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1 ty that the parasite activates the host cell glutamate transporter.
2 amate and are not known to have a functional glutamate transporter.
3  by the gene expression for type 1 vesicular glutamate transporter.
4 a2b, which encodes Eaat2b, a plasma membrane glutamate transporter.
5  colabeled with antibodies against VGluT2, a glutamate transporter.
6 smission due to their abundant expression of glutamate transporters.
7 ly via expression and function of astroglial glutamate transporters.
8 eir highly branched morphologies and express glutamate transporters.
9 h necessitates the proper function of active glutamate transporters.
10 substance, cytochrome oxidase, and vesicular glutamate transporters.
11 domain movements in a bacterial homologue of glutamate transporters.
12 r glutamate, which is regulated primarily by glutamate transporters.
13 mino acids are kept low by the action of the glutamate transporters.
14 g also altered the expression of glucose and glutamate transporters.
15 l binding of two Na(+) ions in Na(+)-coupled glutamate transporters.
16 agnitude slower than those observed in other glutamate transporters.
17 ons contribute to the functional dynamics of glutamate transporters.
18 ls near astrocytes and tightly gated by Glt1 glutamate transporters.
19 1), voltage-gated sodium channels (Nav ) and glutamate transporters.
20 ine-induced suppression of the high-affinity glutamate transporter 1 (EAAT2/GLT-1) in the nucleus acc
21 e via specialized transporters such as glial glutamate transporter 1 (excitatory amino-acid transport
22 seeking while increasing the function of the glutamate transporter 1 (GLT-1) and system xC- (Sxc) in
23 his behavioral effect has been attributed to glutamate transporter 1 (GLT-1) and xCT (a catalytic sub
24  glutamate-aspartate transporter (GLAST) and glutamate transporter 1 (GLT-1) are reduced.
25 t TX enhanced the expression and function of glutamate transporter 1 (GLT-1) in rat astrocytes, an ef
26 ea, to promote the upregulation of the glial glutamate transporter 1 (GLT-1) on astrocytes and to red
27 hat astroglial glutamate transporter subtype glutamate transporter 1 (GLT1) and glutamate uptake is s
28 illary acidic protein, glutamine synthetase, glutamate transporter 1 (GLT1), aquaporin-4, aldehyde de
29 g the tibial nerve (TN), and using Vesicular GLUtamate Transporter 1 (VGLUT1) and the 65 kDa isoform
30 ectors to target expression of the vesicular glutamate transporter 1 (VGLUT1) following injection int
31  or NL2 after IUEP does not affect vesicular glutamate transporter 1 (vGlut1) in the glutamatergic co
32 for an increased expression of the vesicular glutamate transporter 1 (vGluT1) in the striatum of PD p
33  shown by using antibodies against vesicular glutamate transporter 1 [labeling all ON and OFF bipolar
34 tion with synaptic vesicle protein vesicular glutamate transporter 1 in stratum radiatum.
35 porter Sv2 (now known as SV2A) and Vesicular glutamate transporter 1 in the outer molecular layer of
36 ate transporter levels were higher and glial glutamate transporter 1 levels were lower in the DH of f
37 droxylase in periglomerular cells, vesicular glutamate transporter 1, a presynaptic protein, in mitra
38  microscopic level, the density of vesicular glutamate transporter 1-positive (i.e. cortico-subthalam
39 y synapses, defined by overlapping vesicular glutamate transporter 1-positive (VGlut1+) and postsynap
40 ory synapses (i.e., the overlap of vesicular glutamate transporter 1-positive [VGlut1+] puncta and po
41 studies showing that the number of vesicular glutamate transporter 1-positive terminals and of axon t
42 orm, neurofilament light chain and vesicular glutamate transporter 1.
