ライフサイエンスコーパス: glutamine synthetase

1 rocyte-restricted glutamate transporters and glutamine synthetase).

2 was the specific activity of the key enzyme, glutamine synthetase.

3 was most easily studied in strains that lack glutamine synthetase.

4 enases, respectively; and (iv) GLN1 encoding glutamine synthetase.

5 amate dehydrogenase, glutamate synthase, and glutamine synthetase.

6 and the second also had a partially impaired glutamine synthetase.

7 of TnrA is required for the interaction with glutamine synthetase.

8 o regulate the AmtB ammonium transporter and glutamine synthetase.

9 es of mutants that altered the regulation by glutamine synthetase.

10 e site to bring about feedback inhibition of glutamine synthetase.

11 ystem, a putative formate transporter, and a glutamine synthetase.

12 effects on the catalytic characteristics of glutamine synthetase.

13 e acid-labile substrates such as apyrase and glutamine synthetase.

14 were labeled with GFAP and Muller cells with glutamine synthetase.

15 response, including the ammonia-assimilating glutamine synthetase.

16 ression was confirmed by colocalization with glutamine synthetase.

17 ied with antibodies directed against S100 or glutamine synthetase.

18 an inactive complex with feedback-inhibited glutamine synthetase.

19 through a protein- protein interaction with glutamine synthetase.

20 tic properties similar to those of wild-type glutamine synthetase.

21 amma polypeptide of the native and denatured glutamine synthetase.

22 t for normal posttranslational regulation of glutamine synthetase.

23 ll maturation markers carbonic anhydrase and glutamine synthetase.

24 tive for glial fibrillary acidic protein and glutamine synthetase.

25 is inhibitor than a representative mammalian glutamine synthetase.

26 ogen-regulated promoters and the activity of glutamine synthetase.

27 tory effect of PKCdelta on the expression of glutamine synthetase.

28 against vimentin, carbonic anhydrase C, and glutamine synthetase.

29 y histology and expression of the HCC marker glutamine synthetase.

30 ease in the Wnt pathway targets cyclin-D1 or glutamine synthetase.

31 ctive astrocytes downregulated expression of glutamine synthetase.

32 o decreased the association between GFAP and glutamine synthetase.

33 ression of the beta-catenin surrogate target glutamine synthetase.

34 eraction between GlnR and feedback-inhibited glutamine synthetase.

35 ding was not activated by feedback-inhibited glutamine synthetase.

36 This constitutive repression did not require glutamine synthetase.

37 tection of the early glial markers GLAST and glutamine synthetase.

38 h bears significant similarity with GSI-type glutamine synthetases.

39 Glutamine synthetase 1 is regulated in different plants

40 a bona fide plastid transit peptide from the glutamine synthetase 2 gene does not restore DMI1 functi

41 atis bacteria expressing the M. tuberculosis glutamine synthetase A (glnA) gene or open reading frame

42 er but had only a minimal effect on cellular glutamine synthetase, a finding consistent with failure

43 We investigated whether a deficiency in glutamine synthetase, a key enzyme in catabolism of extr

44 in phosphorylated STAT3 was colocalized with glutamine synthetase, a Muller cell marker, by immunocyt

45 However, the degradation of chloroplast glutamine synthetase, a potential substrate for the ClpP

46 The corresponding antigens were derived from glutamine synthetase, a transitional endoplasmic reticul

47 in the domains Bacteria and Archaea encodes glutamine synthetase, a universally distributed enzyme t

48 te synthase, trehalose-6-phosphate synthase, glutamine synthetases, a protein (LIM17) that has been i

49 treatment led to a deficiency in hippocampal glutamine synthetase activity by 82-97% versus saline.

