutamate receptors, Homer2, phospholipase C, and/or pho

166 TD in PE animals was rescued by metabotropic glutamate receptor I activation, suggesting that PE did

167 ry synapses is determined by the presence of glutamate receptors (i.e. AMPA, NMDA, and kainate recept

168 Exposure to ionotropic glutamate receptor (iGluR) agonists, kainic acid (KA) or

169 rocytic processes were blocked by ionotropic glutamate receptor (iGluR) antagonists, tetrodotoxin, zi

170 ents from RGCs in the presence of ionotropic glutamate receptor (iGluR) antagonists.

171 Ionotropic glutamate receptor (iGluR) family members are integrated

172 and GluD2 receptors form the GluD ionotropic glutamate receptor (iGluR) subfamily.

173 Loss of specific ionotropic glutamate receptor (iGluR) subunits during interneuron d

174 are classified as members of the ionotropic glutamate receptor (iGluR) superfamily on the basis of s

175 Ionotropic glutamate receptors (iGluRs) mediate neurotransmission a

176 ly encodes homologs of vertebrate ionotropic glutamate receptors (iGluRs) that are distantly related

177 channels that together with other ionotropic glutamate receptors (iGluRs), the NMDA and kainate recep

178 Here, we studied ionotropic glutamate receptors (iGluRs), which are ion channels act

179 role of synaptic trafficking of AMPA-type of glutamate receptors in HSP, Mecp2 KO neurons have lower

180 te patch-clamp experiments verified that the glutamate receptors in question were located on astrocyt

181 the hypothalamus, and during the blockade of glutamate receptors in rRPa.

182 pendence to show that GluA1 subunits of AMPA glutamate receptors in the nucleus accumbens (NAc), a br

183 relationship between the number of AMPA-type glutamate receptors in the PSD and synaptic strength.

184 elf-administration of cocaine increases AMPA glutamate receptors in the VTA, and this effect enhances

185 Stimulating these glutamate receptors increases nitric oxide (NO) producti

186 al measures to demonstrate that metabotropic glutamate receptor-induced sensitization of TRPA1 nocice

187 In contrast, prolonged stimulation of glutamate receptors induces varicosities in dendrites bu

188 the GluA2 subunit of the AMPAR and requires glutamate receptor interacting protein 1 (GRIP1) interac

189 pled to the actin cytoskeleton to facilitate glutamate receptor internalization has not been demonstr

190 ic to activity-dependent versus constitutive glutamate receptor internalization.

191 y to the spine cytoskeleton and facilitating glutamate receptor internalization.

192 oskeleton has been shown to mediate synaptic glutamate receptor internalization.

193 with the classification used for vertebrate glutamate receptor ion channels (iGluRs).

194 and the gating characteristics of tetrameric glutamate receptor ion channels are determined by their

195 t encoding the glutamate receptor subunit B, glutamate receptor ionotropic AMPA 2 (GRIA2), modifies a

196 ting candidate genes on chromosome 1 (GRIK3 (glutamate receptor ionotropic kainate 3)), chromosome 4

197 ble sensory receptor (the variant ionotropic glutamate receptor IR75b) and attraction-mediating circu

198 lutamate receptors to those of AMPA-specific glutamate receptors is decreased, but the postsynaptic c

199 of the FMRP pathway by group I metabotropic glutamate receptors is involved in regulating synaptic p

200 tic current from activation of NMDA-specific glutamate receptors is progressively increased during tr

201 Postsynaptic kainate-type glutamate receptors (KARs) regulate synaptic network act

202 namics simulations of the GluA2 AMPA subtype glutamate receptor ligand-binding domain (LBD) dimers to

203 R2 and excitatory light-activated ionotropic glutamate receptor LiGluR yielded a distribution of expr

204 In plants, genes of the GLUTAMATE RECEPTOR-LIKE (GLR) family have been implicate

205 e ion channel two pore channel 1 (TPC1), and glutamate receptor-like channels GLR3.3 and GLR3.6.