43  oxide synthase, somatostatin, and vesicular glutamate transporters 1 and 2 accounted for a combined
44 imary afferents immunoreactive for vesicular glutamate transporters 1 and 2 and by intraspinal neuron
45  the entire macaque thalamus using vesicular glutamate transporters 1 and 2 to label cortical and sub
46  Glutamate/aspartate transporter (GLAST) and glutamate transporter-1 (GLT-1) are the most abundant su
47                                              Glutamate transporter-1 (GLT-1) is the main glutamate tr
48  glutamate-aspartate transporter (GLAST) and glutamate transporter-1 (GLT-1), which are essential for
49 n that E2 increases astrocytic expression of glutamate transporter-1 (GLT-1), which would prevent exc
50 B) receptor subunits, and a reduction in the glutamate transporter-1 (GLT-1).
51                                   Astrocytic glutamate transporter-1 (GLT-I) is critical to control t
52 aptic vesicle protein-2 (SV2), and vesicular glutamate transporter-1 (VGLUT1).
53                                          The glutamate transporter-1 [GLT-1 (excitatory amino acid tr
54 V1), neurofilament 200 (NF200), or vesicular glutamate transporter 2 (VGluT1).
55     Brains were then processed for vesicular glutamate transporter 2 (VGLUT2) and examined for AAS-in
56  by their immunoreactivity for the vesicular glutamate transporter 2 (VGluT2) and performed unbiased
57 Cre-expressing neurons or inducing vesicular glutamate transporter 2 (VGLUT2) deficiency in Trpv1-Cre
58 -Cre expressing neurons or induced vesicular glutamate transporter 2 (Vglut2) deficiency in Trpv1-Cre
59 ffect of deleting the gene for the vesicular glutamate transporter 2 (Vglut2) from neurons in the PB.
60                 Based on PTH2R and vesicular glutamate transporter 2 (VGlut2) immunolabeling in anima
61      We have previously shown that vesicular glutamate transporter 2 (VGLUT2) is the predominant VGLU
62 s, including the expression of the vesicular glutamate transporter 2 (VGluT2) mRNA in Ipc neurons, ha
63 n the transcription factor Phox2b, vesicular glutamate transporter 2 (VGLUT2) mRNA, and a subset cont
64 r bouton size, and the presence of vesicular glutamate transporter 2 (Vglut2) or parvalbumin (PV).
65 he VTA has many neurons expressing vesicular glutamate transporter 2 (VGluT2) that also project to LH
66 onprimary boutons that express the vesicular glutamate transporter 2 (VGLUT2), and form asymmetrical
67 N-->CeA CGRP projections coexpress vesicular glutamate transporter 2 (VGLUT2), providing evidence tha
68 rojected to MDmc and expressed the vesicular glutamate transporter 2 (VGLUT2), which is found in high
69 atic rings of terminals expressing vesicular glutamate transporter 2 (VGLUT2)--to subdivide IC GABAer
70 H)-ir and the same proportion were vesicular glutamate transporter 2 (VGLUT2)-ir.
71 a, with or without the presence of vesicular glutamate transporter 2 (VGLUT2)-mediated glutamatergic
72 e, ionic zinc, neurofilaments, and vesicular glutamate transporter 2 (VGluT2).
73 utamatergic neurons expressing the vesicular glutamate transporter 2 (VGluT2).
74 tamate activity was examined using vesicular glutamate transporter 2 (VGLUT2).
75 rome oxidase, Nissl bodies, or the vesicular glutamate transporter 2 (vGluT2).
76 glutamatergic neurons that express vesicular glutamate transporter 2 (VgluT2).
77  markers [neurofilament, NeuN, and vesicular glutamate transporter 2 (VGlut2)], and cultures exhibite
78  (to visualize Purkinje cells) and vesicular glutamate transporter 2 (VGluT2, to visualize climbing f
79 lation of dopamine neurons express vesicular glutamate transporter 2 and make glutamatergic connectio
80 re GABAergic and were contacted by vesicular glutamate transporter 2-containing somatic terminals, as
81 cted in transgenic mice expressing vesicular glutamate transporter 2-enhanced green fluorescent prote
82 kers for both glutamate signaling (vesicular glutamate transporter 2; VGluT2) and GABA signaling (glu
83 with glutamate neurons (expressing vesicular glutamate transporter 2; VGluT2), which play roles in re
84 ricosities were immunoreactive for vesicular glutamate transporter-2 (78%).