50 glnA1 expression, suggesting that decreased glutamine synthetase activity contributes to glutamate a

51 rotein, was stable only in a strain in which glutamine synthetase activity is not inhibited by NH(4)(

52 cDNA is reported as well as the finding that glutamine synthetase activity is present in liver but no

53 growth in MN medium is apparently due to low glutamine synthetase activity, because a DeltaglnD strai

54 n of Tyr-397 is central to the regulation of glutamine synthetase activity, via esterification of the

55 and that this inhibition was dependent upon glutamine synthetase activity.

56 of glnE, potentially encoding a regulator of glutamine synthetase activity.

57 ment are partially consistent with losses in glutamine synthetase activity.

58 Glutamine synthetase adenylyltranferase (ATase, EC 2.7.7

59 gulating the activities of the PII receptors glutamine synthetase adenylyltransferase (ATase) and the

60 ability of PII to control the activities of glutamine synthetase adenylyltransferase (ATase) but did

61 Glutamine synthetase adenylyltransferase (ATase) regulat

62 ith similar AMP-transferring enzymes such as glutamine synthetase adenylyltransferase or kanamycin nu

63 tion of several transcripts, including XDH1, glutamine synthetase, alanine aminotransferase, catalase

64 Thus, glutamine synthetase, an enzyme of central metabolism, d

65 colytic enzyme; HSP72, a stress protein; and glutamine synthetase, an excitotoxicity-related protein.

66 or that controls TnrA activity, a complex of glutamine synthetase and a feedback inhibitor, such as g

67 bition of caspases-3 and -11 also attenuated glutamine synthetase and fibroblast growth factor-2 expr

68 h zVAD significantly attenuated increases in glutamine synthetase and fibroblast growth factor-2 in t

69 luding stellate morphology and expression of glutamine synthetase and fibroblast growth factor-2.

70 There was a progressive loss of glutamine synthetase and GFAP content and a coincident i

71 ler cell changes in culture included loss of glutamine synthetase and GFAP, with coincident gains in

72 Finally, our results reveal that two genes, glutamine synthetase and glutamate synthase, which poten

73 ve and glutamate-induced injury and restored glutamine synthetase and glutamate transporter expressio

74 his was enhanced by inhibition of astrocytic glutamine synthetase and reversed or prevented by exogen

75 proteins, including aquaporin 4, actin, and glutamine synthetase and serine racemase.

76 n and inactivation of glial-related enzymes (glutamine synthetase and the glutamate transporter) know

77 cooperative binding sets the basal level for glutamine synthetase and the regulators of the Ntr respo

78 fibrillary acidic protein (GFAP), vimentin, glutamine synthetase, and alpha smooth muscle actin (alp

79 The cells expressed CRALBP, EGF-R, glutamine synthetase, and alpha-SMA, as judged by confoc

80 histochemical markers (PD-1, cytokeratin 19, glutamine synthetase, and beta-catenin expression).

81 e, an energy-requiring reaction catalyzed by glutamine synthetase, and found that at pH 7, constituti

82 acid residues in bovine serum albumin (BSA), glutamine synthetase, and insulin in the presence of a m

83 hologically normal when stained with an anti-glutamine synthetase antibody.

84 r cells mature in stages: HNK-1 labeling and glutamine synthetase arise earlier than carbonic anhydra

85 and other bacteria that indirectly regulates glutamine synthetase at the transcriptional and post-tra

86 ess nitrogen, the feedback-inhibited form of glutamine synthetase binds to TnrA and blocks DNA bindin

87 from adenosine 5'-triphosphate (ATP) in the glutamine synthetase biosynthetic assay.

88 ons, residual inhibition lost sensitivity to glutamine synthetase blockade, whereas exogenous glutami

89 ransferase (ATase) regulates the activity of glutamine synthetase by adenylylation and deadenylylatio

90 nK in the activation of the adenylylation of glutamine synthetase by ATase.