206 ates that expression of GRM3, a metabotropic glutamate receptor mainly expressed in mammalian central

207 erefore, our results suggest that ionotropic glutamate receptors may have been conserved throughout p

208 At the same time, metabotropic glutamate receptors mediate 20-hydroxyeicosatetraenoic a

209 N-methyl-d-aspartate (NMDA)-type ionotropic glutamate receptors mediate excitatory neurotransmission

210 Glutamate receptors mediate excitatory synaptic transmis

211 isoxazole propionic acid)-subtype ionotropic glutamate receptors mediate fast excitatory neurotransmi

212 AMPA subtype ionotropic glutamate receptors mediate fast excitatory neurotransmi

213 Accordingly, metabotropic glutamate receptor-mediated long-term depression (mGluR-

214 Glutamate receptor-mediated recruitment of GABAergic inh

215 he spatiotemporal profiles of GABA, ACh, and glutamate receptor-mediated synaptic activity in DSGCs e

216 lutamate receptor GLR-1 and the metabotropic glutamate receptor MGL-1 in one of the primary circuits

217 anslation downstream of group I metabotropic glutamate receptors (mGlu1/5) is a core pathophysiology

218 ation of one particular GPCR, a metabotropic glutamate receptor (mGluR), can reduce cone synaptic tra

219 receptors, specifically via the metabotropic glutamate receptor (mGluR).

220 licited by activation of type-I metabotropic glutamate receptors (mGluR-LTD).

221 onist activation of the group I metabotropic glutamate receptor mGluR1 increases the strength of this

222 er1a and signaling from group I metabotropic glutamate receptors mGluR1/5.

223 Type 1 metabotropic glutamate receptor (mGluR1)-dependent signaling at paral

224 ional signalling by the group I metabotropic glutamate receptors, mGluR1 and mGluR5, occurs through G

225 irects GAD67 expression via the metabotropic glutamate receptor mGluR1beta on GABApre terminals and r

226 Group II metabotropic glutamate receptors (mGluR2 and mGluR3) may control rela

227 Group II metabotropic glutamate receptors (mGluR2/3), which couple to Gi/Go, h

228 mine the significance of type 5 metabotropic glutamate receptors (mGluR5s) for behavioral and circuit

229 Upon binding glutamate, the metabotropic glutamate receptor mGluR6 activates the heterotrimeric G

230 alization of beta-DG with dystrophin and the glutamate receptor mGluR6 is disrupted, and the post-syn

231 ransmission is initiated by the metabotropic glutamate receptor, mGluR6, that signals via the G-prote

232 execution, EAAC1 limits group I metabotropic glutamate receptor (mGluRI) activation, facilitates D1 d

233 that EAAC1 limits activation of metabotropic glutamate receptors (mGluRIs) in the striatum and, by do

234 Activation of Group I metabotropic glutamate receptors (mGluRs) activates signaling cascade

235 ivation of postsynaptic group I metabotropic glutamate receptors (mGluRs) and Ca(2+) -permeable AMPA

236 Metabotropic glutamate receptors (mGluRs) are mainly known for regula

237 Metabotropic glutamate receptors (mGluRs) are mandatory dimers playin

238 ses, we show that activation of metabotropic glutamate receptors (mGluRs) by general and group I-spec

239 notropic glutamate receptors or metabotropic glutamate receptors (mGluRs) or orthosteric agonists of

240 Group I metabotropic glutamate receptors (mGluRs) play important roles in var

241 Stimulation of synaptic metabotropic glutamate receptors (mGluRs) reactivates translation of

242 the visual system, presynaptic metabotropic glutamate receptors (mGluRs) regulate cone photoreceptor

243 Activating Group 1 (Gp1) metabotropic glutamate receptors (mGluRs), including mGluR1 and mGluR

244 ule subunit counting on class C metabotropic glutamate receptors (mGluRs), we map dimerization determ

245 create a family of light-gated metabotropic glutamate receptors (mGluRs).

246 duced at excitatory synapses by metabotropic glutamate receptors (mGluRs).

247 posited to result from N-methyl-D-aspartate glutamate receptor (NMDAR) hypofunction.

248 ngthening by increasing N-methyl D-aspartate glutamate receptor (NMDAR) internalization through depho

249 protein kinase II (CaMKII) to the NMDA-type glutamate receptor (NMDAR) subunit GluN2B.

250 on targeted at the N-methyl-D-aspartate type glutamate receptor (NMDAR).

251 Both also require the NMDA class of glutamate receptor (NMDAR).

252 converge on regulation of NMDA and AMPA-type glutamate receptors (NMDAR, AMPAR), including long-term

253 al synaptic responses, mediated by NMDA-type glutamate receptor (NMDARs) activation, form the cellula

254 th synaptic functions by depleting NMDA-type glutamate receptors (NMDARs) from the neuronal surface a

255 s the N-methyl-D-aspartate (NMDA) subtype 2B glutamate receptor (NR2B).