85 , acetylcholinesterase (AChE), and vesicular glutamate transporter-2 (VGluT2) preparations, received
86  glutamatergic neurons--expressing vesicular glutamate transporter-2 (VGluT2)--project to limbic and
87 rformed antibody stainings against vesicular glutamate transporter-2, which suggested that cytochrome
88 -originating pathway consisting of vesicular glutamate transporter 3 (VGluT3) containing neurons that
89                                The vesicular glutamate transporter 3 (VGLUT3) is expressed by striata
90                                    Vesicular glutamate transporter 3 (VGLUT3) loads glutamate into se
91 sent in mutants that lacked either Vesicular glutamate transporter 3 (Vglut3) or Ca(V)1.3.
92 dings to characterize responses of vesicular glutamate transporter 3 (VGluT3)-expressing amacrine cel
93 ch depend on the expression of the vesicular glutamate transporter 3 (VGLUT3).
94 of basket cells expressing CCK and vesicular glutamate transporter 3 (VGlut3).
95 transporter 1(+), somatostatin(+), vesicular glutamate transporter 3 (VGLUT3)/cholecystokinin/CB(1) c
96              Moreover, there is no vesicular glutamate transporter 3-immunoreactive bouton, specific
97                                    Vesicular glutamate transporter 3-positive (VGluT3(+)) primary aff
98                   Mice lacking the vesicular glutamate transporter-3 (VGLUT3) are congenitally deaf d
99                                    The EAAT2 glutamate transporter, accounts for >90% of hippocampal
100 cise, controlled manner in response to glial glutamate transporter activation.
101                                              Glutamate transporters actively take up glutamate into t
102  suggest that the Kir4.1 conductance affects glutamate transporter activity in a dual manner: (1) by
103 t mutants to explore the effects of impaired glutamate transporter activity on locomotor network func
104 mate tone in RVH rats was not due to blunted glutamate transporter activity.
105                                              Glutamate transporters also display an anion conductance
106 nd glutamate transporter expression (glial L-glutamate transporter and L-glutamate/L-aspartate transp
107                                    EAATs are glutamate transporters and anion-selective ion channels,
108 y (MNTB) are stimulated by the activation of glutamate transporters and consequently release glutamin
109 rgic neurotransmission (astrocyte-restricted glutamate transporters and glutamine synthetase).
110 toward the majority of previously identified glutamate transporters and permeability pathways.
111                       Although mitochondrial glutamate transporters and transporters for neutral amin
112 none oxidoreductase 1 (NQO1), Bach1, cystine/glutamate transporter, and glutamate cysteine ligase.
113 pic and metabotropic glutamate receptors and glutamate transporters, and altered neuronal excitabilit
114 /H(+) exchanger, ClC-7 H(+)/Cl(-) exchanger, glutamate transporters, and neutral amino acid transport
115 ound in episodic ataxia of the dual-function glutamate transporter/anion channel EAAT1, and discovere
116                                              Glutamate transporters are essential for recovery of the
117                                              Glutamate transporters are integral membrane proteins th
118 intained in physiological solutions and that glutamate transporters are ready to quickly bind glutama
119 nal-glial microenvironment, as well as glial glutamate transporters, are expected to affect eNMDAR-me
120 ique piston-like quaternary rearrangement in glutamate transporters, as evidenced by the difference b
121   Pharmacological blockade of GLT1 astrocyte glutamate transporters, as well as the gliotoxin alpha-a
122                                            A glutamate transporter-associated chloride channel blocke
123                      By expressing vesicular glutamate transporters at high levels in plasma membrane
124 tamate binding can be isolated in the mutant glutamate transporter because reactions, such as glutama
125 d EPSCs in afferent fibers are unaffected by glutamate transporter blockade, suggesting that transmit
126 ncreased (or decreased) in the presence of a glutamate transporter blocker (or a competitive glutamat
127     DVGLUT functions not only as a vesicular glutamate transporter but also serves as an acid-extrudi
128           They are not only secondary active glutamate transporters but also function as anion channe
129          EAATs are not only secondary active glutamate transporters but also function as anion channe
130 n synaptic vesicles is mediated by vesicular glutamate transporters called VGLUTs.