91 Inhibition of glutamine synthetase by methionine sulfoximine led to a

92 hypothesis that a deficiency in hippocampal glutamine synthetase causes recurrent seizures, even in

93 created a novel animal model of hippocampal glutamine synthetase deficiency by continuous (approxima

94 primary pulmonary hypertension, and hepatic glutamine synthetase deficiency.

95 2, SAG19, MT1 (metallothionein), and Atgsr2 (glutamine synthetase), did not show enhanced transcript

96 ort here that the feedback-inhibited form of glutamine synthetase directly interacts with TnrA and bl

97 ibrates across the cytoplasmic membrane, and glutamine synthetase does not manifest an isotope effect

98 Five mutants containing feedback-resistant glutamine synthetases (E65G, S66P, M68I, H195Y, and P318

99 The activity of glutamine synthetase (EC 6.3.1.2) from Escherichia coli

100 ining ammonia is assimilated into protein by glutamine synthetase (EC 6.3.1.2), which catalyzes the r

101 probably correlated with high expression of glutamine synthetase, enzymes utilizing nitrite/nitrate,

102 ular retinaldehyde binding protein (CRALBP), glutamine synthetase, epidermal growth factor receptor (

103 00beta, and CD44, but low immunostaining for glutamine synthetase, excitatory amino-acid transporter

104 mistry was used to study the distribution of glutamine synthetase, expressed by Muller cells, and zon

105 Glutamine synthetase expression was also increased in th

106 flagellar synthesis, sigma54 is involved in glutamine synthetase expression.

107 athogenic mycobacteria, all of which release glutamine synthetase extracellularly, but had no effect

108 terial microorganisms, none of which release glutamine synthetase extracellularly.

109 e conserved with active-site residues of the glutamine synthetase family of enzymes.

110 suggest that Dop and PafA are members of the glutamine synthetase fold family of proteins.

111 own with excess nitrogen, feedback-inhibited glutamine synthetase forms a protein-protein complex wit

112 own with excess nitrogen, feedback-inhibited glutamine synthetase forms a protein-protein complex wit

113 f the beta and gamma subunit polypeptides of glutamine synthetase from bean (Phaseolus vulgaris L.) r

114 TgTrx-Px2 protected glutamine synthetase from inactivation by Fe(3+)/DTT, sh

115 ne, suggesting a role for pita in protecting glutamine synthetase from inhibition.

116 High-level expression was achieved using the Glutamine Synthetase Gene Amplification System, and the

117 e show that the upstream enhancer of the rat glutamine synthetase gene is also active, specifically i

118 assay for open reading frame 1 (ORF1) of the glutamine synthetase gene of Neisseria gonorrhoeae was a

119 ression of nif (nitrogen-fixation) and glnA (glutamine synthetase) gene expression in vivo.

120 Here, we show that the enzyme glutamine synthetase (Gln1) forms filaments at low pH an

121 Similarly, glutamine synthetase (Gln1-GFP) foci cycled reversibly i

122 N-limited condition and mutants deficient in glutamine synthetase, gln1-3 and gln1-4.

123 n: asparagine synthetase (ASN1 and ASN2) and glutamine synthetase (GLN2).

124 ixation (nif) gene transcription occurs, and glutamine synthetase (glnA) gene transcription falls to

125 utants which synthesize low levels of active glutamine synthetase (glnA).

126 The model predicts the responses of glutamine synthetase, GlnB, and GlnK under time-varying

127 odA), L-alanine dehydrogenase (AlaDH), and L-glutamine synthetase (GlnS) proteins.

128 de the VZ also express the astroglial marker glutamine synthetase (Glns).