256 We here demonstrate that metabotropic glutamate receptors of subtype 5 (mGluR5) contribute to

257 n in AMPK-Thr(P)(172) levels was mediated by glutamate receptors of the N-methyl-d-aspartate (NMDA) s

258 emic injections of antagonists of ionotropic glutamate receptors or metabotropic glutamate receptors

259 ion of PNNs in regulating the trafficking of glutamate receptors places them in a critical position t

260 ns as a primary site of persistent AMPA-type glutamate receptor plasticity by two widely used psychos

261 are a large subfamily of variant ionotropic glutamate receptors present across Protostomia.

262 resynaptic calcium channels and postsynaptic glutamate receptor proteins across the synaptic cleft.

263 basis of allosteric inhibition in ionotropic glutamate receptors, providing key insights into how iGl

264 -in-colorectal-cancer', and the postsynaptic glutamate-receptor-related proteins GluD1 and GluD2.

265 accelerates the turnover of GluA2-containing glutamate receptors, revealing a novel mechanism that co

266 ct excess "gain of function" of metabotropic glutamate receptor signaling at an important cerebellar

267 We demonstrated previously that CeA glutamate receptor signaling mediates cisplatin-induced

268 that the activation of group I metabotropic glutamate receptor signaling though the fragile X mental

269 n cytokine-cytokine receptor interaction and glutamate receptor signaling.

270 on of genes associated with neurogenesis and glutamate receptor signaling.

271 Glutamate-receptor signaling initiates the activity-depe

272 emerging evidence that altered metabotropic glutamate receptor signalling and disrupted calcium home

273 tion of the Gq-coupled, Group 1 metabotropic glutamate receptors, specifically mGlu5, is implicated i

274 vel concept that a breakdown of metabotropic glutamate receptor subtype mGluR5 and endocannabinoid si

275 overy of allosteric ligands for metabotropic glutamate receptor subtypes 1-5 and 7 (mGlu1-5,7) highli

276 coincides with a differential activation of glutamate receptor subtypes.

277 one adenosine in the transcript encoding the glutamate receptor subunit B, glutamate receptor ionotro

278 ct of EphB2 may be mediated by the AMPA-type glutamate receptor subunit GluA2, which can become assoc

279 itution in the homologous but distinct mouse glutamate receptor subunit Grid2 is associated with Lurc

280 rent site substitution (p.A636T) occurs in a glutamate receptor subunit, GRIA1.

281 rally required for dendritic development and glutamate receptor surface expression, core-glycosylated

282 NMDA receptors (NMDARs) are ionotropic glutamate receptors that are crucial for neuronal develo

283 NMDA receptors are ionotropic glutamate receptors that function as heterotetramers com

284 Rs) are a subtype of postsynaptic ionotropic glutamate receptors that function as molecular coinciden

285 -aspartate-receptors (NMDARs) are ionotropic glutamate receptors that function in synaptic transmissi

286 ess kainate receptors (KARs), a subfamily of glutamate receptors that modulate neurite outgrowth and

287 s the remodeling of the ionotropic AMPA-type glutamate receptors that underlie fast excitatory synapt

288 itatory and inhibitory connectivity and GABA/Glutamate receptor time constant based on neural mass mo

289 ing proteins is critical for localization of glutamate receptors to synapses.

290 ission in the brain requires localization of glutamate receptors to synapses.

291 ctive excitatory synapses by recruiting AMPA glutamate receptors to the postsynaptic cell surface.

292 o of postsynaptic responses of NMDA-specific glutamate receptors to those of AMPA-specific glutamate

293 specific antibody antagonist of metabotropic glutamate receptor type 1.

294 rity of the transferrin receptor and several glutamate receptor types, resulting in their appearance

295 ignaling through ionotropic and metabotropic glutamate receptors, ultimately resulting in synaptic dy

296 Synaptic transmission mediated by AMPA-type glutamate receptors was potentiated in the NAc shell 10-

297 GRIA2 subunits leads to a calcium-permeable glutamate receptor, which can promote cell migration and

298 tors (KARs) consist of a class of ionotropic glutamate receptors, which exert diverse pre- and postsy

299 Correspondingly, AMPA-subtype ionotropic glutamate receptors, which mediate the majority of excit

300 ulating lateral diffusion and positioning of glutamate receptors within the postsynaptic density (PSD