131                            Whereas mammalian glutamate transporters can translocate both glutamate an
132 cone during electrical stimulation activates glutamate transporter Cl(-) conductances on neighboring
133                                              Glutamate transporters clear synaptically released gluta
134              Astrocytes also show diminished glutamate transporter current, are significantly depolar
135 or voltage-clamped astrocytes and respective glutamate transporter currents (GTCs) were induced by ph
136  examined intracellular Na(+) transients and glutamate transporter currents as the most telling indic
137  rescued by increasing the expression of the glutamate transporter dEaat1.
138 elates with the upregulation of an astrocyte glutamate transporter, delayed and reduced glutamate acc
139 ate the dynamic regulation and importance of glutamate transporters during development.
140 /Na(+) exchange via the Drosophila vesicular glutamate transporter (DVGLUT).
141      These findings indicate that astrocytic glutamate transporter dysfunction may play an important
142 oduced two cysteine residues in the neuronal glutamate transporter EAAC1 at positions predicted to be
143 g debate on the contribution of the neuronal glutamate transporter EAAC1 to the onset of compulsive b
144 plicate Slc1a1, a gene encoding the neuronal glutamate transporter EAAC1, with obsessive-compulsive d
145  increase the expression and activity of the glutamate transporter (EAAT(2)) on glial cells, blocks m
146 e up-regulated proteins (GFAP, high affinity glutamate transporter (EAAT-2), apo-J (Clusterin), and p
147                                        Glial glutamate transporter EAAT2 plays a major role in glutam
148 Previous literature has indicated that glial glutamate transporter EAAT2 plays an essential role in c
149            Riluzole is known to increase the glutamate transporter EAAT2's ability to scavenge excess
150 lies, the DA transporter (DAT) and the glial glutamate transporter EAAT2, and we identified a conserv
151 s internalization of AQP4 and the associated glutamate transporter EAAT2, leading to glutamate excito
152                                          The glutamate transporter EAAT2, which is primarily localize
153 -181A > C) SNP in the promoter of astroglial glutamate transporter (EAAT2) and the same approach was
154  stabilization of the Purkinje cell-specific glutamate transporter EAAT4 at the plasma membrane.
155 c vesicle machinery, including the vesicular glutamate transporter eat-4/VGLUT, induction of neuropep
156 ture of glutamatergic neurons, the vesicular glutamate transporter EAT-4/VGLUT, is expressed in 38 of
157 tumor mediated by the system x(c)(-) cystine-glutamate transporter (encoded by Slc7a11).
158 tatory amino acid transporter 2)] subtype of glutamate transporter ensures crisp excitatory signaling
159    Perisynaptic astrocytes express important glutamate transporters, especially excitatory amino acid
160              Moreover, we show that neuronal glutamate transporters, especially those localized at th
161 ous glutamate levels controlled by astrocyte glutamate transporters, evokes a transient and reversibl
162 uated glutamate uptake and expression of the glutamate transporter excitatory amino acid transporter
163                                   Astrocytic glutamate transporter excitatory amino acid transporter
164 mily 1 (SLC1), which also includes the human glutamate transporters (excitatory amino acid transporte
165                       Spinal expression of a glutamate transporter, excitatory amino acid carrier 1 (
166                                   EAAT1 is a glutamate transporter expressed by astrocytes and other
167 ted with increases in Cx43, Cx30, Panx1, and glutamate transporter expression (glial L-glutamate tran
168 ssociated membrane protein 1), and astrocyte glutamate transporter expression (glutamate/aspartate tr
169 duced conflicting results and the changes in glutamate transporter expression have not yet been exami
170       Hypoxia and JAK/STAT inhibition reduce glutamate transporter expression in astrocytes, but unli
171 dition, ENT1 inhibition or knockdown reduces glutamate transporter expression in cultured astrocytes.