129 ipal glutamate and GABA-metabolizing enzymes glutamine synthetase, glutamate dehydrogenase, alpha-ket

130 enteric bacteria consists of three enzymes: glutamine synthetase, glutamate synthase (GOGAT), and gl

131 pression of glial fibrillary acidic protein, glutamine synthetase, glutamate transporter 1 (GLT1), aq

132 The glutamine synthetase-glutamate synthase (GOGAT) pathway

133 at both the tricarboxylic acid cycle and the glutamine synthetase/glutamate synthase cycles are linke

134 Ammonia assimilation by the plastidic glutamine synthetase/glutamate synthase system requires

135 as the nitrogen is scavenged by the urea and glutamine synthetase/glutamine 2-oxoglutarate aminotrans

136 s, which was identified as soybean cytosolic glutamine synthetase GS(1)beta1 by mass spectrometry.

137 to a single monomer of the protein substrate glutamine synthetase (GS(m)), as well as that of unligan

138 s the first step in that recovery, we report glutamine synthetase (GS) activity in highly purified Ar

139 roxynitrite to modify amino acid residues in glutamine synthetase (GS) and BSA is greatly influenced

140 e in Wnt-1, beta-catenin, and known targets, glutamine synthetase (GS) and cyclin-D1, along with a co

141 ression of the ammonium-assimilating enzymes glutamine synthetase (GS) and glutamate dehydrogenase (G

142 of ammonia through the concerted activity of glutamine synthetase (GS) and glutamate synthase (GOGAT)

143 r cells and subsequently in the promotion of glutamine synthetase (GS) and L-Glutamate/L-Aspartate Tr

144 An activity that inhibited both glutamine synthetase (GS) and nitrate reductase (NR) was

145 Glutamine synthetase (GS) and superoxide dismutase (SOD)

146 nit binds directly to the astrocytic protein glutamine synthetase (GS) and that this interaction dete

147 se brings about the short-term regulation of glutamine synthetase (GS) by catalyzing the adenylylatio

148 Induction of the enzyme glutamine synthetase (GS) by corticosteroids correlates

149 The regulation of Escherichia coli glutamine synthetase (GS) by reversible adenylylation ha

150 Glutamine synthetase (GS) catalyzes condensation of ammo

151 Glutamine synthetase (GS) catalyzes the ATP-dependent co

152 Glutamine synthetase (GS) catalyzes the ATP-dependent co

153 , which is deactivated by feedback-inhibited glutamine synthetase (GS) during nitrogen excess and sta

154 ruminicola 23 genome encodes three different glutamine synthetase (GS) enzymes: glutamine synthetase

155 In bacteria and yeast, glutamine synthetase (GS) expression is tightly regulate

156 reductase (NR), nitrite reductase (NiR), and glutamine synthetase (GS) from leaves of diploid barley

157 The crystal structure of glutamine synthetase (GS) from Mycobacterium tuberculosi

158 We present the first cloning and study of glutamine synthetase (GS) genes in wheat (Triticum aesti

159 ure of PA5508 from Pseudomonas aeruginosa, a glutamine synthetase (GS) homologue, has been determined

160 genetic analyses suggest that genes encoding glutamine synthetase (GS) III in the prasinophytes evolv

161 ere correlated with changes in expression in glutamine synthetase (GS) in astrocyte-like glia and in

162 The modification of glutamine synthetase (GS) in these glnB mutants appears

163 This study focuses on the mechanism of how glutamine synthetase (GS) inhibits TnrA function in resp

164 Escherichia coli glutamine synthetase (GS) is a dodecameric assembly of i

165 Here we report that glutamine synthetase (GS) is an endogenous substrate of

166 Glutamine synthetase (GS) is an enzyme localized predomi

167 The expression of glutamine synthetase (GS) is induced in rat skeletal mus

168 Glutamine synthetase (GS) is the central enzyme for nitr

169 Glutamine synthetase (GS) is the key enzyme in ammonia a

170 ammonia or glutamate as the nitrogen source; glutamine synthetase (GS) levels were also affected in t

171 non-native conformations, such as denatured glutamine synthetase (GS) monomers, preventing their agg

172 We generated four individual glutamine synthetase (GS) mutants (DeltaglnA1, DeltaglnA

173 Glutamine synthetase (GS) plays an essential role in met

174 We previously have shown that glutamine synthetase (GS) provides the glutamine needed

175 engsin is an eye lens-specific member of the glutamine synthetase (GS) superfamily.

