ライフサイエンスコーパス: glutarate

1 ot find evidence of peroxisomal oxidation of glutarate.

2 abbit (rb) differ in their ability to handle glutarate.

3 and was stimulated in oocytes preloaded with glutarate.

4 s trans-stimulated in oocytes preloaded with glutarate.

5 m-coupled transport of proline, glucose, and glutarate.

6 either bis(3,5-dibromosalicyl)-succinate or -glutarate.

7 ersus NADH, indicating the formation of a E:(glutarate)2 complex as a result of occupying both the ly

8 with methoxypolyethylene glycol succinimidyl glutarate 5000 (MEGC-PEG).

9 d to methoxypolyethylene glycol succinimidyl glutarate-5000 in order to prolong the half-life of rMET

10 with methoxypolyethylene glycol succinimidyl glutarate-5000 with a molar ratio of PEG:rMETase of 15:1

11 omobifunctional cross-linker, disuccinimidyl glutarate (7.7 A).

12 In addition, OAA and glutarate also bind to free enzyme as does lysine at hig

13 ng dual-label competitive uptakes with (14)C-glutarate and (3)H-succinate to calculate the transport

14 External glutarate and alpha-ketoglutarate (1 mM), both counterio

15 an activatable bond in this manner, such as glutarate and citrate, are excluded from catalysis and a

16 wo-electrode voltage clamp, were similar for glutarate and succinate in Xenopus oocytes expressing mN

17 , fumarate, and the inhibitors oxaloacetate, glutarate, and 3-nitropropionate.

18 2,3,4-butanetetracarboxylate, tricarballate, glutarate, and acetate to a C(3)(v) symmetric metallo-ho

19 angeable dicarboxylates alpha-ketoglutarate, glutarate, and adipate, but not by succinate, a nonexcha

20 h of P. aeruginosa PAO1 on both alpha-KG and glutarate (another C5-dicarboxylate).

21 ve state and as a complex with the inhibitor glutarate at 1.85 A and 2.4 A resolution, respectively.

22 wo isoreticular three-dimensional copper(II) glutarate-based pillared-layered metal-organic framework

23 Oxaloacetate (OAA), pyruvate, and glutarate behave as dead-end analogues of lysine, which

24 ulated when oocytes were preloaded with 2 mM glutarate but not glutamate.

25 hippurate and the dicarboxylates adipate and glutarate (but not succinate or malonate) inhibited indi

26 he NAC-Hg(2+) conjugate was cis-inhibited by glutarate, but not by methylsuccinate, paralleling their

27 e addition of RP(OTMS)(2) to alpha-methylene glutarate containing a chiral auxiliary resulted in only

28 xo-acid dehydrogenase (BCOADC-E2), and 2-oxo-glutarate dehydrogenase (OGDC-E2).

29 drogenase complex (BCOADC-E2), and the 2-oxo-glutarate dehydrogenase complex (OGDC-E2).

30 drogenase complex, the E2 subunit of the oxo-glutarate dehydrogenase complex, four additional inner m

31 phogluconate dehydrogenase, 2-(hydroxymethyl)glutarate dehydrogenase, and phenylserine dehydrogenase,

32 ed chain 2-oxo-acid dehydrogenase, and 2-oxo-glutarate dehydrogenase.

33 The iron(II)- and 2-(oxo)glutarate-dependent (Fe/2OG) oxygenases catalyze an arra

34 Iron(II)- and 2-(oxo)-glutarate-dependent oxygenases catalyze diverse oxidativ

35 valuated: the palmitate (C16), the octadecyl glutarate diester (C18-C5) and the decyl carbamate (CB10

36 apenem synthase (CarC), an Fe(II) and 2-(oxo)glutarate (Fe/2OG)-dependent oxygenase, then inverts the

37 Glutarate gives S-parabolic noncompetitive inhibition ve

38 of dianions from succinate (suc(2-)) through glutarate (glu(2-)), alpha-ketoglutarate (kglu(2-)), adi

39 )), malonate (mal(2-)), succinate (suc(2-)), glutarate (glu(2-)), maleate (male(2-)) and fumarate (fu

40 es in the presence of either pyruvate, 3-oxo-glutarate, glutamate, isocitrate, dihydroorotate, alpha-

41 with this interpretation, an imposed outward glutarate gradient stimulated 2,4-D uptake in the absenc

42 olism of dodecanedioate (DODA), azelate, and glutarate in perfused rat livers, using a combination of

43 In contrast, currents evoked by glutarate in rbNaDC1 were only about 5% of the succinate

44 s tissue in vitro was stimulated by external glutarate in the presence of sodium.

45 sodium gradient but only in the presence of glutarate, indicating the presence of apical dicarboxyla

46 Poly(dimethyltin glutarate) is presented as the first organometallic poly

47 d to methoxypolyethylene glycol succinimidyl glutarate (MEGC-PEG-5000).

48 e-gauche enantiomeric interconversion of the glutarate moieties.

49 All ligands demonstrate an invariant glutarate moiety within the S1' pocket of the enzyme.

50 bic functions as substituents of a canonical glutarate moiety.

51 ented binding mode in which the putative P1' glutarate occupies the spacious entrance funnel rather t

52 f TM 3-4 from mNaDC1, had a decreased K(0.5)(glutarate) of 4 mM compared with 15 mM in wild-type rbNa

53 or ophthalmic acid (a GSH analog) but not by glutarate or N-acetylcysteine, suggesting that GSH deriv

54 A number of brefeldin A succinates, glutarates, oxidation products, and sulfone derivatives

55 1-expressing oocytes was trans-stimulated by glutarate, PAH, NAC, DMPS, MeHg-NAC, MeHg-DMPS, and a me

56 res of synthetic discrete mass poly(butylene glutarate) (PBG) oligomers of known structure having deg

57 the formation of sodium malonate and sodium glutarate salts resulted by HCl evaporation from dehydra

58 xy HbA modified with bis(3,5-dibromosalicyl)-glutarate showed that cross-linking only occurred betwee

59 sing rat CP tissue, which showed both sodium/glutarate-stimulated 2,4-D (tissue/medium (T/M) approxim

60 Glutarate stimulation was abolished by 5 mM LiCl.

61 e desired homoallylic derivative, a putative glutarate surrogate.

62 in cross-linking studies with disuccinimidyl glutarate this subunit's most reactive neighbor, and (4)

63 ion modes of citrate, acetate, succinate and glutarate to AuNPs is obtained by (13)C and (23)Na solid

64 affected individuals, urine molar ratios of glutarate to its derivatives (3-hydroxyglutarate, glutar

65 le transmembrane helices (TM) participate in glutarate transport, the most important contribution is

66 To identify domains involved in glutarate transport, we constructed a series of chimeric

67 and F, as well as E and G by disuccinimidyl glutarate was obtained, while in the free V(1) domain, c

68 s trans-stimulated in oocytes preloaded with glutarate, whereas the dicarboxylate methylsuccinate, wh