172 vation and lysosomal pathology, and restored glutamate transporter expression to levels observed in W
173 trocytes, as well as compromise in astrocyte glutamate transporter expression.
174 GLUT3 is an atypical member of the vesicular glutamate transporter family.
175 s study enhances our understanding as to how glutamate transporters function as both amino-acid trans
176    Additionally, the expression of two known glutamate transporters, genderblind and excitatory amino
177 dates include non-recurrent deletions at the glutamate transporter gene SLC1A1, a CNV locus recently
178     The gene for EAAT2, the major astrocytic glutamate transporter, generates two carrier isoforms (E
179 ontrast levels of the predominant cerebellar glutamate transporter GLAST, expressed in Bergmann glia,
180   The compound did not affect the astrocytic glutamate transporter GLAST, nor did it block glutamate
181 s that increase expression of the astroglial glutamate transporter GLT-1 (N-acetylcysteine and ceftri
182 te carrier family 1 member 2), also known as glutamate transporter GLT-1 and excitatory amino acid tr
183 ed to increased expression of the astrocytic glutamate transporter GLT-1 and to attenuated changes in
184 in hemichannels and glutamate uptake via the glutamate transporter GLT-1 in rat glial cells.
185 e; then, using an antisense strategy against glutamate transporter GLT-1, we found that restored tran
186 ed a progressive depletion of the astroglial glutamate transporters GLT-1 and GLAST.
187 efrontal cortical blockade of the astrocytic glutamate transporter (GLT-1) induces anhedonia and c-Fo
188  removing glutamate from the synapse via the glutamate transporter (GLT-1).
189                     Impairment of astrocytic glutamate transporter (GLT-1; EAAT2) function is associa
190 g glutamate spillover by blocking astroglial glutamate transporters (GLT-1) had no effect on reinstat
191 ease in expression and function of the major glutamate transporter, GLT-1.
192 , presumably through dysregulated astroglial glutamate transporter GLT1 and impaired glutamate uptake
193 , likely through the dysregulated astroglial glutamate transporter GLT1 expression and impaired gluta
194       We found that functional expression of glutamate transporter GLT1 is 40% decreased in i-astro-F
195 on of important functional proteins, such as glutamate transporter GLT1.
196 ficantly reduced expression of the major CNS glutamate transporter, GLT1, in superficial dorsal horn
197 ransiently increased, and the glial-specific glutamate transporters glutamate-aspartate transporter (
198                               Dysfunction of glutamate transporters has been proposed to promote neur
199                     SLC1A3 encodes the glial glutamate transporter hEAAT1, which removes glutamate fr
200 utward- and inward-facing conformations of a glutamate transporter homolog from archaebacterium Pyroc
201 based on crystal structures of the bacterial glutamate transporter homolog Glt(Ph).