176 ) show that p300/CBP-mediated acetylation of glutamine synthetase (GS) triggers recognition by the CR

177 Glutamine synthetase (GS), a key enzyme in biological ni

178 s we detected homology between gamma-GCS and glutamine synthetase (GS), allowing these proteins to be

179 To assess the role of glutamine synthetase (GS), an enzyme of central importan

180 he feasibility of inhibiting M. tuberculosis glutamine synthetase (GS), an enzyme that plays a key ro

181 l cell adhesion molecule (EpCAM), K19, CD34, glutamine synthetase (GS), and Ki-67.

182 on were assessed by anti-cytokeratin-7, anti-glutamine synthetase (GS), anti-cytochrome P4502E1 (CYP2

183 in metabolic zonation observed as a lack of glutamine synthetase (GS), Cyp1a2, and Cyp2e1.

184 expressed constitutively in mutants lacking glutamine synthetase (GS), GS is required for repression

185 d up-regulation of beta-catenin targets like glutamine synthetase (GS), leukocyte cell-derived chemot

186 that gamma-GCS is a structural homologue of glutamine synthetase (GS), providing the basis to build

187 zes the adenylylation and deadenylylation of glutamine synthetase (GS), regulating GS activity.

188 s glial fibrillary acidic protein (GFAP) and glutamine synthetase (GS), the bipolar cell marker prote

189 , rhodanese, malate dehydrogenase (MDH), and glutamine synthetase (GS), the folding efficiencies, and

190 Glutamine synthetase (GS), which catalyzes the productio

191 The enzymatic activity of Bacillus subtilis glutamine synthetase (GS), which catalyzes the synthesis

192 In contrast, glutamine synthetase (GS)--and nuclear beta-catenin--is

193 mon property of decreasing total activity of glutamine synthetase (GS).

194 reas they exhibited negligible expression of glutamine synthetase (GS).

195 carbamoylphosphate synthetase I (CPS I) and glutamine synthetase (GS).

196 othricin is a potent inhibitor of the enzyme glutamine synthetase (GS).

197 duct) to regulate the adenylylation state of glutamine synthetase (GS).

198 uced the expression of the astrocytic marker glutamine synthetase (GS).

199 a) cell immunopositive for both S100beta and glutamine synthetase (GS).

200 vated astrocytes and increased expression of glutamine synthetase (GS).

201 ractions with the feedback-inhibited form of glutamine synthetase (GS).

202 ylated-beta-catenin (Y654-beta-catenin), and glutamine synthetase (GS).

203 Specifically, coexpression of CD44/Vim and glutamine synthetase (GS)/VGLUT1 reflects glial speciali

204 Glutamine synthetases (GS) are ubiquitous enzymes that p

205 etabolism (glutamate dehydrogenase [GDH] and glutamine synthetase [GS]), axonal damage (SMI 32) and C

206 ing the expression of three proteins (Bcl-2, glutamine synthetase [GS], and glial fibrillar acidic pr

207 ivation (tyrosine aminotransferase, TAT, and glutamine synthetase, GS) and transrepression (IL-6).

208 generally mirror the response of cytoplasmic glutamine synthetase (Gs1) transcript abundance to chang

209 chloroplast by a chloroplastic isoenzyme of glutamine synthetase (GS2), the predominant GS isoform i

210 Bacterial glutamine synthetases (GSs) are complex dodecameric olig

211 o experiments showed that feedback-inhibited glutamine synthetase had a significantly reduced ability

212 Feedback-inhibited glutamine synthetase had a significantly reduced ability

213 Although S186F glutamine synthetase has kinetic properties that are sim

214 dition of PS-ODNs against the transcripts of glutamine synthetase I (glnA1) and alanine racemase (alr

215 different glutamine synthetase (GS) enzymes: glutamine synthetase I (GSI) (ORF02151), GSIII-1 (ORF014

216 Glutamine synthetase I (GSI) enzyme activity in Streptom

217 tified Muller cells with vimentin, GFAP, and glutamine synthetase immunoreactivities.