202                Crystallographic studies of a glutamate transporter homologue from the archaeon Pyroco
203 y proteins defining glutamatergic signaling (glutamate transporter-I [GLT-I], N-methyl-D-aspartate re
204  Ceftriaxone modulated the expression of the glutamate transporter in a critical region of the cortic
205                      We find that a putative glutamate transporter in Drosophila melanogaster, polyph
206 for early expression of the type 2 vesicular glutamate transporter in mesencephalic DA neurons, we hy
207                             GLT-1, the major glutamate transporter in the adult brain, is abundantly
208           GLT-1 (EAAT2; slc1a2) is the major glutamate transporter in the brain, and is predominantly
209  Glutamate transporter-1 (GLT-1) is the main glutamate transporter in the central nervous system, and
210 AT5 may not act as a classical high-capacity glutamate transporter in the retina; rather, it may func
211  results indicate a role for neuron-specific glutamate transporters in AMPAR synaptic localization an
212    Here we elucidated the roles of these two glutamate transporters in cerebellar pathogenesis mediat
213                                              Glutamate transporters in the brain remove the neurotran
214 ssion for the typically astroglial-localized glutamate transporters in the mediodorsal and ventral ti
215 of glial (GLT-1, GLAST) and neuronal (EAAC1) glutamate transporters in these brain areas were assesse
216 se was tightly controlled by plasma membrane glutamate transporters, indicating that clearance of syn
217 orters, the reverse dialysis of ammonia, the glutamate transporter inhibitor, DL-threo-beta-benzyloxy
218 nion conducting conformation of the neuronal glutamate transporter is associated with an early step o
219                  Specifically, the vesicular glutamate transporter is upregulated, leading to over-ac
220 opose that the switch to the dynamic mode in glutamate transporters is due to separation of the trans
221 ific subtypes of receptors, ion channels and glutamate transporters is revealed at peripheral and cen
222                                 The dominant glutamate transporter isoform in the mammalian brain, GL
223 y studying the expression of EAAT1 and EAAT2 glutamate transporters, it was possible to document the
224 uptake is mediated by members of a family of glutamate transporters known as "excitatory amino acid t
225 elated enzymes (glutamine synthetase and the glutamate transporter) known to play critical roles in g
226 ical rat neurons, we show that inhibition of glutamate transporters leads to rapid reduction in AMPAR
227 ate signaling dynamics, increased astrocytic glutamate transporter levels and alleviated multiple sig
228 uggests that abnormalities in the astroglial glutamate transporter localization and function may unde
229 the peptide transporter CstA, PEB1 aspartate/glutamate transporter, LutABC lactate dehydrogenase and
230                                              Glutamate transporters maintain synaptic concentration o
231             Finally, to model dysfunction of glutamate transporters, manganese was used, and G1 was f
232 itochondria and suggest a mechanism by which glutamate transporters might retain mitochondria at site
233 ription polymerase chain reaction to examine glutamate transporter mRNA expression in the hippocampus
234 erlie their capacity to upregulate the glial glutamate transporter on astrocytes through the vascular
235                                              Glutamate transporters play a crucial role in this respe
236 lar glutamate levels influenced by astrocyte glutamate transporters resulted in a significant inhibit
237  calcium pump SERCA, leucine transporter and glutamate transporter shows that ANMPathway yields resul
238 responding serine residue, Ser-364, of human glutamate transporter SLC1A2 (solute carrier family 1 me
239 id application of glutamate to the wild-type glutamate transporter subtype EAAC1 (excitatory amino ac
240                     We found that astroglial glutamate transporter subtype glutamate transporter 1 (G
241     Other studies report that ceftriaxone, a glutamate transporter subtype-1 activator, is neuroprote
242                            Of the five known glutamate transporter subtypes, the retina-specific subt
243 ate in glutathione biosynthesis, the cystine/glutamate transporter (system xc(-)) represents a potent
244                                              Glutamate transporters terminate neurotransmission by cl
245 rter (GLAST) in rodents, is one of two glial glutamate transporters that are responsible for removing
246 mate receptors, GABA receptors (GABARs), and glutamate transporters that have been implicated in pain
247 ino acid transporters (EAATs) are a class of glutamate transporters that terminate glutamatergic syna
248 sociated protein 25, syntaxin, and vesicular glutamate transporter type 1), together with an increase
249                  Immunostaining of vesicular glutamate transporter type 2 demonstrated a decrement in
250                              Using vesicular glutamate transporter type 2 immunohistochemistry, we la
251 napses and were immunoreactive for vesicular glutamate transporter type 2.
252                       The atypical vesicular glutamate transporter type 3 (VGLUT3) is expressed by su
253 inals that lack VMAT2, but contain vesicular glutamate transporters type 2 (VGluT2).
254                                The vesicular glutamate transporter (VGLUT) plays an essential role in
255                                    Vesicular glutamate transporter (VGLUT) proteins regulate the stor
256  study with probes for the lamprey vesicular glutamate transporter (VGLUT) provides an anatomical bas
257 ded into synaptic vesicles via the vesicular glutamate transporter (VGLUT), a mechanism conserved acr
258 eracidification is mediated by the vesicular glutamate transporter (VGLUT).