218 A computational calculation of glutamine synthetase immunoreactivity was developed so w

219 Muller cell morphology was different and glutamine synthetase immunoreactivity was reduced in the

220 -sulfoximine rapidly inhibited extracellular glutamine synthetase in a concentration-dependent manner

221 A deficiency in glutamine synthetase in astrocytes is a possible molecul

222 in their interaction with feedback-inhibited glutamine synthetase in cross-linking experiments.

223 To further explore the role of glutamine synthetase in MTLE we created a novel animal m

224 The amount of glutamine synthetase in the cell and its catalytic activ

225 In western blots, the expression of glutamine synthetase in the hippocampus was 40% lower in

226 the distribution, quantity, and activity of glutamine synthetase in the MTLE hippocampus.

227 an inhibitory effect on the inactivation of glutamine synthetase in the thiol metal-catalyzed oxidat

228 s with the 5'UTR of gifA mRNA, which encodes glutamine synthetase inactivating factor (IF)7.

229 The top 10 homologs were all bacterial glutamine synthetases, including Salmonella typhimurium

230 ich depends on the ATP-dependent activity of glutamine synthetase, increases for many minutes.

231 g response to methionine sulfoximine (Msx, a glutamine synthetase inhibitor).

232 nted here demonstrate that rapamycin and the glutamine synthetase inhibitor, methionine sulfoximine (

233 In normal islets, methionine sulfoximine, a glutamine synthetase inhibitor, suppressed insulin relea

234 eport that local application of glutamate or glutamine synthetase inhibitors induces astrocytic relea

235 Although it is possible that glutamine synthetase inhibitors interact with additional

236 this study, we investigated the influence of glutamine synthetase inhibitors on the growth of pathoge

237 the feedback inhibitors necessary to convert glutamine synthetase into its feedback-inhibited form an

238 Glutamine synthetase is central to nitrogen metabolism i

239 We conclude that glutamine synthetase is critical for nitrogen assimilati

240 orted that the glutamate-metabolizing enzyme glutamine synthetase is deficient in the hippocampus in

241 Genetic studies indicate that glutamine synthetase is required for the regulation of T

242 One major extracellular protein, the enzyme glutamine synthetase, is of particular interest because

243 Two glutamine synthetase isoforms were found to have differe

244 The assay was used to detect glutamine synthetase isoforms, separated by nondenaturin

245 Phospho-STAT3 colocalized with glutamine synthetase-labeled Muller cells.

246 Glutamine synthetase may be more important for islets th

247 needed to define the cause, but the loss of glutamine synthetase may provide a new focus for therape

248 temporal sclerosis, and that restoration of glutamine synthetase may represent a novel approach to t

249 egulons include those for nitrogen fixation, glutamine synthetase, (methyl)ammonia transport, the reg

250 Mycobacterium tuberculosis glutamine synthetase (MtGS) is a promising target for an

251 We find that pharmacological inhibitors of glutamine synthetase or system A transporters cause an a

252 revealed a 34% increase in the expression of glutamine synthetase (p<0.05) with unchanged metallothio

253 We hypothesized that glutamine synthetase plays an unusually critical role in

254 stic foci throughout the hepatic lobule were glutamine synthetase-positive, suggestive of a pericentr

255 methylammonium to gamma-N-methylglutamine by glutamine synthetase precludes its use in assessing conc

256 te plus ammonium, because feedback-inhibited glutamine synthetase (product of glnA) binds to TnrA and

257 biofilm formation and an apparent defect in glutamine synthetase production.