259 press three different types of the vesicular glutamate transporter (VGLUT).
260       Among these SV cargos is the vesicular glutamate transporter (vGlut).
261 e by controlling expression of the vesicular glutamate transporter (VGlut).
262 xide synthase as well as the three vesicular glutamate transporters (VGLUT 1-3) in the locus coeruleu
263  transporter [vGAT]) or glutamate (vesicular glutamate transporter [vGLUT]) into vesicles, as well as
264 logy and increasing levels of the excitatory glutamate transporter VGLUT1.
265 fore mapped boutons expressing the vesicular glutamate transporters VGluT1 and VGluT2, together with
266 aptic vesicles for release via the vesicular glutamate transporters VGLUT1 and VGLUT2.
267 rents and of central neurons (with vesicular glutamate transporters VGLUT1 or VGLUT2, respectively),
268 he two predominant isoforms of the vesicular glutamate transporter (VGLUT1 and VGLUT2) participate in
269 utamate decarboxylase (GAD67), and vesicular glutamate transporters (vGLUT1 and vGLUT2) in postmortem
270 ing in situ hybridization to label vesicular glutamate transporters (vglut1, vglut2.1, vglut3), gluta
271 e reported expression of all three vesicular glutamate transporters (VGLUT1-3) by astrocytes suggests
272                                    Vesicular glutamate transporters (VGLUT1-3) carry glutamate into s
273 res, which can be identified using vesicular glutamate transporters, VGLUT1 and VGLUT2.
274 specific genetic disruption of the vesicular glutamate transporter VGLUT2 or the obligatory NMDA rece
275 unopositive for met-enkephalin and vesicular glutamate transporter VGLUT2, but not for GABAergic mark
276 resynaptic machinery including the vesicular glutamate transporter VGLUT2, several synaptic vesicle m
277 e terminals were demonstrated to express the glutamate transporter VGlut2, the projections are presum
278 fied through the expression of the vesicular glutamate transporter VGLUT2.
279 ed on coexpression of mRNA for the vesicular glutamate transporter (vGlut2) and the GABA synthetic en
280 most neurons in the GT express the vesicular glutamate transporter (VGluT2) mRNA, indicating a glutam
281 ecarboxylase 65 (GAD65) and type 2 vesicular glutamate transporter (VGLUT2) mRNAs, respectively.
282 r the glutamatergic marker, type 2 vesicular glutamate transporter (VGLUT2), and the GABA synthetic e
283 ially labeled by an isoform of the vesicular glutamate transporter, VGluT2.
284 them with staining patterns of the vesicular glutamate transporter, VGLUT2.
285           Interestingly, the third vesicular glutamate transporter (VGLUT3) is primarily found in neu
286                                    Vesicular glutamate transporters (VGLUTs) are essential for fillin
287                                    Vesicular glutamate transporters (VGLUTs) have been extensively st
288                                The vesicular glutamate transporters (VGLUTs) package glutamate into s
289 apse in NTS, we determined whether vesicular glutamate transporters (VGLUTs) were differentially dist
290 ecord currents associated with the vesicular glutamate transporters (VGLUTs), we characterize a chlor
291          Previous work showed that vesicular glutamate transporters (VGluTs; specific markers for glu
292 ecently, a new model for glutamate uptake by glutamate transporters was proposed based on crystal str
293 T), which is a major component of astrocytic glutamate transporters, was reduced by TNR knockdown.
294 for Nissl, cytochrome oxidase, and vesicular glutamate transporters, we investigated the primary soma
295                This effect was enhanced when glutamate transporters were inhibited, and reduced when
296       It belongs to the widespread family of glutamate transporters, which also includes the mammalia
297 hrin-A2 in the cortex colocalized with glial glutamate transporters, which were significantly downreg
298           We found GLT-1c to be an effective glutamate transporter with high affinity for Na(+) and g
299                           SLC25A22 encodes a glutamate transporter with strong expression in the deve
300                              First, block of glutamate transporters with a nonsubstrate antagonist ex

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