258 nd TnrA proteins is not known, the wild-type glutamine synthetase protein is required for the transdu

259 These results are consistent with the glutamine synthetase protein playing a direct role in re

260 relationship to the more fully characterized glutamine synthetase reaction.

261 feedback-inhibited form of Bacillus subtilis glutamine synthetase regulates the activity of the TnrA

262 odel in which the feedback-inhibited form of glutamine synthetase regulates TnrA activity in vivo.

263 presence or absence of MSX, an inhibitor of glutamine synthetase, resulting in a block of Glu/Gln sy

264 A homology model of B. subtilis glutamine synthetase revealed that the five mutated amin

265 Immunohistochemistry (alpha-glutamine synthetase) revealed that approximately 95% of

266 r retinaldehyde-binding protein (CRALBP) and glutamine synthetase showed that the GFP-expressing cell

267 Double labeling of CB2R and glutamine synthetase shows that CB2R is restricted to Mu

268 reticulin, alteration of normal perivenular glutamine synthetase staining (absent or diffuse), and v

269 , because a DeltaglnD strain with an altered glutamine synthetase that cannot be adenylylated can gro

270 Missense mutations in glutamine synthetase that constitutively express the Tnr

271 -protein interaction with feedback-inhibited glutamine synthetase that stabilizes GlnR-DNA complexes.

272 he alsS gene), acidifying the growth medium, glutamine synthetase (the glnA gene), and two surfactin

273 Alanine also inhibits the enzyme glutamine synthetase, the first enzyme in the other path

274 how they conjointly modulate the activity of glutamine synthetase, the key enzyme for nitrogen assimi

275 Elevated Mn(II) reduces the sensitivity of glutamine synthetase to feedback inhibitors, and we sugg

276 Glutamine synthetase transmits the nitrogen regulatory s

277 Two different genes encoding glutamine synthetase type I (GSI) and GSIII were identif

278 eterotetrameric sarcosine oxidase (SoxA) and glutamine synthetase type III (GSIII) respectively.

279 hydrolyzing carboxylate-amine ligases of the glutamine synthetase type.

280 nrA at the gltAB promoter was antagonized by glutamine synthetase under certain growth conditions.

281 The level of glutamine synthetase was 2 orders of magnitude higher th

282 S186F glutamine synthetase was defective in its ability to blo

283 A model of B. subtilis glutamine synthetase was derived from a crystal structur

284 Among other genes, the gene for glutamine synthetase was identified.

285 ns (glnA) in which the catalytic activity of glutamine synthetase was lowered.

286 However, glutamine synthetase was markedly increased in bone marr

287 Loss of glutamine synthetase was particularly pronounced in area

288 Ammonia assimilation gene glnA (glutamine synthetase) was repressed in vivo, while gdhA

289 The Trp pathway enzymes and cytosolic glutamine synthetase were induced under all of the amino

290 The levels of glutamine synthetase were reduced in both mutants.

291 glnA mRNA levels and specific activities of glutamine synthetase were regulated similarly by nitroge

292 Aldehyde dehydrogenase 1 L1 and glutamine synthetase were used as constitutive astrocyti

293 dulin, solute carrier family 1 member 2, and glutamine synthetase) were identified in a phenotype-dep

294 d by expression of immunoreactive Kir4.1 and glutamine synthetase, were closely associated with neuro

295 regulates the AmtB ammonium transporter and glutamine synthetase, which controls the rate of glutama

296 bears a similarity to the mechanism used by glutamine synthetase, which may point to an ancient link

297 rapidly inactivated purified M. tuberculosis glutamine synthetase, which was 100-fold more sensitive

298 A mutation in glutamine synthetase with a phenylalanine replacement at

299 In this study, we inhibited the activity of glutamine synthetase with methionine-sulfoximine (MSO) a

300 ulation of the AmtB ammonium transporter and glutamine synthetase within R. palustris.